Search results for "Microbio"

showing 10 items of 8741 documents

Bacterial Diversity in a Dynamic and Extreme Sub-Arctic Watercourse (Pasvik River, Norwegian Arctic)

2020

Microbial communities promptly respond to the environmental perturbations, especially in the Arctic and sub-Arctic systems that are highly impacted by climate change, and fluctuations in the diversity level of microbial assemblages could give insights on their expected response. 16S rRNA gene amplicon sequencing was applied to describe the bacterial community composition in water and sediment through the sub-Arctic Pasvik River. Our results showed that river water and sediment harbored distinct communities in terms of diversity and composition at genus level. The distribution of the bacterial communities was mainly affected by both salinity and temperature in sediment samples, and by oxygen…

lcsh:Hydraulic engineeringGeography Planning and DevelopmentClimate changesedimentitAquatic ScienceBiochemistryAlgal bloombakteerit03 medical and health scienceslcsh:Water supply for domestic and industrial purposeslcsh:TC1-978Glacial period030304 developmental biologyWater Science and TechnologyPhylotypearktinen aluelcsh:TD201-5000303 health sciences030306 microbiologyEcologyvesiekosysteemitbacterial diversityriver sediment and waterSedimentmikrobiekologiaSalinitymikrobistoTaxonvirtavedetNGSsub-Arctic systemEnvironmental scienceSurface runoffjoetWater
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The Metabolic Sensor GPR43 Receptor Plays a Role in the Control of Klebsiella pneumoniae Infection in the Lung

2018

Pneumonia is one of the leading causes of death and mortality worldwide. The inflammatory responses that follow respiratory infections are protective leading to pathogen clearance but can also be deleterious if unregulated. The microbiota is known to be an important protective barrier against infections, mediating both direct inhibitory effects against the potential pathogen and also regulating the immune responses contributing to a proper clearance of the pathogen and return to homeostasis. GPR43 is one receptor for acetate, a microbiota metabolite shown to induce and to regulate important immune functions. Here, we addressed the role of GPR43 signaling during pulmonary bacterial infection…

lcsh:Immunologic diseases. Allergy0301 basic medicinePhagocytosisshort-chain fatty acids030106 microbiologyImmunologyInflammationBiologyGut floraMicrobiology03 medical and health sciencesImmune systemmicrobiotamedicinepneumoniaImmunology and AllergyPathogenLunglung infectionGPR43biology.organism_classificationmedicine.disease030104 developmental biologymedicine.anatomical_structureinflammationIncreased inflammatory responsemedicine.symptomlcsh:RC581-607Pneumonia (non-human)Frontiers in Immunology
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Baseline Gut Microbiota Composition Is Associated With Schistosoma mansoni Infection Burden in Rodent Models

2020

In spite of growing evidence supporting the occurrence of complex interactions between Schistosoma and gut bacteria in mice and humans, no data is yet available on whether worm-mediated changes in microbiota composition are dependent on the baseline gut microbial profile of the vertebrate host. In addition, the impact of such changes on the susceptibility to, and pathophysiology of, schistosomiasis remains largely unexplored. In this study, mice colonized with gut microbial populations from a human donor (HMA mice), as well as microbiota-wild type (WT) animals, were infected with Schistosoma mansoni, and alterations of their gut microbial profiles at 50 days post-infection were compared to …

lcsh:Immunologic diseases. Allergy0301 basic medicineRodentImmunologyAntibodies ProtozoanSchistosomiasisGut floradigestive systemParasite LoadHost-Parasite InteractionsMicrobiologyImmunomodulationFecesMice03 medical and health sciences0302 clinical medicineimmune-modulationhuman-microbiota associated mouse modelsRNA Ribosomal 16Sbiology.animalLactobacillusmedicineAnimalsImmunology and AllergySchistosomaBacteriabiologyFOS: Clinical medicineComputational BiologyBiodiversitySchistosoma mansonidysbiosismedicine.diseasebiology.organism_classificationSchistosomiasis mansoniGastrointestinal MicrobiomeDisease Models Animal030104 developmental biologyhelminth-gut microbiota interactionsSchistosomaMetagenomicsSchistosoma mansonigut microbial diversityProteobacterialcsh:RC581-607Dysbiosis030215 immunology
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Candida spp. Determination and Th1/Th2 Mixed Cytokine Profile in Oral Samples From HIV+ Patients With Chronic Periodontitis

2019

Background: Chronic periodontitis (CP), caused by bacteria and fungi, appears in up to 66% of HIV-patients. The impact and association of HIV-treatment (HAART) and Candida itself has not been properly evaluated in the development and progression of CP. The immunopathogenesis is characterized by CD4+ T-cells activation and the balance between the T-helper 1 (Th1) and T-helper 2 (Th2) or a mixed cytokine profile. Currently, the associated causes of an immune response in HIV-patients with CP is controversial. Our aims were the determination of Candida spp. and cytokine profile in oral samples from HIV-positive patients with CP, considering the CD4+ T cells levels and HAART use.Methods: From 50…

lcsh:Immunologic diseases. Allergy0301 basic medicineSalivaHAARTmedicine.medical_treatmentImmunologyGroup AGroup BMicrobiology03 medical and health sciences0302 clinical medicinemedicineImmunology and AllergyOriginal ResearchCandida sppmedicine.diagnostic_testbusiness.industryHIVchronic periodontitisImmunosuppressionmedicine.diseaseChronic periodontitiscytokinesCorpus albicans030104 developmental biologyCytokineImmunoassaylcsh:RC581-607business030215 immunologyFrontiers in Immunology
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Heat-Shock Proteins in Autoimmunity

2013

Heat shock proteins (HSPs), also known as “stress proteins,” are among the highly conserved and immunogenic proteins shared among diverse groups of microbial agents and mammals [1]. Heat and other types of stressful stimuli can increase the cellular expression of HSPs. These proteins have been categorized into different families according to their molecular mass, for example, HSP110, HSP90, HSP70, HSP60, HSP40, HSP20-30, and HSP10 [1–3]. For uniformity, guidelines for the nomenclature of various human HSP families have been proposed [4]. Under physiological conditions, the ubiquitously distributed HSPs maintain the integrity and function of other cellular proteins in stressful conditions. H…

lcsh:Immunologic diseases. AllergyArticle SubjectImmunologychemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoimmunity03 medical and health sciences0302 clinical medicineImmune systemImmunology and Microbiology (miscellaneous)Heat shock proteinmedicineImmunology and Allergy030304 developmental biology0303 health sciencesInnate immune systemFOXP3Acquired immune system3. Good healthMolecular mimicryEditorialImmunologyHSP60lcsh:RC581-607030217 neurology & neurosurgeryAutoimmune Diseases
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IL-12 Expands and Differentiates Human Vγ2Vδ2 T Effector Cells Producing Antimicrobial Cytokines and Inhibiting Intracellular Mycobacterial Growth

2019

While IL-12 plays a key role in differentiation of protective CD4+ Th1 response, little is known about mechanisms whereby IL-12 differentiates other T-cell populations. Published studies suggest that predominant Vγ2Vδ2 T cells in humans/nonhuman primates (NHP) are a fast-acting T-cell subset, with capacities to rapidly expand and produce Th1 and cytotoxic cytokines in response to phosphoantigen (E)-4-hydroxy-3-methyl-but-2-enyl pyrophosphate (HMBPP) produced by Mycobacterium tuberculosis (Mtb) or others. However, whether IL-12 signaling pathway mediates fast-acting and Th1 or anti-microbial features of Vγ2Vδ2 T cells remains poorly defined. Here, we show that IL-12, but not other IL-12 fami…

lcsh:Immunologic diseases. AllergyCells1.1 Normal biological development and functioningproliferationImmunologyLymphocyte ActivationV gamma 2V delta 2 T cellsVaccine Related03 medical and health sciencesPhosphatidylinositol 3-Kinases0302 clinical medicineRare DiseasesUnderpinning researchT-Lymphocyte SubsetsImmunology and AllergyTuberculosis2.1 Biological and endogenous factorsHumansAetiologyIntraepithelial LymphocytesCells Cultured030304 developmental biologyOriginal Researchanti-tuberculosis0303 health sciencesCulturedVγ2Vδ2 T cellsTumor Necrosis Factor-alphaInflammatory and immune systemCorrectionCell DifferentiationMycobacterium tuberculosisdifferentiationSTAT4 Transcription FactorTh1 CellsInterleukin-12Organophosphates3. Good healthInfectious DiseasesGood Health and Well BeingMedical MicrobiologyIL-12Infectionlcsh:RC581-607Proto-Oncogene Proteins c-akt030215 immunologySignal TransductionFrontiers in Immunology
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Neutrophils: Between host defence, immune modulation, and tissue injury.

2015

Neutrophils, the most abundant human immune cells, are rapidly recruited to sites of infection, where they fulfill their life-saving antimicrobial functions. While traditionally regarded as short-lived phagocytes, recent findings on long-term survival, neutrophil extracellular trap (NET) formation, heterogeneity and plasticity, suppressive functions, and tissue injury have expanded our understanding of their diverse role in infection and inflammation. This review summarises our current understanding of neutrophils in host-pathogen interactions and disease involvement, illustrating the versatility and plasticity of the neutrophil, moving between host defence, immune modulation, and tissue da…

lcsh:Immunologic diseases. AllergyChemokineCell SurvivalNeutrophilsPhagocytosisImmunologyInflammation610 Medicine & healthDiseaseReviewNeutropeniamedicine.disease_causeInfectionsMicrobiologyExtracellular TrapsImmune systemCell MovementVirologyGeneticsmedicineAnimalsHumansMolecular Biologylcsh:QH301-705.5InflammationMutationbiologyNeutrophil extracellular trapsmedicine.disease3. Good healthCell biologylcsh:Biology (General)Immunologybiology.proteinParasitologymedicine.symptomlcsh:RC581-607
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CD11b Regulates Fungal Outgrowth but Not Neutrophil Recruitment in a Mouse Model of Invasive Pulmonary Aspergillosis

2019

Abstract Background and Aims: In immunosuppressed individuals Aspergillus (A.) fumigatus is a frequent cause of invasive pulmonary aspergillosis (IPA) which is highly associated with relevant morbidity and mortality. Moreover, it often occurs in patients suffering from leukocyte-adhesion deficiency type 1 (LAD1) which is triggered by a functional loss of CD18 in ß2 integrin receptors as these receptors consist of an alpha subunit (CD11a-CD11d) and CD18 as the common beta subunit. ß2 integrin receptors are differentially expressed by leukocytes, and are required for cell-cell interaction, transendothelial migration, uptake of opsonized pathogens, and cell signaling processes. Here, we asked …

lcsh:Immunologic diseases. AllergyChemokineNeutrophilsPhagocytosisImmunology610 MedizinMedizinMacrophage-1 AntigenCD18InflammationKaplan-Meier EstimateBronchoalveolar LavageBiochemistryMicrobiologyAspergillus fumigatusProinflammatory cytokinecomplement receptor 3MicePhagocytosis610 Medical sciencesmedicineAnimalspneumoniaCC-chemokine ligand 5LungOriginal ResearchInflammationInvasive Pulmonary AspergillosisMice KnockoutCD11b Antigenbiologymedicine.diagnostic_testAspergillus fumigatusCD11bpolymorphonuclear neutrophilsCell BiologyHematologybiology.organism_classificationMice Inbred C57BLDisease Models AnimalBronchoalveolar lavageNeutrophil InfiltrationIntegrin alpha Mβ2 integrinsbiology.proteinCytokinesFemalemedicine.symptomlcsh:RC581-607
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Control of Murine Cytomegalovirus Infection by γδ T Cells

2015

Infections with cytomegalovirus (CMV) can cause severe disease in immunosuppressed patients and infected newborns. Innate as well as cellular and humoral adaptive immune effector functions contribute to the control of CMV in immunocompetent individuals. None of the innate or adaptive immune functions are essential for virus control, however. Expansion of γδ T cells has been observed during human CMV (HCMV) infection in the fetus and in transplant patients with HCMV reactivation but the protective function of γδ T cells under these conditions remains unclear. Here we show for murine CMV (MCMV) infections that mice that lack CD8 and CD4 αβ-T cells as well as B lymphocytes can control a MCMV i…

lcsh:Immunologic diseases. AllergyMuromegalovirusAdoptive cell transferCD3 ComplexT cellImmunologyPopulation-MicrobiologyMiceImmune systemT-Lymphocyte SubsetsMedizinische FakultätVirologyGeneticsmedicineAnimalsCytotoxic T cellddc:610educationlcsh:QH301-705.5Molecular BiologyMice Knockouteducation.field_of_studybiologyvirus diseasesHerpesviridae InfectionsFlow CytometryAdoptive TransferVirologyHigh-Throughput Screening AssaysMice Inbred C57BLmedicine.anatomical_structurelcsh:Biology (General)Immunologybiology.proteinParasitologyAntibodyStem celllcsh:RC581-607CD8Research ArticlePLOS Pathogens
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New Insight into Immunity and Immunopathology of Rickettsial Diseases

2011

Human rickettsial diseases comprise a variety of clinical entities caused by microorganisms belonging to the generaRickettsia,Orientia,Ehrlichia, andAnaplasma. These microorganisms are characterized by a strictly intracellular location which has, for long, impaired their detailed study. In this paper, the critical steps taken by these microorganisms to play their pathogenic roles are discussed in detail on the basis of recent advances in our understanding of molecularRickettsia-host interactions, preferential target cells, virulence mechanisms, three-dimensional structures of bacteria effector proteins, upstream signalling pathways and signal transduction systems, and modulation of gene exp…

lcsh:Immunologic diseases. AllergySettore MED/09 - Medicina InternaVirulence FactorsRickettsial diseasesImmunologyRickettsiaceae InfectionsVirulenceImmunopathologyReview ArticleAdaptive ImmunityHost SpecificityMicrobiologyImmune systemBacterial ProteinsImmunityAnimalsHumansImmunology and AllergyAnaplasmaMolecular Targeted TherapyRickettsiaArthropodsPathogenRickettsieaeGeneticsImmunopathology; Rickettsial diseasesbiologyEffectorGeneral Medicinebacterial infections and mycosesbiology.organism_classificationAcquired immune systemOrientiaImmunity InnateGene Expression RegulationHost-Pathogen Interactionslcsh:RC581-607Signal TransductionClinical and Developmental Immunology
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