Search results for "Polymorphism"

showing 10 items of 1968 documents

Recolonization after habitat restoration leads to decreased genetic variation in populations of a terrestrial orchid.

2012

Colonization is crucial to habitat restoration projects that rely on the spontaneous regeneration of the original vegetation. However, as a previously declining plant species spreads again, the likelihood of founder effects increases through recurrent population founding and associated serial bottlenecks. We related Amplified Fragment Length Polymorphism markers genetic variation and fitness to colonization history for all extant populations of the outcrossing terrestrial orchid Dactylorhiza incarnata in an isolated coastal dune complex. Around 1970, D. incarnata suffered a severe bottleneck yet ultimately persisted and gradually spread throughout the spatially segregated dune slacks, aided…

Genetic MarkersConservation of Natural ResourcesModels StatisticalAmplified Fragment Length Polymorphism markersGenetic Variationgenetic diversitydune slacksFounder EffectassignmentGenetics Populationfounding eventsBelgiumrecolonizationDactylorhiza incarnataFSTFranceGenetic FitnessAmplified Fragment Length Polymorphism AnalysisOrchidaceaeEcosystemhabitat restorationMolecular ecology
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Identification of a genetic contamination in a commercial mouse strain using two panels of polymorphic markers

2007

Rapid detection of genetic contamination is critical in mouse studies involving inbred strains. During a Quantitative Trait Locus (QTL) study using simple sequence length polymorphism (SSLP) markers, we noticed heterozygosity at some loci of a commercially available inbred C57BL/6N mouse strain, suggesting a contamination by another mouse strain. A panel of 100 single-nucleotide polymorphism (SNP) markers was used to confirm and specify the genetic contamination suspected. Retrospective analyses demonstrated that the contamination took place as early as autumn 2003 and has persisted ever since at a fairly constant level. Contaminating alleles most probably originated from a DBA strain. Our…

Genetic MarkersGenotypeMice Inbred StrainsBiologyQuantitative trait locusMice03 medical and health sciences0302 clinical medicineInbred strainGenotypeAnimalsGenetic TestingAlleleSimple sequence length polymorphism030304 developmental biologyGeneticsMice Inbred BALB C0303 health sciencesPolymorphism GeneticBase SequenceGeneral VeterinaryGenetic Carrier ScreeningStrain (biology)Mice Inbred C57BLGenetic marker030220 oncology & carcinogenesisAnimal Science and ZoologyGenetic monitoringLaboratory Animals
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Candidate Gene and Genome-Wide Association Studies for Circulating Leptin Levels Reveal Population and Sex-Specific Associations in High Cardiovascul…

2019

Leptin is a hormone crucial in the regulation of food intake and body-weight maintenance. However, the genes and gene variants that influence its plasma levels are still not well known. Results of studies investigating polymorphisms in candidate genes have been inconsistent, and, in addition, very few genome-wide association studies (GWAS) have been undertaken. Our aim was to investigate the genes and gene variants most associated with plasma leptin concentrations in a high-cardiovascular-risk Mediterranean population. We measured plasma leptin in 1011 men and women, and analyzed the genetic factors associated using three approaches: (1) Analyzing the single nucleotide polymorphisms (SNPs) …

Genetic MarkersLeptinMale0301 basic medicineobesityCandidate genePopulation030209 endocrinology & metabolismGenome-wide association studySingle-nucleotide polymorphismBiologyGenetic polymorphismsleptinPolymorphism Single NucleotideFTO geneArticleMediterranean population03 medical and health sciencesSex Factors0302 clinical medicineLeptinasexHumansSNPgeneticsGenetic Predisposition to Diseaseeducationleptin receptorAgedGenetic associationGeneticseducation.field_of_studygenome-wide association studyNutrition and DieteticsLeptin receptorMediterranean RegionPolimorfisme genèticCross-Sectional Studies030104 developmental biologyCardiovascular DiseasesSpainObesitatFemaleheterogeneitypolymorphismsFood ScienceNutrients
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Evidence for a common origin of most Friedreich ataxia chromosomes in the Spanish population

1996

Haplotype analysis is a powerful approach to understand the spectrum of mutations accounting for a disease in a homogeneous population. We show that haplotype variation for 10 markers linked to the Friedreich ataxia locus (FRDA) argues in favor of an important mutation homogeneity in the Spanish population, and positions the FRDA locus in the region where it has been recently isolated. We also report the finding of a new single nucleotide polymorphism called FAD1. The new marker shows a very strong linkage disequilibrium with Friedreich ataxia (FA) in both the Spanish and French populations. suggesting the existence of an ancient and widespread FRDA mutations. Inclusion of FAD1 in the exten…

Genetic MarkersLinkage disequilibriumAtaxiaMolecular Sequence DataPopulationNerve Tissue ProteinsSingle-nucleotide polymorphismLocus (genetics)BiologyLinkage DisequilibriumTrinucleotide RepeatsGeneticsmedicineHumanseducationPhylogenyGenetics (clinical)Adaptor Proteins Signal TransducingGeneticseducation.field_of_studyPolymorphism GeneticBase SequenceHaplotypeIntronChromosome MappingIntronsHaplotypesFriedreich AtaxiaSpainGenetic markerMutationFrancemedicine.symptom
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Suggestive evidence for association of D2S2188 marker (2q31.1) with autism in 143 Sicilian (Italian) TRIO families

2005

We have screened 143 Sicilian (Italian) families with one autistic child to verify, by a linkage disequilibrium approach, the involvement of the 2q31.1 region in the cause of the disease in these families. Our study design includes the use of intrafamilial association to prevent a population stratification bias and ethnic homogeneity of the sample. The results of our analysis provided suggestive evidence of the occurrence of transmission disequilibrium between autism and the D2S2188 polymorphism in Sicilian TRIO families, a finding which provides further and independent support to the hypothesis of the existence of a susceptibility gene (or genes) for autism on chromosome 2q.

Genetic MarkersLinkage disequilibriumDisequilibriumEthnic groupautism ds2188 pcrDiseaseBiologyPopulation stratificationSettore BIO/13 - Biologia ApplicataPolymorphism (computer science)GeneticsmedicineHumansFamilyAutistic DisorderSicilyBiological PsychiatryGenetics (clinical)GeneticsPolymorphism GeneticChromosome Mappingmedicine.diseaselanguage.human_languagePsychiatry and Mental healthChromosomes Human Pair 2languageAutismmedicine.symptomSicilianPsychiatric Genetics
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RAD SNP markers as a tool for conservation of dolphinfish Coryphaena hippurus in the Mediterranean Sea: Identification of subtle genetic structure an…

2016

Dolphinfish is an important fish species for both commercial and sport fishing, but so far limited information is available on genetic variability and pattern of differentiation of dolphinfish populations in the Mediterranean basin. Recently developed techniques allow genome-wide identification of genetic markers for better understanding of population structure in species with limited genome information. Using restriction-site associated DNA analysis we successfully genotyped 140 individuals of dolphinfish from eight locations in the Mediterranean Sea at 3324 SNP loci. We identified 311 sex-related loci that were used to assess sex-ratio in dolphinfish populations. In addition, we identifie…

Genetic MarkersMale2bRAD0301 basic medicineConservation of Natural ResourcesSex Determination AnalysisRestriction MappingPopulationSettore BIO/05 - ZoologiaIntrogressionAquatic ScienceGenetic differentiationPolymorphism Single NucleotideMediterranean Basin03 medical and health sciencesMediterranean seaMediterranean SeaGeneticsAnimalsOutliersSex RatioGenetic variabilityeducation2bRAD; Genetic differentiation; Outliers; Sex determination markers; Aquatic Science; Geneticseducation.field_of_studyCoryphaenabiologyEcologybiology.organism_classificationPerciformes030104 developmental biologyEvolutionary biologyGenetic markerGenetic structureFemaleAnimal Distribution2bRAD Genetic differentiation Outliers Sex determination markersSex determination markersMarine Genomics
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Sex-dependent genetic markers of CYP3A4 expression and activity in human liver microsomes

2007

Objective: To find genetic markers of the individual cytochrome P450 (CYP)3A expression. Methods: A large collection of liver samples phenotyped for CYP3A expression and activity was genotyped for CYP3A variants. Data were analyzed for associations between CYP3A phenotypes and genotypes, and for evidence of recent selection. Results: We report associations between the hepatic CYP3A4 protein expression level, as well as its enzymatic activity, measured as verapamil N-dealkylation, and genetic polymorphisms from two regions within the CYP3A gene cluster. One region is defined by several variants, mostly located within CYP3A7, the other by a single nucleotide polymorphism in intron 7 of CYP3A…

Genetic MarkersMaleGene ExpressionSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideLinkage Disequilibrium03 medical and health sciences0302 clinical medicineCytochrome P-450 Enzyme SystemGene FrequencyPolymorphism (computer science)Gene expressionGenotypeGene clusterGeneticsCytochrome P-450 CYP3AHumansAllele frequencyCYP3A7030304 developmental biologyPharmacologyGeneticsSex Characteristics0303 health sciencesMolecular biologyGenetic markerMultigene Family030220 oncology & carcinogenesisLinear ModelsMicrosomes LiverMolecular MedicineFemalePharmacogenomics
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Mutations in the β-tropomyosin (TPM2) gene – a rare cause of nemaline myopathy

2002

Nemaline myopathy is a clinically and genetically heterogeneous muscle disorder. In the nebulin gene we have detected a number of autosomal recessive mutations. Both autosomal dominant and recessive mutations have been detected in the genes for alpha -actin and alpha -tropomyosin 3. A recessive mutation causing nemaline myopathy among the Old Order Amish has recently been identified in the gene for slow skeletal muscle troponin T. As linkage studies had shown that at least one further gene exists for nemaline myopathy, we investigated another tropomyosin gene expressed in skeletal muscle, the beta -tropomyosin 2 gene. Screening 66 unrelated patients, using single strand conformation polymor…

Genetic MarkersMaleGenetic LinkageProtein ConformationBiopsyMolecular Sequence DataMutation MissenseTropomyosinmacromolecular substancesMuscle disorderMyopathies NemalineTPM203 medical and health sciencesNebulin0302 clinical medicineNemaline myopathymedicineAnimalsHumansAmino Acid SequenceMuscle SkeletalNemaline bodiesPolymorphism Single-Stranded ConformationalGenetics (clinical)DNA Primers030304 developmental biologyGenetics0303 health sciencesSequence Homology Amino AcidbiologyReverse Transcriptase Polymerase Chain Reactionmusculoskeletal systemmedicine.diseaseMolecular biologyTropomyosinCongenital myopathyPedigree3. Good healthHaplotypesNeurologyMutationPediatrics Perinatology and Child Healthbiology.proteinFemaleNeurology (clinical)Sequence Alignment030217 neurology & neurosurgeryCentral core diseaseNeuromuscular Disorders
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CD45 and multiple sclerosis: the exon 4 C77G polymorphism (additional studies and meta-analysis) and new markers

2003

We re-evaluated the association with multiple sclerosis (MS) of the C77G splicing regulatory variation in the CD45 gene and screened for new mutations the three alternatively spliced exons (#4, 5 and 6). No association with C77G was detected in two groups of patients (total=448) and controls (total=559) from Northern and Southern Italy. When excluding the first published study indicating a positive association, a meta-analysis of the five further studies conducted to date (including the present one) led to a non-significant combined odds ratio (OR) of 1.11. None of the four newly identified nucleotide substitutions, namely C77T (Pro59Pro) in exon 4, G69C (Asp121His) in exon 5, T127A (Ile187…

Genetic MarkersMaleGuanineMultiple SclerosisGenotypeImmunologyBiologyCytosineExonGene FrequencymedicineHumansImmunology and AllergyGeneAllelesGeneticsPolymorphism GeneticMultiple sclerosisGenetic VariationExonsOdds ratiomedicine.diseaseMolecular biologyAlternative SplicingNeurologyMeta-analysisRNA splicingLeukocyte Common AntigensFemaleNeurology (clinical)Journal of Neuroimmunology
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The copy number variant involving part of the α7 nicotinic receptor gene contains a polymorphic inversion.

2008

The alpha7 nicotinic acetylcholine receptor gene (CHRNA7) is located at 15q13-q14 in a region that is strongly linked to the P50 sensory gating deficit, an endophenotype of schizophrenia and bipolar disorder. Part of the gene is a copy number variant, due to a duplication of exons 5-10 and 3' sequence in CHRFAM7A, which is present in many but not all humans. Maps of this region show that the two genes are in opposite orientation in the individual mainly represented in the public access human DNA sequence database (Build 36), suggesting that an inversion had occurred since the duplication. We have used fluorescent in situ hybridization to investigate this putative inversion. Analysis of inte…

Genetic MarkersMaleLinkage disequilibriumBipolar DisorderPan troglodytesalpha7 Nicotinic Acetylcholine ReceptorReceptors NicotinicLinkage DisequilibriumExonGene duplicationGeneticsSettore MED/48 -Scienze Infermierist. e Tecn. Neuro-Psichiatriche e Riabilitat.AnimalsHumansCopy-number variationGeneSettore MED/25 - PsichiatriaGenetics (clinical)Sequence DeletionSegmental duplicationChromosomal inversionGeneticsChromosomes Human Pair 15Polymorphism GeneticBase SequencebiologyCHRNA7Chromosome Mappinginversion schizophrenia bipolar disorder 15q13–q14 CHRNA7 segmental duplicationChromosome InversionSchizophreniabiology.proteinFemale
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