Search results for "HYPOXIA"

showing 10 items of 508 documents

Regional cerebral blood flow and regional metabolism in cold induced oedema.

1973

24 hours following a cold induced oedema in cats rCBF was measured in the lesion area, the bluish stained cortex immediately adjacent to the lesion, a cortical area remote from the lesion, and in the contralateral uninjured hemisphere. Thereafter the brain was frozen and the respective tissue areas were removed and analyzed for water and electrolyte content as well as metabolite concentrations. It seems, that in the neighbourhood of a local lesion at least 3 different brain regions can be differentiated with regard to their characteristic pattern of data. In non-oedematous regions either hyperaemia or hypoaemia could be observed. In areas with local brain oedema rCBF was reduced inversely p…

Pathologymedicine.medical_specialtyBrain EdemaPhosphocreatineMicrocirculationLesionHyperaemiachemistry.chemical_compoundAdenosine TriphosphateIschemiaCortex (anatomy)medicineAnimalsHypoxiaPyruvatesCerebrospinal FluidDiminutionCATSbusiness.industrySodiumBrainAnatomyWater-Electrolyte BalanceAdenosine MonophosphateAdenosine DiphosphateCold Temperaturemedicine.anatomical_structurechemistryCerebral blood flowRegional Blood FlowInjections IntravenousCatsLactatesPotassiumSurgeryNeurology (clinical)medicine.symptombusinessActa neurochirurgica
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Hypoxia in Breast Cancer: Pathogenesis, Characterization and Biological/Therapeutic Implications

2002

Nearly 50% of locally advanced breast cancers exhibit hypoxic and/or anoxic tissue areas that are heterogeneously distributed within the tumour mass. Hypoxia is predominantly caused by structural and functional abnormalities of the newly formed tumour vessels arising from neovascularization, by a disturbed microcirculation, by enlarged diffusion distances, and by tumour-associated or therapy-induced anaemia. The extent of pretherapeutically measured hypoxic tissue areas is independent of clinical tumour size and stage, and histological type and grade. Anaemia can substantially worsen tumour O2 depletion. Hypoxia is known to directly or indirectly confer resistance to irradiation and some ch…

Pathologymedicine.medical_specialtyBreast NeoplasmsDisease-Free SurvivalTreatment failureMicrocirculationPathogenesisNeovascularizationBreast cancerHumansMedicineTreatment FailureNeovascularization Pathologicbusiness.industryMicrocirculationGeneral MedicineHypoxia (medical)Prognosismedicine.diseaseCell HypoxiaClinical trialCancer researchFemalemedicine.symptomMalignant progressionbusinessWiener Medizinische Wochenschrift
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Lack of Association Between Tumor Hypoxia, Glut-1 Expression and Glucose Uptake in Experimental Sarcomas

2003

Hypoxia is a common phenomenon in experimental and human tumors which not only limits the efficacy of non-surgical treatment modalities such as irradiation and 02-dependent chemotherapy but also affects the long-term prognosis of cancer patients. Numerous studies have clearly demonstrated that patients with more hypoxic tumors have a reduced probability for survival and disease-free survival’. For this reason it might be helpful to non-invasively identify patients with hypoxic malignancies.

Pathologymedicine.medical_specialtyChemotherapyTumor hypoxiaTreatment modalitymedicine.medical_treatmentGlucose uptakeCancer researchmedicineBiologyHypoxia (medical)medicine.symptom
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CD15 – A new marker of pathological villous immaturity of the term placenta

2014

Abstract Introduction Idiopathic immaturity is one of the main reasons for latent placental insufficiency and antenatal hypoxia. Postnatal identification of the immature placental phenotype may help early stratification of a heterogeneous population of newborns and individually identify risk of disease in the immediate postnatal life. The aim of the study was to determine the relevant diagnostic markers associated with pathological placental immaturity. Methods 111 tissue samples from normal and pathological term placentas with persisting villous immaturity comprised the comparative immunohistochemical study (CD15, CD34). Positive immunohistochemical reactions were quantitatively assessed i…

Pathologymedicine.medical_specialtyEndotheliumLewis X AntigenAntigens CD34Placental insufficiencyBiologyPregnancyChronic VillitisFetal macrosomiamedicineHumansPathologicalPlacental villous immaturityAsphyxiaObstetrics and GynecologyHypoxia (medical)FucosyltransferasesPlacental Insufficiencymedicine.diseasemedicine.anatomical_structureReproductive MedicineCase-Control Studiesembryonic structuresImmunologyFemalemedicine.symptomBiomarkersDevelopmental BiologyPlacenta
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Sudden death in water: Diagnostic challenges

2016

Abstract The authors report a case of sudden death in a breath-holding diver and highlight the forensic diagnostic difficulties in opining the cause of sudden death in water. The autopsy showed increased thickness of the left ventricular wall with a distinct pattern of concentric hypertrophy, evident particularly in the subaortic interventricular septum. Histological examination revealed diffuse interstitial fibrosis and associated findings of multifocal myocyte disarray especially evident in the subaortic interventricular septum. The analysis and discussion of this case made it possible to attribute sudden death to a lethal arrhythmia following myocyte disarray and hypoxia caused by breath…

Pathologymedicine.medical_specialtyHealth (social science)2734Concentric hypertrophyAutopsy030204 cardiovascular system & hematologyBiologySudden deathPathology and Forensic Medicine03 medical and health sciences0302 clinical medicineSettore MED/43 - Medicina LegaleInternal medicinemedicinelcsh:Law in general. Comparative and uniform law. Jurisprudence030216 legal & forensic medicineInterventricular septumcardiovascular diseasesIncreased thicknessHypoxiaHistological examinationCause of deathlcsh:R5-920Sudden deathApneaArrhythmia; Breath-held diving; Hypoxia; Sudden death; 2734; Health (social science); Lawmedicine.anatomical_structurelcsh:K1-7720Cardiologycardiovascular systemBreath-held divingmedicine.symptomlcsh:Medicine (General)LawArrhythmia
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Hypoxia, Lactate Accumulation, and Acidosis: Siblings or Accomplices Driving Tumor Progression and Resistance to Therapy?

2013

This chapter briefly summarizes the most important processes by which hypoxia, lactate accumulation, and acidosis may influence malignant progression and therapeutic resistance of solid malignant tumors. While these phenomena are often elements of an integrated reaction, they may occur independently of each other under certain circumstances. The latter information may be of interest with regard to possible “targeted” therapeutic interventions.

Pathologymedicine.medical_specialtyHypoxic tumorTumor progressionmedicineCancer researchHypoxia (medical)medicine.symptomMalignant progressionBiologyTherapeutic resistanceStem cell phenotypeAcidosis
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Resistance of subventricular neural stem cells to chronic hypoxemia despite structural disorganization of the germinal center and impairment of neuro…

2015

Xavier d'Anglemont de Tassigny,1,* M Salomé Sirerol-Piquer,2,3,* Ulises Gómez-Pinedo,4 Ricardo Pardal,1 Sonia Bonilla,1 Vivian Capilla-Gonzalez,2 Ivette López-López,1 Francisco Javier De la Torre-Laviana,1 José Manuel García-Verdugo,2,3 José López-Barneo1,3 1Medical Physiology and Biophysics Department, Institute of Biomedicine of Seville (IBiS), Virgen del Rocío University Hospital/CSIC/University of Seville, Seville, Spain; 2Cavanilles Institute of Biodiversity and Evolutionary Biology, University of Valencia, Valencia, Spain; 3Network Center of Biomedical Research on Neurodegenerati…

Pathologymedicine.medical_specialtyNeurogenesisSubventricular zoneBiologyHypoxia (medical)ultrastructureNeural stem cellOligodendrocyteHypoxemiachronic hypoxemiamedicine.anatomical_structurenervous systemNeuroblastImmunologymedicinesubventricular germinal nichemedicine.symptomProgenitor cellHypoxianeuronal differentiationoligodendrocyte survivalOriginal Researchneural stem cellsHypoxia
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Targeting of multiple myeloma-related angiogenesis by miR-199a-5p mimics: in vitro and in vivo anti-tumor activity

2014

// Lavinia Raimondi 1 , Nicola Amodio 1 , Maria Teresa Di Martino 1 , Emanuela Altomare 1 , Marzia Leotta 1 , Daniele Caracciolo 1 , Annamaria Gulla 1 , Antonino Neri 2 , Simona Taverna 3 , Patrizia D’Aquila 4 , Riccardo Alessandro 3 , Antonio Giordano 5 , Pierosandro Tagliaferri 1 and Pierfrancesco Tassone 1,5 . 1 Department of Experimental and Clinical Medicine, Magna Graecia University and Medical Oncology Unit, T. Campanella Cancer Center, Salvatore Venuta University Campus, Catanzaro, Italy 2 Department of Medical Sciences University of Milan, Hematology1, IRCCS Policlinico Foundation, Milan, Italy 3 Department of Pathology and Forensic and Medical Biotechnology, Section of Biology and…

Pathologymedicine.medical_specialtyStromal cellAngiogenesisMultiple Myeloma; microRNA AngiogenesisBlotting WesternEnzyme-Linked Immunosorbent AssayMice SCIDIn Vitro TechniquesBiologyReal-Time Polymerase Chain ReactionTransfectionMicemiR-199-5pCell MovementMice Inbred NODSettore BIO/13 - Biologia ApplicataCell Line TumorCell AdhesionmedicineAnimalsHumansHypoxiaCell adhesionProtein kinase BCell ProliferationPlasma cell leukemiaNeovascularization PathologicmicroRNA AngiogenesisMicroRNATransfectionPlasma cell leukemiamedicine.diseaseXenograft Model Antitumor AssaysMolecular medicineCell HypoxiaMicroRNAsmedicine.anatomical_structureOncologyAngiogenesis; Hypoxia; Microenviroment; MicroRNA; miR-199-5p; MiRNA; Multiple myeloma; Plasma cell leukemiaCancer researchFemaleAngiogenesisBone marrowMicroenviromentMiRNAMultiple MyelomaResearch Paper
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The Blood–Brain Barrier as a Target in Traumatic Brain Injury Treatment

2014

Traumatic brain injury (TBI) is one of the most frequent causes of death in the young population. Several clinical trials have unsuccessfully focused on direct neuroprotective therapies. Recently immunotherapeutic strategies shifted into focus of translational research in acute CNS diseases. Cross-talk between activated microglia and blood–brain barrier (BBB) could initiate opening of the BBB and subsequent recruitment of systemic immune cells and mediators into the brain. Stabilization of the BBB after TBI could be a promising strategy to limit neuronal inflammation, secondary brain damage and acute neurodegeneration. This review provides an overview on the pathophysiology of TBI and brain…

Pathologymedicine.medical_specialtyTraumatic brain injuryPeroxisome Proliferator-Activated ReceptorsBrain EdemaInflammationBrain damageBlood–brain barrierNeuroprotectionRosiglitazoneReceptors GlucocorticoidmedicineHumansHypoglycemic AgentsMyosin-Light-Chain KinaseNeuroinflammationInflammationPioglitazoneMicrogliabusiness.industryNeurodegenerationNeurodegenerative DiseasesGeneral Medicinemedicine.diseaseCell HypoxiaNeuroprotective Agentsmedicine.anatomical_structurenervous systemBlood-Brain BarrierBrain InjuriesThiazolidinedionesmedicine.symptombusinessNeuroscienceArchives of Medical Research
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Endogenous Hypoxia Markers: Case Not Proven!

2008

The pivotal role of hypoxia within the pathophysiological framework of solid malignant tumors is now considered to be indisputable. The fact that hypoxia can cause resistance to various cancer therapies and promote malignant progression is reflected in its adverse impact on prognosis which is repeatedly shown for various tumor entities. Knowledge in this area is based on direct assessment of the oxygenation status using O2-sensitive microsensors. However, weaknesses of this standard method are its invasiveness and limitation to accessible tumor entities. Hypoxia-inducible factor (HIF)-1α, the master transcriptional regulator of the hypoxic response, as well as certain downstream genes, e.g.…

Pathologymedicine.medical_specialtyTumor hypoxiaGlucose transporterEndogenyOxygenationHypoxia (medical)BiologyIn vitromedicineTranscriptional regulationCancer researchImmunohistochemistrymedicine.symptom
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