Search results for "La Protein"

showing 10 items of 245 documents

Tenectin is a novel alphaPS2betaPS integrin ligand required for wing morphogenesis and male genital looping in Drosophila.

2010

International audience; Morphogenesis of the adult structures of holometabolous insects is regulated by ecdysteroids and juvenile hormones and involves cell-cell interactions mediated in part by the cell surface integrin receptors and their extracellular matrix (ECM) ligands. These adhesion molecules and their regulation by hormones are not well characterized. We describe the gene structure of a newly described ECM molecule, tenectin, and demonstrate that it is a hormonally regulated ECM protein required for proper morphogenesis of the adult wing and male genitalia. Tenectin's function as a new ligand of the PS2 integrins is demonstrated by both genetic interactions in the fly and by cell s…

MaleMESH: Extracellular Matrix ProteinsMESH: DrosophilaMESH : Immunohistochemistry[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionIntegrinLigandsLooping morphogenesisExtracellular matrixchemistry.chemical_compound0302 clinical medicineMESH: Genitalia MaleMorphogenesisMESH: LigandsDrosophila ProteinsWings AnimalMESH: AnimalsTransgenesIn Situ Hybridization0303 health sciencesExtracellular Matrix ProteinsMESH : Genitalia MaleMESH : LigandsIntegrin alpha ChainsCell adhesion moleculeMESH : In Situ HybridizationImmunohistochemistry3. Good healthCell biologyLarvaMESH : Integrin alpha ChainsAdhesionDrosophilaMESH : MutationMESH : TransgenesTenectinIntegrin alpha ChainsDrosophila ProteinEcdysoneEcdysoneMESH: MutationMESH: Drosophila ProteinsMESH : MaleIntegrinMorphogenesisMESH : WingMESH: TransgenesBiologyGenitalia MaleArticle03 medical and health sciencesMESH : Extracellular Matrix ProteinsMESH: In Situ HybridizationAnimalsMESH : DrosophilaCell adhesionMolecular Biology030304 developmental biologyMESH : LarvaMetamorphosisMESH: Integrin alpha ChainsLeft–right asymmetryMESH: ImmunohistochemistryCell BiologyMESH : Drosophila ProteinsMESH: WingMESH: MaleMESH: MorphogenesischemistryMESH : MorphogenesisMutationbiology.proteinMESH : AnimalsMESH: Larva[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDevelopmental Biology
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Genetic identification of neurons controlling a sexually dimorphic behaviour

2000

0960-9822 (Print) Journal Article Research Support, Non-U.S. Gov't; In the fruit fly Drosophila melanogaster, locomotor activity is sexually dimorphic: female flies constantly modulate their activity pattern whereas males show a steadier, stereotyped walking pace [1]. Here, we mapped the area of the brain controlling this behavioural dimorphism. Adult male Drosophila expressing a dominant feminising transgene in a small cluster of neurons in the pars intercerebralis exhibited a female-like pattern of locomotor activity. Genetic ablation of these neurons prevented the feminisation of the locomotor activity of transgenic males. The results suggest that this cluster of neurons modulates sex-sp…

MaleMESH: NeuronsCourtshipAnimals Genetically ModifiedSexual Behavior Animal0302 clinical medicineMESH: Saccharomyces cerevisiae ProteinsDrosophila ProteinsNervous System Physiological PhenomenaMESH: AnimalsMESH: Sexual Behavior AnimalDrosophila melanogaster/*physiologymedia_commonNeurons0303 health sciencesFungal proteinSex CharacteristicsbiologyAgricultural and Biological Sciences(all)Nuclear ProteinsAnatomyMESH: Transcription FactorsMotor Activity/*physiologyMESH: Motor ActivityDNA-Binding ProteinsFungal Proteins/geneticsNuclear Proteins/*genetics/physiologyDrosophila melanogasterMESH: Fungal Proteins[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]FemaleDrosophila melanogasterGeneral Agricultural and Biological SciencesLocomotionSex characteristicsMESH: Sex CharacteristicsNervous System PhysiologySaccharomyces cerevisiae ProteinsTransgenemedia_common.quotation_subjectRecombinant Fusion ProteinsRecombinant Fusion Proteins/biosynthesisSexual BehaviorMESH: LocomotionTranscription Factors/geneticsGenetically ModifiedMotor ActivityGeneral Biochemistry Genetics and Molecular BiologyMESH: Drosophila melanogasterFungal ProteinsMESH: Animals Genetically Modified03 medical and health sciencesMESH: Recombinant Fusion ProteinsAnimalsDrosophila030304 developmental biologyBiochemistry Genetics and Molecular Biology(all)Animalfungibiology.organism_classificationMESH: MaleSexual dimorphismMale courtship behaviourMESH: Nervous System PhysiologyNeuroscienceMESH: FemaleMESH: Nuclear ProteinsNeurons/*physiology030217 neurology & neurosurgeryTranscription Factors
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Drosophila female courtship and mating behaviors: sensory signals, genes, neural structures and evolution.

2010

International audience; Interest in Drosophila courtship behavior has a long-standing tradition, starting with the works by Sturtevant in 1915, and by Bastock and Manning in the 50s. The neural and genetic base of Drosophila melanogaster courtship behavior has made big strides in recent years, but the studies on males far outnumber those on females. Recent technical developments have made it possible to begin to unravel the biological substrates underlying the complexity of Drosophila female sexual behavior and its decisive effect on mating success. The present review focus more on the female side and summarizes the sensory signals that the male sends, using multiple channels, and which neu…

MaleMESH: Signal Transduction[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH: NeuronsCourtshipSexual Behavior AnimalMESH : Neural PathwaysMESH : Biological EvolutionNeural PathwaysMESH : Drosophila melanogasterDrosophila ProteinsMESH : FemaleMESH: AnimalsMatingMESH: Sexual Behavior Animalmedia_commonNeuronsbiologyGeneral NeuroscienceBiological EvolutionDrosophila melanogasterFemaleDrosophila melanogasterDrosophila ProteinSignal TransductionMESH: Drosophila ProteinsMESH : Malemedia_common.quotation_subjectMESH: CourtshipSensory systemMESH: Biological EvolutionMESH : NeuronsMESH: Drosophila melanogasterBiological neural networkAnimalsDrosophila (subgenus)MESH : Sexual Behavior AnimalMESH : Signal TransductionMESH : CourtshipCourtship displayMESH: Neural PathwaysfungiCourtshipMESH : Drosophila Proteinsbiology.organism_classificationMESH: MaleMESH : AnimalsNeuroscienceMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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TBC1D24-TLDc-related epilepsy exercise-induced dystonia: rescue by antioxidants in a disease model

2019

Genetic mutations in TBC1D24 have been associated with multiple phenotypes, with epilepsy being the main clinical manifestation. The TBC1D24 protein consists of the unique association of a Tre2/Bub2/Cdc16 (TBC) domain and a TBC/lysin motif domain/catalytic (TLDc) domain. More than 50 missense and loss-of-function mutations have been described and are spread over the entire protein. Through whole genome/exome sequencing we identified compound heterozygous mutations, R360H and G501R, within the TLDc domain, in an index family with a Rolandic epilepsy exercise-induced dystonia phenotype (http://omim.org/entry/608105). A 20-year long clinical follow-up revealed that epilepsy was self-limited in…

MaleModels Molecular0301 basic medicineProtein ConformationAmino Acid Motifsalpha-TocopherolMutantCrystallography X-RayPHENOTYPECompound heterozygosityAntioxidantsAnimals Genetically ModifiedEpilepsy0302 clinical medicineCatalytic DomainDrosophila ProteinsMissense mutationoxidative stressChildTLDC DOMAINVITAMIN-EExome sequencingSequence DeletionNeuronsDystoniaGeneticsexercise-induced dystoniaTBC1D24GTPase-Activating ProteinsANNOTATIONSEpilepsy RolandicPhenotypeRecombinant ProteinsPedigree3. Good healthRolandic epilepsyDystoniaDrosophila melanogasterChild PreschoolFemaleSettore MED/26 - NeurologiaSynaptic VesiclesDrosophila melanogasterPROTEIN STABILITYLife Sciences & BiomedicineLocomotionAdolescentPhysical ExertionMutation MissenseClinical NeurologyPREDICTIONSBiology03 medical and health sciencesmedicineAnimalsHumansAmino Acid SequenceCOMPARTMENToxidative streScience & TechnologySequence Homology Amino AcidMUTATIONSNeurosciencesInfantBiological TransportDEGRADATIONmedicine.diseasebiology.organism_classificationAcetylcysteineDisease Models AnimalOxidative Stress030104 developmental biologyrab GTP-Binding ProteinsSEIZURESNeurosciences & NeurologyNeurology (clinical)Reactive Oxygen SpeciesSequence Alignment030217 neurology & neurosurgery
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Evolution of sex chromosomes: dosage compensation of the Lcp1-4 gene cluster on the evolving neo-X chromosome in Drosophila miranda.

2007

In Drosophila miranda the small multigene family of the larval cuticle protein (Lcp1-4) genes resides on the evolving neo-X and neo-Y sex chromosome pair while in the sibling species Drosophila pseudoobscura and Drosophila persimilis the gene cluster is inherited autosomally. The neo-Y chromosomal Lcp1, Lcp2 and Lcp4 genes are, as previously shown by us, not expressed and only Lcp3 is expressed at a strongly reduced level. As a first step in understanding the evolutionary mechanism(s) transforming an autosome into a dosage compensated X we analysed the expression behaviour and promoter structure of the Lcp1-4 genes on the neo-X. The normalized relative expression levels reveal that all four…

MaleMolecular Sequence DataGenes InsectDrosophila pseudoobscuraGenes Y-LinkedGenes X-LinkedDosage Compensation GeneticSequence Homology Nucleic AcidGene clusterGeneticsAnimalsDrosophila ProteinsPromoter Regions GeneticMolecular BiologyGeneX chromosomeDrosophila persimilisGeneticsDosage compensationAutosomebiologyBase Sequencefungibiology.organism_classificationEvolutionary biologyInsect ScienceMultigene FamilyDrosophilaFemaleDrosophila ProteinInsect molecular biology
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Mild mutations in the pan neural gene prospero affect male-specific behaviour in Drosophila melanogaster

2004

0376-6357 (Print) Journal Article Research Support, Non-U.S. Gov't; The fruitfly Drosophila melanogaster is one of the most appropriate model organisms to study the genetics of behaviour. Here, we focus on prospero (pros), a key gene for the development of the nervous system which specifies multiple aspects from the early formation of the embryonic central nervous system to the formation of larval and adult sensory organs. We studied the effects on locomotion, courtship and mating behaviour of three mild pros mutations. These newly isolated pros mutations were induced after the incomplete excision of a transposable genomic element that, before excision, caused a lethal phenotype during larv…

MaleMutantPoint Mutation/*geneticsSexual Behavior AnimalBehavioral NeuroscienceAnimal/*physiologyDrosophila ProteinsGeneticsBehavior AnimalbiologyReproductionHomozygoteNuclear ProteinsGeneral MedicinePhenotypeNerve Tissue Proteins/*geneticshumanitiesDNA Transposable Elements/geneticsDrosophila melanogasterLocomotion/physiologyFemaleDrosophila melanogasterLocomotionHeterozygoteFertility/physiologySexual BehavioreducationNerve Tissue ProteinsTranscription Factors/*geneticsAnimal/physiologyDrosophilidaeNuclear Proteins/*geneticsPoint MutationAnimalsAlleleGeneDrosophilaReproduction/physiologyAllelesBehaviorfungiDrosophila Proteins/*geneticsHeterozygote advantageRepressor Proteins/*geneticsbiology.organism_classificationRepressor ProteinsFertilityDNA Transposable ElementsAnimal Science and ZoologyTranscription Factors
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Control of apterous by vestigial drives indirect flight muscle development in drosophila

2003

0012-1606 (Print) Journal Article Research Support, Non-U.S. Gov't; Drosophila thoracic muscles are comprised of both direct flight muscles (DFMs) and indirect flight muscles (IFMs). The IFMs can be further subdivided into dorsolongitudinal muscles (DLMs) and dorsoventral muscles (DVMs). The correct patterning of each category of muscles requires the coordination of specific executive regulatory programs. DFM development requires key regulatory genes such as cut (ct) and apterous (ap), whereas IFM development requires vestigial (vg). Using a new vg(null) mutant, we report that a total absence of vg leads to DLM degeneration through an apoptotic process and to a total absence of DVMs in the …

MaleNerve Tissue Proteins/genetics/metabolismMuscle Fibers SkeletalMutantTranscription Factors/genetics/*metabolismmedicine.disease_causeMyoblastsTwist transcription factorMyoblasts/physiologyDrosophila ProteinsWings AnimalDevelopmentalCells CulturedRegulator geneRegulation of gene expressionWing/growth & development/physiologyMutationCulturedMusclesGene Expression Regulation DevelopmentalNuclear ProteinsDrosophila Proteins/genetics/*metabolismAnatomyMuscle degenerationCell biologytwistDrosophilacutMuscles/metabolism/pathology/*physiologyIndirect flight musclesCellsLIM-Homeodomain ProteinsMuscle Fibers/pathology/physiologyNerve Tissue ProteinsBiologyvestigialNuclear Proteins/genetics/*metabolismmedicineHomeodomain Proteins/genetics/*metabolismAnimalsDrosophila/*growth & developmentDrosophilaMolecular BiologyHomeodomain ProteinsTwist-Related Protein 1Cell Biologybiology.organism_classificationapterousTwist Transcription FactorGene Expression RegulationMutationEctopic expressionTranscription FactorsDevelopmental BiologyDevelopmental Biology
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Courtship Behavior of Brain Mosaics in Drosophila

2000

0167-7063 (Print) Journal Article Research Support, Non-U.S. Gov't; Sites in the brain that show functional, sexual dimorphism in courtship behavior have been mapped at high resolution in male/female mosaics of Drosophila melanogaster. The sex mosaics were produced by enhancer-trap expression of GAL4 driving the female-spliced form of the transformer gene (tra), revealing sites in the dorsal brain, lateral protocerebrum, suboesophageal, thoracic and abdominal ganglia, and suggesting the importance of cross-talk between these regions in the implementation of the courtship sequence.

MaleNuclear Proteins/analysis/*geneticsProtocerebrumNervous systemDorsumanimal structuresSexual Behaviormedia_common.quotation_subjectGene ExpressionHigh resolutionGenetically ModifiedBiologyNervous SystemAnimals Genetically ModifiedCourtshipSexual Behavior AnimalCellular and Molecular NeuroscienceGeneticsmedicineDrosophila ProteinsAnimalsCluster AnalysisDrosophila melanogaster/*geneticsNervous System/*chemistrymedia_commonBrain ChemistryGeneticsCourtship displayHistocytochemistryMosaicismAnimalfungiNuclear Proteinsbiology.organism_classificationSexual dimorphismDrosophila melanogastermedicine.anatomical_structureEvolutionary biologyGangliaFemaleDrosophila melanogasterGanglia/chemistryJournal of Neurogenetics
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Sequence variation in couch potato and its effects on life-history traits in a northern malt fly, Drosophila montana

2011

Abstract Couch potato ( cpo ) has previously been connected to reproductive diapause in several insect species including Drosophila melanogaster , where it has been suggested to provide a link between the insulin signalling pathway and the hormonal control of diapause. In the first part of the study we sequenced nearly 3.6 kb of this gene in a northern Drosophila species ( Drosophila montana ) with a robust photoperiodically determined diapause and found several types of polymorphisms along the sequenced area. We also found variation among five Drosophila virilis group species in the length of the 5th exon of cpo and in the site of the stop codon at the end of this exon. The second part of …

MalePhysiologyAmino Acid MotifsMolecular Sequence DataPopulationDiapauseExonSpecies SpecificityAnimalsDrosophila ProteinsAmino Acid SequenceeducationGeneConserved SequenceSequence DeletionGeneticseducation.field_of_studyPolymorphism GeneticSequence Homology Amino AcidbiologyWild typeNuclear ProteinsExonsSequence Analysis DNAbiology.organism_classificationIntronsStop codonDrosophila virilisPhenotypeInsect ScienceDrosophilaFemaleDrosophila melanogasterSequence AlignmentJournal of Insect Physiology
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Genetic identification of a network of factors that functionally interact with the nucleosome remodeling ATPase ISWI.

2008

Nucleosome remodeling and covalent modifications of histones play fundamental roles in chromatin structure and function. However, much remains to be learned about how the action of ATP-dependent chromatin remodeling factors and histone-modifying enzymes is coordinated to modulate chromatin organization and transcription. The evolutionarily conserved ATP-dependent chromatin-remodeling factor ISWI plays essential roles in chromosome organization, DNA replication, and transcription regulation. To gain insight into regulation and mechanism of action of ISWI, we conducted an unbiased genetic screen to identify factors with which it interacts in vivo. We found that ISWI interacts with a network o…

MaleProteomicsCancer Researchlcsh:QH426-470Histone Deacetylase 1BiologySettore MED/08 - Anatomia PatologicaChromosomesHistone DeacetylasesChromatin remodelingHistonesHistone H403 medical and health sciences0302 clinical medicineGenetics and Genomics/EpigeneticsGeneticsAnimalsDrosophila ProteinsNucleosomeMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and Systematics030304 developmental biologyAdenosine TriphosphatasesGenetics0303 health sciencesNuclear ProteinsAcetylationChromatin Assembly and DisassemblyChromatinNucleosomesChromatiniswi drosophilaRepressor ProteinsChromatin epigeneticsHDAC Chromatin RemodellingSin3 Histone Deacetylase and Corepressor Complexlcsh:GeneticsDrosophila melanogasterHistoneHistone deacetylase complexbiology.proteinFemaleHistone deacetylaseHistone deacetylase activity030217 neurology & neurosurgeryResearch ArticleTranscription Factors
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