Search results for "clone"

showing 10 items of 312 documents

Cytotoxic T lymphocytes define multiple peptide isoforms derived from the melanoma-associated antigen MART-1/Melan-A

1999

Peptides derived from the melanoma-associated MART-1/Melan-A antigen are currently implemented in immunotherapy for inducing or augmenting T-cell responses directed against peptides expressed by autologous tumor cells in HLA-A2+ patients with melanoma. Here, we describe the specificity of the T-cell clone SK29-FFM1.1, which secretes GM-CSF in response to a panel of synthetic MART-1/Melan-A-derived peptides, including the naturally presented ILTVILGVL32–40, but exhibits cytotoxicity and IFN-γ secretion exclusively to the MART-1/Melan-A derived peptide AAGIGILTV27–35. In addition, cytotoxic T-lymphocyte (CTL) clone SK29-FFM1.1 recognizes 3 different naturally processed and presented peptides …

Cytotoxicity ImmunologicCancer ResearchCellular immunityReceptors Antigen T-Cell alpha-betaT-Lymphocytesmedicine.medical_treatmentBiologyTransfectionEpitopeInterferon-gammaMART-1 AntigenImmune systemAntigenAntigens NeoplasmHLA-A2 AntigenTumor Cells CulturedmedicineHumansProtein IsoformsCytotoxic T cellAmino Acid SequenceMelanomaneoplasmsintegumentary systemReverse Transcriptase Polymerase Chain ReactionImmunotherapyMolecular biologyRecombinant ProteinsClone CellsNeoplasm ProteinsCTL*OncologyImmunologyClone (B-cell biology)T-Lymphocytes CytotoxicInternational Journal of Cancer
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Cytotoxic T cells with reciprocal antigenic peptide presentation function are not generally resistant to mutual lysis

2003

Cytotoxic T cells normally express major histocompatibility complex class I molecules, to which their T cell antigen receptors are restricted. Therefore, a single cytotoxic T cell can not only act as a cytolytic effector cell, but also as an antigen-presenting cell for other cytotoxic T cells of the same or a different clone. In the present paper, we used a murine cytotoxic T cell clone, 10BK.1, recognizing the ovalbumin-derived peptide OVA257-264 in combination with H-2Kb to investigate the consequences of reciprocal antigen presentation by these cytotoxic T cells. These cells proliferate after incubation with the relevant peptide in the absence of added accessory cells, indicating recipro…

Cytotoxicity ImmunologicCell SurvivalOvalbuminImmunologyAntigen presentationDose-Response Relationship ImmunologicBiologyLymphocyte ActivationMiceInterleukin 21AntigenAnimalsImmunology and AllergyCytotoxic T cellAntigen-presenting cellAntigen PresentationLymphokine-activated killer cellAntibodies MonoclonalCell BiologyCytotoxicity Tests ImmunologicFlow CytometryNatural killer T cellMolecular biologyPeptide FragmentsClone CellsCell biologyInterleukin 12Interleukin-2T-Lymphocytes CytotoxicImmunology & Cell Biology
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An Ovalbumin Peptide-Specific Cytotoxic T Cell Clone with Antigen Self-Presentation Capacity Uses Two Distinct Mechanisms to Kill Target Cells

1993

Abstract Cloned 10BK.1 T cells with specificity for the ovalbumin peptide OVA257-264 are representative of a novel cell type within the CD8 + subset of T cells. In the presence and in the absence of added antigen presenting cells these T cells react toward antigen (Ag) by proliferation and lymphokine production. These data suggest self-presentation of the Ag by 10BK.1 cells. Here we present evidence that 10BK.1 cells exhibit cytotoxic activity that involves two different cytotoxic effector mechanisms. (i) One mechanism is fast killing activity, apparent within 4 hr. Constitutive mouse T cell-specific proteinase-1 (MTSP-1) activity, constitutive expression of MTSP-1 RNA, increased by Ag chal…

Cytotoxicity ImmunologicPore Forming Cytotoxic ProteinsOvalbuminImmunologyAntigen presentationAntigen-Presenting CellsBiologyCytoplasmic GranulesLymphocyte ActivationGranzymesCell LineMiceInterleukin 21AntigenAnimalsCytotoxic T cellIL-2 receptorAntigen-presenting cellLymphotoxin-alphaMembrane GlycoproteinsCD40PerforinTumor Necrosis Factor-alphaSerine EndopeptidasesDegranulationMolecular biologyClone Cellsbiology.proteinInterleukin-2T-Lymphocytes CytotoxicCellular Immunology
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Two tyrosinase nonapeptides recognized on HLA-A2 melanomas by autologous cytolytic T lymphocytes

1994

A number of cytolytic T lymphocyte (CTL) clones derived from several melanoma patients have been found to recognize a majority of melanomas from HLA-A2 patients. We have reported previously that two such CTL clones recognize a product of the tyrosinase gene that is presented by HLA-A2. Here we show that one of these CTL clones recognizes a peptide encoded by the first nine amino acids of the putative signal sequence of tyrosinase. The other CTL clone recognizes a different tyrosinase peptide corresponding to amino acids 368-376. Both peptides contain consensus motifs of HLA-A2 binding peptides.

Cytotoxicity ImmunologicSignal peptideTyrosinaseMolecular Sequence DataImmunologyClone (cell biology)Tyrosinase PeptidePeptideIn Vitro TechniquesBiologyHLA-A2 AntigenTumor Cells CulturedConsensus sequenceHumansImmunology and AllergyAmino Acid SequenceMelanomaPeptide sequenceDNA Primerschemistry.chemical_classificationBase SequenceMonophenol MonooxygenaseVirologyRecombinant ProteinsCTL*chemistryPeptidesT-Lymphocytes CytotoxicEuropean Journal of Immunology
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Fine specificity and cytolytic activity of continuously growing alloreactive cytotoxic T lymphocyte clones.

1980

The role of Lyt 1+ T-cell-derived secondard CTL inducing factor allowed the cloning of alloreactive cytotoxic T lymphocytes (CTL) by the limiting dilution approach. Several monoclonal cell lines were established in vitro. The lytic activity of some of the cell lines exceeded that of CTL from bulk cultures; that is, 50% of the target cells were lysed at an effector to target cell ratio of 0.04:1. The fine specificity of individual CTL clones is discussed.

Cytotoxicity ImmunologicT-LymphocytesImmunologyCellchemical and pharmacologic phenomenaCell LineMiceAntibody SpecificitymedicineCytotoxic T cellAnimalsAntigens ViralMice Inbred BALB CChemistryEffectorH-2 Antigenshemic and immune systemsGeneral MedicineVirologyMolecular biologyClone CellsCTL*Cytolysismedicine.anatomical_structureLytic cycleCell cultureMonoclonalMice Inbred CBALymphocyte Culture Test MixedSpleenScandinavian journal of immunology
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T lymphocyte control of autoreactivity: analysis with human T cell clones and limiting dilution culture

1986

To investigate cellular mechanisms controlling activated autoreactive T lymphocytes, a limiting dilution system was established employing cloned autoreactive major his-tocompatibility complex class II specific lymphocytes (a2/7) as stimulator cells for autologous peripheral blood mononuclear cells. At low responder/stimulator ratios, cytotoxic effector cells were generated capable of lysing clone a2/7. Importantly, within the population of cells mediating autocytotoxic effector function, differential specificities were found to exist. The generation of such autocytotoxic T lymphocytes appears to be inhibited by an additional population of cells circulating at lower frequency suggesting that…

Cytotoxicity Immunologiceducation.field_of_studyT-LymphocytesT cellImmunologyPopulationHistocompatibility Antigens Class IICell CommunicationT lymphocyteBiologyT-Lymphocytes RegulatoryPeripheral blood mononuclear cellClone CellsCell biologymedicine.anatomical_structureCell–cell interactionCell cultureImmunologyImmune TolerancemedicineHumansImmunology and AllergyCytotoxic T cellClone (B-cell biology)educationEuropean Journal of Immunology
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Functional Inactivation of pRB Results in Aneuploid Mammalian Cells After Release From a Mitotic Block

2002

AbstractThe widespread chromosome instability observed in tumors and in early stage carcinomas suggests that aneuploidy could be a prerequisite for cellular transformation and tumor initiation. Defects in tumor suppressers and genes that are part of mitotic checkpoints are likely candidates for the aneuploid phenotype. By using flow cytometric, cytogenetic, immunocytochemistry techniques we investigated whether pRB deficiency could drive perpetual aneuploidy in normal human and mouse fibroblasts after mitotic checkpoint challenge by microtubule-destabilizing drugs. Both mouse and human pRB-deficient primary fibroblasts resulted, upon release from a mitotic block, in proliferating aneuploid …

DNA ReplicationCancer ResearchBrief ArticleClone (cell biology)MitosisAneuploidyCre recombinaseSpindle Apparatuslcsh:RC254-282Retinoblastoma ProteinColony-Forming Units AssayMicechemistry.chemical_compoundChromosome instabilitymedicineAnimalsHumanscentrosomesCINGenes RetinoblastomaMitosisCells CulturedIn Situ Hybridization FluorescenceCentrosomeCell cycle controlbiologyColcemidChromosome FragilityCell CycleGINDemecolcineRetinoblastoma proteinAneuploidy; Cell cycle control; Centrosomes; CIN; PRB;FibroblastsCell cyclelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensAneuploidyFlow Cytometrymedicine.diseaseAntineoplastic Agents PhytogenicCell biologyCell Transformation NeoplasticPRBMicroscopy Fluorescencechemistrybiology.proteinFemaleNeoplasia
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Identification of a clone of Escherichia coli O103:H2 as a potential agent of hemolytic-uremic syndrome in France

1993

In a French multicenter study, six verocytotoxin-producing Escherichia coli strains were isolated from the stools of 6 of 69 children suffering from hemolytic-uremic syndrome. All strains belonged to serotype O103:H2, a serotype commonly associated with diarrhea in weaned rabbits in France. To determine whether the strains from humans and rabbits were genetically related, they were compared by analyzing their esterase electropherotypes and the restriction fragment length polymorphisms of the ribosomal DNA regions. A common clonal origin of these pathogenic strains was suggested by their identical esterase electropherotypes and their identical ribotypes, in addition to their identical seroty…

DNA BacterialMicrobiology (medical)SerotypeBacterial ToxinsMolecular Sequence DataClone (cell biology)VirulenceVerocytotoxinShiga Toxin 1medicine.disease_causePolymerase Chain Reactionlaw.inventionMicrobiology03 medical and health scienceschemistry.chemical_compoundlawEscherichia colimedicineHumansSerotyping[SDV.MP] Life Sciences [q-bio]/Microbiology and ParasitologyEscherichia coliEscherichia coli InfectionsComputingMilieux_MISCELLANEOUSPolymerase chain reaction030304 developmental biology0303 health sciencesBase SequenceVirulencebiology030306 microbiologyInfantCorrectionbiology.organism_classificationEnterobacteriaceae3. Good healthBacterial adhesinPOUVOIR PATHOGENE[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologychemistryChild PreschoolHemolytic-Uremic SyndromeFranceResearch ArticleJournal of Clinical Microbiology
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The rise and the fall of a Pseudomonas aeruginosa endemic lineage in a hospital

2021

The biological features that allow a pathogen to survive in the hospital environment are mostly unknown. The extinction of bacterial epidemics in hospitals is mostly attributed to changes in medical practice, including infection control, but the role of bacterial adaptation has never been documented. We analysed a collection of Pseudomonas aeruginosa isolates belonging to the Besançon Epidemic Strain (BES), responsible for a 12year nosocomial outbreak, using a genotype-to-phenotype approach. Bayesian analysis estimated the emergence of the clone in the hospital 5 years before its opening, during the creation of its water distribution network made of copper. BES survived better than the refe…

DNA Bacterialparallel evolutionLineage (genetic)Genomic IslandsPathogens and EpidemiologyBiologymedicine.disease_causeAmoeba (operating system)Disease OutbreaksMicrobiology03 medical and health sciencesAntibiotic resistanceDrug Resistance Multiple BacterialGenomic islandbacterial pathogensmedicineHumansPseudomonas InfectionsPathogenGenome size[SDV.MP] Life Sciences [q-bio]/Microbiology and ParasitologyResearch Articles030304 developmental biology0303 health sciencesoutbreak030306 microbiologyPseudomonas aeruginosahigh-risk cloneOutbreakBayes TheoremSequence Analysis DNAGeneral MedicineHospitals3. Good healthPhenotype[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyPseudomonas aeruginosa
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Land area fractions and population fractions exposed to extreme climate impact events derived from ISIMIP2b output data

2020

This dataset contains the land area fractions and population fractions exposed ('le' for land exposed and 'pe' for population exposed) to the following six extreme climate impact events: crop failures (lec/pec), drought (led/ped), heatwaves (leh/peh), river floods (ler/per), tropical cyclones (let/pet) and wildfire (lew/pew). It is the data behind Lange et al., 2020. The data are provided on a global 0.5° grid and in annual time steps. It was derived from multi-model climate impacts simulations generated within the second round (ISIMIP2b, https://www.isimip.org/protocol/2b, Frieler et al., 2017) of the Intersectoral Impact Model Intercomparison Project (ISIMIP, https://www.isimip.org). The …

EARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > TROPICAL CYCLONESEARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > FAMINEEARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > WILDFIRESEARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > HEATEARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > FLOODSEARTH SCIENCE > HUMAN DIMENSIONS > NATURAL HAZARDS > DROUGHTS
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