Search results for "Reuptake"

showing 10 items of 96 documents

Behavioral analysis indicates benzodiazepine-tolerance mediated by the benzodiazepine binding-site at the GABA(A)-receptor.

2001

Abstract 1. GABA A -receptor induced changes in locomotion and anxiety-like behaviors were studied in rats using an open-field and an elevated plus-maze. Acute and chronic doses of the benzodiazepine diazepam without and in combination with the GABA uptake inhibitor SKF-89976A were investigated. 2. Fifty-six male rats of the strain PVG/OlaHsd (PVG; 180–200g body wt) were used to assess the influence of the benzodiazepine binding-site to the development of tolerance. Rats were divided into six groups: The first receiving saline (0.9%), the second and third diazepam (10.0 mg/kg) daily for 23 days with or without an acute challenge of 2.0 mg/kg diazepam. The fourth group received diazepam (10.…

MaleElevated plus mazemedicine.medical_specialtymedicine.drug_classGABA AgentsNipecotic AcidsOpen fieldchemistry.chemical_compoundOral administrationInternal medicineMedicineAnimalsheterocyclic compoundsMaze LearningBiological PsychiatryPharmacologyBenzodiazepineDiazepamGABAA receptorbusiness.industryReceptors GABA-ARatsEndocrinologychemistryAnti-Anxiety AgentsExploratory BehaviorSKF-89976AbusinessReuptake inhibitorDiazepammedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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Tiagabine, a gamma-amino-butyric acid transporter inhibitor impairs spatial learning of rats in the Morris water-maze.

2002

Abstract γ-Amino-butyric acid (GABA) is cleaved from the synaptic cleft by uptake via specific transporters. Inhibition of such transporters increases the effectiveness of physiologically released GABA. Increased GABAergic neurotransmission has an impact on learning and memory. Therefore, effects of tiagabine, a GABA-transporter inhibitor, were investigated on spatial orientation in the Morris water-maze. Rats were given four training trials per day for 4 days and a probe trial without platform on the 5th day. Compared to saline treated rats, rats treated daily with 20 mg/kg tiagabine showed impaired learning during the acquisition trials. Retrieval was impaired in rats treated only at the …

MaleGABA Plasma Membrane Transport ProteinsSynaptic cleftTiagabinemedicine.medical_treatmentNipecotic AcidsMorris water navigation taskOrganic Anion TransportersPharmacologyBehavioral Neurosciencechemistry.chemical_compoundMemorymedicineGABA transporterAnimalsNeurotransmitterMaze LearningTiagabineSalineGABA AgonistsSwimmingbiologyMembrane ProteinsMembrane Transport ProteinsTransporterRats Inbred StrainsReceptors GABA-ARatschemistrybiology.proteinReuptake inhibitorCarrier ProteinsNeurosciencemedicine.drugBehavioural brain research
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Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Blood-brain barrier penetration of the enantiomers of venlafaxine and its metabolites in mice lacking P-glycoprotein

2010

According to in vitro studies the enantiomers of venlafaxine display different degrees of serotonin and noradrenaline reuptake inhibition. Therefore, clarification of the enantiomeric drug distribution between serum and brain is highly warranted. To elucidate if P-glycoprotein (P-gp) in a stereoselective manner transports venlafaxine and its metabolites out of the brain we used abcb1ab double-knockout mice that do not express P-gp. A single dose of racemic venlafaxine (10 mg/kg bw) was intraperitoneally injected to knockout (-/-) and wildtype (+/+) mice. Serum and brain samples were collected 1, 3, 6 and 9 h following drug administration for analysis by LC/MS/MS. One to six hours post-dose,…

MaleMedicin och hälsovetenskapVenlafaxinePharmacologyBlood–brain barrierMedical and Health SciencesMicemedicineAnimalsPharmacology (medical)ATP Binding Cassette Transporter Subfamily B Member 1Biological PsychiatryP-glycoproteinPharmacologyMice KnockoutbiologyChemistryVenlafaxine HydrochlorideBiological TransportStereoisomerismCyclohexanolsIn vitroPsychiatry and Mental healthmedicine.anatomical_structureNeurologyBlood-Brain BarrierKnockout mousebiology.proteinStereoselectivityNeurology (clinical)SerotoninEnantiomerSelective Serotonin Reuptake Inhibitorsmedicine.drug
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Treatments used for obsessive-compulsive disorder-An international perspective.

2018

Objective The objective of this study was to characterise international trends in the use of psychotropic medication, psychological therapies, and novel therapies used to treat obsessive–compulsive disorder (OCD). Methods Researchers in the field of OCD were invited to contribute summary statistics on the characteristics of their samples. Consistency of summary statistics across countries was evaluated. Results The study surveyed 19 expert centres from 15 countries (Argentina, Australia, Brazil, China, Germany, Greece, India, Italy, Japan, Mexico, Portugal, South Africa, Spain, the United Kingdom, and the United States) providing a total sample of 7,340 participants. Fluoxetine (n = 972; 13…

MaleObsessive-Compulsive DisorderInternationalitymedicine.medical_treatmentDeep Brain StimulationSocial SciencesFluvoxamineBENZODIAZEPINASpharmacotherapyBenzodiazepines0302 clinical medicinePharmacology (medical)TERAPIA PSICOANALITICAPSICOFARMACOLOGIAantipsychotics; benzodiazepines; cross-cultural study; obsessive-compulsive disorder; pharmacotherapy; selective serotonin reuptake inhibitorsMiddle Aged3. Good healthExposure and response preventionantipsychotics; benzodiazepines; cross-cultural study; obsessive–compulsive disorder; pharmacotherapy; selective serotonin reuptake inhibitorsNeurologyPsychiatry and Mental HealthSerotonin Uptake Inhibitorscross-cultural studyAripiprazoleFemalebenzodiazepineSelective Serotonin Reuptake Inhibitorsmedicine.drugPsychosurgeryAntipsychotic AgentsAdultmedicine.medical_specialty:Ciências da Saúde [Ciências Médicas]Ciências Médicas::Ciências da SaúdeSerotonin reuptake inhibitor03 medical and health sciencesANTIPSICOTICOSobsessive–compulsive disorderselective serotonin reuptake inhibitorsmedicinePSICOTROPICOSHumansAntipsychoticPsychiatryFARMACOTERAPIAFluoxetineRisperidoneantipsychotics; benzodiazepines; cross-cultural study; obsessive–compulsive disorder; pharmacotherapy; selective serotonin reuptake inhibitors; Neurology; Neurology (clinical); Psychiatry and Mental Health; Pharmacology (medical)Science & Technologyselective serotonin reuptake inhibitorbusiness.industryTRASTORNO OBSESIVO COMPULSIVO030227 psychiatryantipsychoticPsychosurgeryantipsychoticsNeurology (clinical)business030217 neurology & neurosurgerySEROTONINAHuman psychopharmacology
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Effects of acute and chronic maprotiline administration on inhibitory avoidance in male mice

2000

The effects of acute and chronic administration of maprotiline (5, 10 or 20 mg/kg, intraperitoneally) were assessed on inhibitory avoidance in male mice. Acute administration of maprotiline before training did not effect training phase latencies, but impaired performance (i.e. produced shorter latencies) in the test at doses of 5 and 20 mg/kg. When given after training, the drug did not modify test latencies at any of the doses used. Chronic administration for 21 days (interrupted 24 h before training) also shortened latencies in the test but not in training. An experiment on the acute effects of maprotiline on analgesia (determination of flinch and jump thresholds for increasing electric f…

MalePain ThresholdAnterograde amnesiaRatónInhibitory postsynaptic potentialDrug Administration ScheduleDevelopmental psychologyNorepinephrine (medication)MiceBehavioral NeuroscienceDrug toleranceThreshold of painAvoidance LearningReaction TimemedicineAnimalsMaprotilineDose-Response Relationship DrugBrainNeural InhibitionDrug ToleranceMaprotilineAnesthesiaMental RecallAntidepressive Agents Second-Generationmedicine.symptomPsychologyReuptake inhibitorInjections Intraperitonealmedicine.drugBehavioural Brain Research
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The pathogenetic role of adulterants in 5 cases of drug addicts with a fatal outcome

2013

The purpose of the present study is to determine the role of lidocaine, caffeine and dextromethorphan, used as adulterant substances, in five cases of drug overdose which have come to our attention. Taking into account the pharmacological mechanism, blood concentration and route of administration (intravenous) we evaluated the hypothesis that these substances could act with a synergistic effect - or at least additive - with the illicit drugs on the central nervous system and cardiovascular system.

MalePyrrolidinesDrug ContaminationLidocainePharmacologyKidneyDextromethorphanDrug Userschemistry.chemical_compoundBileAnesthetics LocalForensic PathologyLungAdulterantMorphineDextromethorphanGastrointestinal ContentsLiverFemaleDrug ContaminationCaffeineSelective Serotonin Reuptake Inhibitorsmedicine.drugAdultNarcoticsSubstance-Related DisordersCitalopramDrug overdosePathology and Forensic MedicineForensic ToxicologyRoute of administrationAdulterantsCaffeinemedicineHumansBrain ChemistryMorphine DerivativesCodeineIllicit Drugsbusiness.industryForensic toxicologyAdulterants Lidocaine Caffeine DextromethorphanLidocainemedicine.diseaseVitreous BodyAntitussive AgentschemistryCentral Nervous System StimulantsDrug OverdosebusinessLawMethadoneForensic Science International
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Risk of bleeding related to selective and non-selective serotonergic antidepressants: a case/non-case approach using data from two pharmacovigilance …

2014

There is increasing evidence for an association between treatment with selective serotonin reuptake inhibitors (SSRI) and an increased risk of bleeding events. The most important underlying mechanism appears to be inhibition of serotonin uptake in platelets, an effect that is also present in antidepressants with non-selective serotonin-reuptake inhibition (NSRI). Accordingly, also NSRI may be associated with an increased risk of bleeding. However, there is little data in this regard.Based on data (spontaneous reports of adverse drug reactions) from 2 pharmacovigilance databases (WHO-database/Vigibase™; BfArM/AkdÄ-database in Germany) we used a case/non-case approach and calculated reporting…

MaleSerotonin uptakeDatabases FactualDrug-Related Side Effects and Adverse ReactionsVenlafaxineHemorrhageSerotonergiccomputer.software_genrePharmacovigilanceDiclofenacSerotonin AgentsSerotonin AgentsGermanyPharmacovigilanceMedicineHumansPharmacology (medical)Databasebusiness.industryDepressionGeneral MedicineOdds ratioAntidepressive AgentsPsychiatry and Mental healthFemalebusinessReuptake inhibitorcomputermedicine.drugPharmacopsychiatry
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Neurochemical Substrates of MDMA Reward: Effects of the Inhibition of Serotonin Reuptake on the Acquisition and Reinstatement of MDMA-induced CPP

2013

Different neurotransmitter brain systems have been implicated in the rewarding effects of 3,4-methylenedioxymetamphetamine (MDMA), including dopamine or serotonin. Serotonin selective reuptake inhibitors (SSRI) are a commonly prescribed therapy for psychiatric disorders, and the SSRI fluoxetine is recommended for MDMA users due to its neuroprotective effect against MDMAinduced neurotoxicity. In the present work, we employed the conditioned place preference (CPP) paradigm to study how the inhibition of serotonin reuptake with fluoxetine affected the rewarding and reinstating effects of MDMA in adolescent male mice. Firstly, we evaluated the motivational effects of fluoxetine (1 and 10 mg/kg)…

MaleSerotoninN-Methyl-34-methylenedioxyamphetaminePharmacologyMicechemistry.chemical_compoundNeurochemicalRewardDopamineFluoxetineConditioning Psychologicalmental disordersDrug DiscoveryAnimalsMedicineNeurotransmitterPharmacologyFluoxetineDose-Response Relationship Drugbusiness.industryMDMAConditioned place preferencechemistryHallucinogensSerotoninbusinessReuptake inhibitorSelective Serotonin Reuptake Inhibitorspsychological phenomena and processesmedicine.drugCurrent Pharmaceutical Design
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Effect of chronic citalopram on serotonin-related and stress-regulated genes in the dorsal raphe nucleus of the rat

2007

Using a model of depression in which chronic social stress induces depressive-like symptoms, we investigated effects of the selective serotonin-reuptake inhibitor (SSRI) citalopram on gene expression in the dorsal raphe nucleus of male rats. Expression of tryptophan hydroxylase (TPH) protein was found to be upregulated by the stress and normalized by citalopram, while mRNAs for genes TPH 1 and 2 were differentially affected. Citalopram had no effect on serotonin transporter mRNA but reduced serotonin-1A autoreceptor mRNA in stressed animals. The SSRI prevented the stress-induced upregulation of mRNA for CREB binding protein, synaptic vesicle glycoprotein 2b and the glial N-myc downstream-re…

MaleSerotoninendocrine systemmedicine.medical_specialtyCitalopramTryptophan HydroxylaseBiologyCitalopramPolymerase Chain Reactionbehavioral disciplines and activitiesGene Expression Regulation Enzymologic03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDorsal raphe nucleusStress PhysiologicalInternal medicinemental disordersmedicineAnimalsPharmacology (medical)Rats WistarSocial BehaviorNeurotransmitterBiological PsychiatrySerotonin transporter030304 developmental biologyPharmacology0303 health sciencesTryptophan hydroxylaseRatsPsychiatry and Mental healthEndocrinologyNeurologychemistryModels Animalbiology.proteinSynaptic vesicle glycoprotein 2BRNARaphe Nuclei5-HT1A receptorNeurology (clinical)SerotoninSelective Serotonin Reuptake Inhibitors030217 neurology & neurosurgerymedicine.drugEuropean Neuropsychopharmacology
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