Search results for "hiv-1"

showing 10 items of 177 documents

Binding isotope effects as a tool for distinguishing hydrophobic and hydrophilic binding sites of HIV-1 RT.

2014

The current treatment for HIV-1 infected patients consists of a cocktail of inhibitors, in an attempt to improve the potency of the drugs by adding the possible effects of each supplied compound. In this contribution, nine different inhibitors of HIV-1 RT, one of the three key proteins responsible for the virus replication, have been selected to develop and test a computational protocol that allows getting a deep insight into the inhibitors’ binding mechanism. The interaction between the inhibitors and the protein have been quantified by computing binding free energies through FEP calculations, while a more detailed characterization of the kind of inhibitor–protein interactions is based on …

StereochemistryBinding energyHuman immunodeficiency virus (HIV)Binding energyMolecular Dynamics Simulationmedicine.disease_causeLigandsIsotopesCatalytic DomainKinetic isotope effectDrug DiscoveryMaterials ChemistrymedicinePhysical and Theoretical ChemistryBinding siteBinding isotope effectsIsotopeChemistryWaterHIV Reverse TranscriptaseSurfaces Coatings and FilmsCrystallographyViral replicationHIV-1SolventsQuantum TheoryReverse Transcriptase InhibitorsThermodynamicsFree energiesHydrophobic and Hydrophilic InteractionsProtein BindingThe journal of physical chemistry. B
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Translocation Biosensors – Cellular System Integrators to Dissect CRM1-Dependent Nuclear Export by Chemicogenomics

2009

Fluorescent protein biosensors are powerful cellular systems biology tools for dissecting the complexity of cellular processes with high spatial and temporal resolution. As regulated nucleo-cytoplasmic transport is crucial for the modulation of numerous (patho)physiological cellular responses, a detailed understanding of its molecular mechanism would open up novel options for a rational manipulation of the cell. In contrast to genetic approaches, we here established and employed high-content cellular translocation biosensors applicable for dissecting nuclear export by chemicogenomics. A431 cell lines, stably expressing a translocation biosensor composed of glutathione S-transferase, GFP and…

Systems biologyChemical biologyNanotechnologychemical biologyComputational biologyBiologylcsh:Chemical technologyBiochemistryArticleAnalytical ChemistryGreen fluorescent proteinFlow cytometrychemical biology; cancer; Exportin 1/CRM1; HIV-1 Rev; import; LMB; nucleocytoplasmic transport; nucleoporinimportmedicinecancerlcsh:TP1-1185Electrical and Electronic EngineeringNuclear export signalLMBInstrumentationExportin 1/CRM1HIV-1 Revnucleocytoplasmic transportmedicine.diagnostic_testnucleoporinAtomic and Molecular Physics and OpticsChemical spacecancer ; HIV-1 Rev ; import ; nucleocytoplasmic transport ; LMB ; chemical biology ; Exportin 1/CRM1 ; nucleoporinNucleoporinNuclear transportBiologieSensors
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HIV-1 Adaptation to Antigen Processing Results in Population-Level Immune Evasion and Affects Subtype Diversification

2014

Summary The recent HIV-1 vaccine failures highlight the need to better understand virus-host interactions. One key question is why CD8+ T cell responses to two HIV-Gag regions are uniquely associated with delayed disease progression only in patients expressing a few rare HLA class I variants when these regions encode epitopes presented by ∼30 more common HLA variants. By combining epitope processing and computational analyses of the two HIV subtypes responsible for ∼60% of worldwide infections, we identified a hitherto unrecognized adaptation to the antigen-processing machinery through substitutions at subtype-specific motifs. Multiple HLA variants presenting epitopes situated next to a giv…

T cellT-LymphocytesPopulationMolecular Sequence DataPopulationHIV InfectionsHuman leukocyte antigenBiologyGeneral Biochemistry Genetics and Molecular BiologyEpitopeArticleAfrica Southern03 medical and health sciencesEpitopesImmune systemGene FrequencymedicineHumansAmino Acid Sequenceeducationlcsh:QH301-705.5HLA-A1 Antigen030304 developmental biologyImmune EvasionGenetics0303 health scienceseducation.field_of_studyAntigen processingImmunogenicity030302 biochemistry & molecular biologyAdaptation Physiological3. Good healthEuropemedicine.anatomical_structurelcsh:Biology (General)HIV-1CD8Cell Reports
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Alteration of DNA topoisomerase II activity during infection of H9 cells by human immunodeficiency virus type 1 in vitro: a target for potential ther…

1990

Infection of H9 cells with human immunodeficiency virus type 1 (HIV-1) was found to decrease the phosphorylation of DNA topoisomerase II during the initial phase of infection. Simultaneously, with a later overshoot of phosphorylation and the subsequent activation of DNA topoisomerase II, the production of HIV-1 started. Applying three new protein kinase C inhibitors from the class of O-alkylglycerophospholipids we demonstrated that inhibition of protein kinase C-mediated phosphorylation of DNA topoisomerase II resulted in an inhibition of HIV-1 production. Based on the differential effect of the two protein kinase C activators, phorbol ester and bryostatin, we conclude that phosphorylation …

T-LymphocytesMitogen-activated protein kinase kinaseIn Vitro TechniquesMAP2K7Cell LineLactonesVirologyAnimalsPhosphorylationProtein kinase AProtein kinase CProtein Kinase CPharmacologybiologyCyclin-dependent kinase 2LysophosphatidylcholinesRats Inbred StrainsDNA topoisomerase II activityBryostatinsProtein kinase RMolecular biologyRatsDNA Topoisomerases Type Ibiology.proteinHIV-1Tetradecanoylphorbol AcetateCyclin-dependent kinase 9Electrophoresis Polyacrylamide GelMacrolidesAntiviral research
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NFAT transcription factors control HIV-1 expression through a binding site downstream of TAR region.

2004

NFAT factors control HIV-1 transcription. We show here that, in addition to binding to two NF-kappaB/NFAT sites within the U3 HIV LTR, NFATc1 and NFATc2 bind to an NFAT site within the LTR's U5 region. Mutations in this site which abolish NFAT binding reduce the ability of NFATs to transactivate LTR-mediated transcription. Mutations in all three NFAT sites strongly interfered with LTR induction, but affected moderately the stimulatory effect of Tat.

Transcription GeneticvirusesImmunologyTransfectionJurkat cellsJurkat CellsTranscription (biology)Immunology and AllergyHumansNuclear proteinBinding siteTranscription factorHIV Long Terminal RepeatBinding SitesNFATC Transcription FactorsChemistryNuclear ProteinsNFATHematologyU937 CellsNFATC Transcription FactorsMolecular biologyDNA-Binding Proteinscardiovascular systemHIV-1HIV Long Terminal RepeatTranscription FactorsImmunobiology
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Evolución de la resistencia transmitida y adquirida a fármacos antirretrovirales en pacientes infectados con VIH-1

2018

Introducción: La adquisición y transmisión de resistencia amenaza el éxito del tratamiento antirretroviral. En este estudio evaluamos el comportamiento epidemiológico de las mutaciones de resistencia (MR) a fármacos antirretrovirales del VIH-1 según factores de riesgo y clínicos específicos. Metodología: Este es un estudio retrospectivo observacional de series transversales acumuladas, realizado en la Unidad de Enfermedades Infecciosas de un hospital universitario en España (CHGUV) entre los años 2003 y 2014. Nuestro objetivo fue estimar la evolución de la frecuencia anual de MR a fármacos antirretrovirales en el genoma de la proteasa y transcriptasa inversa del VIH-1 durante el periodo de …

UNESCO::CIENCIAS MÉDICASmen who have sex with menusuarios de droga por vía parenteralhombres que tienen sexo con otros hombres:CIENCIAS MÉDICAS [UNESCO]HIV-1 drug resistanceresistencia antirretroviral VIH-1intravenous drug usersCD4resistencia antirretroviral
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In-Depth Characterization of Viral Isolates from Plasma and Cells Compared with Plasma Circulating Quasispecies in Early HIV-1 Infection

2012

Background The use of in vitro models to unravel the phenotypic characteristics of circulating viral variants is key to understanding HIV-1 pathogenesis but limited by the availability of primary viral isolates from biological samples. However, overall in vivo genetic variability of HIV-1 within a subject may not be reflected in the viable viral population obtained after isolation. Although several studies have tried to determine whether viral populations expanded in vitro are representative of in vivo findings, the answer remains unclear due to the reduced number of clonal sequences analyzed or samples compared. In order to overcome previous experimental limitations, here we applied Deep P…

Viral DiseasesHeredityGenotypePopulationlcsh:MedicineHIV InfectionsViral quasispeciesMicrobiology03 medical and health sciencesPredictive Value of TestsVirologyGenotypeGenetic variationGeneticsHumansGenetic variabilitylcsh:ScienceeducationBiologyPhylogeny030304 developmental biologyGenetics0303 health scienceseducation.field_of_studyMultidisciplinarybiology030306 microbiologylcsh:RHIVGenetic VariationReproducibility of ResultsGenomicsSequence Analysis DNAVirology3. Good healthIntegraseInfectious DiseasesPhenotypeViral replicationDNA ViralHIV-1Leukocytes Mononuclearbiology.proteinMedicineRNA Virallcsh:QRNA extractionResearch ArticlePLoS ONE
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Expression and cellular localization of the Nef protein from human immunodeficiency virus-1 in stably transfected B-cells.

1992

Nef protein, encoded by the regulatory nef gene of human immunodeficiency virus type 1 (HIV-1), was expressed in the B-cell line Raji. The cells were stably transfected with plasmids containing the nef transcriptional cassette. They expressed Nef with an Mr of 27,000; the yield could be augmented by incubation with the tumor promoter 12-O-tetradecanoylphorbol-13-acetate. The intracellular localization of Nef was analyzed applying immunofluorescence microscopy using a confocal laser scanning microscope. The antigen was stained with a monoclonal antibody directed against the N-terminal part of Nef. The experiments revealed that in non-dividing cells Nef is present both in the cytoplasm and th…

Viral proteinvirusesGenetic VectorsFluorescent Antibody TechniqueBiologymedicine.disease_causeTransfectionVirusGene Products nefGene productAntigenVirologyGene expressionmedicineTumor Cells CulturedHumansnef Gene Products Human Immunodeficiency VirusCellular localizationB-LymphocytesMicroscopyvirus diseasesGeneral MedicineTransfectionVirologyMolecular biologyCytoplasmHIV-1Tetradecanoylphorbol AcetateArchives of virology
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Cordycepin analogues of 2',5'-oligoadenylate inhibit human immunodeficiency virus infection via inhibition of reverse transcriptase.

1991

Analogues of 2',5'-oligoadenylates (2-5A), the cordycepin (3'-deoxyadenosine) core trimer (Co3) and its 5'-monophosphate derivative (pCo3), were shown to display pronounced anti-human immunodeficiency virus type 1 (HIV-1) activity in vitro. Treatment of HIV-1 infected H9 cells with 1 microM Co3 or pCo3 resulted in an almost 100% inhibition of virus production. The compounds were encapsulated in liposomes targeted by antibodies specific for the T-cell receptor molecule CD3. Substitution of one or two cordycepin units in Co3 or pCo3 decreased the antiviral activity of the compounds. pCo3 did not stimulate 2-5A-dependent ribonuclease L activity and displayed no effect on the amount of cellular…

Virus ReplicationBiochemistryAntiviral AgentsVirusCell Linechemistry.chemical_compoundStructure-Activity RelationshipDeoxyadenosineHumansPolymeraseNucleic Acid Synthesis InhibitorsOligoribonucleotidesbiologyCordycepinDeoxyadenosines2'-5'-OligoadenylateAdenine NucleotidesRNAMolecular biologyReverse transcriptaseBiochemistrychemistryRNA RibosomalLiposomesbiology.proteinHIV-1RNA Transfer LysReverse Transcriptase InhibitorsRibonuclease LBiochemistry
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Progettazione e sintesi di 2,3-diaril-1,3-tiazolidin-4-oni nuovi HIV-1 NNRTIs.

2002

agenti HIV-1
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