Search results for "PATHOGEN"

showing 10 items of 1657 documents

Modulation of immune responses ofRhynchophorus ferrugineus(Insecta: Coleoptera) induced by the entomopathogenic nematodeSteinernema carpocapsae(Nemat…

2014

Aim of this study was to investigate relationships between the red palm weevil (RPW) Rhynchophorus ferrugineus (Olivier) and the entomopathogenic nematode Steinernema carpocapsae (EPN); particularly, the work was focused on the immune response of the insect host in naive larvae and after infection with the EPN. Two main immunological processes have been addressed: the activity and modulation of host prophenoloxidase-phenoloxidase (proPO) system, involved in melanization of not-self and hemocytes recognition processes responsible for not-self encapsulation. Moreover, immune depressive and immune evasive strategies of the parasite have been investigated. Our results suggest that RPW possess a…

biologymedia_common.quotation_subjectWeevilEntomopathogenic nematodeInsectbiology.organism_classificationGeneral Biochemistry Genetics and Molecular BiologyMicrobiologyRhynchophorusImmune systemInsect ScienceBotanyParasite hostingAgronomy and Crop ScienceRhabditidaEcology Evolution Behavior and SystematicsSymbiotic bacteriamedia_commonInsect Science
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Latent measles virus infection in Vero cells depending on a temperature-sensitive phenomenon.

1978

A latent infection by measles virus in a line of Vero cells could be maintained only at 37 degrees C. The conditions of temperature nonpermissiveness were associated with some block in virus production and/or release and with the establishment of an autointerference phenomenon. Reduction of the incubation temperature to 33.5 degrees C induced a rather rapid transition from the latent to a lytical infection with a recue of virus. The rescued virus exhibited a restricted capacity to grow at 37 degrees C.

biologyvirusesTemperatureGeneral MedicineHaplorhinibiology.organism_classificationVirus ReplicationVirologyVirusMicrobiologyCell LineMeasles virusIncubation temperatureCytopathogenic Effect ViralMeasles virusVirologyViral InterferenceVero cellAnimalsTemperature sensitiveAntigens ViralArchives of virology
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Animal models: Murine cytomegalovirus

2002

Publisher Summary This chapter focuses on murine cytomegalovirus (CMV) animal models. Multiple-organ cytomegalovirus disease, interstitial pneumonia in particular, is a major concern in the therapy of hematopoietic malignancies by hematoablative treatment and bone marrow transplantation (BMT). Human CMV (hCMV) is the prototype member of the subfamily, Betaherpesvirinae, of the virus family, Herpesviridae . Its genome is a linear, double-stranded DNA with a coding capacity of ca. 165 open reading frames. During an aeon of co-evolution, CMVs have adapted themselves to their respective hosts; therefore, CMV biology is most reliably studied in a natural virus-host combination. Even though hCMV …

biologyvirusesViral pathogenesisvirus diseasesCytomegalovirusmedicine.disease_causebiology.organism_classificationVirologyHerpesviridaeVirusImmune systemViral replicationBetaherpesvirinaeImmunologymedicineCytotoxic T cell
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Does autoimmunity play a part in the pathogenesis of glaucoma?

2013

Glaucoma is a chronic neurodegenerative disease and one of the leading causes of blindness. Several risk factors have been described, e.g. an elevated intraocular pressure (IOP), oxidative stress or mitochondrial dysfunction. Additionally, alterations in serum antibody profiles of glaucoma patients, upregulation (e.g. anti-HSP60, anti-MBP) and downregulation (e.g. anti-14-3-3), have been described, but it still remains elusive if the autoantibodies seen in glaucoma are an epiphenomenon or causative. However, it is known that elicited autoimmunity causes retinal ganglion cell loss resulting in glaucomatous-like damage and according to the autoaggressive nature of some autoantibodies we found…

business.industryAutoantibodyGlaucomaAutoimmunityGlaucomaDiseasemedicine.diseasemedicine.disease_causeNeuroprotectionRetinaSensory SystemsAutoimmune DiseasesBiomarker (cell)AutoimmunityPathogenesisDisease Models AnimalOphthalmologyImmunologyProtective autoimmunitymedicineAnimalsHumanssense organsbusinessAutoantibodiesProgress in Retinal and Eye Research
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Neuroprotection and autoimmunity in glaucoma

2012

In glaucoma, the elevated intraocular pressure cannot explain the disease in all patients. However, the pathogenesis of the disease is widely unknown. Biomarker research could be help to understand the disease process. Beside some genetic and proteomic biomarkers, immunoproteomics could play a significant role. Several studies could provide hints for an involvement of autoantibodies in the pathogenesis of the disease. The complex profiles of natural occurring autoantibodies were analyzed by Western Blotting and mass spectrometry based techniques combined with multivariate statistics and artificial neural networks. In several studies we could demonstrate consistent up- and down-regulations o…

business.industryAutoantibodyGlaucomaGeneral MedicineDiseasemedicine.diseasemedicine.disease_causeNeuroprotectionImmunoproteomicsAutoimmunityPathogenesisOphthalmologyImmunologyBiomarker (medicine)MedicinebusinessActa Ophthalmologica
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OP0081 Aberrant Expression of IL-22RA1 on Hematopoietic Cells as Immunologically Signature of Primary Sjogren’s Syndrome and Sjogren-Associated Non-H…

2013

Background Interleukin (IL)-22 is a potent mediator of cellular inflammatory responses that has been recently reported to play a role in the pathogenesis of primary Sjogren’s Syndrome (p-SS) (1, 2) and of T and B lymphomas. IL-22 biological activity is initiated by binding to a cell-surface complex composed of two subunits, IL-22R1 and IL-10R2 receptor chains, and further regulated by interactions with a soluble binding protein, IL-22BP. Unlike the IL-10R2, which is constitutively expressed in many human tissues, IL-22R1 is not detectable in immune cells. Objectives Aim of this study was to better characterize the role of IL-22 axis in the pathogenesis of p-SS and p-SS-associated lymphomas.…

business.industryCD68ImmunologyInterleukinmedicine.diseasePeripheral blood mononuclear cellGeneral Biochemistry Genetics and Molecular BiologyLymphomaInterleukin 22PathogenesisHaematopoiesisImmune systemRheumatologyImmunologyCancer researchImmunology and AllergyMedicinebusinessAnnals of the Rheumatic Diseases
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Functional relevance of soluble TNF-alpha, transmembrane TNF-alpha and TNF-signal transduction in gastrointestinal diseases with special reference to…

2002

As a result of extensive clinical and basic research, the pivotal role of tumour necrosis factor (TNF) in the pathogenesis of chronic inflammatory diseases such as inflammatory bowel disease (IBD) has now generally been acknowledged. This has led to promising clinically effective anti-TNF-strategies. Of note, there is more and more evidence that TNF seems to play a key role in other gastrointestinal diseases including Helicobacter pylori infection, pancreatitis, viral hepatitis and toxic liver damage, too. The action of TNF at the cellular level is mediated by two cell surface receptors, TNF-R1 (p60) and TNF-R2 (p80). The function of these receptors and the downstream intracellular signal t…

business.industryGastrointestinal DiseasesTumor Necrosis Factor-alphamedicine.medical_treatmentGastroenterologyAntibodies MonoclonalProteinsDiseasemedicine.diseaseInflammatory Bowel DiseasesTNF Receptor-Associated Factor 2Inflammatory bowel diseaseTNF Receptor-Associated Factor 1PathogenesisTransduction (genetics)CytokineImmunologyMedicineHumansTumor necrosis factor alphaSignal transductionbusinessReceptorSignal TransductionZeitschrift fur Gastroenterologie
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Immune pathogenesis of hepatitis A

1992

In an effort to elucidate the mechanism of liver damage resulting from Hepatitis A virus (HAV) infection, we have studied infected skin fibroblasts and autologous lymphocytes from HAV patients. We report here that HLA-restricted virus-specific T cells play an essential role in HAV-related hepatocellular injury.

business.industryMechanism (biology)virusesfungivirus diseasesHepatitis AAutologous lymphocytebiochemical phenomena metabolism and nutritionmedicine.diseaseVirologydigestive system diseasesHepatitis a virusImmune pathogenesismedicineLiver damageHepatocellular injurybusiness
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Heat Shock Proteins in Multiple Sclerosis Pathogenesis: Friend or Foe?

2015

Multiple Sclerosis is a complex chronic inflammatory, neurodegenerative disease conditioned by genetic, epigenetic and environmental factors. Main pathological features of MS include areas of focal demyelination of white matter characterized by gliosis, neuron and oligodendrocyte loss. Neurodegenerative as well as immune-mediated processes play a role in the pathogenesis of this disease. One of these immunogenic factors could be represented by the heat shock proteins. HSP exhibit cytoprotective and cytostimulatory effects due to their molecular chaperones role, in many brain model misfolding diseases such as Alzheimer’s and Parkinson’s diseases, whereas still no unambiguous results have bee…

business.industryMultiple sclerosisCentral nervous systemDiseasemedicine.diseasemedicine.disease_causeOligodendrocyteAutoimmunityPathogenesismedicine.anatomical_structureGliosisAutoimmunity Central nervous system Chaperone activity Demyelination Heat shock proteins Multiple sclerosisHeat shock proteinmedicinemedicine.symptomSettore BIO/06 - Anatomia Comparata E CitologiabusinessNeuroscience
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NF-κB in inflammatory bowel disease

2008

Apart from genetic and environmental factors, the mucosal immune system of the gut plays a central role in the pathogenesis of inflammatory bowel disease (IBD). In the healthy gut, the mucosal immune system ensures the balance between pro- and anti-inflammatory mediators and thereby allows an effective defence against luminal pathogens but at the same time prevents an overwhelming immune reaction directed against the huge amount of harmless luminal antigens (for example, components of food or nonpathological bacteria). In both entities of IBD (Crohn's disease and ulcerative colitis) this immunological balance is severely impaired and shifted towards the pro-inflammatory side. The chronic mu…

business.industryNF-κBInflammationmedicine.diseaseInflammatory bowel diseaseUlcerative colitisdigestive system diseasesPathogenesischemistry.chemical_compoundImmune systemchemistryAntigenImmunityImmunologyInternal Medicinemedicinemedicine.symptombusinessJournal of Internal Medicine
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