Search results for "Generation"

showing 10 items of 3050 documents

Intergenerational continuity in parents’ and adolescents’ externalizing problems: The role of life events and their interaction with GABRA2.

2015

We examine whether parental externalizing behavior has an indirect effect on adolescent externalizing behavior via elevations in life events, and whether this indirect effect is further qualified by an interaction between life events and adolescents’ GABRA2 genotype (rs279871). We use data from 2 samples: the Child Development Project (CDP; n = 324) and FinnTwin12 (n = 802). In CDP, repeated measures of life events, mother-reported adolescent externalizing, and teacher-reported adolescent externalizing were used. In FinnTwin12, life events and externalizing were assessed at age 14. Parental externalizing was indexed by measures of antisocial behavior and alcohol problems or alcohol dependen…

MaleParentsExternalizationAdolescentGenotypeTwinsPolymorphism Single NucleotideArticleDevelopmental psychologyLife Change EventsLife eventsmedicineHumansGenetic Predisposition to DiseaseParent-Child RelationsChildta515AllelesBiological PsychiatryAggressionAntisocial personality disorderAlcohol dependenceAntisocial Personality DisorderReceptors GABA-Amedicine.diseaseModerationChild developmentTwin studyGene-environment interactionExternalizingAggressionAlcoholismIntergenerational continuityClinical PsychologyPsychiatry and Mental healthGABRA2FemaleGene-Environment Interactionmedicine.symptomPsychologyPsychopathologyJournal of Abnormal Psychology
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Personal exposure to radio-frequency electromagnetic fields in Europe: Is there a generation gap?

2018

Background: Exposure to radiofrequency electromagnetic fields (RF-EMF) from mobile communication technologies is changing rapidly. To characterize sources and associated variability, we studied the differences and correlations in exposure patterns between children aged 8 to 18 and their parents, over the course of the day, by age, by activity pattern, and for different metrics of exposure. Methods: Using portable RF-EMF measurement devices, we collected simultaneous real-time personal measurements of RF-EMF over 24 to 72 h in 294 parent-child pairs from Denmark, the Netherlands, Slovenia, Switzerland, and Spain. The devices measured the power flux density (mW/m(2)) in 16 different frequency…

MaleParentsGeneration gapWireless technologyRadio Wavesescuelashumanosadolescente010501 environmental sciences01 natural sciencesSmart phones0302 clinical medicineTelecommunications linkBackground exposureCordless phoneMedicine030212 general & internal medicineChildWorkplacemediana edadlcsh:Environmental sciencesGeneral Environmental Sciencelcsh:GE1-350TravelSchoolsadultoMiddle Aged3. Good healthadulto jovenEuropeondas de radioIntergenerational RelationsFemalePower fluxAdultEveningAdolescentCell phonespadres03 medical and health sciencesYoung Adultrelaciones intergeneracionalescampos electromagnéticosHumans0105 earth and related environmental sciencesbusiness.industryDigital Enhanced Cordless TelecommunicationsElectromagnetic fieldsEnvironmental ExposurePersonal exposureHousingexposición ambientalbusinessviajeCell PhoneDemography
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Sox-2 Positive Neural Progenitors in the Primate Striatum Undergo Dynamic Changes after Dopamine Denervation.

2013

The existence of endogenous neural progenitors in the nigrostriatal system could represent a powerful tool for restorative therapies in Parkinson's disease. Sox-2 is a transcription factor expressed in pluripotent and adult stem cells, including neural progenitors. In the adult brain Sox-2 is expressed in the neurogenic niches. There is also widespread expression of Sox-2 in other brain regions, although the neurogenic potential outside the niches is uncertain. Here, we analyzed the presence of Sox-2(+) cells in the adult primate (Macaca fascicularis) brain in naïve animals (N = 3) and in animals exposed to systemic administration of 1-methyl-4-phenyl-1,2,3,6 tetrahydropyridine to render th…

MalePathologyDopamineFluorescent Antibody Techniquelcsh:MedicineDopaminaStriatumchemistry.chemical_compoundNeural Stem CellsNeurobiology of Disease and RegenerationSox-2 PositiveNeurocièncieslcsh:Scienceeducation.field_of_studyMultidisciplinaryMPTPStem CellsCell DifferentiationNeurochemistryNeurodegenerative DiseasesParkinson DiseaseAnimal ModelsDopamine DenervationDenervationSubstantia NigraAdult Stem CellsNeurologyembryonic structuresMedicineNeural ProgenitorsCalretininNeurochemicalsMacaqueAdult stem cellmedicine.drugResearch Articlemedicine.medical_specialtyendocrine systemNeurogenesisPopulationSubstantia nigraModel OrganismsDevelopmental NeuroscienceDopamineInternal medicinemedicineAnimalsProgenitor celleducationBiologyurogenital systemSOXB1 Transcription Factorslcsh:RCorrectionCorpus StriatumMacaca fascicularisEndocrinologychemistrynervous systemlcsh:QDevelopmental BiologyNeurosciencePLoS ONE
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SIL1 mutations and clinical spectrum in patients with Marinesco-Sjogren syndrome.

2013

Marinesco-Sjogren syndrome is a rare autosomal recessive multisystem disorder featuring cerebellar ataxia, early-onset cataracts, chronic myopathy, variable intellectual disability and delayed motor development. More recently, mutations in the SIL1 gene, which encodes an endoplasmic reticulum resident co-chaperone, were identified as the main cause of Marinesco-Sjogren syndrome. Here we describe the results of SIL1 mutation analysis in 62 patients presenting with early-onset ataxia, cataracts and myopathy or combinations of at least two of these. We obtained a mutation detection rate of 60% (15/25) among patients with the characteristic Marinesco-Sjogren syndrome triad (ataxia, cataracts, m…

MalePathologymedicine.medical_specialtyAtaxiaultrastructure [Muscle Skeletal]SIL1 protein humanAdolescentMarinesco–Sjögren syndromeDNA Mutational Analysisgenetics [Mutation]Bioinformaticsmedicine.disease_causepathology [Muscle Skeletal]physiopathology [Spinocerebellar Degenerations]Cataractspathology [Brain]Intellectual disabilitymedicineGuanine Nucleotide Exchange FactorsHumansddc:610MyopathyMuscle SkeletalCells CulturedRetrospective StudiesSpinocerebellar DegenerationsFamily HealthMutationB-LymphocytesCerebellar ataxiabusiness.industryBrainmedicine.diseasegenetics [Guanine Nucleotide Exchange Factors]Magnetic Resonance Imaging10124 Institute of Molecular Life Sciencesgenetics [Spinocerebellar Degenerations]2728 Neurology (clinical)pathology [Spinocerebellar Degenerations]Mutationultrastructure [Brain]570 Life sciences; biologyAllelic heterogeneityFemaleNeurology (clinical)Neurosciences & Neurologymedicine.symptombusinessBrain : a journal of neurology
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The mechanism of epithelial shedding after ischemic damage to the small intestinal mucosa

1979

The intestinal mucosa of the rat was examined by light and electron microscopy 15, 30, 60 and 120 min after complete ligation of the vessel arcades of the proximal jejunum. The characteristic sign of ischemic damage to the small intestinal mucosa and the reason for epithelial shedding is the appearance of membrane enclosed cytoplasmic blebs which arise at the cell base of the enterocytes and detach the epithelium from the basement membrane. This process begins at the tip of the villi before the enterocytes display signs of irreversible damage and progress to the base of the villi with continuation of the ischemia.

MalePathologymedicine.medical_specialtyCellIschemiaBiologyBasement MembranePathology and Forensic Medicinelaw.inventionIntestinal mucosaIschemialawIntestine SmallmedicineAnimalsRegenerationIntestinal MucosaBasement membraneAnatomymedicine.diseaseEpitheliumRatsMicroscopy Electronmedicine.anatomical_structureCytoplasmElectron microscopeLigationVirchows Archiv B Cell Pathology
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DJ-1 mutations and parkinsonism-dementia-amyotrophic lateral sclerosis complex.

2005

Mutations in DJ-1 gene have been recently shown to cause autosomal recessive early-onset Parkinson’s disease (EOPD) in a large Dutch family and in a small consanguineous Italian family.1 Subsequent to this initial finding, several additional DJ-1 mutations were identified in subjects with EOPD.2–6 We describe a family from southern Italy with three brothers affected by a complex disorder characterized by early-onset parkinsonism-dementia-amyotrophic lateral sclerosis (EOPD-D-ALS). The analysis of the DJ-1 gene showed a novel homozygous mutation (E163K) in exon 7 and a novel homozygous mutation (g.168_185dup) in the promoter region of this gene in living affected subjects

MalePathologymedicine.medical_specialtyDNA Mutational AnalysisProtein Deglycase DJ-1Glutamic AcidGene mutationParkinsonismmedicine.disease_causeDISEASEPARK7GUAMExonMucoproteinsDegenerative diseaseParkinsonian DisordersmedicineHumansDementiaRNA MessengerAmyotrophic lateral sclerosisGeneFamily HealthOncogene ProteinsGeneticsMutationReverse Transcriptase Polymerase Chain Reactionbusiness.industryParkinsonismAmyotrophic Lateral SclerosisIntracellular Signaling Peptides and ProteinsExonsDEGENERATIONBlotting Northernmedicine.diseaseGENEINCLUSIONSNeurologyMutationAmyotrophic LateralFemaleDementiaNeurology (clinical)TAUbusiness
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3-Acetylpyridine-induced degeneration and regeneration in the adult lizard brain: a qualitative and quantitative analysis

1997

Abstract The neurotoxin 3-acetylpyridine (3AP) produces highly selective neuronal damage in specific areas of the lizard brain. Following 3AP intoxication, proliferation and migration of replacement neurons born in the ventricular walls lead to regeneration of the lesioned areas. Earlier studies established the time course of 3AP-induced degeneration and subsequent regeneration in the medial cerebral cortex of adult lizards (Font, E., Garcia-Verdugo, J.M., Alcantara, S. and Lopez-Garcia, C., Neuron regeneration reverses 3-acetylpyridine-induced cell loss in the cerebral cortex of adult lizards, Brain Res. , 551 (1991) 230–235 [13] ). Complementary to our previous studies, we now provide a q…

MalePathologymedicine.medical_specialtyDendritic spinePyridinesNeurotoxinsBiologyTransneuronal degenerationsymbols.namesakeCortex (anatomy)medicineAnimalsMolecular BiologyCell NucleusCerebral CortexNeuronsCerebrumGeneral NeuroscienceNeurogenesisBrainLizardsDNAAnatomyNerve Regenerationmedicine.anatomical_structureCerebral cortexNerve DegenerationNissl bodysymbolsFemaleNeurology (clinical)NeuronThymidineDevelopmental BiologyBrain Research
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Changes in pulmonary calcitonin gene-related peptide and protein gene product 9.5 innervation in rats infected with Mycoplasma pulmonis.

1996

Changes in the expression of calcitonin gene-related peptide (CGRP) and polyneural protein gene product 9.5 (PGP) in hilar peribronchial innervation was investigated by immunohistochemistry in specific pathogen-free rats chronically infected with Mycoplasma pulmonis. Image analysis of immunostained sections revealed a reduction of approximately 62% in the amount of CGRP- and PGP-immunoreactive innervation of the peribronchial area in the infected animals. The portion of the total bronchial perimeter occupied by bronchus-associated lymphoid tissue was increased six-fold. The decrease in the CGRP-immunoreactive area could be the result either of an enhanced CGRP release or of a loss of nerve …

MalePathologymedicine.medical_specialtyHistologyLymphoid TissueCalcitonin Gene-Related PeptideNeuropeptideBronchiNerve Tissue ProteinsCalcitonin gene-related peptideBiologyPathology and Forensic MedicineGene productRodent DiseasesmedicineAnimalsMycoplasma InfectionsLungRespiratory Tract Infectionsintegumentary systemCell BiologyPathophysiologyRatsSpecific Pathogen-Free OrganismsLymphatic systemCalcitoninRats Inbred LewNerve DegenerationMycoplasma pulmonisImmunohistochemistryThiolester HydrolasesUbiquitin ThiolesteraseBiomarkersCell and tissue research
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Addition of blood to a phycogenic bone substitute leads to increased in vivo vascularization

2015

The present study aimed to analyze the effects of the addition of blood to the phycogenic bone substitute Algipore(®) on the severity of in vivo tissue reaction. Initially, Fourier-transform infrared spectroscopy (FTIR) of the bone substitute was conducted to analyze its chemical composition. The subcutaneous implantation model in Balb/c mice was then applied for up to 30 d to analyze the tissue reactions on the basis of specialized histochemical, immunohistochemical, and histomorphometrical methods. The data of the FTIR analysis showed that the phycogenic bone substitute material is mainly composed of hydroxyapatite with some carbonate content. The in vivo analyses revealed that the additi…

MalePathologymedicine.medical_specialtyMaterials scienceAngiogenesisBiomedical EngineeringNeovascularization PhysiologicBioengineeringBone tissueBiomaterialsNeovascularizationMiceImmune systemIn vivoMaterials TestingmedicineAnimalsBlood TransfusionMice Inbred BALB CRegeneration (biology)medicine.anatomical_structureConnective TissueGiant cellBone SubstitutesImmunohistochemistrymedicine.symptomBiomedical Materials
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Liver regeneration induced by a designer human IL‐6/ sIL‐6R fusion protein reverses severe hepatocellular injury

2000

The cytokine IL-6 plays a significant role in liver regeneration in conjunction with additional growth factors (HGF, TNF-α, and TGF-α). Many IL-6 effects depend on a naturally occurring soluble IL-6 receptor (sIL-6R). Here, the chimeric protein hyper-IL-6, constructed from the human IL-6 protein fused to a truncated form of its receptor, was found to have superagonistic IL-6 properties, and as such, enhanced liver cell regeneration. Hyper-IL-6 reversed the state of hepatotoxicity and enhanced the survival rates of rats suffering from fulminant hepatic failure after D-galactosamine administration. The hyper-IL-6 protein has a significant potential for use in the treatment of severe human liv…

MalePathologymedicine.medical_specialtyRecombinant Fusion Proteinsmedicine.medical_treatmentApoptosisGalactosamineThioacetamideBiochemistryFulminant hepatic failureGeneticsmedicineAnimalsHumansReceptorInterleukin 6Molecular BiologybiologyInterleukin-6ChemistryLiver cellRegeneration (biology)Receptors InterleukinReceptors Interleukin-6Fusion proteinRats Inbred F344Liver regenerationLiver RegenerationRatsDisease Models AnimalCytokineCancer researchbiology.proteinCell DivisionLiver FailureBiotechnologyThe FASEB Journal
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