Search results for "Neurotrophic factor"

showing 10 items of 153 documents

Group II metabotropic glutamate receptor activation by agonist LY379268 treatment increases the expression of brain derived neurotrophic factor in th…

2009

A number of in vitro and in vivo studies using selective agonists have indicated a neuroprotective role for group-II metabotropic glutamate (mGlu2/3) receptors in various models of neuronal injury. Although an interplay among neurotrophic factors and mGlu2/3 receptors signalling system has been suggested as possible mechanism involved on neuroprotection, at present poor information are available concerning the in vivo regulation by mGlu2/3 receptors activation of specific neurotrophic factors. By using in situ hybridization and western blotting methods the aim of present study was to analyse the potential regulatory role of selective mGluR2/3 agonist LY379268 treatment on brain derived neur…

Malemedicine.medical_specialtyTime Factorsmedicine.drug_classTropomyosin receptor kinase BBiologyReceptors Metabotropic GlutamateHippocampusSettore BIO/09 - FisiologiaMiceNeurotrophic factorsInternal medicineExcitatory Amino Acid AgonistsmedicineAnimalsRNA MessengerAmino Acidsneurotrophic factorNeuronsBrain-derived neurotrophic factorDose-Response Relationship DrughippocampuBrain-Derived Neurotrophic FactorGeneral NeuroscienceGlutamate receptorBrainLY379268Bridged Bicyclo Compounds HeterocyclicReceptor antagonistUp-RegulationMice Inbred C57BLMetabotropic receptorEndocrinologyBDNFXanthenesMetabotropic glutamate receptorcerebral cortexMetabotropic glutamate receptor 2Excitatory Amino Acid AntagonistsmGlu2/3 receptor
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Changes in cortico-spinal excitability following uphill versus downhill treadmill exercise

2017

International audience; An acute bout of aerobic exercise induces neuroplasticity in the motor cortex. Moreover, paired associative stimulation (PAS) is known to induce neuroplasticity in M1. However, the possible influence of the type of exercise on the neuroplastic changes remains unknown. The present study investigated the effects of two different modes of muscle contraction produced during locomotor exercise on changes in corticospinal (CS) excitability. Subjects performed two 30-min treadmill exercises at an intensity corresponding to 60% of their maximal heart rate with either a +10% (uphill) or -10% (downhill) slope. These exercises were followed or not by paired associative stimulat…

Malemedicine.medical_treatmentPyramidal TractsStimulationBehavioral Neuroscience0302 clinical medicineHeart RateTreadmillelbow flexorsneurotrophic factoreccentric cycle exerciseRespirationDownhillHealthy Volunteersmedicine.anatomical_structureTreadmill runningFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]medicine.symptomPsychologyTreadmill walkingLocomotionMotor cortexMuscle contractionAdultpaired associative stimulationmedicine.medical_specialtyneuroplasticitylengthening contractionsYoung Adult03 medical and health sciencesPhysical medicine and rehabilitationmotor cortexIsometric ContractionNeuroplasticityHeart ratemedicineHumansAerobic exerciseKneeExerciseElectromyographysingle bout030229 sport sciencesEvoked Potentials Motorhuman quadricepsMedian NerveTranscranial magnetic stimulation[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Exercise Testparkinsons-diseaseUphill030217 neurology & neurosurgeryTranscranial magnetic stimulation
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Lactate Threshold Training Program on Patients with Multiple Sclerosis: A Multidisciplinary Approach

2021

Physical activity could play a key role in improving the quality of life, particularly in patients with nervous system diseases such as multiple sclerosis (MS). Through lactacid anaerobic training, this study aims to investigate the effects at a bio-psycho-physical level to counteract the chronic fatigue associated with the pathology, and to improve mental health at a psychological and neurotrophic level. Eight subjects (age: 34.88 ± 4.45 years) affected by multiple sclerosis were involved. A lactate threshold training program was administered biweekly for 12 weeks at the beginning of the study (T0), at the end of the study (T1) and at 9 months after the end of the study (T2), with physical…

Malephysical activityDiseaseneurotrophinschemistry.chemical_compoundMedicineTX341-641FatigueNeuronal PlasticityNutrition and DieteticsneurotrophinMiddle AgedNeuroprotectionSelf EfficacyExercise TherapyTreatment Outcomemultiple sclerosis; physical activity; diet habits; neurotrophins; BDNF; DHEASmultiple sclerosiEating Attitudes TestFemalediet habitsAnaerobic exerciseAdultmedicine.medical_specialtyMultiple SclerosisNormal dietDHEASPhysical exerciseArticleYoung AdultDehydroepiandrosterone sulfateInternal medicineHumansLactic AcidExercisePatient Care TeamMotivationNutrition. Foods and food supplybusiness.industryBrain-Derived Neurotrophic FactorMultiple sclerosisLactate thresholdDehydroepiandrosteroneFeeding Behaviormedicine.diseaseBDNFchemistrydiet habitbusinessBiomarkersFood ScienceNutrients; Volume 13; Issue 12; Pages: 4284
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Missense variants in DPYSL5 cause a neurodevelopmental disorder with corpus callosum agenesis and cerebellar abnormalities

2021

International audience; The collapsin response mediator protein (CRMP) family proteins are intracellular mediators of neurotrophic factors regulating neurite structure/spine formation and are essential for dendrite patterning and directional axonal pathfinding during brain developmental processes. Among this family, CRMP5/DPYSL5 plays a significant role in neuronal migration, axonal guidance, dendrite outgrowth, and synapse formation by interacting with microtubules. Here, we report the identification of missense mutations in DPYSL5 in nine individuals with brain malformations, including corpus callosum agenesis and/or posterior fossa abnormalities, associated with variable degrees of intel…

Models MolecularMale0301 basic medicineHydrolases[SDV]Life Sciences [q-bio]Hippocampal formationMedical and Health Sciences0302 clinical medicineNeurodevelopmental disorderTubulinModelsNeurotrophic factorsCerebellumIntellectual disability2.1 Biological and endogenous factorsMissense mutationAetiologyChilddendrite branchingGenetics (clinical)de novo missense variantsPediatricGenetics & HeredityDPYSL5Biological Sciences[SDV] Life Sciences [q-bio]corpus callosum agenesisMental HealthChild PreschoolNeurologicalFemaleMicrotubule-Associated ProteinsAdultNeuriteIntellectual and Developmental Disabilities (IDD)primary neuronal culturesMutation MissenseBiologyYoung Adult03 medical and health sciencesRare DiseasesMediatorReportIntellectual DisabilityGeneticsmedicineHumansPreschoolCorpus Callosum Agenesisbrain malformationNeurosciencesMolecularmedicine.diseaseneurodevelopmental disorderBrain Disorders030104 developmental biologyNeurodevelopmental DisordersMutationMissenseAgenesis of Corpus CallosumNeuroscience030217 neurology & neurosurgery
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Interleukin-6 and Soluble Interleukin-6 Receptor: Direct Stimulation of gp130 and Hematopoiesis

1998

T HE INTERLEUKIN-6 (IL-6) family of cytokines acts via receptor complexes that contain at least one subunit of the signal transducing protein gp130.[1][1] The family comprises IL-6, IL-11, ciliary neurotrophic factor (CNTF), cardiotrophin-1 (CT-1), leukemia inhibitory factor (LIF), and oncostatin M

Models Molecularendocrine systemmedicine.medical_specialtyRecombinant Fusion ProteinsImmunologyMice TransgenicLeukemia inhibitory factor receptorOncostatin MBiologyCiliary neurotrophic factorBiochemistryDesigner DrugsMiceStructure-Activity RelationshipAntigens CDInternal medicineCytokine Receptor gp130medicineAnimalsHumansInterleukin 6Membrane GlycoproteinsInterleukin-6Oncostatin MOncostatin M receptorCell DifferentiationReceptors InterleukinCell BiologyHematologyHematopoietic Stem CellsGlycoprotein 130Receptors Interleukin-6Molecular biologyHematopoiesisEndocrinologyLiverSolubilitybiology.proteinSignal transductionPeptidesLeukemia inhibitory factorSpleenBlood
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Reduced presynaptic efficiency of excitatory synaptic transmission impairs LTP in the visual cortex of BDNF-heterozygous mice

2006

The neurotrophin brain-derived neurotrophic factor (BDNF) plays an important role in neuronal survival, axonal and dendritic growth and synapse formation. BDNF has also been reported to mediate visual cortex plasticity. Here we studied the cellular mechanisms of BDNF-mediated changes in synaptic plasticity, excitatory synaptic transmission and long-term potentiation (LTP) in the visual cortex of heterozygous BDNF-knockout mice (BDNF(+/-)). Patch-clamp recordings in slices showed an approximately 50% reduction in the frequency of miniature excitatory postsynaptic currents (mEPSCs) compared to wild-type animals, in the absence of changes in mEPSC amplitudes. A presynaptic impairment of excita…

N-MethylaspartatePatch-Clamp TechniquesTime FactorsLong-Term PotentiationPresynaptic TerminalsAMPA receptorIn Vitro TechniquesSynaptic TransmissionMicePostsynaptic potentialQuinoxalinesExcitatory Amino Acid AgonistsAnimalsalpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic AcidVisual CortexMice KnockoutNeuronsBrain-derived neurotrophic factorDose-Response Relationship DrugPost-tetanic potentiationChemistryBrain-Derived Neurotrophic FactorGeneral NeuroscienceDose-Response Relationship RadiationLong-term potentiationElectric StimulationSynaptic fatigueAnimals Newbornnervous systemSynaptic plasticityExcitatory postsynaptic potentialCalciumExcitatory Amino Acid AntagonistsNeuroscienceEuropean Journal of Neuroscience
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Updated overview on interplay between physical exercise, neurotrophins, and cognitive function in humans

2019

Highlights • Physical exercise maintains or improves cognitive functions, and neurotrophin receptor signaling may play an important role. • A positive relationship exists between physical exercise and circulating brain-derived neurotrophic factor levels. • Less clear evidence has been found for a relationship between physical exercise and other neurotrophin levels, such as nerve growth factor, neurotrophin-3, and neurotrophin-4. • The postexercise variation of brain-derived neurotrophic factor might be associated with improvement of neurocognitive functioning. • Physical exercise may be an inexpensive and safe strategy for boosting brain-derived neurotrophic factor release, thus preserving …

Nerve Tissue ProteinsPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseReceptors Nerve Growth Factorneurotrophins03 medical and health sciencesCognition0302 clinical medicineNeurotrophin 3Neurotrophic factorsphysical exerciseRegular PaperHumansMedicineAerobic exerciseOrthopedics and Sports MedicineNerve Growth Factors030212 general & internal medicineExercisephysical exercise neurotrophins cognitive functioncognitive functionSportbiologybusiness.industryBrain-Derived Neurotrophic FactorCognition030229 sport sciencesNerve growth factorSynaptic plasticitybiology.proteinbusinessNeuroscienceNeurocognitiveNeurotrophin
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Regulation of neurogenesis by neurotrophins in developing spinal sensory ganglia.

2002

Neurons and glia in spinal sensory ganglia derive from multipotent neural crest-derived stem cells. In contrast to neural progenitor cells in the central nervous system, neural crest progenitors coexist with differentiated sensory neurons all throughout the neurogenic period. Thus, developing sensory ganglia are advantageous for determining the possible influence of cell-cell interactions in the regulation of precursor proliferation and neurogenesis. Neurotrophins are important regulators of neuronal survival in the developing vertebrate nervous system and, in addition, they appear to influence precursor behavior in vitro. Studies in mice carrying mutations in neurotrophin genes provide a g…

Nervous systemCentral nervous systemSensory systemReceptors Nerve Growth FactorBiologyMiceNeurotrophic factorsGanglia SpinalmedicineAnimalsNerve Growth FactorsNeurons AfferentGeneral NeuroscienceStem CellsNeurogenesisNeural crestCell DifferentiationNeural stem cellmedicine.anatomical_structurenervous systemNeural Crestbiology.proteinNeuroscienceNeurogliaCell DivisionNeurotrophinBrain research bulletin
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Interleukin-6 (IL-6) and its soluble receptor support survival of sensory neurons

1999

The cytokine interleukin-6 (IL-6) has multiple functions in the immune and hematopoietic systems. IL-6 is related to ciliary neurotrophic factor (CNTF), a trophic factor for motoneurons, sensory dorsal root ganglion (DRG) neurons, and other neuronal subpopulations. Both act via related receptor complexes, consisting of one ligand-specific α-receptor subunit (IL-6R and CNTFR, respectively) and two signal-transducing receptor components. Even though IL-6 is expressed by neurons and glia, the functions of IL-6 in the nervous system are poorly understood. Here, we report that exogenous human IL-6 promotes the survival of dissociated newborn rat DRG neurons in vitro if supplemented with soluble …

Nervous systemCytoplasmCiliary neurotrophic factorCellular and Molecular NeuroscienceDorsal root ganglionNeurotrophic factorsGanglia SpinalNerve Growth FactormedicineAnimalsHumansCiliary Neurotrophic FactorNeurons AfferentReceptorAutocrine signallingInterleukin 6Cells CulturedCell DeathDose-Response Relationship DrugbiologyInterleukin-6Receptors Interleukin-6RatsCell biologyAutocrine Communicationmedicine.anatomical_structureAnimals Newbornnervous systembiology.proteinNeuroscienceNeurotrophinJournal of Neuroscience Research
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Sensing life: regulation of sensory neuron survival by neurotrophins

2002

Neurotrophins are a family of structurally and functionally related neurotrophic factors which, in mammals, include: nerve growth factor, brain-derived neurotrophic factor, neurotrophin-3 (NT-3), and NT-4/5. In addition to their canonical role in promoting neuronal survival, these molecules appear to regulate multiple aspects of the development of the nervous system in vertebrates, including neuronal differentiation, axon elongation and target innervation, among others. Actions of neurotrophins and of their receptors in vivo are being analyzed by loss-of-function or gain-of-function experiments in mice. Here, we review the phenotypes of the primary sensory system in these mutant mouse strai…

Nervous systemGenetically modified mouseCell SurvivalMice TransgenicSensory systemReceptors Nerve Growth FactorMiceCellular and Molecular NeuroscienceNeurotrophic factorsmedicineAnimalsReceptor trkCNerve Growth FactorsNeurons AfferentAxonMolecular BiologyMice KnockoutPharmacologyMembrane GlycoproteinsbiologyBrain-Derived Neurotrophic FactorCell BiologyAnatomyProtein-Tyrosine KinasesSensory neuronmedicine.anatomical_structureNerve growth factornervous systembiology.proteinMolecular MedicineNeuroscienceSignal TransductionNeurotrophinCellular and Molecular Life Sciences
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