Search results for "ferroptosis"

showing 10 items of 13 documents

Artesunate Inhibits Growth of Sunitinib-Resistant Renal Cell Carcinoma Cells through Cell Cycle Arrest and Induction of Ferroptosis

2020

Although innovative therapeutic concepts have led to better treatment of advanced renal cell carcinoma (RCC), efficacy is still limited due to the tumor developing resistance to applied drugs. Artesunate (ART) has demonstrated anti-tumor effects in different tumor entities. This study was designed to investigate the impact of ART (1&ndash

0301 basic medicineCancer ResearchTraditional Chinese Medicine (TCM) growth inhibition ferroptosis reactive oxygen species (ROS)Cell cycle checkpointBiologyurologic and male genital diseasesreactive oxygen species (ROS)lcsh:RC254-282Articlegrowth inhibition03 medical and health scienceschemistry.chemical_compound0302 clinical medicinerenal cell carcinoma (RCC)medicineClonogenic assayCytotoxicityartesunate (ART)SunitinibTraditional Chinese Medicine (TCM)Cell cyclelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensferroptosissunitib resistance030104 developmental biologyOncologychemistryCell cultureApoptosis030220 oncology & carcinogenesisCancer researchGrowth inhibitionmedicine.drugCancers
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General Aspects of Metal Ions as Signaling Agents in Health and Disease

2020

This review focuses on the current knowledge on the involvement of metal ions in signaling processes within the cell, in both physiological and pathological conditions. The first section is devoted to the recent discoveries on magnesium and calcium-dependent signal transduction—the most recognized signaling agents among metals. The following sections then describe signaling pathways where zinc, copper, and iron play a key role. There are many systems in which changes in intra- and extra-cellular zinc and copper concentrations have been linked to important downstream events, especially in nervous signal transduction. Iron signaling is mostly related with its homeostasis. However, it is also …

0301 basic medicineCell signalingProgrammed cell deathMetal ions in aqueous solutionCelllcsh:QR1-502DiseaseReviewBiochemistrySynaptic Transmissionlcsh:Microbiology03 medical and health sciences0302 clinical medicinemedicinecell signaling; metal homeostasis; ferroptosiscell signalingAnimalsFerroptosisHumansMolecular BiologyChemistryFerroptosisCell biology030104 developmental biologymedicine.anatomical_structureMetalsmetal homeostasisSignal transduction030217 neurology & neurosurgeryHomeostasisSignal TransductionBiomolecules
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Comparative Proteomics Unveils LRRFIP1 as a New Player in the DAPK1 Interactome of Neurons Exposed to Oxygen and Glucose Deprivation

2020

Altres ajuts: The group has received funding from 'la Caixa Foundation' CI15-00009, from the European Institute of Innovation and Technology (EIT) PoC-2016-SPAIN-04, which receives support from the European Union's Horizon 2020 research and innovation program, and from the 'Fundación para la Innovación y la Prospectiva en Salud en España (FIPSE)' program 3594-18. Death-associated protein kinase 1 (DAPK1) is a pleiotropic hub of a number of networked distributed intracellular processes. Among them, DAPK1 is known to interact with the excitotoxicity driver NMDA receptor (NMDAR), and in sudden pathophysiological conditions of the brain, e.g., stroke, several lines of evidence link DAPK1 with t…

0301 basic medicinePhysiologyClinical BiochemistryExcitotoxicitymedicine.disease_causeProteomicsBiochemistryInteractomeNeuroprotectionArticle03 medical and health sciences0302 clinical medicinemedicineDAPK1Protein kinase AMolecular Biologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950OGDROSCell BiologyneuronferroptosisCell biology030104 developmental biologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistrynervous systemNMDANeuronLRRFIP1MCAO030217 neurology & neurosurgeryIntracellularAntioxidants
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Oxytosis/Ferroptosis—(Re-) Emerging Roles for Oxidative Stress-Dependent Non-apoptotic Cell Death in Diseases of the Central Nervous System

2018

Although nerve cell death is the hallmark of many neurological diseases, the processes underlying this death are still poorly defined. However, there is a general consensus that neuronal cell death predominantly proceeds by regulated processes. Almost 30 years ago, a cell death pathway eventually named oxytosis was described in neuronal cells that involved glutathione depletion, reactive oxygen species production, lipoxygenase activation, and calcium influx. More recently, a cell death pathway that involved many of the same steps was described in tumor cells and termed ferroptosis due to a dependence on iron. Since then there has been a great deal of discussion in the literature about wheth…

0301 basic medicineProgrammed cell deathCell typebrain diseasesCentral nervous systemReviewoxytosisBiologymedicine.disease_causelcsh:RC321-57103 medical and health sciencesironmedicineoxidative stresslcsh:Neurosciences. Biological psychiatry. Neuropsychiatryprogrammed cell deathchemistry.chemical_classificationReactive oxygen speciesGeneral NeuroscienceFerroptosisBrain Diseases ; Ferroptosis ; Iron ; Oxidative Stress ; Oxytosis ; Programmed Cell Deathferroptosis030104 developmental biologymedicine.anatomical_structurechemistryApoptotic cell deathNeuroscienceCalcium influxOxidative stressNeuroscienceFrontiers in Neuroscience
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A naturally occuring triterpene saponin ardisiacrispin B displayed cytotoxic effects in multi-factorial drug resistant cancer cells via ferroptotic a…

2018

WOS: 000432722700010

0301 basic medicineProgrammed cell deathCytotoxicitySaponinPharmaceutical ScienceApoptosisFlow cytometryCell Cycle Distribution03 medical and health sciencesArdisiacrispin BCell Line TumorDrug DiscoverymedicineFerroptosisHumansCytotoxic T cellOleanolic AcidCytotoxicityCaspaseMembrane Potential MitochondrialPharmacologybiologymedicine.diagnostic_testMitochondrial Membrane PotentialChemistryHep G2 CellsSaponinsHCT116 Cellsmedicine.diseaseAntineoplastic Agents PhytogenicDrug Resistance MultipleLeukemia030104 developmental biologyComplementary and alternative medicineDoxorubicinDrug Resistance NeoplasmApoptosisCaspasesCancer cellbiology.proteinCancer researchMolecular MedicineReactive Oxygen SpeciesPhytomedicine
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The alkaloid, soyauxinium chloride, displays remarkable cytotoxic effects towards a panel of cancer cells, inducing apoptosis, ferroptosis and necrop…

2020

Abstract The cytotoxic potential of a naturally occurring indoloquinazoline alkaloid, soyauxinium chloride (SCHL), was determined on a broad panel of animal and human cancer cell lines, including various sensitive and drug-resistant phenotypes. The cytotoxicity, SCHL-induced autophagic, ferroptotic, and necroptotic cell death were evaluated by the resazurin reduction assay (RRA). Caspase-Glo assay was used to detect the activity of caspases using spectrophotometric analysis. Flow cytometry was applied for cell cycle analysis (PI staining), apoptosis (annexin V/PI staining), mitochondrial membrane potential (MMP) (JC-1) and reactive oxygen species (ROS) (H2DCFH-DA). SCHL and doxorubicin (ref…

0301 basic medicineProgrammed cell deathNecroptosisAntineoplastic AgentsApoptosisToxicology03 medical and health sciences0302 clinical medicineCell Line TumorCytotoxic T cellFerroptosisHumansRegulated Cell DeathCytotoxicityCaspasebiologyChemistryCell CycleGeneral MedicineMolecular biology030104 developmental biologyCell cultureApoptosis030220 oncology & carcinogenesisCancer cellMitochondrial MembranesNecroptosisbiology.proteinReactive Oxygen SpeciesChemico-biological interactions
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Ferroptosis and Its Potential Role in Human Diseases

2020

Ferroptosis is a novel regulated cell death pattern discovered when studying the mechanism of erastin-killing RAS mutant tumor cells in 2012. It is an iron-dependent programmed cell death pathway mainly caused by an increased redox imbalance but with distinct biological and morphology characteristics when compared to other known cell death patterns. Ferroptosis is associated with various diseases including acute kidney injury, cancer, and cardiovascular, neurodegenerative, and hepatic diseases. Moreover, activation or inhibition of ferroptosis using a variety of ferroptosis initiators and inhibitors can modulate disease progression in animal models. In this review, we provide a comprehensiv…

0301 basic medicineProgrammed cell deathReviewdegenerative diseasesBiologyHepatic Diseases03 medical and health sciences0302 clinical medicineRegulated cell deathmedicinePharmacology (medical)Pharmacologyreactive oxygen speciesMechanism (biology)Ferroptosislcsh:RM1-950Disease progressionCancermedicine.diseaseferroptosissignaling pathwayslcsh:Therapeutics. Pharmacology030104 developmental biologypharmacology design030220 oncology & carcinogenesisCancer researchSignal transductionFrontiers in Pharmacology
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Multiple modes of cell death in neuroendocrine tumors induced by artesunate.

2020

Abstract Background The paucity of effective treatment in neuroendocrine tumors (NETs) encouraged us to investigate the therapeutic value of artesunate (ART) promised by its inhibitory effect against various tumors and broad safety profile. Methods We evaluated the impact of ART on three NET cell lines, BON-1, QGP-1 and NCI-H727 on cellular and molecular levels. Results Our results showed that ART induced endoplasmic reticulum (ER) stress through phosphorylation of eIF2α, which further gave rise to autophagy in all three NET cell lines. Specifically, apoptosis and ferroptosis were also observed in BON-1 cells, which made BON-1 cell line more vulnerable upon ART treatment. The different sens…

Cyclin-Dependent Kinase Inhibitor p21NiacinamideProgrammed cell deathPharmaceutical ScienceArtesunateAntineoplastic AgentsApoptosisNeuroendocrine tumorsBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationCell Line TumorDrug DiscoveryAntineoplastic Combined Chemotherapy ProtocolsmedicineAutophagyFerroptosisHumans030304 developmental biologyPharmacology0303 health sciencesEndoplasmic reticulumPhenylurea CompoundsAutophagymedicine.diseaseEndoplasmic Reticulum StressNeuroendocrine TumorsComplementary and alternative medicinechemistryCell cultureApoptosisArtesunate030220 oncology & carcinogenesisCancer researchMolecular MedicinePhytomedicine : international journal of phytotherapy and phytopharmacology
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COVID-19, Cation Dysmetabolism, Sialic Acid, CD147, ACE2, Viroporins, Hepcidin and Ferroptosis: A Possible Unifying Hypothesis.

2022

Background: iron and calcium dysmetabolism, with hyperferritinemia, hypoferremia, hypocalcemia and anemia have been documented in the majority of COVID-19 patients at later/worse stages. Furthermore, complementary to ACE2, both sialic acid (SA) molecules and CD147 proved relevant host receptors for SARS-CoV-2 entry, which explains the viral attack to multiple types of cells, including erythrocytes, endothelium and neural tissue. Several authors advocated that cell ferroptosis may be the core and final cell degenerative mechanism. Methods: a literature research was performed in several scientific search engines, such as PubMed Central, Cochrane Library, Chemical Abstract Service. More than 5…

General Immunology and MicrobiologySARS-CoV-2virusesvirus diseasesCOVID-19Endothelial CellsGeneral Medicinebiochemical phenomena metabolism and nutritionGeneral Biochemistry Genetics and Molecular BiologyN-Acetylneuraminic AcidViroporin ProteinsHepcidinsCationsferroptosis cations sialic acid iron ferritin calcium viroporins voltage-gated calcium channels cell membrane CD147 ACE2 hepcidin red blood cells hemoglobin mitochondriaFerroptosisHumansAngiotensin-Converting Enzyme 2General Pharmacology Toxicology and PharmaceuticsF1000Research
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Ferroptosis Meets Cell–Cell Contacts

2021

Ferroptosis is a regulated form of cell death characterized by iron dependency and increased lipid peroxidation. Initially assumed to be selectively induced in tumour cells, there is increasing evidence that ferroptosis plays an important role in pathophysiology and numerous cell types and tissues. Deregulated ferroptosis has been linked to human diseases, such as neurodegenerative diseases, cardiovascular disorders, and cancer. Along these lines, ferroptosis is a promising pathway to overcoming therapy resistance of cancer cells. It is therefore of utmost importance to understand the cellular signalling pathways and the molecular mechanisms underlying ferroptosis regulation, including cont…

Programmed cell deathCell typeSomatic cellQH301-705.5ReviewCell CommunicationBiologyepithelial–mesenchymal transitionNeoplasmsmedicineAnimalsHumansEpithelial–mesenchymal transitionBiology (General)Calcium signalingFerroptosisCancerNeurodegenerative DiseasesGeneral Medicinecell–cell contactsmedicine.diseaseferroptosisCardiovascular DiseasesCancer cellcancer therapyNeuroscienceCells
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