Search results for "disease model"

showing 10 items of 1116 documents

The anti-inflammatory fungal compound (S)-curvularin reduces proinflammatory gene expression in an in vivo model of rheumatoid arthritis.

2012

In previous studies, we identified the fungal macrocyclic lactone (S)-curvularin (SC) as an anti-inflammatory agent using a screening system detecting inhibitors of the Janus kinase/signal transducer and activator of transcription pathway. The objective of the present study was to investigate whether SC is able to decrease proinflammatory gene expression in an in vivo model of a chronic inflammatory disease. Therefore, the effects of SC and dexamethasone were compared in the model of collagen-induced arthritis (CIA) in mice. Total genomic microarray analyses were performed to identify SC target genes. In addition, in human C28/I2 chondrocytes and MonoMac6 monocytes, the effect of SC on proi…

ArthritisMice TransgenicBiologyProinflammatory cytokineArthritis RheumatoidMiceIn vivomedicineAnimalsHumansCells CulturedCell Line TransformedPharmacologyRegulation of gene expressionAnti-Inflammatory Agents Non-SteroidalCurvularinmedicine.diseaseCompound sDisease Models AnimalGene Expression RegulationMice Inbred DBAImmunologyCancer researchSTAT proteinMolecular MedicineZearalenoneInflammation MediatorsJanus kinaseThe Journal of pharmacology and experimental therapeutics
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A Simple Cardiovascular Model for the Study of Hemorrhagic Shock

2020

Hemorrhagic shock is the number one cause of death on the battlefield and in civilian trauma as well. Mathematical modeling has been applied in this context for decades; however, the formulation of a satisfactory model that is both practical and effective has yet to be achieved. This paper introduces an upgraded version of the 2007 Zenker model for hemorrhagic shock termed the ZenCur model that allows for a better description of the time course of relevant observations. Our study provides a simple but realistic mathematical description of cardiovascular dynamics that may be useful in the assessment and prognosis of hemorrhagic shock. This model is capable of replicating the changes in mean …

Article SubjectSwineComputer sciencemedia_common.quotation_subjectComputer applications to medicine. Medical informaticsR858-859.7Context (language use)Cardiovascular ModelShock HemorrhagicExperimental laboratorySettore ING-INF/01 - ElettronicaGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineBattlefieldHemorrhagic ShockmedicineAnimalsHumansComputer Simulation030212 general & internal medicineSimplicitySettore MAT/07 - Fisica MatematicaSimple (philosophy)media_commonMathematical modelsGeneral Immunology and MicrobiologyApplied MathematicsHemodynamicsModels CardiovascularComputational Biology030208 emergency & critical care medicineMathematical ConceptsGeneral MedicinePrognosisAnimal modelsDisease Models AnimalMilitary PersonnelRisk analysis (engineering)Modeling and SimulationShock (circulatory)Settore ING-INF/06 - Bioingegneria Elettronica E InformaticaTime courseHemorrhagic shockCardiovascular Dynamicsmedicine.symptomResearch Article
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Deferiprone and idebenone rescue frataxin depletion phenotypes in a Drosophila model of Friedreich's ataxia

2013

Friedreich's ataxia (FRDA), the most common inherited ataxia, is a neurodegenerative disease caused by a reduction in the levels of the mitochondrial protein frataxin, the function of which remains a controversial matter. Several therapeutic approaches are being developed to increase frataxin expression and reduce the intramitochondrial iron aggregates and oxidative damage found in this disease. In this study, we tested separately the response of a Drosophila RNAi model of FRDA ( Llorens et al., 2007) to treatment with the iron chelator deferiprone (DFP) and the antioxidant idebenone (IDE), which are both in clinical trials. The FRDA flies have a shortened life span and impaired motor coord…

AtaxiaPyridonesUbiquinoneIronLife spanHyperoxiaBiologyPharmacologyMitochondrionmedicine.disease_causeAconitaseAntioxidantsAconitasechemistry.chemical_compoundIron-Binding ProteinsGeneticsmedicineAnimalsIdebenoneDeferiproneAconitate HydrataseHyperoxiaFrataxinClimbing capabilityGeneral MedicineMitochondriaDisease Models AnimalOxidative StressPhenotypechemistryFriedreich AtaxiaOxidative stressMutationFrataxinbiology.proteinDrosophilamedicine.symptomDeferiproneOxidative stressmedicine.drugGene
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Effects of cimetidine, atropine and prostaglandin E2 on rat mucosal erosions produced by intragastric distension

1980

Abstract The effects of three typical antisecretory agents: cimetidine, atrophine and prostaglandin E2 were compared on an acute rat gastric ulcer model which consisted of perfusing the stomach continuously, at a high intraluminal pressure (120 mm H2O), with a simulated gastric juice (0.1 M HCl plus 600 mg pepsin/1). As the acid and pepsin are given exogenously the inhibitory action of the antisecretory drugs is obviated in this model. Cimetidine and atropine failed to reduce gastric erosions, whereas prostaglandin E2 markedly reduced the severity of the mucosal lesions with respect to control values. Long-term treatment with cimetidine also failed to increase the resistance of the gastric …

AtropineMalemedicine.medical_treatmentPharmacologyGuanidinesPepsinmedicineGastric mucosaAnimalsStomach UlcerProstaglandin E2CimetidinePharmacologyGastric Juicebiologybusiness.industryProstaglandins EGastric distensionStomachdigestive oral and skin physiologyRatsDisease Models AnimalAtropinemedicine.anatomical_structureAnesthesiabiology.proteinmedicine.symptomCimetidinebusinessProstaglandin Emedicine.drugEuropean Journal of Pharmacology
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The brainstem efferent acoustic chiasm in pigmented and albino rats

2015

Abstract The present study examined whether structural peculiarities in the brain-efferent pathway to the organ of Corti may underlie functional differences in hearing between pigmented and albino individuals of the same mammalian species. Pigmented Brown-Norway rats and albino Wistar rats received unilateral injections of an aqueous solution of the retrograde neuronal tracer Fluorogold (FG) into the scala tympani of the cochlea to identify olivocochlear neurons (OCN) in the brainstem superior olivary complex. After five days, brains were perfusion-fixed and brainstem sections were cut and analyzed with respect to retrogradely labeled neurons. Intrinsic neurons of the lateral system were lo…

Auditory PathwaysStilbamidinesAlbinismEfferentOlivary NucleusBiologyInjections03 medical and health sciences0302 clinical medicineRats Inbred BNotorhinolaryngologic diseasesmedicineAnimalsTrapezoid bodyRats Wistar030223 otorhinolaryngologyCochlear NerveOrgan of CortiNeuronal Tract-TracersCochleaAnatomySensory SystemsCochleaNeuroanatomical Tract-Tracing TechniquesNeuronal tracingDisease Models Animalmedicine.anatomical_structureOrgan of CortiSuperior olivary complexsense organsBrainstemNucleus030217 neurology & neurosurgeryBrain StemHearing Research
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The matricellular protein SPARC supports follicular dendritic cell networking toward Th17 responses.

2011

Abstract Lymphnode swelling during immune responses is a transient, finely regulated tissue rearrangement, accomplished with the participation of the extracellular matrix. Here we show that murine and human reactive lymph nodes express SPARC in the germinal centres. Defective follicular dendritic cell networking in SPARC-deficient mice is accompanied by a severe delay in the arrangement of germinal centres and development of humoral autoimmunity, events that are linked to Th17 development. SPARC is required for the optimal and rapid differentiation of Th17 cells, accordingly we show delayed development of experimental autoimmune encephalomyelitis whose pathogenesis involves Th17. Not only h…

Autoimmune diseases; Extracellular matrix; Germinal centre reaction; Th17 cellsEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisImmunologyCell CommunicationBiologyfollicular dendritic cellExtracellular matrixAnimals Genetically ModifiedMiceImmune systemSPARC; follicular dendritic cell; Th17Autoimmune diseasemedicinegerminal centre reactionImmunology and AllergyAnimalsHumansautoimmune diseasesOsteonectinMice KnockoutB-LymphocytesCD40Follicular dendritic cellsExperimental autoimmune encephalomyelitisMatricellular proteinGerminal centerSPARCCell Differentiationmedicine.diseaseCell biologyExtracellular MatrixImmunity HumoralMice Inbred C57BLCrosstalk (biology)Disease Models AnimalImmunologybiology.proteinDisease ProgressionTh17 CellsImmunizationMyelin-Oligodendrocyte GlycoproteinTh17autoimmune diseases; extracellular matrix; germinal centre reaction; th17 cellsDendritic Cells FollicularMyelin ProteinsJournal of autoimmunity
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A novel function of Huntingtin in the cilium and retinal ciliopathy in Huntington's disease mice

2015

Huntington's disease (HD) is a neurodegenerative disorder caused by the toxic expansion of polyglutamine in the Huntingtin (HTT) protein. The pathomechanism is complex and not fully understood. Increasing evidence indicates that the loss of normal protein function also contributes to the pathogenesis, pointing out the importance of understanding the physiological roles of HTT. We provide evidence for a novel function of HTT in the cilium. HTT localizes in diverse types of cilia — including 9 + 0 non-motile sensory cilia of neurons and 9 + 2 motile multicilia of trachea and ependymal cells — which exert various functions during tissue development and homeostasis. In the photoreceptor cilium,…

AxonemeMalecongenital hereditary and neonatal diseases and abnormalitiesHuntingtinCentrioleMice TransgenicNerve Tissue ProteinsBiologyMicrotubulesPhotoreceptor cellRetinalcsh:RC321-571MiceHuntington's diseaseIntraflagellar transportmental disordersmedicineAnimalsHumansPhotoreceptor CellsHuntingtinCilialcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSHuntingtin ProteinPhotoreceptorCiliumNuclear ProteinsHuntington's diseasemedicine.diseaseCell biologyCiliopathyDisease Models Animalmedicine.anatomical_structureHEK293 CellsHuntington DiseaseNeurologyFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]sense organs
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Role of oxygen and nitrogen species in experimental uveitis: anti-inflammatory activity of the synthetic antioxidant ebselen.

2002

This study was aimed at examining the role of oxygen and nitrogen reactive species in a model of experimental uveitis upon intravitreal injection of bacterial endotoxin to albino New Zealand rabbits. The inflammatory response was evaluated in terms of: (i) the integrity of the blood aqueous barrier (protein and cell content in samples of aqueous humor), (ii) histopathological changes of the eyes, (iii) clinical evaluation (with a score index based on clinical symptoms), and (iv) the concentration of malondialdehyde (MDA), in aqueous humor, as a marker of oxidative stress. Betamethasone was used as reference treatment, superoxide dismutase as quencher of superoxide anion, L-N(G)-nitro-L-argi…

AzolesFree RadicalsChlorpromazineAnti-Inflammatory AgentsPharmacologyIsoindolesmedicine.disease_causeBiochemistryAntioxidantsSuperoxide dismutaseUveitischemistry.chemical_compoundPhysiology (medical)MalondialdehydeOrganoselenium CompoundsmedicineAnimalsEnzyme InhibitorsReactive nitrogen specieschemistry.chemical_classificationReactive oxygen speciesbiologyChemistrySuperoxideEbselenSuperoxide DismutaseGlutathione peroxidaseMalondialdehydeReactive Nitrogen SpeciesDisease Models AnimalNG-Nitroarginine Methyl EsterBiochemistrybiology.proteinLipid PeroxidationRabbitsNitric Oxide SynthaseReactive Oxygen SpeciesOxidative stressFree radical biologymedicine
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Invasion Ability and Disease Dynamics of Environmentally Growing Opportunistic Pathogens under Outside-Host Competition

2014

Most theories of the evolution of virulence concentrate on obligatory host-pathogen relationship. Yet, many pathogens replicate in the environment outside-host where they compete with non-pathogenic forms. Thus, replication and competition in the outside-host environment may have profound influence on the evolution of virulence and disease dynamics. These environmentally growing opportunistic pathogens are also a logical step towards obligatory pathogenicity. Efficient treatment methods against these diseases, such as columnaris disease in fishes, are lacking because of their opportunist nature. We present a novel epidemiological model in which replication and competition in the outside-hos…

Bacterial Diseases0106 biological sciencesPopulation ModelingDisease01 natural sciencesTheoretical EcologyMedicine and Health SciencesPathogenPOPULATIONmedia_common0303 health scienceseducation.field_of_studyPREDATIONMultidisciplinaryEcologyTransmission (medicine)EcologySimulation and ModelingQRCHANNEL CATFISHEVOLUTIONARY DYNAMICSCOMMUNITYInfectious DiseasesHost-Pathogen Interactions1181 Ecology evolutionary biologyMedicineAlgorithmsResearch ArticleTRANSMISSIONSciencemedia_common.quotation_subjecteducationPopulationVirulenceOpportunistic InfectionsBiologyResearch and Analysis Methods010603 evolutionary biologyCompetition (biology)03 medical and health sciences14. Life underwaterParasite EvolutioneducationEvolutionary dynamicsta413030304 developmental biologyEvolutionary BiologyMathematical ModelingSTABILITYMORTALITYEcology and Environmental SciencesBiology and Life SciencesComputational BiologyFLAVOBACTERIUM-COLUMNAREOutbreakModels TheoreticalEmerging Infectious DiseasesEvolutionary Ecologyta1181VIRULENCEParasitologyInfectious Disease ModelingPLoS ONE
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Bile acid receptor TGR5 is critically involved in preference for dietary lipids and obesity

2020

International audience; We investigated the implication of Takeda G protein-coupled receptor 5 (TGR5) in fat preference and fat sensing in taste bud cells (TBC) in C57BL/6 wild-type (WT) and TGR5 knock out (TGR5-/-) male mice maintained for 20 weeks on a high-fat diet (HFD). We also assessed the implication of TGR5 single nucleotide polymorphism (SNP) in young obese humans. The high-fat diet (HFD)-fed TGR5-/- mice were more obese, marked with higher liver weight, lipidemia and steatosis than WT obese mice. The TGR5-/- obese mice exhibited high daily food/energy intake, fat mass and inflammatory status. WT obese mice lost the preference for dietary fat, but the TGR5-/- obese mice exhibited n…

Blood GlucoseLipopolysaccharidesMale0301 basic medicine[SDV]Life Sciences [q-bio]Endocrinology Diabetes and MetabolismClinical BiochemistryBiochemistryReceptors G-Protein-CoupledMice0302 clinical medicineInsulinReceptorMice Knockout2. Zero hungerchemistry.chemical_classificationNutrition and DieteticsLipidsG protein-coupled bile acid receptor[SDV] Life Sciences [q-bio]medicine.anatomical_structuremedicine.medical_specialtyMice Transgenic030209 endocrinology & metabolismSingle-nucleotide polymorphismDiet High-FatPolymorphism Single NucleotideBile Acids and SaltsFood Preferences03 medical and health sciencesInternal medicineTaste budmedicineAnimalsObesityMolecular BiologyInflammationbusiness.industryTaste budFatty acidFatty acidmedicine.diseaseDietary FatsObesityIn vitroFatty LiverMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologychemistryFatCalciumSteatosisbusinessThe Journal of Nutritional Biochemistry
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