Search results for "Pyrazole"

showing 10 items of 315 documents

CB1 Cannabinoid Receptors and On-Demand Defense Against Excitotoxicity

2003

Abnormally high spiking activity can damage neurons. Signaling systems to protect neurons from the consequences of abnormal discharge activity have been postulated. We generated conditional mutant mice that lack expression of the cannabinoid receptor type 1 in principal forebrain neurons but not in adjacent inhibitory interneurons. In mutant mice,the excitotoxin kainic acid (KA) induced excessive seizures in vivo. The threshold to KA-induced neuronal excitation in vitro was severely reduced in hippocampal pyramidal neurons of mutants. KA administration rapidly raised hippocampal levels of anandamide and induced protective mechanisms in wild-type principal hippocampal neurons. These protecti…

MaleCannabinoid receptorReceptors Drugmedicine.medical_treatment2-ArachidonoylglycerolExcitotoxicityHippocampal formationmedicine.disease_causeHippocampusMicechemistry.chemical_compoundPiperidinesCannabinoid receptor type 1Excitatory Amino Acid AgonistsReceptors Cannabinoidgamma-Aminobutyric AcidMice KnockoutNeuronsKainic AcidMultidisciplinaryBrainEndocannabinoid systemNeuroprotective AgentsMitogen-Activated Protein KinasesRimonabantSignal Transductionmedicine.medical_specialtyKainic acidPolyunsaturated AlkamidesGlutamic AcidMice TransgenicArachidonic AcidsIn Vitro TechniquesBiologyGlyceridesProsencephalonInternal medicinemedicineAnimalsFuransGenes Immediate-EarlyEpilepsyCannabinoidsBrain-Derived Neurotrophic FactorExcitatory Postsynaptic PotentialsMice Inbred C57BLEndocrinologyGene Expression Regulationnervous systemchemistryMutationPyrazolesCannabinoidNeuroscienceEndocannabinoidsScience
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Peripheral and central CB1 cannabinoid receptors control stress-induced impairment of memory consolidation

2016

Stressful events can generate emotional memories linked to the traumatic incident, but they also can impair the formation of nonemotional memories. Although the impact of stress on emotional memories is well studied, much less is known about the influence of the emotional state on the formation of nonemotional memories. We used the novel object-recognition task as a model of nonemotional memory in mice to investigate the underlying mechanism of the deleterious effect of stress on memory consolidation. Systemic, hippocampal, and peripheral blockade of cannabinoid type-1 (CB1) receptors abolished the stress-induced memory impairment. Genetic deletion and rescue of CB1 receptors in specific ce…

MaleEstrès0301 basic medicineIndolesCannabinoid receptormedicine.medical_treatmentPopulationDopamine beta-HydroxylaseHippocampal formation03 medical and health sciences0302 clinical medicinePiperidinesReceptor Cannabinoid CB1Cannabinoides -- ReceptorsmedicineAnimalsMemory impairmentReceptoreducationMemory ConsolidationMice KnockoutNeuronsElectroshockMemory Disorderseducation.field_of_studyMultidisciplinaryBiological SciencesEndocannabinoid system3. Good health030104 developmental biologyHindlimb SuspensionPyrazolesMemory consolidationCannabinoidRimonabantPsychologyNeuroscienceAnisomycinStress Psychological030217 neurology & neurosurgeryMemòriaProceedings of the National Academy of Sciences
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A Polymorphism in the Crhr1 Gene Determines Stress Vulnerability in Male Mice

2014

Chronic stress is a risk factor for psychiatric disorders but does not necessarily lead to uniform long-term effects on mental health, suggesting modulating factors such as genetic predispositions. Here we address the question whether natural genetic variations in the mouse CRH receptor 1 (Crhr1) locus modulate the effects of adolescent chronic social stress (ACSS) on long-term stress hormone dysregulation in outbred CD1 mice, which allows a better understanding of the currently reported genes × environment interactions of early trauma and CRHR1 in humans. We identified 2 main haplotype variants in the mouse Crhr1 locus that modulate the long-term effects of ACSS on basal hypothalamic-pitui…

MaleHypothalamo-Hypophyseal SystemGenotypeGene ExpressionPituitary-Adrenal SystemLocus (genetics)Single-nucleotide polymorphismRegulatory Sequences Nucleic AcidBiologyBinding CompetitivePolymorphism Single NucleotideReceptors Corticotropin-Releasing HormoneMiceEndocrinologyGene FrequencyGenetic predispositionAnimalsHumansGenetic Predisposition to DiseaseChronic stressCRHR1 GeneGeneIn Situ HybridizationSocial stressGeneticsBehavior AnimalTriazinesHaplotypeHaplotypesPituitary GlandPyrazolesFemaleGene-Environment InteractionCorticosteroneStress PsychologicalSignal TransductionEndocrinology
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Synthesis and pharmacological study of ethyl 1-methyl-5-(substituted 3,4-dihydro-4-oxoquinazolin-3-yl)-1H-pyrazole-4-acetates

2001

Several new ethyl 1-methyl-5-(substituted 3,4-dihydro-4-oxoquinazolin-3-yl)-1H-pyrazole-4-acetates 2, substituted at 2 and, alternatively at, 6, 7 or 8 positions of the quinazolinone nucleus, were synthesised. The compounds were screened for their analgesic and antiinflammatory activities, acute toxicity and ulcerogenic effect. Substitution in the benzene moiety of the quinazolinone ring did not show any advantage for the analgesic activity, whereas it improved in some cases the antiinflammatory activity. Some compounds showed appreciable antiinflammatory activity and, at the same time, very low ulcerogenic index.

MaleMagnetic Resonance SpectroscopySpectrophotometry InfraredStereochemistryAnalgesicAnti-Inflammatory AgentsPeritonitisPyrazoleChemical synthesisLethal Dose 50Rats Sprague-DawleyMicechemistry.chemical_compoundDrug DiscoveryBenzoquinonesAnimalsEdemaMoietyStomach UlcerQuinazolinonePharmacologyAnalgesicsBicyclic moleculeOrganic ChemistryGeneral MedicineAcute toxicityRatschemistryQuinazolinesLactamPyrazolesEuropean Journal of Medicinal Chemistry
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Bone marrow characteristics predict outcome in a multicenter cohort of primary immune thrombocytopenia patients treated with thrombopoietin analogs

2019

It is well established that immune thrombocytopenia (ITP) results from increased immune mediated platelet destruction (anti-platelets antibodies, autoreactive T cells, and reduction of regulatory T cells) along with impaired production in the bone marrow.1 The latter has been attributed to both cellular and humoral mediators that cause suppression of megakaryocyte production and maturation.2 Current standard first line therapy consists of corticosteroids, with or without intravenous Ig, achieving about 70-80% response rate. However, a consistent proportion of patients would relapse after corticosteroid discontinuation or tapering, and requires further therapy. ...

MaleOncologymedicine.medical_specialtyRecombinant Fusion ProteinsEltrombopagBone Marrow CellsImmune Thrombocytopenic PurpuraReceptors FcBenzoateschemistry.chemical_compoundInternal medicinemedicineHumansOnline Only ArticlesThrombopoietinRetrospective StudiesPurpura Thrombocytopenic IdiopathicRomiplostimbusiness.industryRetrospective cohort studyromiplostimHematologyMiddle AgedImmune thrombocytopeniaClinical trialHydrazinesmedicine.anatomical_structureThrombopoietinchemistryCohortPyrazolesFemaleBone marroweltrombopagbusinessFollow-Up Studiesmedicine.drugHaematologica
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Disseminated tuberculosis in a patient treated with a JAK2 selective inhibitor: a case report

2012

Abstract Background Primary myelofibrosis is a myeloproliferative disorder characterized by bone marrow fibrosis, abnormal cytokine expression, splenomegaly and anemia. The activation of JAK2 and the increased levels of circulating proinflammatory cytokines seem to play an important role in the pathogenesis of myelofibrosis. Novel therapeutic agents targeting JAKs have been developed for the treatment of myeloproliferative disorders. Ruxolitinib (INCB018424) is the most recent among them. Case presentation To our knowledge, there is no evidence from clinical trials of an increased risk of tuberculosis during treatment with JAK inhibitors. Here we describe the first case of tuberculosis in a…

MaleOncologymedicine.medical_specialtyRuxolitinibTuberculosisSettore MED/17 - Malattie InfettiveAnemiaAntitubercular AgentsMyelofibrosislcsh:MedicineCase ReportGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineMyeloproliferative DisordersInternal medicineNitrilesmedicineHumansTuberculosisMyelofibrosislcsh:Science (General)lcsh:QH301-705.5Medicine(all)Janus kinase 2biologyLatent tuberculosisBiochemistry Genetics and Molecular Biology(all)business.industryTuberculosis Myelofibrosis Ruxolitiniblcsh:RGeneral MedicineJanus Kinase 2medicine.diseasePyrimidinesRuxolitiniblcsh:Biology (General)Primary MyelofibrosisImmunologybiology.proteinPyrazolesbusinessmedicine.druglcsh:Q1-390BMC Research Notes
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The hMTH1 paradox: antioxidants recommended in cancer?

2014

Summary Activated Ras GTPase signalling is a critical driver of oncogenic transformation and malignant disease. Cellular models of RAS-dependent cancers have been used to identify experimental small-molecules, such as SCH51344, but their molecular mechanism of action remains generally enigmatic. Here, using a chemical proteomic approach we identify the target of SCH51344 as the human mutT homologue MTH1, a nucleotide pool sanitising enzyme. Loss-of-function of MTH1 impaired growth of KRAS tumour cells whereas MTH1 overexpression mitigated sensitivity toward SCH51344. Searching for more drug-like inhibitors, we identified the kinase inhibitor crizotinib as a nanomolar suppressor of MTH1 acti…

MalePyridinesMEDLINEDNA repairAntineoplastic AgentsAntioxidantesSaludBiologyBioinformaticsstereoselectivityBiochemistryArticleText miningNeoplasmsmedicineAnimalsHumanscancerMolecular BiologyProtein Kinase Inhibitorscrizotinibbusiness.industryNucleotidesCancerdrugCell BiologyCáncermedicine.diseasePhosphoric Monoester HydrolasesMTH1DNA Repair EnzymesPyrazolesFemalebusiness
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Discovery and SAR Evolution of Pyrazole Azabicyclo[3.2.1]octane Sulfonamides as a Novel Class of Non-Covalent N-Acylethanolamine-Hydrolyzing Acid Ami…

2021

Inhibition of intracellular N-acylethanolamine-hydrolyzing acid amidase (NAAA) activity is a promising approach to manage the inflammatory response under disabling conditions. In fact, NAAA inhibition preserves endogenous palmitoylethanolamide (PEA) from degradation, thus increasing and prolonging its anti-inflammatory and analgesic efficacy at the inflamed site. In the present work, we report the identification of a potent, systemically available, novel class of NAAA inhibitors, featuring a pyrazole azabicyclo[3.2.1]octane structural core. After an initial screening campaign, a careful structure–activity relationship study led to the discovery of endo-ethoxymethyl-pyrazinyloxy-8-azabicyclo…

MaleStereochemistryAnti-Inflammatory AgentsPeptides and proteinsPyrazoleArticleAmidohydrolasesAmidaseRats Sprague-DawleyStructure-Activity Relationshipchemistry.chemical_compoundIn vivoN-AcylethanolamineDrug DiscoverymedicineAnimalsHumansSulfonesEnzyme InhibitorsIC50InhibitionInflammationchemistry.chemical_classificationPalmitoylethanolamideMolecular StructureInhibitorsSulfonamideMice Inbred C57BLMolecular Docking SimulationMechanism of actionchemistryMicrosomes LiverInhibitorsInhibitionSulfonesPeptides and proteinsInflammationPyrazolesMolecular Medicinemedicine.symptomProtein BindingTropanesJournal of Medicinal Chemistry
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Short-term adaptation of conditioned fear responses through endocannabinoid signaling in the central amygdala

2010

International audience; Both, the cannabinoid receptor type 1 (CB1) and the central nucleus of the amygdala (CeA) are known to play crucial roles in the processing of fear and anxiety, whereby they appear to be especially involved in the control of fear states. However, in contrast to many other brain regions including the cortical subregions of the amygdala, the existence of CB1 in the CeA remains enigmatic. Here we show that CB1 is expressed in the CeA of mice and that CB1 in the CeA mediates short-term synaptic plasticity, namely depolarization-induced suppression of excitation (DSE) and inhibition (DSI). Moreover, the CB1 antagonist AM251 increased both excitatory and inhibitory postsyn…

MaleTime FactorsAction PotentialsAnxietyExtinction PsychologicalGABA AntagonistsPropanolaminesMice0302 clinical medicinePiperidinesReceptor Cannabinoid CB1Adaptation PsychologicalConditioning PsychologicalMoodFear conditioningHabituationStress DisordersMice Knockout0303 health sciencesBehavior AnimalCentral nucleus of the amygdalaValineFearExtinctionAmygdalaPyridazinesPsychiatry and Mental healthmedicine.anatomical_structureOriginal ArticlePsychologypsychological phenomena and processesSignal TransductionSensory Receptor CellsNeurophysiologyIn Vitro TechniquesInhibitory postsynaptic potentialAmygdala03 medical and health sciencesQuinoxalinesCannabinoid Receptor ModulatorsmedicineAnimalsMaze Learning030304 developmental biologyPharmacologyFear processing in the brainLearning & MemoryCannabinoidsExtinction (psychology)Phosphinic AcidsElectric StimulationMice Inbred C57BLGene Expression Regulationnervous systemSynaptic plasticityPyrazolesNeuroscienceExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryEndocannabinoidsConditioning
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Effects of aspirin, nimesulide, and SC-560 on vasopressin-induced contraction of human gastroepiploic artery and saphenous vein.

2007

Objective: The present experiments were designed to evaluate differences in the effects of cyclooxygenase (COX)-1 and COX-2 inhibition on contractile responses of human gastroepiploic artery and saphenous vein elicited by vasopressin. Design: Laboratory investigation. Setting: University laboratory. Subjects: Rings of human gastroepiploic artery were obtained from 32 patients undergoing gastrectomy, and rings of saphenous vein were obtained from 30 patients undergoing coronary artery bypass surgery. Interventions: The rings were suspended in organ baths for isometric recording of tension. We studied the responses to vasopressin in the absence and in the presence of either the vasopressin V …

MaleVasopressinVasopressinsNeuropeptideGastroepiploic ArteryPharmacologyCritical Care and Intensive Care MedicineMuscle Smooth VascularCoronary artery bypass surgeryIntensive careMedicineHumansCyclooxygenase InhibitorsSaphenous VeinAgedAged 80 and overAspirinSulfonamidesbiologyAspirinDose-Response Relationship Drugbusiness.industryMiddle AgedAnesthesiabiology.proteinPyrazolesFemaleCyclooxygenasebusinessGastroepiploic ArteryNimesulidemedicine.drugMuscle ContractionCritical care medicine
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