Search results for "Mast Cells"

showing 10 items of 99 documents

Genetic Variation Determines Mast Cell Functions in Experimental Asthma

2011

Abstract Mast cell-deficient mice are a key for investigating the function of mast cells in health and disease. Allergic airway disease induced as a Th2-type immune response in mice is employed as a model to unravel the mechanisms underlying inception and progression of human allergic asthma. Previous work done in mast cell-deficient mouse strains that otherwise typically mount Th1-dominated immune responses revealed contradictory results as to whether mast cells contribute to the development of airway hyperresponsiveness and airway inflammation. However, a major contribution of mast cells was shown using adjuvant-free protocols to achieve sensitization. The identification of a traceable ge…

ImmunologyCongenicCell CountInflammationImmunoglobulin EMiceMice CongenicTh2 CellsImmune systemmedicineAnimalsImmunology and AllergyMast CellsSensitizationAsthmaInflammationPolymorphism Geneticbiologymedicine.diseaseMast cellAsthmaInterleukin 33medicine.anatomical_structureImmunologybiology.proteinBronchial Hyperreactivitymedicine.symptomThe Journal of Immunology
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Specific and Redundant Roles for NFAT Transcription Factors in the Expression of Mast Cell-Derived Cytokines

2006

Abstract By virtue of their ability to express a plethora of biologically highly active mediators, mast cells (MC) are involved in both adaptive and innate immune responses. MC-derived Th2-type cytokines are thought to act as local amplifiers of Th2 reactions, including chronic inflammatory disorders such as allergic asthma, whereas MC-derived TNF-α is a critical initiator of antimicrobial defense. In this study, we demonstrate that the transcription factors NFATc1 and NFATc2 are part of a MC-specific signaling network that regulates the expression of TNF-α and IL-13, whereas NFATc3 is dispensable. Primary murine bone marrow-derived MC from NFATc2−/− mice, activated by either ionomycin or I…

ImmunologyDown-RegulationImmunoglobulin EMicechemistry.chemical_compoundTh2 CellsCell Line TumormedicineAnimalsImmunology and AllergyMast CellsTranscription factorCells CulturedMice KnockoutMice Inbred BALB CGene knockdownInterleukin-13Innate immune systemNFATC Transcription FactorsbiologyTumor Necrosis Factor-alphaDegranulationNFATMast cellUp-RegulationCell biologymedicine.anatomical_structurechemistryIonomycinImmunologybiology.proteinCytokinesThe Journal of Immunology
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Mast cells are crucial for early inflammation, migration of Langerhans cells, and CTL responses following topical application of TLR7 ligand in mice.

2007

Abstract Until recently, IgE-activated mast cells have been regarded merely as effector cells of adaptive immune responses, involved in allergic reactions and mucosal immunity to parasites. Herein, we report that murine dermal mast cells, activated by local administration of a cream containing the synthetic TLR7 ligand imiquimod, are essential to initiate an early inflammatory reaction. The mast-cell–derived cytokines TNF-α and IL-1β play an important role in this process. Furthermore, TLR7-activated mast cells are also able to promote the emigration of Langerhans cells, which partly depends on the expression of mast-cell–derived IL-1β. We have previously shown that TLR7 ligation enhances t…

ImmunologyInterleukin-1betaInflammationImmunoglobulin ELigandsBiochemistryMiceImmune systemAdjuvants ImmunologicCell MovementmedicineCytotoxic T cellAnimalsMast CellsAntigensSkinInflammationImmunity CellularMice Inbred BALB CVaccinesImiquimodMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaDegranulationCell BiologyHematologyTLR7Immunoglobulin EAcquired immune systemImmunity InnateInterleukin 33Toll-Like Receptor 7Langerhans CellsImmunologybiology.proteinAminoquinolinesImmunizationmedicine.symptomAgranulocytosisT-Lymphocytes CytotoxicBlood
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Mast Cells Are Key Promoters of Contact Allergy that Mediate the Adjuvant Effects of Haptens

2011

SummaryA prominent feature of sensitizing environmental compounds that cause allergic contact dermatitis is the rapid induction of an innate inflammatory response that seems to provide danger signals for efficient T cell priming. We generated mouse models of mast cell deficiency, mast cell-specific gene inactivation, and mast cell reporter mice for intravital imaging and showed that these adjuvant effects of contact allergens are mediated by mast cells and histamine. Mast cell deficiency resulted in impaired emigration of skin DCs to the lymph node and contact hypersensitivity was dramatically reduced in the absence of mast cells. In addition, mast cell-specific inactivation of the Il10 gen…

ImmunologyMedizinPriming (immunology)BiologyMicechemistry.chemical_compoundImmune systemAdjuvants ImmunologicCell MovementmedicineAnimalsImmunology and AllergyMast CellsInterleukin 5Allergic contact dermatitisNeovascularization PathologicDendritic CellsHypertrophymedicine.diseaseMast cellImmunity InnateMice Inbred C57BLInterleukin 33Interleukin 10medicine.anatomical_structureInfectious DiseaseschemistryDermatitis Allergic ContactMutationImmunologyLymph NodesHaptensHistamineHistamineImmunity
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What is the physiological function of mast cells?

2003

Under physiological conditions, skin mast cells preferentially localize around nerves, blood vessels and hair follicles. This observation, which dates back to Paul Ehrlich, intuitively suggests that these enigmatic, multifacetted protagonists of natural immunity are functionally relevant to many more aspects of tissue physiology than just to the generation of inflammatory and vasodilatory responses to IgE-dependent environmental antigens. And yet, for decades, mainstream-mast cell research has been dominated by a focus on the -undisputedly prominent and important - mast cell functions in type I immune responses and in the pathogenesis and management of allergic diseases. Certainly, it is ha…

InflammationAllergyWound HealingInnate immune systemCellInflammationDermatologyBiologymedicine.diseaseMast cellBiochemistryModels Biologicalmedicine.anatomical_structureImmune systemAntigenImmune SystemImmunoglobulin GImmunologymedicineAnimalsHumansMast Cellsmedicine.symptomWound healingMolecular BiologyExperimental dermatology
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Classical and alternative pathways of mast cell activation.

2002

It has long since been recognized that mast cells are critical effectors of anaphylactic reactions, and the existence of these potentially hazardous cells has solely been justified due to their beneficial role in some infections with extracellular parasites. A novel understanding of mast cells as sentinels of the immune system has been made possible by taking advantage of mast cell-deficient mice in order to study the roles of mast cells in vivo and by detailed analyses of mast cell activation in vitro. Collectively, these experiments have revealed a variety of IgE-independent stimuli, which lead to the activation of mast cells as crucial initiators of an inflammatory response. Besides thei…

InflammationCell typeAdenosinePolymers and PlasticsEndothelin-1EffectorReceptors IgEBiologyInfectionsNeurosecretory SystemsIn vitroCell DegranulationCell biologyDisease Models AnimalImmune systemGene Expression RegulationIn vivoImmune SystemImmunoglobulin GExtracellularAnimalsMast CellsReceptorFunction (biology)General Environmental ScienceCritical reviews in immunology
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Exploring a regulatory role for mast cells: 'MCregs'?

2010

Regulatory cells can mould the fate of the immune response by direct suppression of specific subsets of effector cells, or by redirecting effectors against invading pathogens and infected or neoplastic cells. These functions have been classically, although not exclusively, ascribed to different subsets of T cells. Recently, mast cells have been shown to regulate physiological and pathological immune responses, and thus to act at the interface between innate and adaptive immunity assuming different functions and behaviors at discrete stages of the immune response. Here, we focus on these poorly defined, and sometimes apparently conflicting, functions of mast cells.

InflammationEffectorMast cell; Regulatory cells; cell-cell crosstalkImmunologyRegulatory cellModels ImmunologicalAutoimmunityAdaptive ImmunityBiologybiochemical phenomena metabolism and nutritionAcquired immune systemT-Lymphocytes RegulatoryImmunity InnateClassical complement pathwaycell-cell crosstalkImmune systemRegulatory cellsNeoplasmsImmunologyImmune ToleranceMAST CELLAnimalsHumansImmunology and AllergyMast Cells
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Estrogens control inflammation in experimental colitis.

2014

There is now a wealth of experimental evidence indicating that the deficit in endogenous estrogen facilitates the onset of inflammation that can be antagonized by estrogen replacement therapy. This work investigated the role of estrogen in the control of intestinal inflammation in a panel of colitis models, focusing on the morphological changes, the activity of mast cells, the expression of cytokines (IL-1beta, IL-6, and TNF-alpha), fibronectin and reactive oxygen species. Two hundred adult male rats were divided into 4 groups: colitis was induced in Group I and Group II but only the latter was treated with estrogen; Group III received estrogen only, and Group IV saline. Colitis was induced…

InflammationMaleRats Sprague-DawleyDisease Models AnimalColonestrogens colitis inflammation.AnimalsCytokinesEstrogensMast CellsColitisFibronectinsRats
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Kalanchoe pinnata inhibits mast cell activation and prevents allergic airway disease

2011

Aqueous extract of Kalanchoe pinnata (Kp) have been found effective in models to reduce acute anaphylactic reactions. In the present study, we investigate the effect of Kp and the flavonoid quercetin (QE) and quercitrin (QI) on mast cell activation in vitro and in a model of allergic airway disease in vivo. Treatment with Kp and QE in vitro inhibited degranulation and cytokine production of bone marrow-derived mast cells following IgE/FcɛRI crosslinking, whereas treatment with QI had no effect. Similarly, in vivo treatment with Kp and QE decreased development of airway hyperresponsiveness, airway inflammation, goblet cell metaplasia and production of IL-5, IL-13 and TNF. In contrast, treatm…

KalanchoeOvalbuminmedicine.medical_treatmentBasophil Degranulation TestPharmaceutical ScienceInflammationImmunoglobulin EMiceIn vivoDrug DiscoverymedicineAnimalsMast CellsPharmacologyMetaplasiaMice Inbred BALB CGoblet cellInterleukin-13biologyPlant ExtractsTumor Necrosis Factor-alphabusiness.industryDegranulationIn vitroCytokinemedicine.anatomical_structureComplementary and alternative medicineImmunologybiology.proteinMolecular MedicineQuercetinTumor necrosis factor alphaGoblet CellsBronchial HyperreactivityInterleukin-5medicine.symptombusinessPhytotherapyPhytomedicine
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Genetic ablation of mast cells redefines the role of mast cells in skin wound healing and bleomycin-induced fibrosis.

2014

Conclusive evidence for the impact of mast cells (MCs) in skin repair is still lacking. Studies in mice examining the role of MC function in the physiology and pathology of skin regenerative processes have obtained contradictory results. To clarify the specific role of MCs in regenerative conditions, here we used a recently developed genetic mouse model that allows conditional MC ablation to examine MC-specific functions in skin. This mouse model is based on the cell type–specific expression of Cre recombinase in connective tissue–type MCs under control of the Mcpt5 promoter and the Cre-inducible diphtheria toxin receptor–mediated cell lineage ablation by diphtheria toxin. In response to ex…

KeratinocytesPathologymedicine.medical_specialtymedicine.medical_treatmentCellCre recombinaseMice TransgenicDermatologyBiologyBleomycinBiochemistrySkin Diseaseschemistry.chemical_compoundBleomycinMiceFibrosismedicineLeukocytesAnimalsMast CellsMolecular BiologyDiphtheria toxinSkin repairWound HealingAntibiotics AntineoplasticGranulation tissueCell BiologyAblationmedicine.diseaseFibrosisDisease Models Animalmedicine.anatomical_structurechemistryGranulation TissueThe Journal of investigative dermatology
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