Search results for "guano"

showing 10 items of 193 documents

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome

2010

Effect of nut consumption on oxidative stress and the endothelial function in metabolic syndrome BACKGROUND & AIMS: Oxidative stress has a key role in atherosclerosis, cancer and other chronic diseases. Some bioactive compounds in nuts have been implicated in antioxidant activities. OBJECTIVE: We assessed how nut consumption affected several markers of oxidation and endothelial function (EF) in metabolic syndrome (MetS) patients. PATIENTS AND METHODS: A randomized, controlled, parallel feeding trial was conducted on 50 MetS adults who were recommended a healthy diet supplemented or not with 30 g of mixed nuts (Nut and Control groups, respectively) every day for 12 weeks. The plasma anti…

MaleAntioxidantmedicine.medical_treatmentantioxidant capacityIsoprostanesCritical Care and Intensive Care Medicinemedicine.disease_causeAntioxidantsLipid peroxidationchemistry.chemical_compoundendothelial functionMedicineNutschemistry.chemical_classificationMetabolic SyndromeBioquímica y tecnologíaNutrition and Dieteticsdigestive oral and skin physiologyfood and beveragesArteriesMiddle Aged0261-5614Lipoproteins LDLBiochemistry and technologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineFatty Acids UnsaturatedPolyunsaturated fatty acidAdultmedicine.medical_specialtyEndotheliumAdolescentDNA damageEstrès oxidatiuFruita seca -- Aspectes nutritiusBioquímica i biotecnologiaYoung AdultLipid oxidationInternal medicineHumansAgedbusiness.industryDeoxyguanosineEndoteli vascularmedicine.diseaseDietOxidative StressEndocrinologychemistryDNA damageVascular ResistanceEndothelium VascularMetabolic syndromebusinessOxidative stressBiomarkers
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Amylase release from streptolysin O-permeabilized pancreatic acinar cells. Effects of Ca2+, guanosine 5'-[gamma-thio]triphosphate, cyclic AMP, tetanu…

1992

The molecular requirements for amylase release and the intracellular effects of botulinum A toxin and tetanus toxin on amylase release were investigated using rat pancreatic acinar cells permeabilized with streptolysin O. Micromolar concentrations of free Ca2+ evoked amylase release from these cells. Maximal release was observed in the presence of 30 microM free Ca2+. Ca(2+)-stimulated, but not basal, amylase release was enhanced by guanosine 5′-[gamma-thio]triphosphate (GTP[S]) (3-4 fold) or cyclic AMP (1.5-2 fold). Neither the two-chain forms of botulinum A toxin and tetanus toxin, under reducing conditions, nor the light chains of tetanus toxin, inhibited amylase release triggered by Ca2…

MaleBotulinum ToxinsCell Membrane PermeabilityClostridium tetanimedicine.disease_causeBiochemistryNorepinephrineBacterial ProteinsTetanus ToxinAcinar cellmedicineCyclic AMPNeurotoxinAnimalsAmylaseMolecular BiologyPancreasbiologyToxinProteolytic enzymesRats Inbred StrainsCell BiologyRatsBiochemistryGuanosine 5'-O-(3-Thiotriphosphate)AmylasesStreptolysinsbiology.proteinClostridium botulinumStreptolysinCalciumResearch Article
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Characterization of DNA adducts at the bay region of dibenz[a,h]anthracene formed in vitro

1993

Bay region diolepoxide-DNA adducts of dibenz[a,h]anthracene (DBA) formed in vitro were identified and their absolute stereochemistry was assigned. After activation of [5,12-14C]DBA with liver microsomes obtained from Aroclor 1254 treated male Sprague-Dawley rats in the presence of calf thymus DNA for 1 h, the amount of DNA adducts was found to be 9.9 +/- 2.4 pmol/mg DNA, calculated on the basis of the portion of radioactivity eluted from the HPLC reversed-phase column with a water/acetonitrile gradient. Bay region diolepoxide-DNA adducts represented 27.5% of radioactivity associated with DNA adducts. The absolute configuration of the various adducts was determined from the reaction of the (…

MaleCancer ResearchAnthraceneMetaboliteAbsolute configurationStereoisomerismDNAGeneral MedicineIn Vitro TechniquesHigh-performance liquid chromatographyMedicinal chemistryRatsAdductRats Sprague-DawleyDNA Adductschemistry.chemical_compoundchemistryBiochemistryDeoxyadenosineBenz(a)AnthracenesMicrosomes LiverAnimalsDeoxyguanosineDibenz(ah)anthraceneBiotransformationCarcinogenesis
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Oxidative stress in bisphosphonate-related osteonecrosis of the jaws

2013

Objectives To analyze whether oxidative stress (OS) changes are present in patients with bisphosphonate-related osteonecrosis of the jaw (BRONJ) versus controls. Materials and Methods Oxidative stress was analyzed in serum and unstimulated saliva of three groups: Group 1 consisted of 24 patients who had been treated with intravenous bisphosphonates (ivBPs) and developed BRONJ, group 2 consisted of 20 patients who had received ivBPs and did not develop BRONJ, and group 3 comprised 17 control subjects. Reduced glutathione (GSH), malondialdehyde (MDA), oxidized glutathione (GSSG), and 8–oxo-7,8-dihydro-2-deoxyguanosine (8-oxo-dG) levels, as well as the GSSG/GSH ratio, were measured. Results Me…

MaleCancer ResearchSalivaOral Hygiene Indexmedicine.medical_treatmentmedicine.disease_causeGastroenterologychemistry.chemical_compoundfluids and secretionsAdrenal Cortex HormonesMalondialdehydeDiphosphonatesGlutathione DisulfideDental Plaque IndexMiddle AgedMalondialdehydeGlutathione8-Hydroxy-2'-DeoxyguanosinePeriodonticsAdministration IntravenousBisphosphonate-Associated Osteonecrosis of the JawFemaleOral SurgeryMultiple Myelomamedicine.medical_specialtyAntineoplastic AgentsBreast NeoplasmsPathology and Forensic MedicineSex FactorsInternal medicinemedicineHumansSalivaAgedBisphosphonate-associated osteonecrosis of the jawDMF Indexbusiness.industryDeoxyguanosineGlutathioneBisphosphonatemedicine.diseaseSurgeryOxidative StressOtorhinolaryngologychemistryCase-Control StudiesGlutathione disulfideOsteonecrosis of the jawbusinessBiomarkersOxidative stressJournal of Oral Pathology & Medicine
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Gender- and age-related distinctions for the in vivo prooxidant state in Fanconi anaemia patients.

2004

Abstract Some selected oxidative stress parameters were measured in 56 Fanconi anaemia (FA) patients (42 untransplanted and 14 transplanted), 54 FA heterozygotes (parents) and 173 controls. Untransplanted FA patients showed a highly significant increase in leukocyte 8-hydroxy-2’-deoxyguanosine (8-OHdG) (p = 0.00003) and a borderline increase (p = 0.076) in urinary levels of 8-OHdG vs. child controls. These increases were more pronounced in female FA patients (p = 0.00005 for leukocyte 8-OHdG, and p = 0.021 for urinary 8-OHdG). Female FA patients also displayed a highly significant excess of spontaneous chromosomal breaks vs. male patients (p = 0.00026), in the same female:male ratio (≅ 1.4)…

MaleCancer Researchmedicine.medical_treatmentTransplantsUrineAscorbic Acidmedicine.disease_causechemistry.chemical_compoundLeukocytesChromosomes HumanVitamin EChildRespiratory BurstGlutathione DisulfideAge FactorsChromosome BreakageGeneral MedicinePyruvaldehydeGlutathioneBiochemistry8-Hydroxy-2'-DeoxyguanosineChild PreschoolFemaleOxidation-ReductionAdultmedicine.medical_specialtyHeterozygoteAdolescentUrinary systemBiologySex FactorsInternal medicinemedicineHumansVitamin CVitamin EDeoxyguanosineInfantGlutathioneDNAAscorbic acidUric AcidOxidative StressEndocrinologyFanconi AnemiachemistryCase-Control StudiesUric acidReactive Oxygen SpeciesOxidative stressCarcinogenesis
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Enhanced Functional Activity of the Cannabinoid Type-1 Receptor Mediates Adolescent Behavior.

2015

Adolescence is characterized by drastic behavioral adaptations and comprises a particularly vulnerable period for the emergence of various psychiatric disorders. Growing evidence reveals that the pathophysiology of these disorders might derive from aberrations of normal neurodevelopmental changes in the adolescent brain. Understanding the molecular underpinnings of adolescent behavior is therefore critical for understanding the origin of psychopathology, but the molecular mechanisms that trigger adolescent behavior are unknown. Here, we hypothesize that the cannabinoid type-1 receptor (CB1R) may play a critical role in mediating adolescent behavior because enhanced endocannabinoid (eCB) sig…

MaleCannabinoid receptorAdolescentmedicine.medical_treatmentIn Vitro TechniquesImpulsivityMediatorRisk-TakingCocaineReceptor Cannabinoid CB1Sulfur IsotopesmedicineAnimalsHumansMaze LearningRadionuclide ImagingSocial BehaviorCannabinoid Receptor AntagonistsBehavior AnimalGeneral NeuroscienceNovelty seekingAge FactorsBrainArticlesPhenotypeEndocannabinoid systemCorpus StriatumRats Inbred F344RatsAdolescent BehaviorGuanosine 5'-O-(3-Thiotriphosphate)Models AnimalMutationExploratory BehaviorCannabinoid receptor antagonistCannabinoidmedicine.symptomRats TransgenicPsychologyNeuroscienceEndocannabinoidsThe Journal of neuroscience : the official journal of the Society for Neuroscience
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Different impacts of cardiovascular risk factors on oxidative stress.

2011

The objective of the study was to evaluate oxidative stress (OS) status in subjects with different cardiovascular risk factors. With this in mind, we have studied three models of high cardiovascular risk: hypertension (HT) with and without metabolic syndrome, familial hypercholesterolemia (FH) and familial combined hyperlipidemia (FCH) with and without insulin resistance. Oxidative stress markers (oxidized/reduced glutathione ratio, 8-oxo-deoxyguanosine and malondialdehide) together with the activity of antioxidant enzyme triad (superoxide dismutase, catalase, glutathione peroxidase) and activation of both pro-oxidant enzyme (NAPDH oxidase components) and AGTR1 genes, as well as antioxidant…

MaleGPX1Antioxidantmedicine.medical_treatmentGlutathione reductaseHyperlipidemia Familial Combinedmedicine.disease_causelcsh:Chemistrychemistry.chemical_compoundRisk FactorsMalondialdehydeoxidative stressglutathione peroxidaselcsh:QH301-705.5Spectroscopychemistry.chemical_classificationbiologyfamilial hypercholesterolemiaChemistryGlutathione peroxidaseGeneral MedicineMiddle AgedCatalaseGlutathioneComputer Science ApplicationsGlutathione Reductase8-Hydroxy-2'-DeoxyguanosineCardiovascular DiseasesFemaleThioredoxinAdultmedicine.medical_specialtyhypertensionmRNACatalysisGlutathione SynthaseArticleInorganic ChemistrySuperoxide dismutaseHyperlipoproteinemia Type IIInternal medicinemedicineHumansPhysical and Theoretical ChemistryMolecular BiologySuperoxide DismutaseGene Expression ProfilingOrganic ChemistryDeoxyguanosineNADPH OxidasesGlutathionesuperoxide dismutasesPhosphoproteinscombined familial dyslipidemiaEndocrinologylcsh:Biology (General)lcsh:QD1-999biology.proteinOxidative stressBiomarkersInternational journal of molecular sciences
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Increased Oxidative Damage Associated with Unfavorable Cytogenetic Subgroups in Chronic Lymphocytic Leukemia

2014

Oxidative stress contributes to genomic instability in chronic lymphocytic leukemia (CLL), but its relationship with the acquisition of specific chromosomal abnormalities is unknown. We recruited 55 untreated CLL patients and assessed 8-oxo-2′-deoxyguanosine (8-oxo-dG), glutathione, and malondialdehyde (MDA) levels, and we compared them among the cytogenetic subgroups established using fluorescence in situ hybridization (FISH). Significant increases in 8-oxo-dG and/or MDA were observed in patients with unfavorable cytogenetic aberrations (17p and 11q deletions) compared to the 13q deletion group.TP53deletion patients exhibited a diminished DNA repair efficiency. Finally, cases with normal F…

MaleGenome instabilityArticle SubjectDNA RepairDNA damageDNA repairChronic lymphocytic leukemialcsh:MedicineBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCohort Studieschemistry.chemical_compoundMalondialdehydemedicineHumansLymphocytesIn Situ Hybridization FluorescenceAgedAged 80 and overChromosome AberrationsGeneral Immunology and Microbiologymedicine.diagnostic_testlcsh:RDeoxyguanosineGeneral MedicineGlutathioneMiddle AgedMalondialdehydemedicine.diseaseGlutathioneLeukemia Lymphocytic Chronic B-CellOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineImmunologyFemaleLipid PeroxidationReactive Oxygen SpeciesGene DeletionOxidative stressDNA DamageResearch ArticleFluorescence in situ hybridizationBioMed Research International
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The ineffectiveness of the NO-cyclic GMP signaling pathway in the atrial myocardium

1995

1. This study was performed to determine whether nitric oxide (NO) has direct effects on force of contraction (Fc) in atrial myocardium from rats, rabbits, guinea-pigs, frogs, and man. 2. Glyceryl trinitrate, isosorbide dinitrate, 3-morpholino-sydnonimine hydrochloride (SIN-1), and S-nitroso-N-acetylpenicillamine (SNAP) did not significantly reduce Fc in the various preparations investigated, either given alone or after stimulation of alpha- or beta-adrenoceptors. 3. SNAP did not change the time course of contractions in rat, guinea-pig and human preparations. 4. 8-Bromo-guanosine-3':5'-cyclic monophosphate (8-Br-cyclic GMP) produced a negative inotropic effect in rat, guinea-pig and human …

MaleInotropemedicine.medical_specialtyContraction (grammar)RanidaeVasodilator AgentsGuinea PigsGuanosine MonophosphateArginineNitric OxideNitric oxideRats Sprague-DawleyContractilitychemistry.chemical_compoundInternal medicinemedicineAnimalsHumansHeart AtriaEnzyme InhibitorsPharmacologyomega-N-MethylargininebiologyChemistrySnapAtrial FunctionMyocardial ContractionAcetylcholineRatsNitric oxide synthaseEndocrinologybiology.proteinOmega-N-MethylarginineCarbacholFemaleRabbitsNitric Oxide SynthaseIsosorbide dinitrateSignal TransductionResearch Articlemedicine.drugBritish Journal of Pharmacology
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The basal levels of 8-oxoG and other oxidative modifications in intact mitochondrial DNA are low even in repair-deficient (Ogg1(-/-)/Csb(-/-)) mice.

2007

Abstract Mitochondrial DNA (mtDNA) is assumed to be highly prone to damage by reactive oxygen species (ROS) because of its location in close proximity to the mitochondrial electron transport chain. Accordingly, mitochondrial oxidative DNA damage has been hypothesized to be responsible for various neurological diseases, ageing and cancer. Since 7,8-dihydro-8-oxoguanine (8-oxoG), one of the most frequent oxidative base modifications, is removed from the mitochondrial genome by the glycosylase OGG1, the basal levels of this lesion are expected to be highly elevated in Ogg1−/− mice. To investigate this hypothesis, we have used a mtDNA relaxation assay in combination with various repair enzymes …

MaleMitochondrial DNADNA RepairDNA repairHealth Toxicology and MutagenesisOxidative phosphorylationBiologyMitochondrionDNA MitochondrialDNA Glycosylaseschemistry.chemical_compoundMiceGeneticsAnimalsPoly-ADP-Ribose Binding ProteinsMolecular BiologyMice KnockoutGuanosinePlant ExtractsCorticoviridaeMolecular biologyNuclear DNAMice Inbred C57BLDNA Repair EnzymeschemistryDNA glycosylaseDNA ViralFemaleDNANucleotide excision repairDNA DamageMutation research
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