Search results for "Phenotype"

showing 10 items of 1875 documents

Adipose stromal cells promote the transition of colorectal cancer cells toward a mesenchymal-like phenotype

2021

ABSTRACT Colon cancer progression is among the risks that increase with obesity. We have recently unveiled the molecular mechanism by which adipose tissue-released molecules, HGF and IL-6, make colorectal cancer (CRC) cells acquiring mesenchymal traits. Targeting of adipose-derived factors abrogate the metastatic potential of CRC stem cells (CR-CSCs) in obese patients.

cancer stem cellCancer ResearchStromal cellTransition (genetics)Colorectal cancerbusiness.industryadipose stromal cellMesenchymal stem cellAdipose tissuecolorectal cancermedicine.diseasePhenotypedigestive system diseaseshybrid EMTCommentaryMolecular mechanismCancer researchmedicineMolecular MedicineSettore MED/46 - Scienze Tecniche Di Medicina Di LaboratorioStem cellbusinessobesity.Article CommentaryMolecular & Cellular Oncology
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Instruction of haematopoietic lineage choices, evolution of transcriptional landscapes and cancer stem cell hierarchies derived from an AML1-ETO mous…

2013

The t(8;21) chromosomal translocation activates aberrant expression of the AML1-ETO (AE) fusion protein and is commonly associated with core binding factor acute myeloid leukaemia (CBF AML). Combining a conditional mouse model that closely resembles the slow evolution and the mosaic AE expression pattern of human t(8;21) CBF AML with global transcriptome sequencing, we find that disease progression was characterized by two principal pathogenic mechanisms. Initially, AE expression modified the lineage potential of haematopoietic stem cells (HSCs), resulting in the selective expansion of the myeloid compartment at the expense of normal erythro- and lymphopoiesis. This lineage skewing was foll…

cancer stem cellsCancer stem cells; Core binding factor acute myeloid leukaemia; Preclinical mouse model; Therapy target validation; Whole transcriptome sequencingMyeloidtherapy target validationOncogene Proteins FusionCloseupsBiologyGranulocyte-Macrophage Progenitor CellsTranslocation Geneticwhole transcriptome sequencingImmunophenotypingMiceGranulocyte-Macrophage Progenitor CellsCancer stem cellhemic and lymphatic diseasesmedicineAML1-ETOAnimalsCell Lineageacute myeloid leukaemiaLymphopoiesisProgenitor cellt(8;21)Research Articlespreclinical mouse modelGeneticsRegulation of gene expressionAntibiotics AntineoplasticSequence Analysis RNAcore binding factor acute myeloid leukaemiainducible mouse-modelHematopoietic Stem CellsMice Inbred C57BLDisease Models AnimalLeukemia Myeloid AcuteHaematopoiesisPhenotypemedicine.anatomical_structureGene Expression RegulationDoxorubicinCancer researchNeoplastic Stem CellsMolecular MedicineStem cell
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Genetic and Molecular Characterization of The Human Osteosarcoma 3AB-OS Cancer Stem Cell Line: A Possible Model For Studying Osteosarcoma Origin and …

2013

Finding new treatments targeting cancer stem cells (CSCs) within a tumor seems to be critical to halt cancer and improve patient survival. Osteosarcoma is an aggressive tumor affecting adolescents, for which there is no second-line chemotherapy. Uncovering new molecular mechanisms underlying the development of osteosarcoma and origin of CSCs is crucial to identify new possible therapeutic strategies. Here, we aimed to characterize genetically and molecularly the human osteosarcoma 3AB-OS CSC line, previously selected from MG63 cells and which proved to have both in vitro and in vivo features of CSCs. Classic cytogenetic studies demonstrated that 3AB-OS cells have hypertriploid karyotype wit…

cancer stem cellsPhysiologyClinical Biochemistrymedicine.disease_causePolymerase Chain ReactionOsteosarcoma cancer stem cellSettore BIO/10 - BiochimicaChromosomes HumanGene Regulatory NetworksCopy-number variationOligonucleotide Array Sequence AnalysisGeneticsComparative Genomic HybridizationOsteosarcomabiologychromosomal aberrationGene Expression Regulation NeoplasticPhenotypemiRNAsNeoplastic Stem CellsOsteosarcomaMitosisBone NeoplasmsHMGA2Cancer stem cellCell Line TumormicroRNABiomarkers Tumorgene expression profilingmedicineHumansOsteosarcoma cancer stem cells; karyotype; chromosomal aberrations; gene expression profiling; miRNAsCell LineageGenetic Predisposition to DiseaseRNA MessengerCell NucleusChromosome AberrationsPloidiesModels GeneticComputational BiologyCancerCell Biologymedicine.diseasekaryotypeMicroRNAsKaryotypingbiology.proteinCancer researchCarcinogenesisComparative genomic hybridization
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Butyrate-Induced Growth Arrest of GH3-Cells is not Linked to a Distinct Morphological Phenotype

1983

N-butyric acid is known to be a potent proliferation-inhibitor of a great number of cell types, both normal and neoplastic (1). In many cases growth arrest is accompanied by striking changes in morphology, e.g. formation of cell processes or increased spreading (1). These changes can be traced back to altered glycolipide and glycoprotein patterns of the plasma membrane and to a reorganization of the cytoskeleton (2, 3).

chemistry.chemical_classificationCell typeChemistryCellSodium butyrateButyratePhenotypeCell biologychemistry.chemical_compoundmedicine.anatomical_structureMembranemedicinesense organsCytoskeletonGlycoprotein
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Peripheral innervation of the heart

1987

The present immunohistochemical study demonstrates the multiplicity, histotopography and origin of peptidergic innervation in the mammalian heart. Neuropeptide Y (NPY) is the major representative of peptides in cardiac sympathetic efferents. Sympathetic afferents are characterized by the presence of tachykinins, calcitonin gene-related peptide and apparently also some opioid peptides. Predominant peptides of the vagal system are tachykinins. The intrinsic peptidergic system predominantly consists of vasoactive intestinal polypeptide/peptide histidine isoleucine. Paracrine systems are merely opioid-ergic. Target relations of extrinsic and intrinsic peptidergic nerves were found to be more di…

chemistry.chemical_classificationParacrine signallingchemistryCalcitoninVasoactive intestinal peptideImmunohistochemistryPeptideBiologyNeuropeptide Y receptorOpioid peptideNeurosciencePhenotype
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Identification of the 3-amino-3-carboxypropyl (acp) transferase enzyme responsible for acp3U formation at position 47 in Escherichia coli tRNAs

2019

AbstracttRNAs from all domains of life contain modified nucleotides. However, even for the experimentally most thoroughly characterized model organism Escherichia coli not all tRNA modification enzymes are known. In particular, no enzyme has been found yet for introducing the acp3U modification at position 47 in the variable loop of eight E. coli tRNAs. Here we identify the so far functionally uncharacterized YfiP protein as the SAM-dependent 3-amino-3-carboxypropyl transferase catalyzing this modification and thereby extend the list of known tRNA modification enzymes in E. coli. Similar to the Tsr3 enzymes that introduce acp modifications at U or m1Ψ nucleotides in rRNAs this protein conta…

chemistry.chemical_classificationTRNA modificationAlkyl and Aryl TransferasesNucleic Acid EnzymesNucleotidesRNASaccharomyces cerevisiaeBiologymedicine.disease_causePhenotypeEnzymechemistryBiochemistryBacterial ProteinsRNA TransferTransfer RNAGeneticsmedicineEscherichia coliTransferaseNucleic Acid ConformationNucleotideEscherichia coliNucleic Acids Research
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Involvement of Peroxynitrite on the Early Loss of P450 in Short-Term Hepatocyte Cultures

2001

The biological chemistry of nitric oxide (NO) in the oxygenated cellular environment is extremely complex. It involves the direct interaction of NO with specific biomolecules and the so-called indirect effects, due to secondary more potent oxidant species derived from NO which are also able to react with DNA, lipids, thiols and transition metals (Wink et al. 1996; Nathan, 1992). In addition to its regulatory role as a signalling molecule (Nathan, 1992; Moncada and Palmer, 1991) it has become evident that NO (or NO-derived species) is a critical factor involved in various toxicological mechanisms (Wink et al. 1996; Wang et al. 1998; Estevez et al. 1999; Wink et al. 1999). Some controversy ex…

chemistry.chemical_compoundPeroxynitrous acidmedicine.anatomical_structureChemistryHepatocyteCellmedicineCarbohydrate metabolismPhenotypeFunction (biology)PeroxynitriteNitric oxideCell biology
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The ATXN1 and TRIM31 genes are related to intelligence in an ADHD background: evidence from a large collaborative study totaling 4,963 subjects

2010

Contains fulltext : 96400.pdf (Publisher’s version ) (Closed access) Intelligence is a highly heritable trait for which it has proven difficult to identify the actual genes. In the past decade, five whole-genome linkage scans have suggested genomic regions important to human intelligence; however, so far none of the responsible genes or variants in those regions have been identified. Apart from these regions, a handful of candidate genes have been identified, although most of these are in need of replication. The recent growth in publicly available data sets that contain both whole genome association data and a wealth of phenotypic data, serves as an excellent resource for fine mapping and …

cognitionCandidate genegenetic associationUbiquitin-Protein LigasesEuropean Continental Ancestry GroupIntelligencePopulationMedizinNerve Tissue ProteinsSingle-nucleotide polymorphismGenomic disorders and inherited multi-system disorders Functional Neurogenomics [IGMD 3]Quantitative trait locusBiologyPolymorphism Single NucleotideGenomeWhite PeopleNuclear FamilyGenomic disorders and inherited multi-system disorders [IGMD 3]Tripartite Motif ProteinsCohort Studies03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineMeta-Analysis as TopicADHDHumansddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendalterseducationAtaxin-1Genetics (clinical)030304 developmental biologyGenetic associationGeneticsMental Health [NCEBP 9]0303 health scienceseducation.field_of_studyIntelligence quotientHuman intelligenceNuclear ProteinsALSPACPsychiatry and Mental healthPhenotypeAtaxinsAttention Deficit Disorder with Hyperactivitycandidate genesFunctional Neurogenomics [DCN 2]030217 neurology & neurosurgeryResearch Article
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Polygenic association between attention-deficit/hyperactivity disorder liability and cognitive impairments.

2022

AbstractBackgroundA recent genome-wide association study (GWAS) identified 12 independent loci significantly associated with attention-deficit/hyperactivity disorder (ADHD). Polygenic risk scores (PRS), derived from the GWAS, can be used to assess genetic overlap between ADHD and other traits. Using ADHD samples from several international sites, we derived PRS for ADHD from the recent GWAS to test whether genetic variants that contribute to ADHD also influence two cognitive functions that show strong association with ADHD: attention regulation and response inhibition, captured by reaction time variability (RTV) and commission errors (CE).MethodsThe discovery GWAS included 19 099 ADHD cases …

cognitionTrastorns per dèficit d'atenció amb hiperactivitat en els infantsMedizinSocial SciencesGenome-wide association studyAttention deficit disorder with hyperactivity in children3202 Applied Psychology2738 Psychiatry and Mental Health0302 clinical medicineAtencióDUPLICATIONS2.1 Biological and endogenous factorsPsychologyAetiologyGenetic riskChildPOPULATIONApplied PsychologyResponse inhibitionPsychiatryREACTION-TIME VARIABILITYCognition10058 Department of Child and Adolescent PsychiatryinhibitionPsychiatry and Mental healthPhenotypeMental Healthpolygenic risk scoresreaction time variabilityCognicióPublic Health and Health Services/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingTrastorns per dèficit d'atenció amb hiperactivitat en els adultsRESPONSE-INHIBITIONClinical psychologyAdultAdolescentDEFICIT HYPERACTIVITY DISORDER610 Medicine & healthGENETIC RISKbehavioral disciplines and activitiesYoung Adult03 medical and health sciencesWORKING-MEMORYSDG 3 - Good Health and Well-beingmental disordersReaction TimeGeneticsmedicineHumansAttention deficit hyperactivity disorderADHDCognitive DysfunctionGENOME-WIDE ASSOCIATIONAssociation (psychology)business.industryPreventionHuman GenomeNeurosciencesGenetic variantsPERFORMANCEmedicine.diseaseAttention Deficit Hyperactivity Disorder (ADHD)030227 psychiatryattentionAttention Deficit Disorder with HyperactivityInhibicióCase-Control StudiesAttention deficit disorder with hyperactivity in adultsPolygenic risk scorebusiness030217 neurology & neurosurgeryGenome-Wide Association StudyPsychological medicine
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Psychiatrists' Attitudes Toward Disruptive New Technologies: Mixed-Methods Study

2018

Background Recent discoveries in the fields of machine learning (ML), Ecological Momentary Assessment (EMA), computerized adaptive testing (CAT), digital phenotype, imaging, and biomarkers have brought about a new paradigm shift in medicine. Objective The aim of this study was to explore psychiatrists’ perspectives on this paradigm through the prism of new clinical decision support systems (CDSSs). Our primary objective was to assess the acceptability of these new technologies. Our secondary objective was to characterize the factors affecting their acceptability. Methods A sample of psychiatrists was recruited through a mailing list. Respondents completed a Web-based survey. A quantitative…

computerized adaptive testingStandardizationEmerging technologiesApplied psychologySample (statistics)digital phenotypeClinical decision support systemprofessional culture03 medical and health sciences0302 clinical medicineacceptabilityMailing listclinical decision support systemsOriginal Papermobile phonebusiness.industryecological momentary assessmentUsability030227 psychiatryTherapeutic relationshipPsychiatry and Mental healthmachine learningComputerized adaptive testingPsychologybusiness030217 neurology & neurosurgeryJMIR Mental Health
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