Search results for "Synapses"

showing 10 items of 183 documents

Brain choline has a typical precursor profile.

1998

Choline is product and precursor to both acetylcholine and membrane phospholipids, and, in the brain, is ultimately provided by the circulation. The brain is protected from excess choline and choline deprivation by a refined system of homeostatic mechanisms that maintain a level of extracellular choline that, for its role as precursor, meets saturation criteria under normal conditions. The kinetic and activity profiles of choline are typical for a biosynthetic precursor.

ChemistryGeneral NeuroscienceBrainCholine acetyltransferaseAcetylcholineCholinechemistry.chemical_compoundKineticsMembraneBiochemistryPhysiology (medical)SynapsesmedicineExtracellularCholinergicCholineAnimalsHomeostasisHumansCholinergic mechanismsAcetylcholineHomeostasisPhospholipidsmedicine.drugJournal of physiology, Paris
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An exon junction complex‐independent function of Barentsz in neuromuscular synapse growth

2021

The exon junction complex controls the translation, degradation, and localization of spliced mRNAs, and three of its core subunits also play a role in splicing. Here, we show that a fourth subunit, Barentsz, has distinct functions within and separate from the exon junction complex in Drosophila neuromuscular development. The distribution of mitochondria in larval muscles requires Barentsz as well as other exon junction complex subunits and is not rescued by a Barentsz transgene in which residues required for binding to the core subunit eIF4AIII are mutated. In contrast, interactions with the exon junction complex are not required for Barentsz to promote the growth of neuromuscular synapses.…

ChemistryTransgeneProtein subunitMutantRNA-Binding ProteinsTranslation (biology)ExonsBiochemistryNeuromuscular junctionCell biologySynapsemedicine.anatomical_structureRNA splicingEukaryotic Initiation Factor-4ASynapsesGeneticsmedicineExon junction complexAnimalsDrosophila ProteinsDrosophilaMolecular BiologyReports
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Sparse Distributed Representation of Odors in a Large-scale Olfactory Bulb Circuit

2013

In the olfactory bulb, lateral inhibition mediated by granule cells has been suggested to modulate the timing of mitral cell firing, thereby shaping the representation of input odorants. Current experimental techniques, however, do not enable a clear study of how the mitral-granule cell network sculpts odor inputs to represent odor information spatially and temporally. To address this critical step in the neural basis of odor recognition, we built a biophysical network model of mitral and granule cells, corresponding to 1/100th of the real system in the rat, and used direct experimental imaging data of glomeruli activated by various odors. The model allows the systematic investigation and g…

Circuit ModelsMaleNerve net0302 clinical medicineLateral inhibitionOdorlcsh:QH301-705.5NeuronsFeedback PhysiologicalCoding Mechanisms0303 health sciencesNeuronal PlasticityEcologyAnatomyOlfactory BulbSynapseSensory Systemsmedicine.anatomical_structureComputational Theory and MathematicsModeling and SimulationExcitatory postsynaptic potentialResearch ArticleModels NeurologicalBiologyInhibitory postsynaptic potential03 medical and health sciencesCellular and Molecular NeuroscienceGeneticNeuroplasticityGeneticsmedicineAnimalsComputer SimulationBiologyMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyComputational NeuroscienceOlfactory SystemAnimalComputational BiologyNeuronEcology Evolution Behavior and SystematicRatsOlfactory bulbOdorlcsh:Biology (General)OdorantsSynapsesSynaptic plasticityRatNerve NetNeuroscience030217 neurology & neurosurgeryNeuroscience
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A modeling study suggesting how a reduction in the context-dependent input on CA1 pyramidal neurons could generate schizophrenic behavior.

2011

The neural mechanisms underlying schizophrenic behavior are unknown and very difficult to investigate experimentally, although a few experimental and modeling studies suggested possible causes for some of the typical psychotic symptoms related to this disease. The brain region most involved in these processes seems to be the hippocampus, because of its critical role in establishing memories for objects or events in the context in which they occur. In particular, a hypofunction of the N-methyl-D-aspartate (NMDA) component of the synaptic input on the distal dendrites of CA1 pyramidal neurons has been suggested to play an important role for the emergence of schizophrenic behavior. Modeling st…

Computer scienceCognitive Neurosciencemedia_common.quotation_subjectSchizophrenia Realistic model CA1 Hippocampus Object recognition Synaptic integrationCentral nervous systemModels NeurologicalCa1 neuronHippocampusHippocampal formationSynapse03 medical and health sciences0302 clinical medicineArtificial IntelligencePerceptionmedicineAnimalsHumansInvariant (mathematics)CA1 Region Hippocampal030304 developmental biologymedia_common0303 health sciencesRecallArtificial neural networkPyramidal NeuronSynaptic integrationPyramidal CellsCognitive neuroscience of visual object recognitionDendritesmedicine.diseasemedicine.anatomical_structurenervous systemSchizophreniaSynapsesSchizophreniaNMDA receptorNeuronNerve NetNeuroscience030217 neurology & neurosurgeryNeural networks : the official journal of the International Neural Network Society
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Coincident glutamatergic depolarizations enhance GABAA receptor-dependent Cl- influx in mature and suppress Cl- efflux in immature neurons.

2021

The impact of GABAergic transmission on neuronal excitability depends on the Cl--gradient across membranes. However, the Cl--fluxes through GABAA receptors alter the intracellular Cl- concentration ([Cl-]i) and in turn attenuate GABAergic responses, a process termed ionic plasticity. Recently it has been shown that coincident glutamatergic inputs significantly affect ionic plasticity. Yet how the [Cl-]i changes depend on the properties of glutamatergic inputs and their spatiotemporal relation to GABAergic stimuli is unknown. To investigate this issue, we used compartmental biophysical models of Cl- dynamics simulating either a simple ball-and-stick topology or a reconstructed CA3 neuron. Th…

Databases FactualPhysiologyNervous SystemBiochemistrySynaptic TransmissionAnimal CellsMedicine and Health SciencesCl effluxBiology (General)Receptorgamma-Aminobutyric AcidNeuronsNeuronal PlasticityEcologyNeuronal MorphologyGABAA receptorChemistryPyramidal CellsNeurochemistryNeurotransmittersCA3 Region HippocampalElectrophysiologymedicine.anatomical_structureComputational Theory and MathematicsModeling and SimulationGABAergicAnatomyCellular TypesReceptor PhysiologyIntracellularResearch ArticleCell PhysiologyQH301-705.5Models NeurologicalNeurophysiologyMembrane PotentialCellular and Molecular NeuroscienceGlutamatergicChloridesGeneticsmedicineAnimalsMolecular BiologyEcology Evolution Behavior and SystematicsBiology and Life SciencesComputational BiologyCell BiologyNeuronal DendritesReceptors GABA-ACellular NeuroscienceSynapsesCa3 pyramidal neuronDepolarizationNeuronNeuroscienceNeurosciencePLoS Computational Biology
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Synaptic release of zinc from brain slices: factors governing release, imaging, and accurate calculation of concentration.

2006

Cerebrocortical neurons that store and release zinc synaptically are widely recognized as critical in maintenance of cortical excitability and in certain forms of brain injury and disease. Through the last 20 years, this synaptic release has been observed directly or indirectly and reported in more than a score of publications from over a dozen laboratories in eight countries. However, the concentration of zinc released synaptically has not been established with final certainty. In the present work we have considered six aspects of the methods for studying release that can affect the magnitude of zinc release, the imaging of the release, and the calculated concentration of released zinc. We…

Diagnostic ImagingPyridinesColoring agentschemistry.chemical_elementZincIn Vitro TechniquesRats Sprague-DawleyPregnancyAnimalsAcido edeticoPolycyclic CompoundsRats WistarColoring AgentsEdetic AcidFluorescent DyesNeuronsExtramuralChemistryGeneral NeuroscienceTemperatureBrainOriginal dataRatsSprague dawleyZincDentate GyrusMossy Fibers HippocampalSynapsesFemaleSynaptic VesiclesNeuroscienceJournal of neuroscience methods
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Apical dendritic spines and axonic terminals in the bipyramidal neurons of the dorsomedial cortex of lizards (Lacerta).

1984

Gold-toned bipyramidal neurons of the dorsomedial cortex of Lacerta have been studied using light and electron microscopy. The spines have been classified as stubby, mushroom-shaped or thin. Thin and mushroom-shaped spines are only found on proximal and intermediate dendritic segments, whereas stubby spines are found on distal dendritic segments. A Timm's method modification for electron microscopy (sulphide-osmium procedure) has been used. Timm-positive axonal endings usually synapse on thin and mushroom-shaped spines, whereas Timm-negative axonal endings usually synapse on stubby spines. Timm-positive afferents and their post-synaptic spines on bipyramidal neurons of Lacerta's dorsomedial…

EmbryologyDendritic spineHippocampusPodarcis hispanicalaw.inventionSynapselawCortex (anatomy)medicineLacertaAnimalsCerebral CortexMammalsNerve EndingsNeuronsbiologyLizardsCell BiologyAnatomyDendritesbiology.organism_classificationAxonsMicroscopy Electronmedicine.anatomical_structurenervous systemCerebral cortexSynapsesAnatomyElectron microscopeDevelopmental BiologyAnatomy and embryology
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Identification of the cannabinoid receptor type 1 in serotonergic cells of raphe nuclei in mice.

2007

The endocannabinoid system (ECS) possesses neuromodulatory functions by influencing the release of various neurotransmitters, including GABA, noradrenaline, dopamine, glutamate and acetylcholine. Even though there are studies indicating similar interactions between the ECS and the serotonergic system, there are no results showing clear evidence for type 1 cannabinoid receptor (CB1) location on serotonergic neurons. In this study, we show by in situ hybridization that a low but significant fraction of serotonergic neurons in the raphe nuclei of mice contains CB1 mRNA as illustrated by the coexpression with the serotonergic marker gene tryptophane hydroxylase 2, the rate limiting enzyme for t…

Genetic MarkersSerotoninSerotonin uptakeBiologyTryptophan HydroxylaseSerotonergicHippocampuschemistry.chemical_compoundMiceNerve FibersReceptor Cannabinoid CB1Cannabinoid receptor type 1AnimalsRNA MessengerNeurotransmitterIn Situ HybridizationSerotonin Plasma Membrane Transport ProteinsMicroscopy ConfocalTPH2General NeuroscienceAmygdalaEndocannabinoid systemImmunohistochemistryIsoenzymesMice Inbred C57BLnervous systemchemistryDentate GyrusSynapsesRaphe NucleiFemaleSerotoninRaphe nucleiNeuroscienceNeuroscience
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Usher syndrome: molecular links of pathogenesis, proteins and pathways.

2006

Contains fulltext : 50437.pdf (Publisher’s version ) (Closed access) Usher syndrome is the most common form of deaf-blindness. The syndrome is both clinically and genetically heterogeneous, and to date, eight causative genes have been identified. The proteins encoded by these genes are part of a dynamic protein complex that is present in hair cells of the inner ear and in photoreceptor cells of the retina. The localization of the Usher proteins and the phenotype in animal models indicate that the Usher protein complex is essential in the morphogenesis of the stereocilia bundle in hair cells and in the calycal processes of photoreceptor cells. In addition, the Usher proteins are important in…

Genetics and epigenetic pathways of disease [NCMLS 6]Usher syndromeCell Cycle ProteinsNerve Tissue ProteinsBiologyRetinaAdherens junctionMiceHair Cells AuditoryCell polarityGeneticsmedicineotorhinolaryngologic diseasesNeurosensory disorders [UMCN 3.3]AnimalsHumansProtein IsoformsCell Cycle ProteinMolecular BiologyGenetics (clinical)Renal disorder [IGMD 9]Adaptor Proteins Signal TransducingStereociliumMembrane ProteinsSignal transducing adaptor proteinGeneral MedicineActin cytoskeletonmedicine.diseaseeye diseasesCell biologyCytoskeletal ProteinsGenetic defects of metabolism [UMCN 5.1]Ear InnerMultiprotein ComplexesCateninSynapsessense organsUsher SyndromesPhotoreceptor Cells Vertebrate
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Pineal 'synaptic' ribbon numbers and melatonin synthesis of rat are resistant to guanethidine sympathectomy.

1995

Chemical sympathectomy is widely used to study the impact of the noradrenergic system on neuronal and neuroendocrine circuits. We tested the effects of intraperitoneal injections of guanethidine, an adrenergic neuron blocking agent, on selected functional parameters of the rat pineal gland which are known to be under sympathetic influence. The reliability of the method was demonstrated by the clear enophthalmus developed by experimental animals. However, neither the numbers of 'synaptic' ribbons nor melatonin synthesis differed between treated and control rats, both parameters exhibiting the nocturnal increase seen in intact animals. These results are in striking contrast to those obtained …

GuanethidineMalemedicine.medical_specialtymedicine.medical_treatmentAdrenergicPineal GlandMelatoninRats Sprague-DawleyCellular and Molecular NeuroscienceInternal medicineMedicineAnimalsMelatonin synthesisGanglionectomyMolecular BiologyGuanethidineMelatoninPharmacologySynaptic ribbonbusiness.industrySympathectomy ChemicalCell BiologyRatsEndocrinologymedicine.anatomical_structureSympathectomySynapsesMolecular MedicineNeuronbusinessmedicine.drugExperientia
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