Search results for "Receptor"

showing 10 items of 6990 documents

A mutation in the second intracellular loop of the pituitary adenylate cyclase activating polypeptide type I receptor confers constitutive receptor a…

2000

AbstractThe pituitary adenylate cyclase activating polypeptide (PACAP) type I receptor belongs to the glucagon/secretin/vasoactive intestinal polypeptide (VIP) receptor family. We mutated and deleted an amino acid residue (E261) which is located within the second intracellular loop of the rat PACAP type I receptor and which is highly conserved among the receptor family. The wild-type receptor and the mutant receptors were efficiently expressed at the surface of COS-7 cells at nearly the same level and revealed the same high affinity for the agonist PACAP-27. The cAMP contents of COS cells transfected with the E261A, E261Q, and the deletion mutant receptor were 4.6-, 5.7-, and 6.7-fold highe…

endocrine systemGrowth-hormone-releasing hormone receptorMolecular Sequence DataReceptors Pituitary Adenylate Cyclase-Activating PolypeptideBiophysicsGlutamic AcidSignal transductionTransfectionBiochemistryBeta-1 adrenergic receptorConstitutive activityStructural BiologycAMPCyclic AMPGeneticsEnzyme-linked receptorAnimals5-HT5A receptorAmino Acid SequenceReceptors Pituitary HormoneMolecular BiologySequence DeletionPeptide hormone receptorSite-directed mutagenesisPituitary adenylate cyclase activating polypeptideChemistryLiver receptor homolog-1Cell BiologyMolecular biologyRatsInterleukin-21 receptorCOS CellsMutagenesis Site-DirectedEstrogen-related receptor gammaSequence AlignmentGlucagon receptor familyhormones hormone substitutes and hormone antagonistsAdenylyl CyclasesReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type IFEBS Letters
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Human interleukin-6 facilitates hepatitis B virus infection in vitro and in vivo.

2000

Abstract Background and aim. Research on hepatitis B virus (HBV) infection in vivo has been limited due to the absence of a suitable animal model. We have developed a human–mouse radiation chimera in which normal mice, preconditioned by lethal total body irradiation and radioprotected with SCID mouse bone marrow cells, are permissive for engraftment of human hematopoietic cells and solid tissues. This resulting human–mouse model, which comprises three genetically disparate sources of tissue, is therefore termed Trimera. This study was aimed at assessing the effect of human IL-6 on HBV infection in vivo in Trimera mice. Methods. Trimera mice were transplanted with human liver tissue fragment…

endocrine systemHepatitis B virusMice SCIDmedicine.disease_causeVirus ReplicationMiceIn vivoVirologymedicineAnimalsHumansHepatitis B virusbiologychimeric miceInterleukin-6Hepatitis BVirologyMolecular biologydigestive system diseasesIn vitroTransplantationDisease Models Animalmedicine.anatomical_structureCell cultureRadiation Chimerabiology.proteinviral infectionBone marrowAntibodyviral receptorEx vivoVirology
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Autoimmune thyroid disease: new models of cell death in autoimmunity

2002

Autoimmunity to thyroid antigens leads to two distinct pathogenic processes with opposing clinical outcomes: hypothyroidism in Hashimoto's thyroiditis and hyperthyroidism in Graves' disease. The high frequency of these diseases and easy accessibility of the thyroid gland has allowed the identification of key pathogenic mechanisms in organ-specific autoimmune diseases. In early investigations, antibody- and T-cell-mediated death mechanisms were proposed as being responsible for autoimmune thyrocyte depletion. Later, studies on apoptosis have provided new insights into autoimmune target destruction, indicating the involvement of death receptors and cytokine-regulated apoptotic pathways in the…

endocrine systemHistoryProgrammed cell deathFas Ligand Proteinendocrine system diseasesImmunologyThyroid GlandApoptosisAutoimmunityDiseasemedicine.disease_causeThyroiditisEducationAutoimmunityPathogenesisAntigenSettore MED/04 - PATOLOGIA GENERALEHumansMedicinefas ReceptorMembrane Glycoproteinsbiologybusiness.industryThyroidThyroiditis Autoimmunemedicine.diseaseGraves DiseaseComputer Science Applicationsmedicine.anatomical_structureModels AnimalImmunologybiology.proteinAntibodybusinessT-Lymphocytes CytotoxicNature Reviews Immunology
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Germinal center B cells govern their own fate via antibody feedback

2013

High-affinity antibodies reenter germinal centers (GCs) and limit antigen access, thus causing sustained directional evolution in GCs toward higher-affinity antibody production.

endocrine systemImmunologyB-cell receptorAntibody AffinityPlasma cellBiologyAntibodiesAffinity maturationMice03 medical and health sciences0302 clinical medicinehealth services administrationpolycyclic compoundsmedicineAnimalsImmunology and AllergyCell LineageAntigen-presenting cell030304 developmental biologyB-Lymphocytes0303 health sciencesB cell selectionBrief Definitive ReportGerminal centerGerminal CenterMolecular biology3. Good healthMice Inbred C57BLB-1 cellmedicine.anatomical_structurePolyclonal B cell responsesense organshormones hormone substitutes and hormone antagonistsDendritic Cells Follicular030215 immunologyJournal of Experimental Medicine
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Tumor de células de la granulosa extraovárico con mutación FOXL2. Diagnóstico diferencial morfológico e inmunohistoquímico

2020

Extraovarian granulosa cell tumor is a very uncommon tumor and the identification of a recurrent mutation in FOXL2 may be used as another diagnostic tool along with the classical morphological and immunohistochemical findings. Here, we report a new case of extraovarian granulosa cell tumor in a 57 years old female patient presented with a sub-hepatic mass and abdominal pain. Histopathological examination of the excised mass showed features of adult-type granulosa cell tumor with α-inhibin, calretinin, WT1, S100, CD99 and progesterone receptor immunoreactivity. A FOXL2 mutation was detected on molecular biology study. A final diagnosis was an extraovarian adult-type granulosa cell tumor. We …

endocrine systemPathologymedicine.medical_specialtyAbdominal painurogenital systemGranulosa cellCD99BiologyPathology and Forensic Medicine03 medical and health sciences0302 clinical medicine030220 oncology & carcinogenesisFemale patientProgesterone receptormedicineImmunohistochemistry030211 gastroenterology & hepatologyDifferential diagnosisCalretininmedicine.symptomRevista Española de Patología
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Somatostatin and Somatostatin Receptors: From Signaling to Clinical Applications in Neuroendocrine Neoplasms

2021

Neuroendocrine neoplasms (NENs) are heterogeneous neoplasms which arise from neuroendocrine cells that are distributed widely throughout the body. Although heterogenous, many of them share their ability to overexpress somatostatin receptors (SSTR) on their cell surface. Due to this, SSTR and somatostatin have been a large subject of interest in the discovery of potential biomarkers and treatment options for the disease. The aim of this review is to describe the molecular characteristics of somatostatin and somatostatin receptors and its application in diagnosis and therapy on patients with NENs as well as the use in the near future of somatostatin antagonists.

endocrine systemQH301-705.5CellMedicine (miscellaneous)ReviewGeneral Biochemistry Genetics and Molecular BiologymedicineBiology (General)Tumorspeptide receptor radionuclide therapyReceptors d'hormonesneuroendocrine neoplasmssomatostatin analoguesSomatostatin receptorbusiness.industryTreatment optionsLU-DOTA-TATEmedicine.anatomical_structureSomatostatinPotential biomarkerssomatostatin receptorssomatostatin antagonistsCancer research68Ga PETbusinesshormones hormone substitutes and hormone antagonistsBiomedicines
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EZH2 mutations are frequent and represent an early event in follicular lymphoma

2013

Gain of function mutations in the H3K27 methyltransferase EZH2 represent a promising therapeutic target in germinal center lymphomas. In this study, we assessed the frequency and distribution of EZH2 mutations in a large cohort of patients with follicular lymphoma (FL) (n = 366) and performed a longitudinal analysis of mutation during the disease progression from FL to transformed FL (tFL) (n = 33). Mutations were detected at 3 recurrent mutation hot spots (Y646, A682, and A692) in 27% of FL cases with variant allele frequencies (VAF) ranging from 2% to 61%. By comparing VAF of EZH2 with other mutation targets (CREBBP, MLL2, TNFRSF14, and MEF2B), we were able to distinguish patients harbori…

endocrine systemTime FactorsMethyltransferasemedicine.medical_treatmentDNA Mutational AnalysisImmunologyFollicular lymphomaKaplan-Meier Estimatemacromolecular substancesBiologymedicine.disease_causeBiochemistryTargeted therapyCohort StudiesGene Frequencyhemic and lymphatic diseasesBiomarkers TumormedicineHumansEnhancer of Zeste Homolog 2 ProteinLymphoma FollicularAllele frequencyMutationLymphoid NeoplasiaMEF2 Transcription FactorsGene Expression ProfilingEZH2Polycomb Repressive Complex 2Germinal centerCell BiologyHematologymedicine.diseaseCREB-Binding ProteinLymphomaMutationDisease ProgressionCancer researchReceptors Tumor Necrosis Factor Member 14Blood
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Vasopressin and oxytocin receptors (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database

2019

Vasopressin (AVP) and oxytocin (OT) receptors (nomenclature as recommended by NC-IUPHAR [92]) are activated by the endogenous cyclic nonapeptides vasopressin and oxytocin. These peptides are derived from precursors which also produce neurophysins (neurophysin I for oxytocin; neurophysin II for vasopressin). Vasopressin and oxytocin differ at only 2 amino acids (positions 3 and 8). There are metabolites of these neuropeptides that may be biologically active [67].

endocrine systemVasopressinChemistryNeurophysin INeuropeptideNeurophysinsPharmacologyOxytocin receptorOxytocinNeurophysin IImedicineReceptorhormones hormone substitutes and hormone antagonistsmedicine.drugIUPHAR/BPS Guide to Pharmacology CITE
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Thyroid Stimulating Hormone Receptor Antibodies in Thyroid Eye Disease-Methodology and Clinical Applications.

2018

Background Thyroid stimulating hormone receptor antibodies (TSHR-Ab) cause autoimmune hyperthyroidism and are prevalent in patients with related thyroid eye disease (TED). Purpose To provide a historical perspective on TSHR-Ab and to present evidence-based recommendations for clinical contemporary use. Methods The authors review the recent literature pertaining to TSHR-Ab in patients with TED and describe the various immunoassays currently used for detecting TSHR-Ab and their clinical applications. Results We provide a historical summary and description of the various methods used to detect TSHR-Ab, foremost, the functional TSHR-Ab. Increasing experimental and clinical data demonstrate the …

endocrine systemendocrine system diseasesEye disease030209 endocrinology & metabolismAutoimmunitymedicine.disease_causeBioinformaticsAutoimmunity03 medical and health sciences0302 clinical medicineMedicineAnimalsHumansClinical severityReceptorAutoantibodiesbiologybusiness.industryThyroidReceptors ThyrotropinGeneral Medicinemedicine.diseaseClinical diseaseeye diseasesThyroid stimulating hormone receptorGraves OphthalmopathyOphthalmologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinSurgeryAntibodybusinesshormones hormone substitutes and hormone antagonistsOphthalmic plastic and reconstructive surgery
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Review article: a comparison of glucagon-like peptides 1 and 2.

2013

Summary Background Recent advancements in understanding the roles and functions of glucagon-like peptide 1 (GLP-1) and 2 (GLP-2) have provided a basis for targeting these peptides in therapeutic strategies. Aim To summarise the preclinical and clinical research supporting the discovery of new therapeutic molecules targeting GLP-1 and GLP-2. Methods This review is based on a comprehensive PubMed search, representing literature published during the past 30 years related to GLP-1 and GLP-2. Results Although produced and secreted together primarily from L cells of the intestine in response to ingestion of nutrients, GLP-1 and GLP-2 exhibit distinctive biological functions that are governed by t…

endocrine systemmedia_common.quotation_subjectIncretinPharmacologyintestinal peptides GLP-1 GLP-2 incretin intestinal motilitySettore BIO/09 - FisiologiaIntestinal mucosaGlucagon-Like Peptide 1Glucagon-Like Peptide 2Receptors GlucagonAnimalsHumansMedicinePharmacology (medical)Molecular Targeted TherapyReceptormedia_commonClinical Trials as TopicHepatologybusiness.industrydigestive oral and skin physiologyGastroenterologyAppetiteReview articleDisease Models Animalmedicine.anatomical_structureGlucagon-Like PeptidesbusinessPancreashormones hormone substitutes and hormone antagonistsFunction (biology)
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