Search results for "Fibril"

showing 10 items of 836 documents

Ultramicro-trauma in the endometrial-myometrial junctional zone and pale cell migration in adenomyosis

2015

Objective To determine if ultrastructural tissue trauma occurs in the junctional zone in uteri in adenomyosis. Design A case-control experimental study. Setting Endometriosis research center. Patient(s) Twelve uteri with adenomyosis, and 9 uteri without adenomyosis, were gained during laparoscopy-assisted vaginal hysterectomy. Intervention(s) Transmission electron microscopic study of the junctional zone, as well as immunohistochemical staining for epithelial cadherin, and van Gieson staining and immunofluorescence for CD45 and CD68. Main Outcome Measure(s) Analysis of the electron microscopy photos and the immunoreactive scores of the staining. Result(s) The inner myometrial muscle fibers …

PathologyEndometriosisFluorescent Antibody TechniqueEndometriumBasal (phylogenetics)EndometriumMyofibrilsCell MovementVan Gieson's stainHysterectomy VaginalMedicine (all)MyometriumObstetrics and GynecologyAntigens CD45AnatomyMiddle AgedCadherinsultrastructureImmunohistochemistrymedicine.anatomical_structurePhenotypeMyometriumImmunohistochemistryFemaleAdenomyosis pathogenesiCase-Control StudieAdenomyosisHumanAdultmedicine.medical_specialtyAntigens Differentiation MyelomonocyticBiologypale cellMicroscopy Electron TransmissionAntigens CDmedicineHumansAdenomyosisendometrial-myometrial junctional zoneEpithelial CellCadherinEpithelial CellsBiomarkermedicine.diseaseReproductive MedicineCase-Control StudiesCadherinLeukocyte Common AntigensLaparoscopyAdenomyosiBiomarkersMyofibril
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Expression of C1q, a subcomponent of the rat complement system, is dramatically enhanced in brains of rats with either Borna disease or experimental …

1995

In situ hybridization, RT-PCR and Northern blot analysis as well immunohistochemistry were used to examine the expression of C1q, a subcomponent of the rat complement system, in brains of rats infected with Borna disease virus (BDV) and rats afflicted with experimental allergic encephalomyelitis (EAE) induced by the adoptive transfer of myelin basic protein specific T cells. C1q mRNA, which was not detected in normal brain, became clearly detectable using RT-PCR analysis by d14 post infection (p.i.) with BDV. Maximal levels of C1q mRNA were reached 21 days p.i. when inflammatory reactions in the brain were also at a peak. Similarly, C1q mRNA was elevated when the clinical symptoms of EAE be…

Pathologymedicine.medical_specialtyAdoptive cell transferEncephalomyelitis Autoimmune ExperimentalEncephalomyelitisMolecular Sequence Datachemical and pharmacologic phenomenaIn situ hybridizationBiologyHippocampusPolymerase Chain Reactionimmune system diseasesGlial Fibrillary Acidic ProteinmedicineAnimalsNorthern blotRNA MessengerIn Situ HybridizationBrain ChemistryBorna diseaseMicrogliaBase SequenceComplement C1qRNA-Directed DNA Polymerasemedicine.diseaseBlotting NorthernImmunohistochemistryMyelin basic proteinComplement systemRatsUp-RegulationBlotting Southernmedicine.anatomical_structureNeurologyBorna Diseasebiology.proteinFemaleNeurology (clinical)MicrogliaJournal of the neurological sciences
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Immunohistochemical investigation of the brain of aged dogs. I. Detection of neurofibrillary tangles and of 4-hydroxynonenal protein, an oxidative da…

2001

In the aging dog brain lesions develop spontaneously. They share some morphological characteristics with those of Alzheimer 's disease in man. Diffuse and primitive plaques are well known, whereas neuritic plaques rarely develop. Neurofibrillary tangles have not been seen in the canine. The aim of the present investigation was to study major age-related changes of the dog's brain using paraffin sections with respect to cross-immunoreactivity of tau, A beta protein and other immunoreactive components including hydroxynonenal protein, which is a marker for oxidative damage. The occurrence of neurofibrillary tangles and of the protein tau therein was studied in serial brain sections of two dog…

Pathologymedicine.medical_specialtyAgingAmyloidmedicine.drug_classTau proteinModels NeurologicalNerve Tissue ProteinsPlaque AmyloidMonoclonal antibodymedicine.disease_causeDogsAlzheimer DiseaseInternal MedicinemedicineAnimalsHumansSenile plaquesDog DiseasesAldehydesbiologyChemistryBrainNeurofibrillary Tanglesmedicine.diseaseImmunohistochemistryOxidative StressPolyclonal antibodiesbiology.proteinImmunohistochemistryAlzheimer's diseaseOxidative stressAmyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis
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Unusual Intranuclear Inclusions in Malignant Fibrous Histiocytoma: Presence in Primary Tumor

1982

We describe previously unreported intranuclear inclusions in 2 cases of malignant fibrous histiocytoma. The inclusions were found in 2-10% of the tumor cells removed from the patients and in 2-10% of the cells examined in tumor tissue xenotransplanted in nude mice. By stereo electron microscopy the inclusions are closely packed undulating fibrils 18-23 nm in diameter. They are sometimes associated with fibrillary bodies. They closely resemble the inclusions reported in some animals inoculated with serum from patients with non-A non-B hepatitis; however, their nature at present is unknown.

Pathologymedicine.medical_specialtyChemistryIntranuclear InclusionsTumor cellsmedicine.diseaseFibrilPrimary tumorTumor tissuePathology and Forensic Medicinelaw.inventionTransplantationStructural BiologylawmedicineNon b hepatitisElectron microscopeUltrastructural Pathology
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Primary cutaneous biphasic sarcomatoid basal cell carcinoma with myoepithelial carcinoma differentiation: A new variant

2019

Isolated cases of basal cell carcinoma (BCC) with partial myoepithelial component have been described. However, myoepithelial differentiation has not been described in sarcomatoid basal cell carcinomas, which usually show features resembling osteosarcoma, chondrosarcoma, or leiomyosarcoma. We report a case of an 87-year-old man with a forehead lesion that histologically showed a minor component of conventional nodular BCC in transition with a major biphasic sarcomatoid growth composed of invasive spindle-cell and epithelial-like components, the latter with a reticular pattern and scattered ductal structures. Both components showed cytological atypia and high mitotic rate (26/10HPF), with at…

Pathologymedicine.medical_specialtyHistologyintegumentary systembiologyGlial fibrillary acidic proteinCalponinMyoepithelial cellDermatologymedicine.diseasePathology and Forensic Medicine030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisbiology.proteinmedicineAtypiaOsteosarcomaBasal cell carcinomaChondrosarcomaImmunostainingJournal of Cutaneous Pathology
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Preservation of glial cytoarchitecture from ex vivo human tumor and non-tumor cerebral cortical explants: A human model to study neurological diseases

2007

For the human brain, in vitro models that accurately represent what occurs in vivo are lacking. Organotypic models may be the closest parallel to human brain tissue outside of a live patient. However, this model has been limited primarily to rodent-derived tissue. We present an organotypic model to maintain intraoperatively collected human tumor and non-tumor explants ex vivo for a prolonged period of time (similar to 11 days) without any significant changes to the tissue cytoarchitecture as evidenced through immunohistochemistry and electron microscopy analyses. The ability to establish and reliably predict the cytoarchitectural changes that occur with time in an organotypic model of tumor…

Pathologymedicine.medical_specialtyIndolesTime FactorsbrainMatrix (biology)BiologyModels BiologicalStatistics NonparametricArticleOrgan Culture TechniquesMicroscopy Electron TransmissionIn vivoGlial Fibrillary Acidic ProteinmedicineHumanshumanorganotypicCerebral Cortexelectron microscopyBrain NeoplasmsGeneral NeuroscienceexplantReproducibility of ResultsCell migrationHuman brainMiddle AgedImmunohistochemistrymedicine.anatomical_structureCytoarchitectureImmunohistochemistryFemaleTissue PreservationNervous System DiseasesNeurogliaEx vivoExplant culture
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Neonatal form of nemaline myopathy, muscle immaturity, and a microvascular injury.

1990

An infant with a neonatal form of nemaline myopathy showed ultrastructural features of muscle immaturity. Immaturity was characterized by an abnormal presence of myotubes, as well as cells in clusters within a common basement membrane and a great number of satellite cells adhering to very small muscle fibers. In addition, degenerative changes and a severe microvascular lesion were observed. The pathologic findings in the muscle of this patient were those of neonatal nemaline myopathy complicating severe microvascular injury, possibly induced by an unknown toxic agent. ( J Child Neurol 1990;5:122-126).

Pathologymedicine.medical_specialtyMuscle HypotoniaBiopsyIschemiaBiologyMuscle Smooth VascularLesion03 medical and health sciences0302 clinical medicineNemaline myopathyMyofibrilsIschemia030225 pediatricsBiopsymedicineHumansBasement membraneInclusion BodiesAsphyxia NeonatorumRespiratory Distress Syndrome Newbornmedicine.diagnostic_testMyogenesisMicrocirculationMusclesInfant NewbornDisseminated Intravascular Coagulationmedicine.diseaseMicroscopy ElectronMuscular Atrophymedicine.anatomical_structurePediatrics Perinatology and Child HealthMuscle HypotoniaFemaleNeurology (clinical)medicine.symptomMyofibril030217 neurology & neurosurgeryJournal of child neurology
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The disruption of myofibre structures in skeletal muscle after forced lengthening contractions

1998

Specific antibodies against structural proteins (actin, desmin, dystrophin, fibronectin) of muscle fibres were used to study the effect of forced lengthening contractions on muscle microarchitecture. Tibialis anterior (TA) muscle of male Wistar rats were subjected to 240 forced lengthening contractions. At consecutive time points (0, and 6 h, 2, 4, and 7 days) after stimulation, the TA muscle was excised for biochemical and histological assays. β-Glucuronidase activity, a quantitative indicator of muscle damage, showed increased values 2–7 days after the lengthening, peaking on day 4 (11.7-fold increase). A typical course of histopathological changes (myofibre swelling, necrosis and regener…

Pathologymedicine.medical_specialtyNecrosisSkeletal muscleAnatomyBiologyPathology and Forensic Medicinemedicine.anatomical_structurePhysiology (medical)medicinebiology.proteinDesminmedicine.symptomMyofibrilIntermediate filamentDystrophinCytoskeletonActinPathophysiology
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Silver Impregnation of Alzheimer's Neurofibrillary Changes Counterstained for Basophilic Material and Lipofuscin Pigment

1988

A method is described in which selective silver staining of Alzheimer's neurofibrillary changes is combined with staining of cell nuclei, Nissl material, and lipofuscin granules. Formalin fixed, paraffin embedded sections of human autopsy tissue are silver stained according to a method proposed by Gallyas. Lipofuscin is stained by crotonaldehyde fuchsin following performic acid oxidation. Nissl substance is visualized by either Darrow red or gallocyanin-chrome alum staining. Architectonic units showing the specific pathology and the neuronal types prone to develop the neurofibrillary changes can be recognized using this technique.

Pathologymedicine.medical_specialtySilverALIZARIN REDLipofuscinLipofuscinSilver stainPigmentsymbols.namesakechemistry.chemical_compoundAlzheimer DiseasemedicineHumansPerformic acidStaining and LabelingChemistryMiddle AgedStainingBasophilicnervous systemNissl Bodiesvisual_artNeurofibrilsvisual_art.visual_art_mediumNissl bodysymbolsFemalesense organsAnatomyStain Technology
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Filamin C accumulation is a strong but nonspecific immunohistochemical marker of core formation in muscle.

2002

Filamin C is the muscle isoform of a group of large actin-crosslinking proteins. On the one hand, filamin C is associated with the Z-disk of the myofibrillar apparatus and binds to myotilin; on the other hand, it interacts with the sarcoglycan complex at the sarcolemma. Filamin C may be involved in reorganizing the cytoskeleton in response to signalling events and in muscle it may, in addition, fulfill structural functions at the Z-disk. An examination of biopsies from patients with multi-minicore myopathy, central core myopathy and neurogenic target fibers with core-like target formations (TF) revealed strong reactivity of all the cores and target formations with two different anti-filamin…

Pathologymedicine.medical_specialtyanimal structuresBiopsyFilaminsmacromolecular substancesBiologyFilamin03 medical and health sciences0302 clinical medicineContractile ProteinsMuscular DiseasesReference ValuesmedicineMyotilinHumansProtein IsoformsCytoskeletonMyopathyMicroscopy ImmunoelectronMuscle Skeletal030304 developmental biology0303 health sciencesSarcolemmaMicrofilament Proteinsmedicine.diseaseImmunohistochemistryCell biologybody regionsNeurologyDesminNeurology (clinical)medicine.symptomMyofibrilCarrier Proteins030217 neurology & neurosurgeryCentral core diseaseBiomarkersJournal of the neurological sciences
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