Search results for "Myocyte"

showing 10 items of 248 documents

Über den Zusammenhang zwischen histologischer Struktur und funktionellem Verhalten des Skelettmuskels

1956

A stimultaneous investigation of the function and the histological structure of two different muscles of the rat showed no relation between the arrangement of the fibrilles in the muscle cell and the function of the muscle. Such a relation between structure and function was assumed byKruger on the basis of histological investigations, but our findings do not support this hypothesis.

PharmacologyCellular and Molecular NeuroscienceChemistryMolecular MedicineMyocyteCell BiologyFunction (mathematics)AnatomyMolecular BiologyStructure and functionExperientia
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Extracellular ATP Increases <i>L</i>-Carnitine Transport and Content in C2C12 Cells

2008

Extracellular ATP regulates cell proliferation, muscle contraction and myoblast differentiation. ATP present in the muscle interstitium can be released from contracting skeletal muscle cells. <i>L</i>-Carnitine is a key element in muscle cell metabolism, as it serves as a carrier for fatty acid through mitochondrial membranes, controlling oxidation and energy production. Treatment of C2C12 cells with 1 mmol/l of ATP induced a marked increase in <i>L</i>-carnitine uptake that was associated with an increase in <i>L</i>-carnitine content in these cells. These effects were found to be dependent on the density of the cultured cells and on the dose of ATP. The…

PharmacologyP2Y receptorChemistrySkeletal muscleGeneral MedicineMetabolismCell biologymedicine.anatomical_structureBiochemistrymedicineExtracellularMyocytemedicine.symptomITGA7ActinMuscle contractionPharmacology
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Beneficial effects of l-carnitine in myoblastic C2C12 cells

2003

L-Carnitine is a key molecule in the transfer of fatty acid across mitochondrial membranes. Bioavailable L-carnitine is either provided by an endogeneous biosynthesis or after intestinal absorption of dietary items containing L-carnitine. After intestinal absorption or hepatic biosynthesis, L-carnitine is transferred to organs whose metabolism is dependent upon fatty acid oxidation, such as skeletal muscle. To cross the muscle plasma membrane, there are several transporters involved. Among those transporters, OCTN2 is actually the only one to have been clearly characterized. Zidovudine is a commonly used inhibitor of human immunodeficiency virus (HIV) replication. Zidovudine has many side e…

PharmacologySkeletal muscleBiologyMitochondrionPharmacologyBiochemistryIntestinal absorptionZidovudinemedicine.anatomical_structureBiochemistrymedicineMyocyteCarnitinemedicine.symptomMyopathyBeta oxidationmedicine.drugBiochemical Pharmacology
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Dose-dependent biphasic leptin-induced proliferation is caused by non-specific IL-6/NF-κB pathway activation in human myometrial cells

2015

Background and Purpose Leptin, an adipokine synthesized by the placenta during pregnancy, has been proposed for the management of preterm labour (PTL), as it is able to prevent in vitro uterine contractility and remodelling associated with labour onset. Another common feature of labour onset is the phenotypic switch of myometrial smooth muscle cells from a proliferative to a hypertrophic state. As proliferative effects have been demonstrated for leptin in other tissues, we aimed to investigate its ability to induce myometrial proliferation and thus to maintain uterine quiescence. Experimental Approach We stimulated human primary myometrial smooth muscle cells with leptin in the presence or …

Pharmacologymedicine.medical_specialtyLeptin receptorLeptindigestive oral and skin physiologyMyometriumAdipokineStimulationBiologyEndocrinologyInternal medicinemedicineMyocyteSignal transductionReceptorBritish Journal of Pharmacology
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Comparison of complex fractionated atrial electrograms at cellular scale using numerical and experimental models.

2010

This study investigates the existence of the pseudo complex fractionated atrial electrogram (CFAE) at cellular level. Our assumptions are based on the fact that CFAEs are linked to the generation of the spiral waves. These are created using a numerical model and an experimental model of in vitro culture of neonatal rats cardiac cells. Pseudo bipolar electrograms resulting from these two models are compared qualitatively and some patterns could be identified as CFAE signature.

PhysicsScale (ratio)Experimental modelModels CardiovascularAction PotentialsNumerical modelsCellular levelElectrocardiographyBiological ClocksHeart Conduction SystemAtrial FibrillationAnimalsHumansComputer SimulationMyocytes CardiacHeart AtriaBiological systemCellular biophysicsBiomedical engineeringAnnual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference
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The metalloproteinase-disintegrin ADAM10 is exclusively expressed by type I muscle fibers.

2008

ADAM10 (Kuzbanian) is a member of a recently discovered family of membrane-anchored metalloproteinases with a complex and conserved domain structure. In part, these metalloproteinases have been implicated in muscle formation. Herein the expression pattern of ADAM10 in human skeletal muscle was studied. ADAM10 was found to be present in human myoblasts and to be exclusively expressed in type I fibers, suggesting that it may be critical in muscle fiber differentiation.

PhysiologyADAM10Matrix metalloproteinaseCellular and Molecular NeuroscienceADAM10 ProteinPhysiology (medical)DisintegrinmedicineMyocyteHumansAdenosine TriphosphatasesMetalloproteinasebiologyMyosin Heavy ChainsMyogenesisChemistrySkeletal muscleMembrane ProteinsCell biologyADAM Proteinsmedicine.anatomical_structureMuscle Fibers Slow-TwitchBiochemistrybiology.proteinNeurology (clinical)Amyloid Precursor Protein SecretasesITGA7Musclenerve
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Versuche zur Bestimmung des wahren K-Efflux im Rattenzwerchfell

1960

In an isolated muscle loaded with 42K the potassium efflux through the muscle fibre membrane can be calculated from the rate constant of the loss of 42K into an inactive bathing solution. The calculation can only be valid if the amount of 42K leaving all the individual fibres is equivalent to the amount of 42K entering the bathing solution from the surface of the whole muscle. It seems possible that 42K ions which have already left a muscle cell can be taken up again into a muscle fibre before diffusing into the bathing solution. Thus the calculated potassium efflux might be smaller than the real efflux.

PhysiologyPotassiumClinical Biochemistryfood and beverageschemistry.chemical_elementHuman physiologyMembraneReaction rate constantchemistryBiochemistryPhysiology (medical)BiophysicsMyocyteEffluxMuscle fibrePfl�gers Archiv f�r die Gesamte Physiologie des Menschen und der Tiere
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<i>In vitro</i> Modeling of Ryanodine Receptor 2 Dysfunction Using Human Induced Pluripotent Stem Cells

2011

Background/Aims: Induced pluripotent stem (iPS) cells generated from accessible adult cells of patients with genetic diseases open unprecedented opportunities for exploring the pathophysiology of human diseases in vitro. Catecholaminergic polymorphic ventricular tachycardia type 1 (CPVT1) is an inherited cardiac disorder that is caused by mutations in the cardiac ryanodine receptor type 2 gene (RYR2) and is characterized by stress-induced ventricular arrhythmia that can lead to sudden cardiac death in young individuals. The aim of this study was to generate iPS cells from a patient with CPVT1 and determine whether iPS cell-derived cardiomyocytes carrying patient specific RYR2 mutation recap…

PhysiologyRyanodine receptorCellular differentiationPharmacologyBiologyCatecholaminergic polymorphic ventricular tachycardiamedicine.diseaseRyanodine receptor 2Calcium imagingcardiovascular systemmedicineMyocytePatch clampInduced pluripotent stem cellCellular Physiology and Biochemistry
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De Novo prion aggregates trigger autophagy in skeletal muscle

2014

ABSTRACT In certain sporadic, familial, and infectious prion diseases, the prion protein misfolds and aggregates in skeletal muscle in addition to the brain and spinal cord. In myocytes, prion aggregates accumulate intracellularly, yet little is known about clearance pathways. Here we investigated the clearance of prion aggregates in muscle of transgenic mice that develop prion disease de novo . In addition to neurodegeneration, aged mice developed a degenerative myopathy, with scattered myocytes containing ubiquitinated, intracellular prion inclusions that were adjacent to myocytes lacking inclusions. Myocytes also showed elevated levels of the endoplasmic reticulum chaperone Grp78/BiP, su…

PrionsAutophagosome maturationanimal diseasesBlotting WesternImmunologyMice TransgenicBiologyProtein degradationPolymerase Chain ReactionMedical and Health SciencesMicrobiologyTransgenicPrion DiseasesMiceVirologyAutophagymedicineAnimalsMyocyteMuscle SkeletalEndoplasmic Reticulum Chaperone BiPHeat-Shock ProteinsDNA PrimersMuscle CellsAgricultural and Veterinary SciencesBlottingEndoplasmic reticulumNeurodegenerationAutophagySkeletal muscleSkeletalBiological Sciencesmedicine.diseaseImmunohistochemistryMolecular biologynervous system diseasesmedicine.anatomical_structureInsect ScienceChaperone (protein)biology.proteinMuscleWestern
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Calcitonin gene-related peptide partly protects cultured smooth muscle cells from apoptosis induced by an oxidative stress via activation of ERK1/2 M…

2003

Abstract Oxidative stress induced by a glucose/glucose oxidase (G/GO) generator system dose-dependently decreased the viability of cultured vascular smooth muscle cells (VSMC) as estimated by MTT assay. Cell death was induced in 40% of cells exposed to 0.2 IU/ml of the free radical generating mixture. Annexin-V labeling, Hoechst staining together with DNA laddering demonstrated that apoptosis was responsible for this cell loss. Pretreatment of the cells with 10−8 M calcitonin gene-related peptide (CGRP) significantly attenuated the damaging effect of the oxidative stress. Indeed, cell viability was estimated to be 80% in CGRP-treated group, instead of 60% in absence of CGRP treatment. This …

Programmed cell deathVascular smooth musclep38 mitogen-activated protein kinasesCalcitonin Gene-Related PeptideMyocytes Smooth MuscleApoptosisBiologyDNA ladderingCalcitonin gene-related peptidemedicine.disease_causeProtective AgentsMuscle Smooth VascularmedicineAnimalsHumansCGRPViability assayRats WistarMolecular BiologyCells CulturedMitogen-Activated Protein Kinase 3integumentary systemSAPKCell BiologyHydrogen PeroxideMAPKMolecular biologyRatsUp-RegulationNeuropeptideOxidative StressMitogen-activated protein kinaseVascular smooth muscle cellbiology.proteinMitogen-Activated Protein KinasesOxidative stressReceptors Calcitonin Gene-Related PeptideSignal TransductionBiochimica et biophysica acta
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