Search results for "Homeostasis"

showing 10 items of 630 documents

The histone acetyltransferase MOF activates hypothalamic polysialylation to prevent diet-induced obesity in mice

2014

Overfeeding causes rapid synaptic remodeling in hypothalamus feeding circuits. Polysialylation of cell surface molecules is a key step in this neuronal rewiring and allows normalization of food intake. Here we examined the role of hypothalamic polysialylation in the long-term maintenance of body weight, and deciphered the molecular sequence underlying its nutritional regulation. We found that upon high fat diet (HFD), reduced hypothalamic polysialylation exacerbated the diet-induced obese phenotype in mice. Upon HFD, the histone acetyltransferase MOF was rapidly recruited on the St8sia4 polysialyltransferase-encoding gene. Mof silencing in the mediobasal hypothalamus of adult mice prevented…

medicine.medical_specialtyobesityfood intake[ SDV.BA ] Life Sciences [q-bio]/Animal biology03 medical and health sciences0302 clinical medicineInternal medicineBiologie animalemedicineGene silencinghypothalamusMolecular BiologyGene030304 developmental biology2. Zero hungerAnimal biology0303 health sciencessynaptic plasticitybiology[SDV.BA]Life Sciences [q-bio]/Animal biologypolysialylationNeurosciencesCell BiologyHistone acetyltransferasePhenotypeChromatinEndocrinologyHypothalamus[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurons and CognitionSynaptic plasticitybiology.proteinchromatinOriginal Articlehypothalamus;polysialylation;synaptic plasticity;obesity;food intake;chromatin[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]030217 neurology & neurosurgeryHomeostasis
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Immune-Inflammatory Responses and Oxidative Stress in Alzheimers Disease: Therapeutic Implications

2010

Alzheimer's disease (AD) is a heterogeneous and progressive neurodegenerative disease which in Western society mainly accounts for clinical dementia. AD has been linked to inflammation and oxidative stress. Neuro-pathological hallmarks are senile plaques, resulting from the accumulation of several proteins and an inflammatory reaction around deposits of amyloid, a fibrillar protein, Abeta, product of cleavage of a much larger protein, the beta-amyloid precursor protein (APP) and neurofibrillary tangles. Inflammation clearly occurs in pathologically vulnerable regions of AD and several inflammatory factors influencing AD development, i.e. environmental factors (pro-inflammatory phenotype) an…

medicine.medical_treatmentCellular homeostasisInflammationmedicine.disease_causeImmune systemAlzheimer DiseaseDrug DiscoverymedicineAnimalsHumansSettore MED/05 - Patologia ClinicaSenile plaquesInflammationSettore MED/04 - Patologia GeneralePharmacologychemistry.chemical_classificationReactive oxygen speciesbusiness.industrymedicine.diseaseOxidative StressCytokinechemistryImmunologyInflammation MediatorsAlzheimer's disease curcuminIL-6 inflammation oxidative stressAlzheimer's diseasemedicine.symptombusinessOxidative stressCurrent Pharmaceutical Design
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Concept of an extracellular regulation of muscular metabolic rate during heavy exercise in humans by psychophysiological feedback.

1996

Efferent motor signals to skeletal muscles concern not only the space/ time pattern of motion, but also the setting of muscular performance and through this the control of the current metabolic rate. For an optimal adjustment of metabolic rate during heavy exercise-e.g. in athletic competitions-a feedback control system must exist, including a programmer that takes into consideration a finishing point (teleoanticipation). The presented experiments, using Borg's scale, indicate the existence and functioning of a system for optimal adjustment of performance during heavy exercise and the relevance of teleoanticipatory effects. Thus motor learning includes not only somatosensory control, but al…

medicine.medical_treatmentEfferentPhysical ExertionModels PsychologicalSomatosensory systemBiofeedbackModels BiologicalRunningCellular and Molecular NeuroscienceStress PhysiologicalmedicineHomeostasisHumansMuscle SkeletalMolecular BiologySwimmingPharmacologyMotor controlBiofeedback PsychologyCell BiologyTime perceptionMetabolic control analysisTime PerceptionExercise TestMolecular MedicineRegression AnalysisMotor learningPsychologyNeuroscienceCentral governorExperientia
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Transcription factors controlling development and function of innate lymphoid cells.

2014

Abstract Innate lymphoid cells (ILCs) are a heterogeneous group of lymphocytes, which play an important role in tissue homeostasis at epithelial surfaces. They are scarce in spleen and lymph nodes, but substantial numbers can be found in the intestinal mucosa even at steady state. There, they represent the first line of defence against invading pathogens and contribute to lymphorganogenesis, tissue repair and, when inappropriately activated, immune pathology. Lineage-specific development, function and maintenance of these cells depend on a restricted set of transcription factors that partially emerged as a result of diversification and selection during vertebrate evolution. The differential…

medicine.medical_treatmentImmunologyBiologyLymphocyte ActivationIntestinal mucosaRAR-related orphan receptor gammamedicineTranscriptional regulationImmunology and AllergyAnimalsHomeostasisHumansCell LineageLymphopoiesisLymphocytesIntestinal MucosaTranscription factorTissue homeostasisInnate lymphoid cellGene Expression Regulation DevelopmentalCell DifferentiationGeneral MedicineBiological EvolutionImmunity InnateCytokineImmunologyHost-Pathogen InteractionsCytokinesInterleukin Receptor Common gamma SubunitTranscription FactorsInternational immunology
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Differential requirements for antigen or homeostatic cytokines for proliferation and differentiation of human Vgamma9Vdelta2 naive, memory and effect…

2005

We have compared four human subsets of Vgamma9Vdelta2 T cells, naive (T(naive), CD45RA(+)CD27(+)), central memory (T(CM), CD45RA(-)CD27(+)), effector memory (T(EM), CD45RA(-)CD27(-)) and terminally differentiated (T(EMRA), CD45RA(+)CD27(-)), for their capacity to proliferate and differentiate in response to antigen or homeostatic cytokines. Cytokine responsiveness and IL-15R expression were low in T(naive) cells and progressively increased from T(CM) to T(EM) and T(EMRA) cells. In contrast, the capacity to expand in response to antigen or cytokine stimulation showed a reciprocal pattern and was associated with resistance to cell death and Bcl-2 expression. Whereas antigen-stimulated cells a…

medicine.medical_treatmentT cellCellular differentiationImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationAntigenimmune system diseasesT-Lymphocyte SubsetsmedicineImmunology and AllergyHomeostasisHumansAntigensReceptorCells CulturedInterleukin-15Receptors Interleukin-15virus diseaseshemic and immune systemsCell DifferentiationReceptors Antigen T-Cell gamma-deltaReceptors Interleukin-2In vitroCell biologyTumor Necrosis Factor Receptor Superfamily Member 7Cytokinemedicine.anatomical_structureInterleukin 15CytokinesLeukocyte Common AntigensImmunologic MemoryEx vivoEuropean journal of immunology
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CD40 activity on mesenchymal cells negatively regulates OX40L to maintain bone marrow immune homeostasis under stress conditions

2021

BackgroundWithin the bone marrow (BM), mature T cells are maintained under homeostatic conditions to facilitate proper hematopoietic development. This homeostasis depends upon a peculiar elevated frequency of regulatory T cells (Tregs) and immune regulatory activities from BM-mesenchymal stem cells (BM-MSCs). In response to BM transplantation (BMT), the conditioning regimen exposes the BM to a dramatic induction of inflammatory cytokines and causes an unbalanced T-effector (Teff) and Treg ratio. This imbalance negatively impacts hematopoiesis, particularly in regard to B-cell lymphopoiesis that requires an intact cross-talk between BM-MSCs and Tregs. The mechanisms underlying the ability of…

mesenchymal cellAdultMaleCancer ResearchTransplantation ConditioningT cellbone marrow transplantationImmunologyBone Marrow CellsOX40 LigandBiologySettore MED/08 - Anatomia PatologicaLymphocyte ActivationMesenchymal Stem Cell TransplantationT-Lymphocytes RegulatoryMiceYoung AdultImmune systemBone MarrowStress PhysiologicalmedicineCD40AnimalsHomeostasisHumansImmunology and AllergyLymphopoiesisCD40 AntigensOriginal ResearchAgedCD40B-cell developmentMesenchymal Stem Cellshemic and immune systemsRC581-607Middle AgedOX40LCell biologyTransplantationHaematopoiesismedicine.anatomical_structureGene Expression Regulationbiology.proteinFemaleBone marrowImmunologic diseases. AllergyStem cellB-cell developmentbone marrow transplantation CD40 mesenchymal cell OX40L
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Bakteryjne strategie wykorzystania jonów metali – kulisy chemii koordynacyjnej

2022

n the Biological Inorganic Chemistry Group we are inspired to better understand metal ions acquisition and homeostasis in pathogenic bacteria, and in this review we present three different approaches to the role of these processes. The growing importance of a full understanding of the iron transport system in pathogens prompted us to study synthetic analogs of siderophores, used as structural probes in the process of iron uptake by microorganisms. The ferrichrome biomimetic analogs allowed efficient Fe(III) chelation under biological conditions and were recognized better by P. putida. than E. coli, suggesting differences in uptake mechanisms. Addition of a fluorescent probe to the compound …

metaloproteinymetal toxicitmetalloproteinsmetal homeostasishomeostaza metalimetaloforytoksyczność metalimetalophoresWiadomości Chemiczne
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Posttraumatic Propofol Neurotoxicity Is Mediated via the Pro–Brain-Derived Neurotrophic Factor-p75 Neurotrophin Receptor Pathway in Adult Mice*

2016

Objectives:The gamma-aminobutyric acid modulator propofol induces neuronal cell death in healthy immature brains by unbalancing neurotrophin homeostasis via p75 neurotrophin receptor signaling. In adulthood, p75 neurotrophin receptor becomes down-regulated and propofol loses its neurotoxic effect. H

musculoskeletal diseases0301 basic medicineBrain-derived neurotrophic factorProgrammed cell deathbiologybusiness.industryNeurotoxicityCaspase 3PharmacologyCritical Care and Intensive Care Medicinemedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinenervous systemAnesthesiamedicinebiology.proteinLow-affinity nerve growth factor receptorReceptorbusiness030217 neurology & neurosurgeryHomeostasisNeurotrophinCritical Care Medicine
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Adipose Stromal/Stem Cell-Derived Extracellular Vesicles: Potential Next-Generation Anti-Obesity Agents

2022

Over the last decade, several compounds have been identified for the treatment of obesity. However, due to the complexity of the disease, many pharmacological interventions have raised concerns about their efficacy and safety. Therefore, it is important to discover new factors involved in the induction/progression of obesity. Adipose stromal/stem cells (ASCs), which are mostly isolated from subcutaneous adipose tissue, are the primary cells contributing to the expansion of fat mass. Like other cells, ASCs release nanoparticles known as extracellular vesicles (EVs), which are being actively studied for their potential applications in a variety of diseases. Here, we focused on the importance …

obesityAdipogenesisQH301-705.5Organic ChemistrySubcutaneous FatMesenchymal Stem CellsGeneral Medicinemetabolic disease/syndromeSettore BIO/09 - FisiologiaCatalysisComputer Science Applicationsadipose tissueInorganic ChemistryChemistrySettore BIO/14 - Farmacologiaadipose stromal/stem cells (ASCs)Adipose stromal/stem cells (ASCs) Adipose tissue Extracellular vesicles Metabolic disease/syndrome ObesityHomeostasisHumansPhysical and Theoretical ChemistryBiology (General)extracellular vesiclesMolecular BiologyQD1-999SpectroscopyInternational Journal of Molecular Sciences
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Taste of Fat and Obesity: Different Hypotheses and Our Point of View

2022

Obesity results from a temporary or prolonged positive energy balance due to an alteration in the homeostatic feedback of energy balance. Food, with its discriminative and hedonic qualities, is a key element of reward-based energy intake. An alteration in the brain reward system for highly palatable energy-rich foods, comprised of fat and carbohydrates, could be one of the main factors involved in the development of obesity by increasing the attractiveness and consumption of fat-rich foods. This would induce, in turn, a decrease in the taste of fat. A better understanding of the altered reward system in obesity may open the door to a new era for the diagnosis, management and treatment of th…

obesityNutrition and Dieteticsfood intakeRewardreward systemNutrition. Foods and food supplyTasteHomeostasisHumansTX341-641Energy MetabolismFood Sciencefat tasteNutrients
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