Search results for "Inflammatory cytokine"

showing 10 items of 464 documents

Salivary Inflammatory Molecules as Biomarkers of Sleep Alterations: A Scoping Review.

2021

Poor sleep quality and sleep disorders are the most common problems in people, affecting health-related quality of life. Various studies show an association between sleep disorders and altered levels of stress hormones and inflammatory cytokines measured in saliva. The main objective of this article is to provide an analysis of the current evidence related to changes in inflammatory markers in the saliva and their associations with sleep quality measurement (both objective and subjective methods) in healthy subjects and in sleep-related disorders. To that end, a scoping review was carried out, following the PRISMA criteria in the bibliographic search in several databases: PubMed, EBSCO, and…

0301 basic medicineSalivaClinical BiochemistryPopulationAlpha (ethology)InflammationReviewBioinformaticsProinflammatory cytokine03 medical and health sciences0302 clinical medicineQuality of lifecytokineMedicinesleepeducationeducation.field_of_studyChronobiologylcsh:R5-920salivabusiness.industrybiomarkersSleep in non-human animals030104 developmental biologyinflammationmedicine.symptombusinesslcsh:Medicine (General)030217 neurology & neurosurgeryDiagnostics (Basel, Switzerland)
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Interaction between ROR1 and MuSK activation complex in myogenic cells

2017

The ROR family of receptor tyrosine kinases, ROR1 and ROR2, is known to play an important role during skeletal muscle regeneration. ROR1 has a critical role in regulating satellite cell (SC) proliferation during muscle regeneration, and proinflammatory cytokines such as TNF-α and IL-1β can induce expression of ROR1 in myogenic cells via NF-κB activation. While searching for ROR1-interacting proteins in myogenic cells, we identified MuSK as a ROR1-binding protein. MuSK interacts with and phosphorylates ROR1 at the cytoplasmic proline-rich domain. ROR1 also interacts with the MuSK activator Dok-7 independently of MuSK interaction. Collectively, our results identified ROR1 as a new interacting…

0301 basic medicineSatellite Cells Skeletal MuscleBiophysicsMuscle ProteinsReceptor Tyrosine Kinase-like Orphan ReceptorsBiochemistryReceptor tyrosine kinaseCell LineProinflammatory cytokineMice03 medical and health sciencesProtein DomainsStructural BiologyChlorocebus aethiopsGeneticsAnimalsHumansReceptors CholinergicProtein phosphorylationPhosphorylationMolecular BiologyCell ProliferationBinding SitesbiologyKinaseChemistryActivator (genetics)Receptor Protein-Tyrosine KinasesCell DifferentiationROR2Cell BiologyCell biologyHEK293 Cells030104 developmental biologyCOS CellsROR1biology.proteinPhosphorylationProtein BindingFEBS Letters
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Monocyte-derived inflammatory Langerhans cells and dermal dendritic cells mediate psoriasis-like inflammation

2016

Dendritic cells (DCs) have been implicated in the pathogenesis of psoriasis but the roles for specific DC subsets are not well defined. Here we show that DCs are required for psoriasis-like changes in mouse skin induced by the local injection of IL-23. However, Flt3L-dependent DCs and resident Langerhans cells are dispensable for the inflammation. In epidermis and dermis, the critical DCs are TNF-producing and IL-1β-producing monocyte-derived DCs, including a population of inflammatory Langerhans cells. Depleting Ly6Chi blood monocytes reduces DC accumulation and the skin changes induced either by injecting IL-23 or by application of the TLR7 agonist imiquimod. Moreover, we find that IL-23-…

0301 basic medicineSciencePopulationGeneral Physics and Astronomychemical and pharmacologic phenomenaInflammationInterleukin-23ArticleMonocytesGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineMajor Histocompatibility ComplexMice03 medical and health sciencesAdjuvants ImmunologicPsoriasismedicineInterleukin 23AnimalsHumansPsoriasiseducationSkinInflammationMice Knockouteducation.field_of_studyImiquimodMultidisciplinaryFollicular dendritic cellsbusiness.industryMonocyteQMembrane Proteinshemic and immune systemsDendritic CellsGeneral ChemistryTLR7medicine.disease3. Good health030104 developmental biologymedicine.anatomical_structureGene Expression RegulationLangerhans CellsImmunologyAminoquinolinesDrug Eruptionsmedicine.symptombusinessNature Communications
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Anti-inflammatory and cognitive effects of interferon-β1a (IFNβ1a) in a rat model of Alzheimer’s disease

2018

Background: Aβ 1-42 peptide abnormal production is associated with the development and maintenance of neuroinflammation and oxidative stress in brains from Alzheimer disease (AD) patients. Suppression of neuroinflammation may then represent a suitable therapeutic target in AD. We evaluated the efficacy of IFNβ1a in attenuating cognitive impairment and inflammation in an animal model of AD. Methods: A rat model of AD was obtained by intra-hippocampal injection of Aβ 1-42 peptide (23 μg/2 μl). After 6 days, 3.6 μg of IFNβ1a was given subcutaneously (s.c.) for 12 days. Using the novel object recognition (NOR) test, we evaluated changes in cognitive function. Measurement of pro-inflammatory or …

0301 basic medicineTime Factorsmedicine.medical_treatmentHippocampusCell CountPharmacologymedicine.disease_causeHippocampuslcsh:RC346-429Superoxide Dismutase-10302 clinical medicineNeuroinflammationNF-kBMicrogliaGeneral NeuroscienceMicrofilament ProteinsROSPro-inflammatory cytokineIFNβ1amedicine.anatomical_structureCytokineNeurologyIL-10CytokinesFemalemedicine.symptomAlzheimer's diseaseInterferon beta-1aPro-inflammatory cytokinesImmunologyAβ 1-42InflammationProinflammatory cytokine03 medical and health sciencesCellular and Molecular NeuroscienceHippocampuAlzheimer DiseaseGlial Fibrillary Acidic ProteinmedicineAnimalsAβ1-42Rats WistarSODMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationInflammationAmyloid beta-PeptidesNeuroscience (all)Superoxide Dismutasebusiness.industryResearchCalcium-Binding ProteinsRecognition Psychologymedicine.diseasePeptide FragmentsRatsDisease Models Animal030104 developmental biologyLipid PeroxidationCognition DisordersReactive Oxygen Speciesbusiness030217 neurology & neurosurgeryOxidative stressJournal of Neuroinflammation
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Calcifediol-loaded liposomes for local treatment of pulmonary bacterial infections.

2017

The influence of vitamin D3 and its metabolites calcifediol (25(OH)D) and calcitriol on immune regulation and inflammation is well described, and raises the question of potential benefit against bacterial infections. In the current study, 25(OH)D was encapsulated in liposomes to enable aerosolisation, and tested for the ability to prevent pulmonary infection by Pseudomonas aeruginosa. Prepared 25(OH)D-loaded liposomes were nanosized and monodisperse, with a negative surface charge and a 25(OH)D entrapment efficiency of approximately 23%. Jet nebulisation of liposomes was seen to yield an aerosol suitable for tracheo-bronchial deposition. Interestingly, 25(OH)D in either liposomes or ethanol…

0301 basic medicineVitaminRMCalcitriolCystic FibrosisPharmaceutical ScienceInflammationBronchiBiologyPharmacologymedicine.disease_causeMicrobiologyProinflammatory cytokineCell Line03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicinePseudomonas infectionAdministration InhalationmedicineAnimalsHumansImmunologic FactorsPseudomonas InfectionsRespiratory Tract InfectionsCalcifediolLiposomePseudomonas aeruginosaEpithelial CellsGeneral Medicinemedicine.disease030104 developmental biology030228 respiratory systemchemistryLiposomesPseudomonas aeruginosaCytokinesNanoparticlesCalcifediolmedicine.symptomBiotechnologymedicine.drug
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Impact of polymer-modified gold nanoparticles on brain endothelial cells: exclusion of endoplasmic reticulum stress as a potential risk factor

2016

A library of polymer-coated gold nanoparticles (AuNPs) differing in size and surface modifications was examined for uptake and induction of cellular stress responses in the endoplasmic reticulum (ER stress) in human brain endothelial cells (hCMEC/D3). ER stress is known to affect the physiology of endothelial cells (ECs) and may lead to inflammation or apoptosis. Thus, even if applied at non-cytotoxic concentrations ER stress caused by nanoparticles should be prevented to reduce the risk of vascular diseases and negative effects on the integrity of barriers (e.g. blood-brain barrier). We exposed hCMEC/D3 to twelve different AuNPs (three sizes: 18, 35, and 65 nm, each with four surface-modif…

0301 basic medicineXBP1BiPCell SurvivalPolymersBiomedical EngineeringMetal NanoparticlesApoptosis02 engineering and technologyBiologyEndoplasmic ReticulumToxicologyArticleCell LineProinflammatory cytokine03 medical and health sciencescell stressDownregulation and upregulationRisk FactorsHeat shock proteinAnimalsHumansHSP70 Heat-Shock ProteinsParticle SizeHeat-Shock ProteinsATF6Endoplasmic reticulumInterleukin-8ATF4Endothelial CellsMembrane Proteinsunfolded protein responseEndoplasmic Reticulum Stress021001 nanoscience & nanotechnologyQPActivating Transcription Factor 4Cell biology030104 developmental biologyBlood-Brain Barriertight junction proteinsImmunologyUnfolded protein responseGold0210 nano-technologyTranscription Factor CHOPNanotoxicology
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Functions and Therapeutic Potential of Extracellular Hsp60, Hsp70, and Hsp90 in Neuroinflammatory Disorders

2021

Neuroinflammation is implicated in central nervous system (CNS) diseases, but the molecular mechanisms involved are poorly understood. Progress may be accelerated by developing a comprehensive view of the pathogenesis of CNS disorders, including the immune and the chaperone systems (IS and CS). The latter consists of the molecular chaperones; cochaperones; and chaperone cofactors, interactors, and receptors of an organism and its main collaborators in maintaining protein homeostasis (canonical function) are the ubiquitin–proteasome system and chaperone-mediated autophagy. The CS has also noncanonical functions, for instance, modulation of the IS with induction of proinflammatory cytokines. …

0301 basic medicineamyotrophic lateral sclerosislcsh:TechnologychaperonopathiesProinflammatory cytokinelcsh:Chemistrys disease03 medical and health sciences0302 clinical medicinechaperone systemmedicineamyotrophic lateral sclerosiGeneral Materials Sciencelcsh:QH301-705.5InstrumentationchaperonotherapyNeuroinflammationFluid Flow and Transfer Processesbiologylcsh:TMechanism (biology)Process Chemistry and Technologymolecular chaperonesNeurodegenerationAutophagyGeneral EngineeringParkinson’S diseasemolecular chaperonemedicine.diseaseHuntington’ s diseaseHsp90lcsh:QC1-999Computer Science Applications030104 developmental biologylcsh:Biology (General)lcsh:QD1-999lcsh:TA1-2040multiple sclerosiChaperone (protein)Alzheimerbiology.proteinHSP60lcsh:Engineering (General). Civil engineering (General)Alzheimer’s diseaseNeurosciencelcsh:Physics030217 neurology & neurosurgeryHuntington’s diseaseApplied Sciences
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Induction of Autophagy by Pterostilbene Contributes to the Prevention of Renal Fibrosis via Attenuating NLRP3 Inflammasome Activation and Epithelial-…

2020

Chronic kidney disease (CKD) is recognized as a global public health problem. NLRP3 inflammasome activation has been characterized to mediate diverse aspect mechanisms of CKD through regulation of proinflammatory cytokines, tubulointerstitial injury, glomerular diseases, renal inflammation, and fibrosis pathways. Autophagy is a characterized negative regulation mechanism in the regulation of the NLRP3 inflammasome, which is now recognized as the key regulator in the pathogenesis of inflammation and fibrosis in CKD. Thus, autophagy is undoubtedly an attractive target for developing new renal protective treatments of kidney disease via its potential effects in regulation of inflammasome. Howe…

0301 basic medicineautophagypterostilbeneATG5epithelial-mesenchymal transitionInflammationProinflammatory cytokine03 medical and health sciencesCell and Developmental Biology0302 clinical medicineFibrosismedicineRenal fibrosisEpithelial–mesenchymal transitionlcsh:QH301-705.5Original Researchbusiness.industryAutophagyInflammasomeCell Biologymedicine.diseaserenal fibrosisNLRP3 inflammasome030104 developmental biologylcsh:Biology (General)030220 oncology & carcinogenesisCancer researchmedicine.symptombusinessDevelopmental Biologymedicine.drugFrontiers in Cell and Developmental Biology
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Protective Effect of Pogostone on 2,4,6-Trinitrobenzenesulfonic Acid-Induced Experimental Colitis via Inhibition of T Helper Cell

2017

Inflammatory bowel disease (IBD) is a chronic immune-related disease mainly caused by the disequilibrium of T helper (Th) cell paradigm? Pogostone (PO) is one of the major chemical constituents of Pogostemon cablin (Blanco) Benth. The present study aims to investigate the potential benefit of PO against IBD in a 2,4,6-trinitrobenzenesulfonic acid (TNBS)-induced experimental colitis model. PO treatment by enema significantly brought down the disease activity index (DAI) of the TNBS-challenged rats, which was manifested by the ameliorated inflammatory features including ulceration, adhesion, and edema. Hematoxylin-eosin (HE) staining and immunohistochemistry analysis showed that PO effectivel…

0301 basic medicineexperimental colitisCellPharmacologyInflammatory bowel diseaseProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound246-Trinitrobenzenesulfonic acidEdemamedicinePharmacology (medical)T helper cellOriginal ResearchPharmacologybiologyCell growthbusiness.industrylcsh:RM1-950pogostoneT helper cellmedicine.diseaseTNBSanti-inflammationdigestive system diseaseslcsh:Therapeutics. Pharmacology030104 developmental biologymedicine.anatomical_structurechemistryMyeloperoxidaseImmunologybiology.proteinmedicine.symptombusinessFrontiers in Pharmacology
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Bioactive Polyphenols from Pomegranate Juice Reduce 5-Fluorouracil-Induced Intestinal Mucositis in Intestinal Epithelial Cells

2020

Intestinal epithelial cells (IECs) play a pivotal role in maintaining intestinal homeostasis. Different noxious agents, among them also anticancer therapies, can impair intestinal epithelial integrity triggering inflammation and oxidative stress. A frequent complication of chemotherapy is gastrointestinal mucositis, strongly influencing the effectiveness of therapy, increasing healthcare costs, and impairing patients&rsquo

0301 basic medicinegastrointestinal mucositionconutraceuticalPhysiologyClinical BiochemistryInflammationPharmacologymedicine.disease_causeBiochemistryArticleProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound0302 clinical medicineMucositisMedicineoxidative stress5-fluorouracilMolecular BiologyPunica granatum L.polyphenolsoxidative strebiologybusiness.industryNitrotyrosinegastrointestinal mucositislcsh:RM1-950intestinal epithelial cellCell BiologyPunica granatum Lmedicine.diseasebiology.organism_classification5‐fluorouracil; Gastrointestinal mucositis; Inflammation; Intestinal epithelial cells; Onconutraceutical; Oxidative stress; Polyphenols; Punica granatum L5‐fluorouracilpolyphenollcsh:Therapeutics. Pharmacology030104 developmental biologychemistryApoptosisPolyphenolinflammation030220 oncology & carcinogenesisPunicaintestinal epithelial cellsmedicine.symptombusinessOxidative stressAntioxidants
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