Search results for "Micronuclei"

showing 10 items of 13 documents

Possible relationship between micronucleated and binucleated cells induced by cisplatin in cultured CHO cells

1993

Abstract Chinese hamster ovary (CHO) cells were treated with a single dose (10 μg/ml) of cis-diammino-dichloroplatinum (II) (cisplatin) for 1 h and the effect of the drug on the kinetics of proliferation of the cultures was studied. It was found that the drug produces a delay in the proliferation rates of the treated cultures. The induction of micronuclei and binucleated cells (BC) at different times after treatment have also been studied, and the ability of these cells to undergo DNA synthesis (measured as the ability to incorporate [3H]thymidine) is shown. It was found that cisplatin induced a particular type of BC that contains one or more micronuclei rather than a pure population of BC.…

Cisplatineducation.field_of_studyDNA synthesisChinese hamster ovary cellBinucleated cellsPopulationHamsterCHO CellsBiologyToxicologycomplex mixturesMolecular biologychemistry.chemical_compoundchemistryCricetinaeMicronucleus testGeneticsmedicineAnimalsCisplatineducationThymidineCell DivisionMicronuclei Chromosome-Defectivemedicine.drugMutation Research/Environmental Mutagenesis and Related Subjects
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Genomic instability induced by α-pinene in Chinese hamster cell line.

2012

Here, we report the effects of exposure of mammalian cells to α-pinene, a bicyclic monoterpene used in insecticides, solvents and perfumes. Morphological analysis, performed in V79-Cl3 cells exposed for 1 h to increasing concentrations (25 up to 50 μM) of α-pinene, indicated a statistically significant increase in micronucleated and multinucleated cell frequencies; apoptotic cells were seen at 40 and 50 μM. This monoterpene caused genomic instability by interfering with mitotic process; in fact, 50% of cells (versus 19% of control cells) showed irregular mitosis with multipolar or incorrectly localised spindles. Cytogenetic analysis demonstrated high-frequency hypodiploid metaphases as well…

DNA damageHealth Toxicology and MutagenesisApoptosisToxicologymedicine.disease_causeChinese hamsterGenomic InstabilityColony-Forming Units AssayImmunoenzyme TechniquesMultinucleateCricetulusGenomic instability hamster cell lines a-pineneCricetinaeGeneticsmedicineAnimalsMitosisGenetics (clinical)Cells CulturedMicronuclei Chromosome-DefectiveBicyclic MonoterpenesChromosome AberrationsMicronucleus Testsbiologybiology.organism_classificationMolecular biologyComet assaySettore BIO/18 - GeneticaOxidative StressCell cultureMicronucleus testMonoterpenesComet AssayReactive Oxygen SpeciesOxidative stressDNA DamageMutagenesis
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Inhibition of clastogenicity of benzo[a]pyrene and of its trans-7,8-dihydrodiol in mice in vivo by fruits, vegetables, and flavonoids.

2003

In the in vivo mouse bone marrow micronucleus assay, homogenates of spinach, artichoke, peaches, and blue grapes as well as commercial concentrates of these vegetables and fruits reduced induction of micronuclei by benzo[a]pyrene (BaP) by 43-50%. Concentrates of strawberries (31% reduction) and of cauliflower (20% reduction) were less potent. Inhibition of genotoxicity by spinach and peaches was not caused by any delay in maturation of micronucleated erythrocytes as shown by experiments with sampling times of 24, 48, and 72 h after dosing of BaP. Pre-treatment of the mice with spinach 48, 24, and 12h before application of BaP resulted in a 44% reduction of micronuclei while peaches generate…

MaleHealth Toxicology and MutagenesisFlavonoidAdministration OralBone Marrow CellsMice Inbred Strainsmedicine.disease_causecomplex mixturesDihydroxydihydrobenzopyreneschemistry.chemical_compoundClastogenMiceVegetablesGeneticsmedicineBenzo(a)pyreneCytochrome P-450 CYP1A1AnimalsFood scienceMicronuclei Chromosome-Defectivechemistry.chemical_classificationMicronucleus TestsbiologyDose-Response Relationship DrugPlant Extractsfood and beveragesAntimutagenic Agentsbiology.organism_classificationDose–response relationshipBenzo(a)pyrenechemistryBiochemistryLiverFruitMicronucleus testCytochrome P-450 CYP2B1SpinachDrug Therapy CombinationQuercetinQuercetinGenotoxicityInjections IntraperitonealMutagensMutation research
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Intra- and extra-cellular DNA damage by harmine and 9-methyl-harmine

2014

It is known that b-carbolines are able to produce photosensitized damage in cell-free DNA, but there is little information on their effects on cellular DNA. Therefore, we have analyzed the DNA damage produced by harmine and 9-methyl-harmine under UVA irradiation in V79 cells, together with the associated generation of micronuclei and photocytotoxicity. The results indicate that the most frequent photoproducts generated in the cellular DNA are modified purines such as 8-oxo-7,8-dihydroguanine. Only relatively few single-strand breaks were observed. CPDs were absent, although they were generated in cell-free DNA irradiated under the same conditions. The overall extent of DNA damage in the cel…

Guanineb-carbolinesUltraviolet RaysDNA damageBiophysicsFree radical damage to DNACHO CellsMicronucleiBiologyPhototoxicitychemistry.chemical_compoundCricetulusHarmineCyclobutane pyrimidine photodimersCricetinaeAnimalsRadiology Nuclear Medicine and imagingPurine metabolismRadiationRadiological and Ultrasound TechnologyCell growthOtras Ciencias QuímicasCiencias QuímicasDNAHydrogen-Ion ConcentrationHarmineQuímica OrgánicaBiochemistrychemistryMicronucleus testBiophysicsPhotosensitizationPhototoxicityCIENCIAS NATURALES Y EXACTASDNADNA DamageJournal of Photochemistry and Photobiology B: Biology
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Human cytochrome P450 reductase can act as a source of endogenous oxidative DNA damage and genetic instability.

2005

Studies with repair-deficient mice and other experiments suggest that oxidative DNA modifications are generated in all types of cells even under physiological conditions and that this type of endogenous DNA damage contributes to spontaneous cancer incidence. However, the cellular sources of reactive oxygen species that are relevant for nuclear oxidative DNA damage are largely unknown. Here, we report that expression of human NADPH-cytochrome P450 reductase (hOR) in cultured V79 Chinese hamster cells gives rise to elevated basal levels of oxidative purine modifications after depletion of glutathione. Also, the basal levels of micronuclei are increased in the hOR-expressing cells, and again t…

Genome instabilityAntioxidantDNA damagemedicine.medical_treatmentGlutathione reductaseEndogenyOxidative phosphorylationCHO CellsBiologyBiochemistryGenomic Instabilitychemistry.chemical_compoundPhysiology (medical)CricetinaemedicineAnimalsHumansMicronuclei Chromosome-DefectiveNADPH-Ferrihemoprotein Reductasechemistry.chemical_classificationReactive oxygen speciesGlutathioneMolecular biologyGlutathionechemistryPurinesReactive Oxygen SpeciesOxidation-ReductionDNA DamageFree radical biologymedicine
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A Putatively Functional Haplotype in the Gene Encoding Transforming Growth Factor Beta-1 as a Potential Biomarker for Radiosensitivity

2011

Purpose To determine whether genetic variability in TGFB1 is related to circulating transforming growth factor-β1 (TGF-β1) plasma concentrations after radiotherapy and to radiosensitivity of lymphoid cells. Patients and Methods Transforming growth factor-β1 plasma concentrations ( n = 79) were measured in patients 1 year after radiotherapy and chromosomal aberrations ( n = 71) ex vivo before therapy start. Furthermore, TGF-β1 secretion and apoptosis were measured in isolated peripheral blood mononuclear cells of 55 healthy volunteers. These phenotypes were analyzed in relation to five germline polymorphisms in the 5′ region of the TGFB1 gene. Because of high linkage disequilibrium, these fi…

AdultMaleCancer ResearchSomatic cellDNA damageApoptosisPolymorphism Single NucleotideRadiation TolerancePeripheral blood mononuclear cellLinkage DisequilibriumCell LineTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineHumansMedicineRadiology Nuclear Medicine and imagingRadiosensitivityGeneMicronuclei Chromosome-DefectiveAged030304 developmental biologyAged 80 and over0303 health sciencesRadiationbiologybusiness.industryHaplotypeTransforming growth factor betaMiddle AgedMolecular biology3. Good healthHaplotypesOncology030220 oncology & carcinogenesisMicronucleus testImmunologyLeukocytes Mononuclearbiology.proteinFemalebusinessBiomarkersDNA DamageInternational Journal of Radiation Oncology*Biology*Physics
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Clastogenic and aneuploidizing effects of antiblastic busulphan revealed by kinetochore immunofluorescence in CHO cells.

1991

We utilized, in CHO cells, the cytoplasm preservation technique to evaluate the micronucleus frequency at different busulphan concentrations, and the indirect immunofluorescence technique, using sera obtained from patients with scleroderma (CREST variant), to analyze if busulphan-induced micronuclei have kinetochores. Results show that this alkylating agent is capable of causing a significant increase of micronuclei in vitro, a great part (40%) of them having CREST-positive kinetochores. These findings confirm the clastogenic effect of busulphan and reveal a considerable capability of this agent to induce aneuploidy. These results are examined taking into account the high incidence of secon…

CentromereAneuploidyFluorescent Antibody TechniqueBiologyImmunofluorescenceCell LineAcetoneClastogenhemic and lymphatic diseasesmedicineHumansBusulfanMicronuclei Chromosome-DefectiveChromosome AberrationsMicronucleus TestsScleroderma Systemicmedicine.diagnostic_testDose-Response Relationship DrugGeneral Medicinemedicine.diseaseAneuploidyMolecular biologyIn vitroCell cultureMicronucleus testMicronucleusBusulfanmedicine.drugMutation research
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Treatment with the anti-tumor drugs, cis-platin and mafosfamide, does not affect the structure of prekinetochores in a human breast cancer cell line.…

1996

Abstract The goal of the present article was to determine whether a nuclear parameter, centromere structure of interphase cells, could serve as an indicator to assess cellular damage caused by anti-tumor drugs. These were cis-platin and mafosfamide, which are widely used for the management of solid tumors. To visualize the centromeres, we probed treated and untreated cells of a human breast cancer cell line, MX-1, with a human anti-centromere serum. The serum was obtained from a scleroderma patient and detects antigens associated with prekinetochores of the decondensed chromosomes. The DNA was simultaneously displayed by a specific fluorescent dye. The cells were grown on coverslips, incuba…

CentromereAntineoplastic AgentsBreast NeoplasmsBiologyImmunofluorescencechemistry.chemical_compoundMultinucleateAntigenMafosfamideTumor Cells CulturedmedicineHumansFluorescent Antibody Technique IndirectKinetochoresCyclophosphamideMicronuclei Chromosome-Defectivemedicine.diagnostic_testTemperatureChromosomeGeneral MedicineCell cycleMolecular biologyMicroscopy ElectronchemistryCytoplasmInterphaseCisplatinAnatomyDevelopmental BiologyAnnals of Anatomy - Anatomischer Anzeiger
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Genotoxicity of selected pesticides in the mouse bone-marrow micronucleus test and in the sister-chromatid exchange test with human lymphocytes in vi…

1996

Selected pesticides (aldicarb, 1,3-dichloropropene, methidathion, parathion, triadimefon, vinclozolin) were tested for their clastogenic and aneugenic activities in the mouse bone-marrow micronucleus (MN) test in vivo and for their sister-chromatid exchange-inducing activities in human lymphocytes in vitro in the presence and absence of an exogenous metabolizing system from rat-liver S9. 1,3-Dichloropropene significantly increased the frequencies of micronucleated polychromatic erythrocytes (PCE) in bone-marrow cells of female mice from 3.3 MN/1000 PCE to 15.3 MN/1000 PCE (187 mg per kg body weight). 1,3-Dichloropropene (100 microM) induced 16.0 SCE/metaphase after 24 h of incubation as com…

AdultMaleSister chromatid exchangeMethidathion010501 environmental sciencesBiologyToxicologymedicine.disease_cause01 natural sciencesMice03 medical and health scienceschemistry.chemical_compoundClastogenBone MarrowHydrocarbons ChlorinatedmedicineAnimalsHumansLymphocytesVinclozolinPesticidesOxazolesMetaphaseCells CulturedMicronuclei Chromosome-Defective030304 developmental biology0105 earth and related environmental sciences0303 health sciencesMicronucleus TestsParathionOrganothiophosphorus CompoundsGeneral MedicineTriazolesMolecular biology3. Good healthAllyl CompoundschemistryImmunologyMicronucleus testFemaleMicronucleusSister Chromatid ExchangeAldicarbGenotoxicityToxicology Letters
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Induction of micronuclei in V79 Chinese hamster cells by tetrachlorohydroquinone, a metabolite of pentachlorophenol

1992

Tetrachlorohydroquinone, a metabolite of the fungicide pentachlorophenol, induced significant dose-related increases in micronuclei in V79 Chinese hamster cells without exogenous metabolic activation. The lowest observed effective dose was 10 microM, where the relative survival was about 62%. At the highest dose tested, 20 microM, the relative survival was about 8% and the frequency of cells with micronuclei was about 6 times the solvent control frequency. The induction of micronuclei by tetrachlorohydroquinone was significantly inhibited by the hydroxyl radical scavenger dimethyl sulfoxide at 5% (v/v).

PentachlorophenolMetaboliteHamsterToxicologycomplex mixturesChinese hamsterchemistry.chemical_compoundCricetulusCricetinaeGeneticsAnimalsDimethyl SulfoxideCells CulturedMicronuclei Chromosome-DefectiveCarcinogenMicronucleus TestsbiologyDimethyl sulfoxidebiology.organism_classificationMolecular biologyEffective dose (pharmacology)HydroquinonesPentachlorophenolchemistryBiochemistryMicronucleus testDNA DamageMutation Research/Genetic Toxicology
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