Search results for "MESH : animals"

showing 10 items of 99 documents

Peroxisome proliferator-activated receptor alpha deficiency impairs regulatory T cell functions: Possible application in the inhibition of melanoma t…

2016

International audience; Regulatory T (Treg) cells are important to induce and maintain immunological self-tolerance. Although the progress accomplished in understanding the functional mechanism of Treg cells, intracellular molecules that control the mechanisms of their suppressive capacity are still on investigation. The present study showed that peroxisome proliferator-activated receptor-alpha deficiency impaired the suppressive activity of Treg cells on CD4(+)CD25(-) and CD8(+) T cell proliferation. In Treg cells, PPARα gene deletion also induced a decrease of migratory abilities, and downregulated the expression of chemokine receptors (CCR-4, CCR-8 and CXCR-4) and p27(KIP1) mRNA. Treg ce…

0301 basic medicineMaleAdoptive cell transferMESH: Tumor BurdenB16 melanoma tumorMelanoma ExperimentalMESH: T-Lymphocyte SubsetsCD4(+)CD25(+) regulatory T cellsBiochemistryMESH: Mice KnockoutImmunotherapy AdoptiveT-Lymphocytes RegulatoryPPARαMESH : T-Lymphocytes RegulatoryCell MovementT-Lymphocyte SubsetsMESH: Reverse Transcriptase Polymerase Chain ReactionMESH : Cell ProliferationMESH : Cell MovementMESH: AnimalsIL-2 receptorMESH: PPAR alphaMESH: Cell MovementCells CulturedMice KnockoutMESH : Melanoma ExperimentalbiologyMESH : Tumor BurdenReverse Transcriptase Polymerase Chain ReactionMESH : Reverse Transcriptase Polymerase Chain ReactionFOXP3hemic and immune systemsGeneral MedicineMESH: Gene Expression Regulation Neoplastic3. Good healthTumor BurdenMESH: Melanoma ExperimentalDNA-Binding ProteinsGene Expression Regulation Neoplasticmedicine.anatomical_structureMESH: Immunotherapy AdoptiveReceptors ChemokineMESH : DNA-Binding ProteinsMESH: Cells Culturedmedicine.medical_specialtyMESH : Receptors ChemokineMESH: Cell Line TumorRegulatory T cellMESH : Gene Expression Regulation NeoplasticT cellMESH : MaleMESH : PPAR alphachemical and pharmacologic phenomenaMESH : Mice Inbred C57BLMESH : Clonal Anergy03 medical and health sciencesMESH: Mice Inbred C57BLInternal medicineMESH: Cell ProliferationCell Line TumorMESH : Cells CulturedmedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyPPAR alpha[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyCell ProliferationClonal AnergyPerforinMESH : Cell Line TumorMESH: T-Lymphocytes RegulatoryMolecular biologyMESH: MaleMESH : T-Lymphocyte SubsetsGranzyme BMice Inbred C57BL030104 developmental biologyEndocrinologyPerforinMESH: Clonal Anergybiology.proteinMESH : Mice KnockoutMESH : AnimalsMESH: Receptors ChemokineCD8MESH: DNA-Binding ProteinsMESH : Immunotherapy Adoptive
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Consequences of organ choice in describing bacterial pathogen assemblages in a rodent population

2017

SUMMARYHigh-throughput sequencing technologies now allow for rapid cost-effective surveys of multiple pathogens in many host species including rodents, but it is currently unclear if the organ chosen for screening influences the number and identity of bacteria detected. We used 16S rRNA amplicon sequencing to identify bacterial pathogens in the heart, liver, lungs, kidneys and spleen of 13 water voles (Arvicola terrestris) collected in Franche-Comté, France. We asked if bacterial pathogen assemblages within organs are similar and if all five organs are necessary to detect all of the bacteria present in an individual animal. We identified 24 bacteria representing 17 genera; average bacterial…

0301 basic medicineOperational taxonomic unitMESH: Sequence Analysis DNAEpidemiologyMESH : PrevalenceMESH : Tissue DistributionRodent DiseasesRNA Ribosomal 16Sbacterial pathogensPrevalenceMESH : DNA BacterialTissue DistributionMESH: AnimalsPathogen[SDV.EE]Life Sciences [q-bio]/Ecology environmenteducation.field_of_studybiologyEcologyArvicolinaeMicrobiotaMESH : Rodent Diseases3. Good healthMESH: RNA Ribosomal 16SInfectious DiseasesArvicolinaeFrancerodent-borne pathogenDNA Bacterial030106 microbiologyPopulationShort ReportZoology[ SDV.EE ] Life Sciences [q-bio]/Ecology environment03 medical and health sciencesAnimalsMESH: MicrobiotaMESH : BacteriaMESH: Tissue DistributionArvicola terrestriseducationMESH : FranceMESH: Prevalence[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyBacteriaHost (biology)tissue selectionBacteria PresentSequence Analysis DNAMESH: Arvicolinaebiology.organism_classificationMESH: DNA BacterialMESH: FranceMESH : ArvicolinaeMESH : RNA Ribosomal 16SMESH: BacteriaHigh-Throughput Sequencing030104 developmental biologyMESH : MicrobiotaSpecies richnessMESH: Rodent DiseasesMESH : Animals[SDE.BE]Environmental Sciences/Biodiversity and EcologyBacteriaMESH : Sequence Analysis DNA
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Stat3 and Gfi-1 Transcription Factors Control Th17 Cell Immunosuppressive Activity via the Regulation of Ectonucleotidase Expression

2012

International audience; Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that in vitro Th17 cells generated with the cytokines IL-6 and TGF-β expressed CD39 and CD73 ectonucleotidases, leading to adenosine release and the subsequent suppression of CD4(+) and CD8(+) T cell effector functions. The IL-6-mediated activation of the transcription factor Stat3 and the TGF-β-driven downregulation of Gfi-1 transcription factor were both essential for the expression of ectonucleotidases during Th17 cell differentiation. Stat3 supported whereas Gfi-1 repressed CD39 and CD73 expression by binding to thei…

Adoptive cell transferMESH : Transcription FactorsCellular differentiationMESH: Th17 CellsT-LymphocytesCellMESH : Promoter Regions GeneticMESH : RNA Small InterferingMESH: Mice KnockoutMice0302 clinical medicineTransforming Growth Factor betaMESH: RNA Small InterferingMESH : STAT3 Transcription FactorImmunology and Allergy[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyEctonucleotidaseMESH: AnimalsRNA Small InterferingSTAT3MESH: Lymphocytes Tumor-InfiltratingPromoter Regions GeneticMESH: Antigens CD5'-NucleotidaseRegulation of gene expressionMice Knockout0303 health sciencesMESH : Gene Expression RegulationApyraseMESH: STAT3 Transcription FactorMESH: Transcription FactorsMESH: Gene Expression RegulationMESH : Mice TransgenicCell biologyMESH : Lymphocytes Tumor-InfiltratingDNA-Binding ProteinsMESH : ApyraseInfectious Diseasesmedicine.anatomical_structure[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : DNA-Binding ProteinsMESH: ApyraseSTAT3 Transcription Factor[SDV.IMM] Life Sciences [q-bio]/ImmunologyMESH : Interleukin-6MESH: Mice TransgenicT cellImmunologyMice TransgenicMESH : Mice Inbred C57BLBiology03 medical and health sciencesLymphocytes Tumor-InfiltratingMESH: Mice Inbred C57BLAntigens CDMESH: Promoter Regions GeneticMESH : 5'-NucleotidaseMESH : MicemedicineMESH : Antigens CDMESH : Th17 CellsAnimalsTranscription factorMESH: MiceMESH: Transforming Growth Factor beta030304 developmental biologyMESH : T-LymphocytesBinding SitesInterleukin-6MESH: Interleukin-6Mice Inbred C57BLMESH: T-LymphocytesMESH : Transforming Growth Factor betaMESH: Binding SitesGene Expression Regulationbiology.proteinMESH : Mice KnockoutTh17 CellsMESH : AnimalsMESH: 5'-NucleotidaseMESH: DNA-Binding ProteinsMESH : Binding Sites030215 immunologyTranscription FactorsImmunity
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Prevalence and genetic diversity of Aichi virus strains in stool samples from community and hospitalized patients.

2008

ABSTRACT Aichi virus has been proposed as a causative agent of gastroenteritis. A total of 457 stool specimens from children hospitalized with acute diarrhea and 566 stool specimens from adults and children involved in 110 gastroenteritis outbreaks were screened for the presence of Aichi virus by reverse transcription-PCR (RT-PCR) amplification of the genomic region of the 3C and 3D (3CD) nonstructural proteins. Our results show a low incidence of Aichi virus in pediatric samples and the existence of mixed infections with other microbiological agents in some cases. From the outbreak survey, it appears that the presence of Aichi virus is an indicator of mixed infections causing gastroenterit…

Aichi virusEpidemiologyMESH : PrevalenceMESH : DiarrheaMESH : KobuvirusDisease OutbreaksFecesMESH : ChildMESH: Picornaviridae InfectionsMESH: ChildMESH: AnimalsMESH: Genetic VariationMESH: PhylogenyChildPhylogeny0303 health sciencesCross InfectionMESH: KobuvirusMESH : Reverse Transcriptase Polymerase Chain ReactionMESH: Fecesvirus diseasesMESH : InfantMESH: Infant3. Good healthMESH : GastroenteritisMESH: DiarrheaMESH: Seafood[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyChild Preschool[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyAichi virusMESH : Cross InfectionMicrobiology (medical)DiarrheaMESH : Community-Acquired InfectionsKobuvirusMolecular Sequence Data[ SDV.MP.VIR ] Life Sciences [q-bio]/Microbiology and Parasitology/VirologyMESH: Ostreidae03 medical and health sciencesMESH : AdolescentHumansMESH : Disease OutbreaksMESH: PrevalenceMESH: AdolescentMESH : SeafoodMESH: HumansMESH: Molecular Sequence DataPicornaviridae Infections030306 microbiologyMESH: Child PreschoolMESH : HumansOutbreakGenetic VariationInfantDNAVirologyMESH: GastroenteritisSeafoodMESH : Sequence Analysis DNAMESH: Sequence Analysis DNAMESH : Molecular Sequence DataMESH : Child Preschool[ SDV.MP ] Life Sciences [q-bio]/Microbiology and ParasitologyMESH: Reverse Transcriptase Polymerase Chain ReactionGenotypePrevalenceMESH: Disease Outbreaks[SDV.MP.VIR] Life Sciences [q-bio]/Microbiology and Parasitology/VirologyMESH : Picornaviridae InfectionsbiologyReverse Transcriptase Polymerase Chain ReactionIncidence (epidemiology)MESH: Infant NewbornGastroenteritisCommunity-Acquired InfectionsDiarrheaMESH: Community-Acquired InfectionsKobuvirusFrancemedicine.symptomSequence AnalysisAdolescentMESH : Infant NewbornMESH : Genetic VariationGenetic variationmedicineAnimalsPreschoolMESH : FranceFeces030304 developmental biologyMESH : OstreidaeInfant NewbornMESH: Cross InfectionMESH : PhylogenySequence Analysis DNAMESH : Fecesbiology.organism_classificationNewbornOstreidaeMESH: FranceMESH : Animals
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Glycogen synthase 2 is a novel target gene of peroxisome proliferator-activated receptors.

2007

International audience; Glycogen synthase 2 (Gys-2) is the ratelimiting enzyme in the storage of glycogen in liver and adipose tissue, yet little is known about regulation of Gys-2 transcription. The peroxisome proliferator-activated receptors (PPARs) are transcription factors involved in the regulation of lipid and glucose metabolism and might be hypothesized to govern glycogen synthesis as well. Here, we show that Gys-2 is a direct target gene of PPARalpha, PPARbeta/delta and PPARgamma. Expression of Gys-2 is significantly reduced in adipose tissue of PPARalpha-/-, PPARbeta/delta-/- and PPARgamma+/- mice. Furthermore, synthetic PPARbeta/delta, and gamma agonists markedly up-regulate Gys-2…

Animals; Chromatin/ultrastructure; DNA Primers; Gene Expression Regulation Enzymologic; Glycogen Synthase/genetics; Hepatocytes/enzymology; Hepatocytes/physiology; Mice; Mice Knockout; Peroxisome Proliferator-Activated Receptors/deficiency; Peroxisome Proliferator-Activated Receptors/genetics; Polymerase Chain Reaction; RNA/genetics; RNA/isolation & purification; Rats; Transcription GeneticTranscription GeneticPeroxisome proliferator-activated receptorMESH : HepatocytesPPREPolymerase Chain Reactionadipose-tissuePPARMESH: HepatocytesMice0302 clinical medicineMESH: Animals610 Medicine & healthchemistry.chemical_classificationRegulation of gene expression0303 health sciencesGlycogenglycogen-synthaseChromatinGlycogen Synthase030220 oncology & carcinogenesisMESH : DNA PrimersmicroarrayMESH: DNA Primersmedicine.medical_specialtyHealth aging / healthy living [IGMD 5]fatty-acid oxidationliverGene Expression Regulation EnzymologicMESH: Chromatin03 medical and health sciencesskeletal-muscleGlycogen synthaseMolecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyHNF4αVLAGPharmacologybeta/deltaMESH: Polymerase Chain Reactionresponse elementsMESH : Peroxisome Proliferator-Activated ReceptorsEndocrinologychemistryMicrobial pathogenesis and host defense [UMCN 4.1]Response elementPeroxisome Proliferator-Activated ReceptorsAdipose tissueMESH: Peroxisome Proliferator-Activated Receptorsin-vivoMESH: Mice KnockoutTransactivationchemistry.chemical_compoundVoeding Metabolisme en GenomicaMESH : RNAMESH : Polymerase Chain ReactionMice KnockoutMESH : ChromatinMESH : RatsMESH: Gene Expression Regulation EnzymologicMetabolism and Genomicsadipose tissueMetabolisme en GenomicaMolecular MedicineNutrition Metabolism and GenomicsMESH : Glycogen SynthaseResearch ArticleMESH: Ratsglycogen synthase 2610 Medicine & healthBiologyMESH : Gene Expression Regulation EnzymologicCellular and Molecular NeuroscienceVoedingMESH: RNAInternal medicineMESH : MicemedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyTranscription factorMESH: Micealpha ppar-alpha030304 developmental biologyNutritionDNA PrimersMESH: Glycogen SynthaseMESH: Transcription GeneticMESH : Transcription GeneticCell BiologyRatsgene transcriptionbiology.proteinHepatocytesRNAMESH : Mice KnockoutgammaMESH : Animalsmetabolism
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A multi-locus inference of the evolutionary diversification of extant flamingos (Phoenicopteridae).

2014

9 pages; International audience; BACKGROUND: Modern flamingos (Phoenicopteridae) occupy a highly specialized ecology unique among birds and represent a potentially powerful model system for informing the mechanisms by which a lineage of birds adapts and radiates. However, despite a rich fossil record and well-studied feeding morphology, molecular investigations of the evolutionary progression among modern flamingos have been limited. Here, using three mitochondrial (mtDNA) markers, we present the first DNA sequence-based study of population genetic variation in the widely distributed Chilean Flamingo and, using two mtDNA and 10 nuclear (nDNA) markers, recover the species tree and divergence…

BillBiogeographyPopulationMESH : Multilocus Sequence TypingZoologyMetapopulationMirandornithesBirdsEvolution MolecularMESH : Bayes TheoremPhylogeneticsMESH : Genetic VariationMESH : Biological EvolutionMESH : PhylogeographyAnimalsMESH : Evolution MolecularMESH : BirdseducationDivergence timeEcology Evolution Behavior and SystematicsPhylogenyCell Nucleuseducation.field_of_studyMESH : Cell NucleusbiologyPhylogenetic treeFossilsGenetic VariationBayes TheoremFossilbiology.organism_classificationBiological EvolutionPhoenicoparrusPhoenicopteridaePhylogeographyFlamingoBiogeographyEvolutionary biologyFilter feedingMESH : FossilsMESH : Animals[ SDV.BID.SPT ] Life Sciences [q-bio]/Biodiversity/Systematics Phylogenetics and taxonomyMultilocus Sequence TypingResearch Article
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Acute activation of cannabinoid receptors by anandamide reduces gastrointestinal motility and improves postprandial glycemia in mice.

2015

International audience; The endocannabinoid system (ECS) is associated with an alteration of glucose homeostasis dependent on cannabinoid receptor-1 (CB1R) activation. However, very little information is available concerning the consequences of ECS activation on intestinal glucose absorption. Mice were injected intraperitoneally with anandamide, an endocannabinoid binding both CB1R and CB2R. We measured plasma glucose and xylose appearance after oral loading, gastrointestinal motility, and glucose transepithelial transport using the everted sac method. Anandamide improved hyperglycemia after oral glucose charge whereas glucose clearance and insulin sensitivity were impaired, pointing out so…

Blood GlucoseMaleIndolesCannabinoid receptorMESH : Piperidines[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMESH: EndocannabinoidsMESH : PyrazolesMESH : Receptors CannabinoidMicechemistry.chemical_compoundPiperidinesMESH : IndolesMESH: Receptors CannabinoidMESH: Reverse Transcriptase Polymerase Chain ReactionMESH : Arachidonic AcidsGlucose homeostasisMESH: Gastrointestinal TransitMESH: AnimalsReceptors CannabinoidMESH: IndolesReverse Transcriptase Polymerase Chain ReactionMESH : RatsMESH : Reverse Transcriptase Polymerase Chain ReactionAnandamidePostprandial PeriodEndocannabinoid systemMESH : Gastrointestinal MotilityPostprandialMESH: PiperidinesMESH: Postprandial PeriodMESH: Gastrointestinal MotilityRimonabantMESH : EndocannabinoidsMESH : Gastrointestinal Transitmedicine.medical_specialtyMESH: RatsPolyunsaturated AlkamidesMESH : MaleArachidonic AcidsMESH : Mice Inbred C57BLMESH : Rats WistarMESH: Mice Inbred C57BLInternal medicineMESH : MiceInternal MedicinemedicineAnimalsMESH: Arachidonic AcidsMESH : Polyunsaturated AlkamidesRats WistarGastrointestinal TransitMESH: MiceGastric emptyingMESH: Polyunsaturated AlkamidesGlucose transporterMESH: Rats WistarMESH : Blood GlucoseMESH: MaleRatsMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologychemistryHyperglycemiaMESH : HyperglycemiaMESH: Blood GlucosePyrazolesMESH : AnimalsCannabinoidMESH : Postprandial PeriodGastrointestinal MotilityMESH: Hyperglycemia[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH: PyrazolesEndocannabinoids
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Obesity alters the gustatory perception of lipids in the mouse: plausible involvement of lingual CD36. : Obesity decreases the fat preference

2013

International audience; A relationship between orosensory detection of dietary lipids, regulation of fat intake, and body mass index was recently suggested. However, involved mechanisms are poorly understood. Moreover, whether obesity can directly modulate preference for fatty foods remains unknown. To address this question, exploration of the oral lipid sensing system was undertaken in diet-induced obese (DIO) mice. By using a combination of biochemical, physiological, and behavioral approaches, we found that i) the attraction for lipids is decreased in obese mice, ii) this behavioral change has an orosensory origin, iii) it is reversed in calorie-restricted DIO mice, revealing an inverse …

CD36 AntigensCD36[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionAdipose tissueMESH : Behavior AnimalBiochemistryCalcium in biologyMice0302 clinical medicineEndocrinologyMESH : Calcium SignalingMESH: Behavior AnimalMESH: ObesityMESH: AnimalsLingual papillaResearch Articles2. Zero hunger0303 health sciencesMESH : Food PreferencesBehavior AnimalMESH : TongueMESH : Diet High-FatMESH: TongueTaste Perceptiontaste sensitivityMESH : Antigens CD36calcium imagingAdipose TissueHealthMESH: Dietary FatsMESH : ObesityFat tasteMESH: Adipose Tissuemedicine.medical_specialtyFood behavior030209 endocrinology & metabolismMESH : Mice Inbred C57BLQD415-436BiologyDiet High-FatMESH: Calcium SignalingMESH : Adipose TissueFood Preferences03 medical and health sciencesCalcium imagingTongueDownregulation and upregulationMESH: Mice Inbred C57BLInternal medicineMESH : MicemedicineAnimalsCalcium SignalingObesityFatty acidsMESH: Food PreferencesMESH: Mice030304 developmental biologyNutritionlong-chain fatty acidsMESH: Antigens CD36MESH : Taste PerceptionCell Biologymedicine.diseaseDietary FatsObesityMice Inbred C57BLMESH: Diet High-FatEndocrinologyMESH: Taste Perceptionbiology.proteinMESH : AnimalsBody mass index[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Dietary Fats
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Decreasing dietary linoleic acid promotes long chain omega-3 fatty acid incorporation into rat retina and modifies gene expression

2011

International audience; Age-related macular degeneration (AMD) may be partially prevented by dietary habits privileging the consumption of ω3 long chain polyunsaturated fatty acids (ω3s) while lowering linoleic acid (LA) intake. The present study aimed to document whether following these epidemiological guidelines would enrich the neurosensory retina and RPE with ω3s and modulate gene expression in the neurosensory retina. Rat progenitors and pups were fed with diets containing low or high LA, and low or high ω3s. After scotopic single flash and 8-Hz-Flicker electroretinography, rat pups were euthanized at adulthood. The fatty acid profile of the neurosensory retina, RPE, liver, adipose tis…

CD36 AntigensMaleMESH : RNA MessengerMESH: 5-Lipoxygenase-Activating ProteinsMESH : Receptors LDLMESH: Electroretinography0302 clinical medicineMESH: Fatty Acids Omega-3MESH: AnimalsMESH : Retinal Ganglion Cellschemistry.chemical_classification0303 health sciencesMESH : Gene Expression RegulationMESH : ElectroretinographyMESH: RetinaMESH: Chromatography GasMESH: Dietary Fats Unsaturateddocosahexaenoic acidpolyunsaturated fatty acidSensory Systems3. Good healthnutritionMESH: Photic StimulationAdipose TissueMESH: Adipose Tissuemedicine.medical_specialtyChromatography Gasmacular degenerationLinoleic acidMESH : Arachidonate 12-LipoxygenaseArachidonate 12-LipoxygenaseMESH : Adipose TissueMESH: Arachidonate 12-Lipoxygenasepufa03 medical and health sciencesMESH : Dietary Fats UnsaturatedlipidElectroretinographyRats Long-EvansRNA MessengerMESH: Linoleic AcidMESH: Antigens CD36MESH : RetinaFatty acidMESH: Retinal Ganglion Cellseye diseasesOphthalmologyEndocrinologychemistryMESH: Receptors LDL030221 ophthalmology & optometryATP-Binding Cassette Transportersn 3MESH: FemalePhotic StimulationMESH: LiverRetinal Ganglion CellsretinaMESH : 5-Lipoxygenase-Activating Proteinsgenetic structures[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionretinal pigment epitheliumelectroretinogramMESH : Photic StimulationAdipose tissueangiogenesischemistry.chemical_compoundMESH : FemaleMESH : Rats Long-Evans2. Zero hungermedicine.diagnostic_testMESH : RatsMESH: Real-Time Polymerase Chain ReactionMESH: Gene Expression RegulationMESH : Antigens CD36medicine.anatomical_structureLiverALOX12BiochemistryMESH: ATP-Binding Cassette TransportersFemaleATP Binding Cassette Transporter 1Polyunsaturated fatty acidMESH : Fatty Acids Omega-3MESH: RatsbrainMESH : Male5-Lipoxygenase-Activating ProteinsMESH : Real-Time Polymerase Chain Reactionrhesus monkeyBiologyReal-Time Polymerase Chain ReactionMESH : Chromatography GasLinoleic AcidCellular and Molecular NeuroscienceDietary Fats UnsaturatedMESH : Linoleic AcidMESH: Rats Long-EvansInternal medicineFatty Acids Omega-3medicineAnimalsMESH : ATP-Binding Cassette TransportersOmega 3 fatty acidMESH: RNA Messenger030304 developmental biologydeficient dietRetinal pigment epitheliumMESH : LiverMESH: MaleRatsGene Expression RegulationReceptors LDLgene expressionMESH : Animalssense organs[SDV.AEN]Life Sciences [q-bio]/Food and NutritionElectroretinographyExperimental Eye Research
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Expression profiling of prospero in the Drosophila larval chemosensory organ: Between growth and outgrowth

2010

AbstractBackgroundThe antenno-maxilary complex (AMC) forms the chemosensory system of theDrosophilalarva and is involved in gustatory and olfactory perception. We have previously shown that a mutant allele of the homeodomain transcription factor Prospero (prosVoila1,V1), presents several developmental defects including abnormal growth and altered taste responses. In addition, many neural tracts connecting the AMC to the central nervous system (CNS) were affected. Our earlier reports on larval AMC did not argue in favour of a role ofprosin cell fate decision, but strongly suggested thatproscould be involved in the control of other aspect of neuronal development. In order to identify these fu…

Central Nervous SystemMESH : Transcription FactorsMESH: DrosophilaOF-FUNCTION SCREEN;MUSCA-DOMESTICA L;HOUSE-FLY LARVA;FINE-STRUCTURE;AXON GUIDANCE;TRANSCRIPTION FACTOR;PATTERN-FORMATION;GENETIC-ANALYSIS;NERVOUS-SYSTEMGenes InsectMESH: Genes InsectAXON GUIDANCEMUSCA-DOMESTICA L0302 clinical medicineMESH: Gene Expression Regulation DevelopmentalCluster AnalysisDrosophila ProteinsMESH: AnimalsTRANSCRIPTION FACTORMESH: Nerve Tissue ProteinsMESH : Nerve Tissue ProteinsOF-FUNCTION SCREENOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesMESH : Central Nervous SystemMicrobiology and ParasitologyMESH : Genes InsectGene Expression Regulation DevelopmentalNuclear ProteinsMESH: Transcription FactorsNull alleleMicrobiologie et ParasitologieMESH : Oligonucleotide Array Sequence Analysis[ SDV.BBM.GTP ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]Larva[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]DrosophilaDrosophila ProteinResearch ArticleBiotechnologylcsh:QH426-470MESH: Drosophila Proteinslcsh:BiotechnologyNerve Tissue ProteinsBiotechnologiesBiology03 medical and health sciencesMESH: Gene Expression ProfilingGENETIC-ANALYSIS[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]lcsh:TP248.13-248.65GeneticsAnimalsMESH : Cluster AnalysisMESH: Central Nervous SystemAlleleMESH : DrosophilaAlleles030304 developmental biologyMESH : LarvaMicroarray analysis techniquesHOUSE-FLY LARVAGene Expression ProfilingMESH : Gene Expression ProfilingMESH: AllelesWild typeMESH : Nuclear ProteinsProsperobiology.organism_classificationMESH : Drosophila ProteinsMESH: Cluster AnalysisNERVOUS-SYSTEMGene expression profilinglcsh:GeneticsMESH: Oligonucleotide Array Sequence AnalysisHomeoboxMESH : AnimalsMESH : Gene Expression Regulation DevelopmentalMESH : AllelesMESH: Nuclear ProteinsMESH: Larva030217 neurology & neurosurgeryTranscription FactorsPATTERN-FORMATIONFINE-STRUCTURE
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