Search results for "connective tissue disease"

showing 10 items of 874 documents

microRNA Expression Profile in Single Hormone Receptor-Positive Breast Cancers is Mainly Dependent on HER2 Status—A Pilot Study

2020

Estrogen (ER) and progesterone (PgR) receptors and HER2 are crucial in the assessment of breast cancer specimens due to their prognostic and predictive significance. Single hormone receptor-positive breast cancers are less common and their clinical course is less favorable than ER(+)/PgR(+) tumors. Their molecular features, especially microRNA (miRNA) profiles, have not been investigated to date. Tumor specimens from 36 chemonaive breast cancer patients with known ER and PgR status (18 ER(+)/PgR(&minus

0301 basic medicineendocrine systemmedicine.drug_classClinical BiochemistryEstrogen receptorBiologyArticleprogesterone receptor03 medical and health sciences0302 clinical medicineBreast cancerbreast cancerHER2Progesterone receptormedicineReceptorskin and connective tissue diseaseslcsh:R5-920microRNAMicroRNA Expression Profilemedicine.diseaseFold change030104 developmental biologyEstrogenHormone receptor030220 oncology & carcinogenesisbreast cancer; estrogen receptor; progesterone receptor; HER2; microRNACancer researchlcsh:Medicine (General)hormones hormone substitutes and hormone antagonistsestrogen receptorDiagnostics
researchProduct

Molecular Signatures Associated with Treatment of Triple-Negative MDA-MB231 Breast Cancer Cells with Histone Deacetylase Inhibitors JAHA and SAHA

2017

Jay Amin Hydroxamic Acid (JAHA; N8-ferrocenylN1-hydroxy-octanediamide) is a ferrocene-containing analogue of the histone deacetylase inhibitor (HDACi) suberoylanilide hydroxamic acid (SAHA). JAHA’s cytotoxic activity on MDA-MB231 triple negative breast cancer (TNBC) cells at 72 h has been previously demonstrated with an IC50 of 8.45 M. JAHA’s lethal effect was found linked to perturbations of cell cycle, mitochondrial activity, signal transduction and autophagy mechanisms. In order to glean novel insights on how MDA-MB231 breast cancer cells respond to the cytotoxic effect induced by JAHA, and to compare the biological effect with the related compound SAHA, we have employed a combination of…

0301 basic medicinemedicine.drug_classAntineoplastic AgentsTriple Negative Breast NeoplasmsBiologyHydroxamic AcidsToxicologyStructure-Activity Relationship03 medical and health sciences0302 clinical medicineCell Line TumormedicineHumansCytotoxic T cellFerrous CompoundsSettore BIO/06 - Anatomia Comparata E Citologiaskin and connective tissue diseasesVorinostatTriple-negative breast cancerVorinostatDose-Response Relationship DrugHistone deacetylase inhibitorComputational BiologyGeneral MedicineTriple Negative Breast NeoplasmsCell cycleHistone Deacetylase InhibitorsSettore BIO/18 - Genetica030104 developmental biologyBiochemistryCell culture030220 oncology & carcinogenesisCancer researchHistone deacetylaseJAHA Comet assay MDA-MB231 Histone Deacetylase InhibitorsDrug Screening Assays Antitumormedicine.drug
researchProduct

Mechanisms underlying the influence of oestrogen on cardiovascular physiology in women.

2019

Women show a lower incidence of cardiovascular diseases than age-matched men, but this benefit disappears after menopause. Oestrogen-mediated vascular actions are mainly attributed to oestradiol and exerted by oestrogen receptors (ERα, ERβ and G protein-coupled oestrogen receptor), through rapid and/or genomic mechanisms, but these effects depend on ageing and inflammation. A cardiovascular approach in women's health has arisen due to controversy regarding oestrogen's beneficial impact as reported in experimental and observational studies and large randomized trials. These can be explained, in part, by two mutually non-exclusive hypotheses. On the one hand, the timing hypothesis, which stat…

0301 basic medicinemedicine.medical_specialtyEndotheliumPhysiologyMedicinaEstrogen receptorInflammationProstacyclin03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansEndothelial dysfunctionReceptorskin and connective tissue diseasesMolecular Structurebusiness.industryEstrogen Receptor alphaEstrogensmedicine.diseaseCardiovascular physiologyPostmenopause030104 developmental biologymedicine.anatomical_structureEndocrinologyAgeingFemaleEndothelium Vascularmedicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Journal of physiologyReferences
researchProduct

Interstitial lung disease in systemic sclerosis: current and future treatment.

2017

Systemic sclerosis (SSc) has the highest fatality rate among connective tissue diseases and is characterized by vascular damage, inflammation and fibrosis of the skin and various internal organs. Interstitial lung disease (ILD) frequently complicates SSc and can be a debilitating disorder with a poor prognosis. ILD is the most frequent cause of death in SSc, and the management of SSc–ILD patients is a great challenge. Early detection of pulmonary involvement based on a recent decline of lung function tests and on the extent of lung involvement at high-resolution computed tomography is critical for the best management of these patients. This article summarizes classification, pathogenesis,…

0301 basic medicinemedicine.medical_specialtyPathologyFibrosiImmunologyInterstitial lung diseaseSclerodermaPulmonary function testingSclerodermaPathogenesis03 medical and health sciencesSystemic sclerosi0302 clinical medicineRheumatologyFibrosisPredictive Value of TestsRisk FactorsInternal medicineCase fatality ratemedicineAnimalsHumansImmunology and AllergyMolecular Targeted Therapyskin and connective tissue diseasesLungCause of death030203 arthritis & rheumatologyScleroderma Systemicintegumentary systembusiness.industryInterstitial lung diseaseHematopoietic Stem Cell Transplantationrespiratory systemmedicine.diseaseRheumatologyRespiratory Function Testsrespiratory tract diseasesTreatmentSettore MED/16 - Reumatologia030104 developmental biologyEarly DiagnosisTreatment OutcomebusinessLung Diseases InterstitialTomography X-Ray ComputedImmunosuppressive AgentsLung Transplantation
researchProduct

Tissue microenvironment dictates the fate and tumor-suppressive function of type 3 ILCs

2017

Nussbaum et al. found that tumor suppression through innate lymphoid cells (ILCs) cannot be predicted solely based on the ILC phenotype and lineage but that their immune properties are shaped both by their ontogeny and by the tissue microenvironment they reside in.

0301 basic medicinemedicine.medical_treatmentImmunology314610 Medicine & healthBiology10263 Institute of Experimental ImmunologyArticle31103 medical and health sciencesMiceRAR-related orphan receptor gammaCell Line TumormedicineImmunology and AllergyAnimalsLymphocytesskin and connective tissue diseasesTranscription factorResearch ArticlesMice Knockout2403 ImmunologyInnate lymphoid cellNeoplasms ExperimentalNuclear Receptor Subfamily 1 Group F Member 3PhenotypeCell biologybody regionsKiller Cells NaturalMice Inbred C57BL030104 developmental biologyCytokineCellular MicroenvironmentCell cultureTumor progressionInterleukin 122723 Immunology and AllergyCytokines570 Life sciences; biologyTranscription Factors
researchProduct

New insights into the pathogenesis of giant cell arteritis

2017

Giant cell arteritis (GCA) is an inflammatory chronic disease occurring exclusively in elderly individuals. Until recently, the disease has been considered a unique disease resulting from the interaction in the walls of susceptible arteries, between an unknown infectious agents with local dendritic cells (DCs), activated CD4 T cells and effector macrophages. Recent evidence has shown that this view was too simplistic and has clarified many of the pathogenetic aspects of the disease. Many genetic studies recently published have identified different new genes, including cytokines, adhesion molecules and regulators of innate immunity, as crucial players in the development and progression of GC…

030203 arthritis & rheumatology0301 basic medicineImmunology and Allergy; ImmunologyInnate immune systemGiant Cell ArteritisImmunologyContext (language use)DiseaseBiologymedicine.diseasePathogenesisSettore MED/16 - Reumatologia03 medical and health sciencesGiant cell arteritis030104 developmental biology0302 clinical medicineImmune systemLymphatic systemAntigenImmunologymedicineAnimalsHumansImmunology and Allergyskin and connective tissue diseases
researchProduct

AB0691 INCREASED CARDIOVASCULAR RISK IN MIXED CONNECTIVE TISSUE DISEASE: EVALUATION OF MACROVASCULAR INVOLVEMENT AND ITS PREDICTORS BY AORTIC PULSE W…

2019

Background: Macrovascular involvement and cardiovascular (CV) risk have not been sufficiently studied in patients with mixed connective tissue disease (MCTD). In particular, the gold standard assessment method of aortic stiffness carotid-femoral pulse wave velocity (cfPWV) (1) has never been evaluated in patients with this disease. Objectives: Aims of the present study were to examine cfPWV in MCTD and to evaluate its associations with MCTD associated parameters and traditional CV risk factors. Methods: cfPWV measurements were performed in 43 MCTD patients and 107 healthy controls. The difference between cfPWV in the two groups was statistically examined and subsequently controlled for the …

030203 arthritis & rheumatology0301 basic medicinemedicine.medical_specialtybusiness.industryGold standardConfoundingmedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicineMixed connective tissue diseaseBlood pressureInternal medicineAssessment methodsmedicineCardiologyAortic stiffnessIn patientbusinessPulse wave velocityAbstracts accepted for Publication
researchProduct

Role of MUC1 in idiopathic pulmonary fibrosis: mechanistic insights

2017

Background: Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and irreversible form of fibrotic interstitial lung disease. MUC1, a membrane-bound O-glycoprotein, is considered as oncogenic molecule by altering signaling pathways involved in cellular processes related to IPF. In previous studies we have observed an up-regulation of MUC1 and its phosphorylated forms in IPF lung tissue. However the exact participation of MUC1 in IPF is currently unknown. Objective: To analyze the mechanism of MUC1-induced lung fibrosis in different cellular and animal models of IPF. Methods: The intracellular mechanism of MUC1 was evaluated by western blot, immunoprecipation and immunofluorescence …

030204 cardiovascular system & hematologyImmunofluorescence03 medical and health sciencesIdiopathic pulmonary fibrosis0302 clinical medicineWestern blot0502 economics and businessmedicineskin and connective tissue diseasesFibroblastneoplasmsMUC1Lungmedicine.diagnostic_testbusiness.industry05 social sciencesInterstitial lung diseaseWild typerespiratory systemmedicine.diseasedigestive system diseasesrespiratory tract diseasesmedicine.anatomical_structureCancer research050211 marketingbusinessDiffuse Parenchymal Lung Disease
researchProduct

Responsiveness and Minimal Clinically Important Difference of the Five Times Sit-to-Stand Test in Patients with Stroke

2021

This study aimed to analyze the responsiveness of the 5STS test among stroke patients and to estimate the MCIDs (minimal clinically important differences) for different severity levels of community ambulation and stages of recovery. The 5STS and comparator instruments (gait speed and Functional Ambulatory Category (FAC)) were evaluated at baseline. These measures were repeated at 4 (Stage 1) and 8 weeks (Stage 2), together with the Global Rating of Change (GROC). The MCIDs were calculated with two anchor-based methods using the GROC as the external criterion. Responsiveness to change for the 5STS was estimated analyzing the correlation with changes in the two comparator instruments and thei…

030506 rehabilitationmedicine.medical_specialtyresponsivenessHealth Toxicology and MutagenesisMinimal Clinically Important Differencelcsh:MedicineWalkingMCIDArticleCorrelation03 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationmedicineHumansIn patientstagesFACskin and connective tissue diseasesStrokebusiness.industryallergologyMinimal clinically important differencelcsh:RPublic Health Environmental and Occupational HealthSit to stand testmedicine.disease5STSstrokeGait speedGlobal Ratingseverity levelAmbulatorysense organs0305 other medical sciencebusinessgait speed030217 neurology & neurosurgeryInternational Journal of Environmental Research and Public Health
researchProduct

Intensified forestry as a climate mitigation measure alters surface water quality in low intensity managed forests

2020

Climate change has led to a focus on forest management techniques to increase carbon (C) sequestration as a mitigation measure. Fertilisation and increased removal of biomass have been proposed. But these and other forest practices may have undesirable effects on surface water quality. In naturally acid-sensitive areas such as much of Fennoscandia a concern is acidification due to acid deposition in combination with forest practices that increase the removal of base cations and leaching of nitrate (NO3). Here we apply the biogeochemical model MAGIC to the coniferous-forested catchment at Birkenes, southernmost Norway, to simulate the effects of forest fertilisation and harvest on soil and s…

040101 forestry010504 meteorology & atmospheric sciencesForest managementBiomassClimate changeForestry04 agricultural and veterinary sciences01 natural sciencesEnvironmental protectionSurface water qualityVDP::Matematikk og Naturvitenskap: 400::Basale biofag: 4700401 agriculture forestry and fisheriesEnvironmental sciencesense organsWater qualityskin and connective tissue diseasesIntensity (heat transfer)0105 earth and related environmental sciences
researchProduct