Search results for "eficiency"

showing 10 items of 1074 documents

Positive Iron Balance in Chronic Kidney Disease: How Much is Too Much and How to Tell?

2017

<b><i>Background:</i></b> Regulation of body iron occurs at cellular, tissue, and systemic levels. In healthy individuals, iron absorption and losses are minimal, creating a virtually closed system. In the setting of chronic kidney disease and hemodialysis (HD), increased iron losses, reduced iron absorption, and limited iron availability lead to iron deficiency. Intravenous (IV) iron therapy is frequently prescribed to replace lost iron, but determining an individual’s iron balance and stores can be challenging and imprecise, contributing to uncertainty about the long-term safety of IV iron therapy. <b><i>Summary:</i></b> Patients on HD recei…

Iron030232 urology & nephrologyPhysiology030204 cardiovascular system & hematologyDirect reduced iron03 medical and health sciences0302 clinical medicineHepcidinmedicineHomeostasisHumansErythropoiesisRenal Insufficiency ChronicHemochromatosischemistry.chemical_classificationbiologybusiness.industryIron deficiencymedicine.diseaseTrace ElementschemistryNephrologyTransferrinToxicitybiology.proteinErythropoiesisAdministration IntravenousbusinessKidney diseaseAmerican journal of nephrology
researchProduct

A genome-wide transcriptional study reveals that iron deficiency inhibits the yeast TORC1 pathway

2019

Iron is an essential micronutrient that participates as a cofactor in a broad range of metabolic processes including mitochondrial respiration, DNA replication, protein translation and lipid biosynthesis. Adaptation to iron deficiency requires the global reorganization of cellular metabolism directed to optimize iron utilization. The budding yeast Saccharomyces cerevisiae has been widely used to characterize the responses of eukaryotic microorganisms to iron depletion. In this report, we used a genomic approach to investigate the contribution of transcription rates to the modulation of mRNA levels during adaptation of yeast cells to iron starvation. We reveal that a decrease in the activity…

IronSaccharomyces cerevisiaeBiophysicsRibosome biogenesisSaccharomyces cerevisiaeMechanistic Target of Rapamycin Complex 1Biochemistry03 medical and health sciencesStructural BiologyRibosomal proteinTranscription (biology)Gene Expression Regulation FungalLipid biosynthesisGeneticsHumansRNA MessengerPhosphorylationMolecular BiologyGene030304 developmental biology0303 health sciencesAnemia Iron-Deficiencybiology030306 microbiologyChemistryIron deficiencyRNA polymerasesRNATORbiology.organism_classificationAdaptation PhysiologicalYeastCell biologyDNA-Binding ProteinsGene Expression RegulationProtein BiosynthesisSignal transductionTranscription
researchProduct

The elemental role of iron in DNA synthesis and repair

2017

Iron is an essential redox element that functions as a cofactor in many metabolic pathways. Critical enzymes in DNA metabolism, including multiple DNA repair enzymes (helicases, nucleases, glycosylases, demethylases) and ribonucleotide reductase, use iron as an indispensable cofactor to function. Recent striking results have revealed that the catalytic subunit of DNA polymerases also contains conserved cysteine-rich motifs that bind iron–sulfur (Fe/S) clusters that are essential for the formation of stable and active complexes. In line with this, mitochondrial and cytoplasmic defects in Fe/S cluster biogenesis and insertion into the nuclear iron-requiring enzymes involved in DNA synthesis a…

Iron-Sulfur Proteins0301 basic medicineDNA RepairDNA polymeraseDNA damageDNA repairIronBiophysicsDNA repairEukaryotic DNA replicationSaccharomyces cerevisiaeBiochemistryDNA GlycosylasesBiomaterials03 medical and health sciencesRibonucleotide ReductasesHumansProtein–DNA interactionRibonucleotide reductaseReplication protein Achemistry.chemical_classificationDNA ligaseDeoxyribonucleasesDNA synthesis030102 biochemistry & molecular biologybiologyIron deficiencyDNA HelicasesMetals and AlloysHelicaseDNAYeast030104 developmental biologyIron cofactorBiochemistrychemistryChemistry (miscellaneous)biology.proteinIron-sulfur clusterMetallomics
researchProduct

Acute pancreatitis in children. An Italian multicentre study

2002

Abstract Aim . To evaluate the clinical, morphological and aetiological aspects of acute pancreatitis in children in Italy. Patients . The hospital records of 50 consecutive patients with acute pancreatitis observed in 5 Italian Pediatric Departments were reviewed. Results . A total of 25 males and 25 females (median age 10.5 years, range 2–17) were studied. Of these patients, 48 (96%) had abdominal pain. The pancreatitis was associated with biliary disease in 10 patients (20%); it was due to viral infection in 6 patients (12%), pancreatic duct abnormalities in 4 (8%), familial chronic pancreatitis in 3 (6%), trauma in 5 (10%) and other causes in 5 (10%); the pancreatitis was of unknown ori…

Italy/epidemiologyMalemedicine.medical_specialtyAbdominal painURLSettore MED/09 - Medicina Internaacute pancreatitisBUNupper reference limitDiseasePancreatitis/epidemiologyGastroenterologyC-reactive proteinBiliary diseaseSettore MED/38 - Pediatria Generale E SpecialisticaInternal medicineMedicineHumansPancreatitis/etiologyChildblood urea nitrogenRetrospective StudiesPancreatic ducthuman immunodeficiency virusHepatologybiologybusiness.industryC-reactive proteinGastroenterologyHIVAcute Disease Child Female Humans Italy/epidemiology Male Pancreatitis/diagnosis Pancreatitis/epidemiology* Pancreatitis/etiology Retrospective Studiesacute pancreatitisBUN; blood urea nitrogen; CRP; C-reactive protein; HIV; human immunodeficiency virus; URL; upper reference limitmedicine.diseaseSurgerymedicine.anatomical_structureItalyPancreatitisAcute DiseaseEtiologybiology.proteinAcute pancreatitisPancreatitisFemalemedicine.symptomCRPbusinessPancreatitis/diagnosiHuman
researchProduct

Structural differences of prebiotic oligosaccharides influence their capability to enhance iron absorption in deficient rats

2014

This study evaluates the influence of novel galacto-oligosaccharides derived from lactulose (GOS-Lu), kojibiose or 4′-galactosyl-kojibiose in hematological parameters of Fe homeostasis using Fe-deficient animals. Liver TfR-2, IL-6, NFκB and PPAR-γ expression (mRNA) were also determined by RT-qPCR analyses, and active hepcidin peptide production and short chain fatty acids by LC coupled to MS/MS or UV detection. Feeding animals with GOS-Lu or kojibiose together with FeCl3 increased hemoglobin (Hb) production (by 17%) and mean Hb concentration into erythrocytes relative to animals administered with FeCl3 alone (14.1% and 19.7%, respectively). Animals administered with prebiotics showed decrea…

Kojibiosemedicine.medical_treatmentPeptideAbsorption (skin)Ferric CompoundsIntestinal absorptionHemoglobinschemistry.chemical_compoundLactuloseChloridesHepcidinsTandem Mass SpectrometryHepcidinReceptors TransferrinmedicineAnimalsHomeostasisMicronutrientsRNA MessengerRats Wistarchemistry.chemical_classificationAnemia Iron-DeficiencybiologyInterleukin-6ChemistryPrebioticNF-kappa BGeneral MedicineFatty Acids VolatileRatsPPAR gammaDisease Models AnimalPrebioticsIntestinal AbsorptionLiverBiochemistryDietary Supplementsbiology.proteinFemaleTrisaccharidesIron DietaryHomeostasisFood Sciencemedicine.drugFood Funct.
researchProduct

HIV/HCV co-infection: putting the pieces of the puzzle together

2003

The most relevant consequence of human immunodeficiency virus (HIV) infection is the exposure of the patient to opportunistic agents. In addition, HIV may also modify the natural history of some non-opportunistic pathogens, which in turn may alter the course of HIV infection.

Kupffer CellsHuman immunodeficiency virus (HIV)HIV InfectionsBiologymedicine.disease_causeTh2 CellsCytokines metabolismImmunityparasitic diseasesmedicineAnimalsHumansMolecular BiologyLiver immunologyImmunity Cellularvirus diseasesCell BiologyHepatitis Cmedicine.diseaseFibrosisHepatitis CVirologyNatural historyLiverModels AnimalImmunologyCytokinesCo infectionCell Death & Differentiation
researchProduct

Recombinant activated factor VII administration in a patient with congenital lack of factor VII undergoing laparoscopic hysterectomy: A case report

2021

Highlights • Patients with lack of factor VIIa, suffering from abnormal uterine bleedings can be treated with laparoscopic hysterectomy. • Technique using bipolar coagulation and non-absorbable clips is safe and not time consuming. • Pre-operative loading with recombinant factor VIIa must be followed by prolonged supplementation in post-operative period. • We present suggested dosage and length of factor VIIa supplementation based on literature review and own experience.

Laparoscopic surgerymedicine.medical_specialtymedicine.medical_treatmentHysterectomySurgical bleeding03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCase reportmedicineotorhinolaryngologic diseasesCLIPSLaparoscopycomputer.programming_languageHysterectomyFactor VIImedicine.diagnostic_testbiologybusiness.industrymedicine.diseaseEndometrial hyperplasiaSurgerychemistryRecombinant factor VIIa030220 oncology & carcinogenesisFactor VII deficiencybiology.protein030211 gastroenterology & hepatologySurgeryPremedicationLaparoscopysense organsbusinesscomputercirculatory and respiratory physiologyInternational Journal of Surgery Case Reports
researchProduct

Head and neck squamous cell carcinoma in people living with HIV in France.

2018

Abstract Objectives Head and neck squamous cell carcinoma (HNSCC), mainly due to smoking, is one of the leading causes of cancer deaths. However, an increasing number of tumors – especially oropharyngeal cancer – are reported in non-smokers in association with the human papillomavirus (HPV). As HIV-infected individuals are particularly at risk of HPV-related disease, we aimed to describe the burden of HNSCC in this population. Methods Retrospective chart review of patients from HIV clinics diagnosed with HNSCC between 2004 and 2014. Case patients were defined using the International Classification of Disease for Oncology (3rd edition). Age at HIV diagnosis and time from HIV diagnosis to HNS…

LarynxOncologyMalemedicine.medical_specialtyPopulationHuman immunodeficiency virus (HIV)HIV InfectionsDiseasemedicine.disease_causeCauses of cancer03 medical and health sciences0302 clinical medicineInternal medicineotorhinolaryngologic diseasesCarcinomamedicineHumans030212 general & internal medicineeducationLaryngeal NeoplasmsComputingMilieux_MISCELLANEOUSRetrospective Studieseducation.field_of_studyHypopharyngeal Neoplasmsbusiness.industrySquamous Cell Carcinoma of Head and Neckvirus diseasesCancerMiddle Agedmedicine.diseaseHead and neck squamous-cell carcinoma[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology3. Good healthstomatognathic diseasesInfectious Diseasesmedicine.anatomical_structure030220 oncology & carcinogenesis[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology[SDV.IMM]Life Sciences [q-bio]/ImmunologyFemaleMouth NeoplasmsFrancebusinessMedecine et maladies infectieuses
researchProduct

Leptin and the central control of feeding behavior.

2012

International audience; The discovery of leptin by Friedman and coll. in 1995 was a major step forward in our comprehensive view of energy homeostasis. Since the original paper, a tremendous amount of work has been performed in laboratories all over the world. Many recent reviews have described this work in details. In the present review, we focus on the role of leptin on food intake. It is accepted by most authors working in this field that the control of food intake can be divided in two closely-related system: the homeostatic system and the hedonic system. Leptin has been shown to act on both systems.

LeptinFood intakemedicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionControl (management)BiologyBiochemistryEnergy homeostasisFeeding behaviorInternal medicinemedicineAnimalsHomeostasisHumansNeuronsLeptin DeficiencyLeptindigestive oral and skin physiologyGeneral MedicineFeeding BehaviorEndocrinologyHomeostatic systemNerve Net[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBrain circuitryCognitive psychology
researchProduct

Fasting enhances the response of arcuate neuropeptide Y-glucose-inhibited neurons to decreased extracellular glucose

2009

0363-6143 (Print) Comparative Study In Vitro Journal Article Research Support, N.I.H., Extramural; Fasting increases neuropeptide Y (NPY) expression, peptide levels, and the excitability of NPY-expressing neurons in the hypothalamic arcuate (ARC) nucleus. A subpopulation of ARC-NPY neurons ( approximately 40%) are glucose-inhibited (GI)-type glucose-sensing neurons. Hence, they depolarize in response to decreased glucose. Because fasting enhances NPY neurotransmission, we propose that during fasting, GI neurons depolarize in response to smaller decreases in glucose. This increased excitation in response to glucose decreases would increase NPY-GI neuronal excitability and enhance NPY neurotr…

LeptinMalemedicine.medical_specialtyArcuate Nucleus/cytology/*metabolismPhysiologyGlucose/*deficiencyAMP-Activated Protein Kinases/metabolismAMP-Activated Protein KinasesIn Vitro TechniquesNeurotransmissionBiologySynaptic TransmissionEnergy homeostasisMembrane PotentialsRats Sprague-Dawley03 medical and health sciences0302 clinical medicineNeuropeptide Y/*metabolismArcuate nucleusInternal medicinemental disordersmedicineAnimalsHomeostasisNeuropeptide YNervous System Cell BiologyFasting/*metabolismNeurons/enzymology/*metabolism030304 developmental biologyNeuronsMembrane potential0303 health sciencesLeptinArcuate Nucleus of HypothalamusLeptin/metabolismNeural InhibitionFastingCell BiologyNeuropeptide Y receptorhumanitiesRatsGlucosemedicine.anatomical_structureEndocrinologyNeuronSprague-DawleyEnergy Metabolism030217 neurology & neurosurgeryHomeostasis
researchProduct