Search results for "N-2"

showing 10 items of 1881 documents

Anandamide inhibits IL-12p40 production by acting on the promoter repressor element GA-12: Possible involvement of the COX-2 metabolite prostamide E 2

2007

The eCB [endoCB (cannabinoid)] system is being considered as a novel therapeutic target for immune disorders. Cytokines of the IL-12 (interleukin-12) family have essential functions in cell-mediated immunity. In the present study, we have addressed the mechanisms of action of the eCB AEA (anandamide) on the regulation of IL-12p40 in activated microglia/macrophages. We demonstrated that AEA can inhibit the expression of p35, p19 and p40 subunits, which form the biologically-active cytokines IL-12 and IL-23 in microglia stimulated with LPS (lipopolysaccharide)/IFNγ (interferon γ). Additionally, we have provided evidence that AEA reduces the transcriptional activity of the IL-12p40 gene in LPS…

Lipopolysaccharidesmedicine.medical_specialtyPolyunsaturated Alkamidesmedicine.medical_treatmentMolecular Sequence DataRepressorArachidonic AcidsBiologyInterleukin-23BiochemistryDinoprostoneInterferon-gammaMicechemistry.chemical_compoundInternal medicinemedicineAnimalsEthanolamidePromoter Regions GeneticReceptors CannabinoidMolecular BiologyCells CulturedRegulation of gene expressionMice Inbred BALB CInterleukin-12 Subunit p40Cell BiologyAnandamideEndocannabinoid systemCell biologyProtein SubunitsEndocrinologyGene Expression RegulationchemistryCyclooxygenase 2lipids (amino acids peptides and proteins)MicrogliaCannabinoidSignal transductionEndocannabinoidsSignal TransductionProstaglandin E
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Evaluation of soluble CD 14 and neopterin as serum parameters of the inflammatory activity of pulmonary sarcoidosis.

1992

CD14 represents the most specific marker for monocytes/macrophages. It has been demonstrated in vitro that monocytes/macrophages lose this antigen upon activation. Results of studies investigating the expression of membrane-bound CD14 on the surface of monocytes/macrophages in sarcoidosis patients are controversial. To investigate whether the soluble form of CD14 reflects monocyte/macrophage activation in sarcoidosis, serum levels of soluble CD14 were determined concurrently with other serum markers of monocyte/macrophage activation (neopterin, angiotensin-converting enzyme) in 50 consecutive patients with bioptically confirmed sarcoidosis. The patients were allocated to three groups accord…

Lung Diseasesmedicine.medical_specialtySarcoidosisCD14CD4-CD8 RatioLipopolysaccharide ReceptorsAntigens Differentiation MyelomonocyticPeptidyl-Dipeptidase ANeopterinSensitivity and SpecificityMonocyteschemistry.chemical_compoundImmune systemAntigenAntigens CDInternal medicineDrug DiscoverymedicineMacrophageHumansGenetics (clinical)Inflammationmedicine.diagnostic_testMonocyteNeopterinGeneral MedicineMacrophage Activationmedicine.diseaseBiopterinBronchoalveolar lavageEndocrinologymedicine.anatomical_structurechemistrySolubilityImmunologyMolecular MedicineInterleukin-2SarcoidosisBronchoalveolar Lavage FluidBiomarkersThe Clinical investigator
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Interferon-α Suppresses cAMP to Disarm Human Regulatory T Cells

2013

Abstract IFN-α is an antineoplastic agent in the treatment of several solid and hematologic malignancies that exerts strong immune- and autoimmune-stimulating activity. However, the mechanisms of immune activation by IFN-α remain incompletely understood, particularly with regard to CD4+CD25highFoxp+ regulatory T cells (Treg). Here, we show that IFN-α deactivates the suppressive function of human Treg by downregulating their intracellular cAMP level. IFN-α–mediated Treg inactivation increased CD4+ effector T-cell activation and natural killer cell tumor cytotoxicity. Mechanistically, repression of cAMP in Treg was caused by IFN-α–induced MAP–ERK kinase (MEK)/extracellular signal-regulated ki…

MAPK/ERK pathwayCancer Researchmedicine.medical_treatmentGraft vs Host DiseaseAutoimmunitychemical and pharmacologic phenomenaBiologyLymphocyte ActivationT-Lymphocytes RegulatoryNatural killer cellMiceImmune systemDownregulation and upregulationT-Lymphocyte SubsetsCyclic AMPmedicineAnimalsHumansIL-2 receptorPhosphorylationExtracellular Signal-Regulated MAP KinasesCells CulturedMitogen-Activated Protein Kinase KinasesInterleukin-2 Receptor alpha SubunitInterferon-alphaFOXP3hemic and immune systemsDNA-Binding ProteinsKiller Cells NaturalSTAT Transcription Factorsmedicine.anatomical_structureCytokineOncologyHumanized mouseImmunologyCancer researchCancer Research
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P38 MAP Kinase Signaling Is Required for the Conversion of CD4+CD25− T Cells into iTreg

2008

CD4+CD25+ regulatory T cells (Treg) are important mediators of immune tolerance. A subset of Treg can be generated in the periphery by TGF-beta dependent conversion of conventional CD4+CD25− T cells into induced Treg (iTreg). In chronic viral infection or malignancy, such induced iTreg, which limit the depletion of aberrant or infected cells, may be of pathogenic relevance. To identify potential targets for therapeutic intervention, we investigated the TGF-beta signaling in Treg. In contrast to conventional CD4+ T cells, Treg exhibited marked activation of the p38 MAP kinase pathway. Inhibition of p38 MAP kinase activity prevented the TGF-beta-dependent conversion of CD4+CD25− T cells into …

MAPK/ERK pathwayp38 mitogen-activated protein kinasesImmunologyBlotting WesternImmunology/Immunomodulationlcsh:MedicineImmunology/Autoimmunitychemical and pharmacologic phenomenaBiologyT-Lymphocytes Regulatoryp38 Mitogen-Activated Protein KinasesImmune toleranceImmunology/Leukocyte Signaling and Gene ExpressionMiceAnimalsIL-2 receptorlcsh:ScienceMultidisciplinarylcsh:RInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsFlow CytometryCell biologyMitogen-activated protein kinaseCD4 Antigensbiology.proteinPhosphorylationlcsh:QSignal transductionResearch ArticleSignal TransductionPLoS ONE
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3,5-Diaizvietotu-4-fenil-2-pirolidonu un to strukturālo analogu sintēze

2017

5-Alkil-4-fenil-3-metilēn-2-pirolidonu un to strukturālo analogu sintēze. Baškevičs V., Darba vadītājs: Dr. ķīm. Maksims Vorona. Maģistra darbs. 102 lappuses, 18 attēli, 30 pielikumi, 32 literatūras avoti. Latviešu valodā. Uz Morita-Baylis-Hillman reakcijas produktu bāzes realizēta to acilēšana un nitroalkānu pievienošanas starpproduktam. Iegūtie starpprodukti tika pakļauti reducēšanas reakcijai un iekšmolekulārai ciklizācijai ar eksometilēn grupu saturošo 2-pirolidonu veidošanu. Šim nolūkam bija izmēģinātas vairākas literatūrā eksistējošas 5-alkil-4-fenil-3-metilēn-2-pirolidonu iegūšanas metodes, no kuriem tika izvēlēta reducēšanas reakcija ar dzelzs pulvera palīdzību ledus etiķskābē. Iegū…

MORITA-BAYLIS-HILLMAN REAKCIJA2-(5-ALKYL-3-METHYLENE-4-PHENYL-2-OXOPYRROLIDIN-1-YL)ACETAMIDES2-(5-ALKIL-4-FENIL-3-METILĒN-2-OKSOPIROLIDIN-1-IL)ACETAMIDI5-ALKIL-4-FENIL-3-METILĒN-2-PIROLIDONIĶīmija5-ALKYL-3-METHYLENE-4-PHENYL-2-PIRROLIDONES
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Macromolecular Prodrugs Based on Synthetic Polyaminoacids: Drug Delivery and Drug Targeting in Antitumor Therapy

2011

In the last twenty years a depth study on potential pharmaceutical applications of synthetic polymers at proteinlike structure as carrier for macromolecular prodrug production has been performed in academia and in industry. In particular α,β-poly(N-2-hydroxyethyl)-DL-aspartamide (PHEA), α,β-polyaspartylhydrazide (PAHy), poly(glutamic acid) (PGA), poly(aspartic acid) (PAA) and polylysine (PLL) have been extensively studied in this field. In the present review, the use of PHEA, PAHy, PGA as starting materials to prepare macromolecular prodrugs is reported and drug delivery and targeting aspects have been considered.

Macromolecular prodrugsStereochemistryMacromolecular SubstancesAntineoplastic AgentsGeneral MedicineGlutamic acidCombinatorial chemistryAntitumor therapyαβ-poly(N-2-hydroxyethyl)-DL-aspartamideαβ-polyaspartylhydrazide poly(glutamic acid) carrierchemistry.chemical_compoundanticancer drugsDrug Delivery SystemschemistryTargeted drug deliverySettore CHIM/09 - Farmaceutico Tecnologico ApplicativoPolylysineDrug DiscoveryAspartic acidDrug deliveryAnimalsHumansProdrugsAmino Acids
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The role of Interleukin-2 during the activation of cytotoxic T-lymphocytes

1983

Der vorliegende Artikel stellt eine Ubersicht dar Uber die derzeitigen Vorstellungen des Zusammenwirkens zellularer und humoraler Faktoren, die zu T-Zell-vermittelten zytotoxischen Immunreaktionen fuhren. Da im Ablauf derartiger Immunreaktionen hormonahnliche Wachstumsfaktoren (interleukine) eine entscheidende Rolle spielen, liegt der Schwerpunkt der Diskussion auf der Beschreibung dieser Mediatoren; insbesondere wird die Bedeutung von Interleukin-2 (Il-2) diskutiert. Il-2 ist ein losliches, nicht antigenspezifisches Glykoprotein mit einem Molekulargewicht von 15 000 Dalton (humanes Il-2) bzw. 30 000 Dalton (murines Il-2). Es wird in vitro von T-Helfer-Lymphozyten sezerniert, die in der Mau…

MacrophagesLymphocyte CooperationDrug DiscoveryHumansInterleukin-2Molecular MedicineT-Lymphocytes Helper-InducerGeneral MedicineReceptors ImmunologicModels BiologicalGenetics (clinical)Interleukin-1T-Lymphocytes CytotoxicKlinische Wochenschrift
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Pegylated nanoparticles based on a polyaspartamide. Preparation, physico-chemical characterization and intracellular uptake

2006

Nanoparticles with different surface PEGylation degree were prepared by using as starting material alpha,beta-poly(N-2-hydroxyethyl)-d,l-aspartamide (PHEA). PHEA was functionalized with a PEG amino-derivative for obtaining PHEA-PEG(2000) copolymer. Both PHEA and PHEA-PEG(2000) were derivatized with methacrylic anhydride (MA) for obtaining poly(hydroxyethylaspartamide methacrylated) (PHM) and poly(hydroxyethylaspartamide methacrylated)-PEGylated (PHM-PEG(2000)), respectively. Nanoparticles were obtained by UV irradiation of an inverse microemulsion, using as internal phase an aqueous solution of PHM alone or of the PHM/PHM-PEG(2000) mixture at different weight ratio and as external phase a m…

Magnetic Resonance SpectroscopyPolymers and PlasticsUltraviolet RaysNanoparticleMethacrylic anhydrideBioengineeringPolyethylene GlycolsBiomaterialschemistry.chemical_compoundMicroscopy Electron TransmissionPEG ratioPolymer chemistrySpectroscopy Fourier Transform InfraredMaterials ChemistryZeta potentialHumansMicroemulsionParticle SizeNanoparticlesalphabeta-poly(N-2-hydroxyethyl)-dl-aspartamide (PHEA)methacrylic anhydride.Aqueous solutionchemistryPropylene carbonatePEGylationMethacrylatesNanoparticlesK562 CellsPeptidesNuclear chemistry
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ATNX2 is not a regulatory gene in Italian amyotrophic lateral sclerosis patients with C9ORF72 GGGGCC expansion

2015

Abstract There are indications that both familial amyotrophic lateral sclerosis (ALS) and sporadic ALS phenotype and prognosis are partly regulated by genetic and environmental factors, supporting the theory that ALS is a multifactorial disease. The aim of this article was to assess the role of ATXN2 intermediate length repeats in a large series of Italian and Sardinian ALS patients and controls carrying a pathogenetic C9ORF72 GGGGCC hexanucleotide repeat. A total of 1972 ALS cases were identified through the database of the Italian ALS Genetic consortium, a collaborative effort including 18 ALS centers throughout Italy. The study population included: (1) 276 Italian and 57 Sardinian ALS ca…

Male0301 basic medicineAgingC9ORF72Genetic Association Studie030105 genetics & heredityBiologySettore MED/03 - GENETICA MEDICA03 medical and health sciences0302 clinical medicineC9orf72medicineAlleleAmyotrophic lateral sclerosisAmyotrophic lateral sclerosiAgedAtaxin-2Regulator geneAmyotrophic lateral sclerosis; ATXN2; C9ORF72; Phenotype; Neuroscience (all); Medicine (all); Aging; Developmental Biology; Geriatrics and Gerontology; Neurology (clinical)GeneticsDNA Repeat ExpansionNeuroscience (all)ProteinMedicine (all)General NeuroscienceATXN2Middle AgedDNA Repeat Expansionmedicine.diseaseAmyotrophic lateral sclerosis3. Good healthC9orf72 ProteinAmyotrophic lateral sclerosis; ATXN2; C9ORF72; Phenotype; Neurology (clinical); Neuroscience (all); Aging; Developmental Biology; Geriatrics and GerontologyPhenotypeItalyPopulation studyFemaleSettore MED/26 - NeurologiaNeurology (clinical)Geriatrics and GerontologyTrinucleotide repeat expansion030217 neurology & neurosurgeryHumanDevelopmental Biology
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Intraperitoneal administration of the anti-IL-23 antibody prevents the establishment of intestinal nematodes in mice

2018

AbstractPrevious studies have established that an increased Th-9 response creates a hostile environment for nematode parasites. Given that IL-23, a cytokine required for maintenance of the IL-17–secreting phenotype, has inhibitory effects on IL-9 production, we hypothesized that reducing circulating IL-23 by treatment with anti-IL-23 antibodies would reduce the establishment and development of parasitic intestinal nematodes. In this study, we show that animals treated with anti-IL-23 monoclonal antibodies showed a drastic reduction in the number of mouse pinworms (Aspiculuris tetraptera) recovered from the intestine (p < 0.001) at 23 days post-infection compared to the untreated animals.…

Male0301 basic medicineChemokinemedicine.drug_classmedicine.medical_treatmentlcsh:MedicineMonoclonal antibodyInterleukin-23Inflammatory bowel diseaseArticleParasite LoadMice03 medical and health sciencesInterleukin 23medicineAnimalsNematode Infectionslcsh:ScienceMice Inbred ICRMultidisciplinarybiologyInterleukinslcsh:RAntibodies Monoclonalmedicine.diseasebiology.organism_classificationIntestinesCCL20030104 developmental biologyNematodeCytokineImmunologybiology.proteinlcsh:QAntibodyInjections Intraperitoneal
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