Search results for "Prefrontal Cortex"

showing 10 items of 323 documents

Prefrontal and temporo-parietal involvement in taking others' perspective: TMS evidence.

2008

INTRODUCTION: Understanding the mental states of others entails a number of cognitive processes known as Theory of Mind (ToM). Behavioural and functional neuroimaging evidence suggests that prefrontal and temporo-parietal cortices are involved in these abilities. The present study was aimed at investigating the role of the dorsolateral prefrontal cortex and temporo-parietal junction in ToM by using a repetitive transcranial magnetic stimulation (rTMS) paradigm. MATERIAL AND METHODS: Eleven healthy subjects participated in the study. The experimental ToM procedure was constituted by false belief and faux-pas written stories. Subjects were evaluated in baseline condition (Sham) and after 1Hz …

AdultMalemedicine.medical_treatmentCulturePrefrontal CortexNeurosciences. Biological psychiatry. NeuropsychiatryTHEORY OF MINDbehavioral disciplines and activitiesFunctional neuroimagingTheory of mindParietal Lobemental disordersmedicineReaction TimeSocial Perception; Culture; Humans; Temporal Lobe; Adult; Prefrontal Cortex; Transcranial Magnetic Stimulation; Parietal Lobe; Male; Female; Reaction TimeHumansBRAINPrefrontal cortexSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaParietal lobeCognitionGeneral MedicineTranscranial Magnetic StimulationTemporal LobeTranscranial magnetic stimulationDorsolateral prefrontal cortexNeuropsychology and Physiological Psychologymedicine.anatomical_structureNeurologyMentalizationnervous systemSocial PerceptionmentalizingSettore MED/26 - NeurologiaFemaleNeurology (clinical)PsychologyNeurosciencepsychological phenomena and processesRC321-571Cognitive psychologyResearch Article
researchProduct

Effects of post-extinction l-DOPA administration on the spontaneous recovery and reinstatement of fear in a human fMRI study

2015

Relapse is a pertinent problem in the treatment of anxiety disorders. In the laboratory, relapse is modeled as return of conditioned fear responses after successful fear extinction and is explained by insufficient retrieval and/or expression of the fear-inhibitory extinction memory that is generated during extinction learning. We have shown in mice and humans that return of fear can be prevented by administration of a single dose of the dopamine precursor l-3,4-dihydroxyphenylalanine (l-DOPA) immediately after extinction. In mice, this effect could be attributed to an enhancement of extinction memory consolidation. In our human study, we could not exclude that l-DOPA might have acted by int…

AdultMalemedicine.medical_treatmentDopamine AgentsSpontaneous recoveryExposure therapyVentromedial prefrontal cortexAmygdalaFear-potentiated startleExtinction PsychologicalDevelopmental psychologyLevodopaRandom AllocationDouble-Blind MethodConditioning PsychologicalmedicineHumansPharmacology (medical)Fear conditioningBiological PsychiatryMemory ConsolidationPharmacologyFear processing in the brainBrain MappingPsychotropic DrugsBrainFearGalvanic Skin Responsesocial sciencesExtinction (psychology)Magnetic Resonance ImaginghumanitiesPsychiatry and Mental healthmedicine.anatomical_structureNeurologyVisual PerceptionNeurology (clinical)CuesPsychologyNeuroscienceEuropean Neuropsychopharmacology
researchProduct

Lateralized effects of self-induced sadness and happiness on corticospinal excitability.

1997

We studied the changes in excitability of the corticospinal projection evoked by self-induced sad and happy thoughts. Corticospinal excitability was probed using focal, single-pulse transcranial magnetic stimulation (TMS) applied to the optimal scalp position for evoking motor potentials in the contralateral first dorsal interosseus muscle. Fourteen right-handed subjects were studied while counting mentally, thinking sad thoughts, or thinking happy thoughts. In each of these three conditions TMS was applied in each subject randomly, 20 times to the right and 20 times to the left hemisphere. Sad thoughts resulted in a significant facilitation of the motor potentials evoked by left-hemispheri…

AdultMalemedicine.medical_treatmentHappinessPyramidal TractsStimulationbehavioral disciplines and activitiesLateralization of brain functionFunctional LateralityMagneticsPhysical StimulationmedicineHumansPrefrontal cortexPyramidal tractsEvoked Potentials MotorTranscranial magnetic stimulationElectrophysiologyAffectmedicine.anatomical_structureScalpCerebral hemisphereFemaleNeurology (clinical)PsychologyNeurosciencepsychological phenomena and processesNeurology
researchProduct

Recognition memory and prefrontal cortex: Dissociating recollection and familiarity processes using rTMS

2008

Recognition memory can be supported by both the assessment of the familiarity of an item and by the recollection of the context in which an item was encountered. The neural substrates of these memory processes are controversial. To address these issues we applied repetitive transcranial magnetic stimulation (rTMS) over the right and left dorsolateral prefrontal cortex (DLPFC) of healthy subjects performing a remember/know task. rTMS disrupted familiarity judgments when applied before encoding of stimuli over both right and left DLPFC. rTMS disrupted recollection when applied before encoding of stimuli over the right DLPFC. These findings suggest that the DLPFC plays a critical role in recog…

AdultMalemedicine.medical_treatmentNeurosciences. Biological psychiatry. NeuropsychiatryContext (language use)Recognition (Psychology)behavioral disciplines and activitiesPrefrontal cortexNORecognition memoryJudgmentRecollectionEncoding (memory)mental disordersmedicineHumansJudgment; Memory; Recognition (Psychology); Humans; Adult; Mental Recall; Prefrontal Cortex; Transcranial Magnetic Stimulation; Male; FemalePrefrontal cortexTMS; MEMORYLeft dorsolateral prefrontal cortexRecognition memoryRecallSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaMEMORYHealthy subjectsRecognition PsychologyGeneral MedicineFamiliarityTranscranial Magnetic StimulationTranscranial magnetic stimulationNeuropsychology and Physiological Psychologynervous systemNeurologyTMSMental RecallFemaleSettore MED/26 - NeurologiaNeurology (clinical)Psychologypsychological phenomena and processesFamiliarity; Prefrontal cortex; Recognition memory; Recollection;Research ArticleRC321-571Cognitive psychology
researchProduct

High frequency repetitive transcranial magnetic stimulation (rTMS) reduces EEG-hypofrontality in patients with schizophrenia.

2015

The reduced metabolic activity in the prefrontal brain lobes, so-called hypofrontality, is associated with increased electrophysiological delta-band activity. Schizophrenia inpatients (N=35) received sham-controlled 10Hz rTMS over the left dorsolateral prefrontal cortex in a randomised design. After treatment, the resting electroencephalography revealed a significant decrease in the delta-band activity, which originated in the right prefrontal cortex and correlated with improvements in facial affect recognition.

AdultMalemedicine.medical_treatmentPrefrontal CortexHypofrontalityElectroencephalography03 medical and health sciences0302 clinical medicinemedicineHumansIn patientPrefrontal cortexBiological PsychiatryFacial affectmedicine.diagnostic_testBrainElectroencephalographymedicine.diseaseTranscranial Magnetic Stimulation030227 psychiatryTranscranial magnetic stimulationPsychiatry and Mental healthElectrophysiologyDelta RhythmSchizophreniaSchizophreniaFemaleSchizophrenic PsychologyPsychologyNeuroscienceFacial Recognition030217 neurology & neurosurgeryPsychiatry research
researchProduct

Excitability regulation in the dorsomedial prefrontal cortex during sustained instructed fear responses: a TMS-EEG study

2018

AbstractThreat detection is essential for protecting individuals from adverse situations, in which a network of amygdala, limbic regions and dorsomedial prefrontal cortex (dmPFC) regions are involved in fear processing. Excitability regulation in the dmPFC might be crucial for fear processing, while abnormal patterns could lead to mental illness. Notwithstanding, non-invasive paradigms to measure excitability regulation during fear processing in humans are missing. To address this challenge we adapted an approach for excitability characterization, combining electroencephalography (EEG) and transcranial magnetic stimulation (TMS) over the dmPFC during an instructed fear paradigm, to dynamica…

AdultMalemedicine.medical_treatmentPrefrontal Cortexlcsh:MedicineElectroencephalographyAmygdalaBrain mappingArticle050105 experimental psychologyYoung Adult03 medical and health sciences0302 clinical medicineDorsomedial Prefrontal Cortex ; Fear Paradigm ; TMS-evoked Potentials (TEPs) ; Fear Network ; Fear ProcessingHeart RateReaction TimemedicineHumans0501 psychology and cognitive scienceslcsh:ScienceEvoked PotentialsBrain MappingElectroshockMultidisciplinarymedicine.diagnostic_test05 social scienceslcsh:RHealthy subjectsStructural integrityElectroencephalographyFearDorsomedial prefrontal cortexTranscranial Magnetic StimulationTranscranial magnetic stimulationmedicine.anatomical_structureFemalelcsh:QPsychologyNeuroscience030217 neurology & neurosurgery
researchProduct

Keeping memory clear and stable--the contribution of human basal ganglia and prefrontal cortex to working memory.

2010

Successful remembering involves both hindering irrelevant information from entering working memory (WM) and actively maintaining relevant information online. Using a voxelwise lesion-behavior brain mapping approach in stroke patients, we observed that lesions of the left basal ganglia render WM susceptible to irrelevant information. Lesions of the right prefrontal cortex on the other hand make it difficult to keep more than a few items in WM. These findings support basal ganglia-prefrontal cortex models of WM whereby the basal ganglia play a gatekeeper role and allow only relevant information to enter prefrontal cortex where this information then is actively maintained in WM.

AdultMalephysiology [Prefrontal Cortex]Interference theoryphysiology [Basal Ganglia]Prefrontal CortexBrain mappingBasal GangliaBasal (phylogenetics)Cortex (anatomy)Basal gangliamedicineHumansphysiopathology [Memory Disorders]complications [Stroke]ddc:610Prefrontal cortexAgedAged 80 and overMemory DisordersBrain Mappingphysiopathology [Stroke]Working memoryGeneral NeurosciencePutamenphysiology [Putamen]Middle AgedMagnetic Resonance Imagingetiology [Memory Disorders]StrokeMemory Short-Termmedicine.anatomical_structurephysiology [Memory Short-Term]pathology [Stroke]Femalediagnosis [Memory Disorders]physiopathology [Putamen]Brief CommunicationsConsumer neurosciencePsychologyNeuroscienceCognitive psychology
researchProduct

Neural correlates of antinociception in borderline personality disorder.

2006

Context A characteristic feature of borderline personality disorder (BPD) is self-injurious behavior in conjunction with stress-induced reduction of pain perception. Reduced pain sensitivity has been experimentally confirmed in patients with BPD, but the neural correlates of antinociceptive mechanisms in BPD are unknown. We predicted that heat stimuli in patients with BPD would activate brain areas concerned with cognitive and emotional evaluation of pain. Objective To assess the psychophysical properties and neural correlates of altered pain processing in patients with BPD. Design Case-control study. Setting A university hospital. Participants Twelve women with BPD and self-injurious behav…

AdultPain Thresholdmedicine.medical_specialtyHot TemperatureDifferential ThresholdPainPrefrontal CortexAudiologybehavioral disciplines and activitiesAmygdalaGyrus CingulimethodsArts and Humanities (miscellaneous)bloodBorderline Personality DisorderPhysical StimulationAdult Amygdala; physiopathology Borderline Personality Disorder; diagnosis/physiopathology/psychology Brain Mapping Brain; physiopathology Case-Control Studies Differential Threshold; physiology Female Gyrus Cinguli; physiopathology Hot Temperature; diagnostic use Humans Magnetic Resonance Imaging Oxygen; blood Pain Measurement; methods Pain Threshold; physiology Pain; diagnosis/physiopathology/psychology Physical Stimulation Prefrontal Cortex; physiopathology Self-Injurious Behavior; diagnosis/physiopathology Thermosensing; physiologymental disordersThreshold of painmedicineHumansThermosensingPrefrontal cortexPsychiatryBorderline personality disorderPain MeasurementBrain MappingBlood-oxygen-level dependentmedicine.diagnostic_testBrainmedicine.diseaseAmygdalaMagnetic Resonance Imagingdiagnosis/physiopathologyFunctional imagingDorsolateral prefrontal cortexOxygenPsychiatry and Mental healthmedicine.anatomical_structureCase-Control StudiesphysiologyFemalediagnosis/physiopathology/psychologyphysiopathologydiagnostic useFunctional magnetic resonance imagingPsychologySelf-Injurious BehaviorArchives of general psychiatry
researchProduct

Neurobiological signature of intimacy in anorexia nervosa

2019

Background Intimacy and psychosexual development represent core problems of anorexia nervosa (AN). Experiential and neurobiological evidence however is scarce. Material and methods Thirty-one female AN patients were compared with 35 non-patients (NP) and 22 recovered participants (REC) by using functional magnetic resonance imaging. Participants viewed pictures of couples in intimate relationships and control stimuli. Results AN patients experienced intimate stimuli with lower valence and dominance. AN showed decreased activation of parietal cortices. NP decreased the prefrontal cortex response, which AN patients did not. REC participants did not differ from NP on a behavioural level, thoug…

AdultSexual BehaviorPICTURE STIMULIDysfunctional familyintimacyanorexia nervosaROMANTIC LOVEArousalACTIVATIONYoung AdultmedicineSEXUALITYHumansValence (psychology)Prefrontal cortexABUSEENVIRONMENTmedicine.diagnostic_testWOMENAROUSALmedicine.diseaseMagnetic Resonance Imagingfunctional magnetic resonance imagingPsychiatry and Mental healthClinical PsychologyEating disordersATTACHMENT REPRESENTATIONAnorexia nervosa (differential diagnoses)Psychosexual developmentFemaleFunctional magnetic resonance imagingPsychologyFEMALESClinical psychologyEuropean Eating Disorders Review
researchProduct

Alterations in the expression of PSA-NCAM and synaptic proteins in the dorsolateral prefrontal cortex of psychiatric disorder patients.

2012

Alterations in the structure and physiology of the prefrontal cortex (PFC) have been found in different psychiatric disorders and some of them involve inhibitory networks, especially in schizophrenia and major depression. Changes in the structure of these networks may be mediated by the polysialylated neural cell adhesion molecule (PSA-NCAM), a molecule related to neuronal structural plasticity, expressed in the PFC exclusively by interneurons. Different studies have found that PSA-NCAM expression in the hippocampus and the amygdala is altered in schizophrenia, major depression and animal models of these disorders, in parallel to changes in the expression of molecules related to inhibitory …

Adultmedicine.medical_specialtyBipolar DisorderSynaptophysinHippocampusPrefrontal CortexNeural Cell Adhesion Molecule L1NeurotransmissionHippocampusmedicineNeuropilHumansPsychiatryPrefrontal cortexAgedDepressive Disorder MajorNeuronal PlasticitybiologyGlutamate DecarboxylaseGeneral NeuroscienceMental DisordersNeural InhibitionMiddle AgedAmygdalaDorsolateral prefrontal cortexmedicine.anatomical_structurenervous systemSynaptic plasticitySynapsesVesicular Glutamate Transport Protein 1Synaptophysinbiology.proteinSchizophreniaSialic AcidsNeural cell adhesion moleculePsychologyNeuroscienceNeuroscience letters
researchProduct