Search results for "phage"

showing 10 items of 1573 documents

Phage-driven loss of virulence in a fish pathogenic bacterium

2012

Parasites provide a selective pressure during the evolution of their hosts, and mediate a range of effects on ecological communities. Due to their short generation time, host-parasite interactions may also drive the virulence of opportunistic bacteria. This is especially relevant in systems where high densities of hosts and parasites on different trophic levels (e.g. vertebrate hosts, their bacterial pathogens, and virus parasitizing bacteria) co-exist. In farmed salmonid fingerlings, Flavobacterium columnare is an emerging pathogen, and phage that infect F. columnare have been isolated. However, the impact of these phage on their host bacterium is not well understood. To study this, four s…

Gliding motilityPathogenesisAquacultureFish DiseasesFlavobacteriaceae InfectionsSalmonphageBacteriophagesPathogenZebrafishGliding motility0303 health sciencesEvolutionary TheoryMultidisciplinarybiologyEcologyVirulenceQRFishesvirulenssiAnimal ModelsBiological EvolutionBacterial PathogensHost-Pathogen InteractionLytic cycleMedicineResearch ArticleScienceVirulenceMicrobiologyFlavobacteriumMicrobiologyMicrobial EcologyHost-Parasite Interactions03 medical and health sciencesModel OrganismsVirologyAnimals14. Life underwaterBiology030304 developmental biologyEvolutionary Biology030306 microbiologyHost (biology)ta1182biology.organism_classificationEvolutionary Ecologyphage resistanceFlavobacterium columnareVirulence Factors and Mechanismsta1181BacteriaFlavobacteriumopportunismi
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The designer cytokine hyper-IL-6 mediates growth inhibition and GM-CSF-dependent rejection of B16 melanoma cells.

2000

The low immunogenic B16 melanoma cell line was transfected with a mammalian expression vector containing the complementary DNA for a sIL-6R/IL-6 fusion protein, termed Hyper-IL-6 (H-IL-6), which was shown to have biological activities at 100-1000-fold lower concentrations than IL-6 in combination with sIL-6R. The secreted p84 glycoprotein was detected in the supernatant of transfected cells and was fully active on BAF3/gp130 cells, which respond to IL-6/sIL-6R but not to IL-6 alone. Administration of recombinant H-IL-6 to C57BL/6 mice resulted in a prolonged acute phase protein gene expression indicating long systemic persistence of the fusion protein. Transfected B16 cells (B16/H-IL6 cells…

Graft RejectionCancer ResearchTumor suppressor geneRatónmedicine.medical_treatmentRecombinant Fusion ProteinsMelanoma ExperimentalMice TransgenicTransfectionchemistry.chemical_compoundMiceGene expressionGeneticsmedicineAnimalsDrug InteractionsInterleukin 6neoplasmsMolecular BiologybiologyInterleukin-6MelanomaGranulocyte-Macrophage Colony-Stimulating FactorReceptors Interleukinmedicine.diseaseReceptors Interleukin-6Growth Inhibitorsrespiratory tract diseasesCytokinechemistryCell cultureReceptors Granulocyte-Macrophage Colony-Stimulating FactorImmunologybiology.proteinCancer researchGrowth inhibitionImmunosuppressive AgentsOncogene
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GM-CSF restores innate, but not adaptive, immune responses in glucocorticoid-immunosuppressed human blood in vitro.

2003

Abstract Infection remains the major complication of immunosuppressive therapy in organ transplantation. Therefore, reconstitution of the innate immunity against infections, without activation of the adaptive immune responses, to prevent graft rejection is a clinically desirable status in transplant recipients. We found that GM-CSF restored TNF mRNA and protein expression without inducing IL-2 production and T cell proliferation in glucocorticoid-immunosuppressed blood from either healthy donors or liver transplant patients. Gene array experiments indicated that GM-CSF selectively restored a variety of dexamethasone-suppressed, LPS-inducible genes relevant for innate immunity. A possible ex…

Graft RejectionLipopolysaccharidesT-LymphocytesCell Cycle ProteinsCell SeparationOrgan transplantationDexamethasoneMiceCDC2-CDC28 KinasesConcanavalin ATumor Cells CulturedImmunology and AllergySkin TransplantationMiddle AgedCyclin-Dependent KinasesUp-RegulationSurvival Ratemedicine.anatomical_structureImmunity ActiveTumor necrosis factor alphaGlucocorticoidCell DivisionCyclin-Dependent Kinase Inhibitor p27Immunosuppressive Agentsmedicine.drugAdultmedicine.medical_specialtyT cellImmunologyDown-RegulationBiologyProtein Serine-Threonine KinasesImmune systemAdjuvants ImmunologicIn vivomedicineAnimalsHumansDexamethasoneAgedSalmonella Infections AnimalInnate immune systemTumor Suppressor ProteinsCyclin-Dependent Kinase 2Granulocyte-Macrophage Colony-Stimulating FactorImmunity InnateGene Expression RegulationImmunologyLeukocytes MononuclearMice Inbred CBAInterleukin-2Interleukin-1Journal of immunology (Baltimore, Md. : 1950)
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In vitro synthesis of factor B of the alternative pathway of complement activation by mouse peritoneal macrophages

1976

Factor B of the alternative pathway of complement activation was shown to be synthesized and secreted by unstimulated mouse peritoneal macrophages. The activity of B in the culture supernatants from macrophage monolayers was detected by consumption of C3 in reaction mixtures containing supernatant and guinea pig factors C3, D and insoluble C3b. Using a monospecific antiserum, factor B in concentrated culture supernatants was shown by immunodiffusion and immunoelectrophoresis to be identical to factor B in mouse plasma and to form a characteristic complex with cobra venom factor in the presence of D. A steady rate of factor B secretion was observed for 4 days providing the medium was changed…

Guinea PigsImmunologyImmunoelectrophoresisCycloheximideBiologyComplement factor BMicechemistry.chemical_compoundmedicineAnimalsAscitic FluidImmunology and AllergyCycloheximideCells CulturedGlycoproteinsAntiserumProperdinmedicine.diagnostic_testMacrophagesComplement C3Complement System ProteinsMolecular biologyIn vitroComplement systemImmunodiffusionKineticschemistryBiochemistryAlternative complement pathwayEuropean Journal of Immunology
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Influence of Macrophage Activation on the Synthesis of Complement Components C2, C3, C4

1982

Macrophages are a major site of complement synthesis. In the guinea pig production of complement components C1, C2, C4, C3, D, B, and P by peritoneal macrophages has been demonstrated (reviewed in Ref. 1). Whereas marked differences exist in biological activity between resident, elicited and activated macrophages (2), we investigated whether this holds true for the synthesis and secretion of complement components C2, C3, and C4.

Guinea pigChemistryMacrophageBiological activitySecretionComplement (complexity)Cell biologyComplement components
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Zur therapie der �sophagusvarizenblutung in der Bundesrepublik Deutschland (West) ? Ergebnisse einer Urnfrage

1991

An 1076 chirurgischen und internistischen Abteilung in der BRD (West) wurde eine Umfrage zur Behandlung der Osophagusvarizenblutung durchgefuhrt. Es uberwiegt die Akutsklerosierung bzw. die Ballonsondenbehandlung mit nachfolgender Sklerosierung bei der aktiven Blutung. Die abdominelle Sperroperation wird bei der konservativ nicht stillbaren Blutung noch vor dem portokavalen Notshunt bevorzugt. Betablocker werden vor allem in internistischen Abteilungen zur Rezidivprophylaxe eingesetzt. Erst nach mehrfacher Rezidivblutung trotz Sklerosierung sehen etwa die Halfte der Abteilungen eine Indikation zum elektiven Shunt. Der distale splenorenale Shunt nach Warren and die portokavale Anastomose dom…

Gynecologymedicine.medical_specialtybusiness.industryGeneral surgeryVascular surgeryCardiac surgeryShunt operationWest germanyCardiothoracic surgeryBleeding esophageal varicesMedicineSurgerybusinessAbdominal surgeryLangenbecks Archiv fur Chirurgie
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Cross-reactivity between tumor MHC class I-restricted antigens and an enterococcal bacteriophage

2020

International audience; Intestinal microbiota have been proposed to induce commensal-specific memory T cells that cross-react with tumor-associated antigens. We identified major histocompatibility complex (MHC) class I-binding epitopes in the tail length tape measure protein (TMP) of a prophage found in the genome of the bacteriophage Enterococcus hirae Mice bearing E. hirae harboring this prophage mounted a TMP-specific H-2Kb-restricted CD8+ T lymphocyte response upon immunotherapy with cyclophosphamide or anti-PD-1 antibodies. Administration of bacterial strains engineered to express the TMP epitope improved immunotherapy in mice. In renal and lung cancer patients, the presence of the ent…

H-2 AntigenProgrammed Cell Death 1 ReceptorCD8-Positive T-LymphocytesEpitopeEpitopesFecesMice0302 clinical medicineEnterococcus hiraeNeoplasmsMonoclonalBacteriophages0303 health sciencesMultidisciplinarybiologyAntibodies MonoclonalViral Tail ProteinsAlkylating3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisCross ReactionEpitopeImmunotherapyHumanT cellAntineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/CancerCross ReactionsMajor histocompatibility complexAntibodiesMicrobiology03 medical and health sciencesAnimals; Antibodies Monoclonal; Antigens Neoplasm; Antineoplastic Agents Alkylating; Bacteriophages; CD8-Positive T-Lymphocytes; Cross Reactions; Cyclophosphamide; Enterococcus hirae; Epitopes; Feces; Gastrointestinal Microbiome; H-2 Antigens; Histocompatibility Antigens Class I; Humans; Immunotherapy; Mice; Neoplasms; Programmed Cell Death 1 Receptor; Viral Tail Proteins[SDV.CAN] Life Sciences [q-bio]/CancerAntigenAntigens NeoplasmMHC class ImedicineAnimalsHumansAntigensBacteriophageAntineoplastic Agents AlkylatingCyclophosphamideProphage030304 developmental biologyEnterococcus hiraeAnimalHistocompatibility Antigens Class IH-2 AntigensCD8-Positive T-Lymphocytebiology.organism_classificationGastrointestinal Microbiomebiology.proteinNeoplasmFeceCD8
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Health-related quality of life (HRQOL) in patients (pts) with advanced gastric cancer/gastroesophageal junction cancer (GC/GEJC) or esophageal adenoc…

2021

4066 Background: CheckMate 649 (NCT02872116) is a randomized, open label phase 3 study in first line (1L) GC/GEJC/EAC. Prespecified interim results showed statistically significant improvement in overall survival (OS) and progression-free survival (PFS) for N+C vs C in all randomized pts and pts whose tumors expressed programmed death-ligand 1 combined positive score (CPS) ≥ 5. We present interim HRQOL results for CPS ≥ 5 pts, included as exploratory in the study. Methods: HRQOL was assessed using EQ-5D-3L (EQ-5D) and Functional Assessment of Cancer Therapy–Gastric Cancer (FACT-Ga). Assessments were performed at baseline (BL), every 6 weeks during treatment, and during follow-up. Change fr…

Health related quality of lifeCancer ResearchChemotherapymedicine.medical_specialtybusiness.industrymedicine.medical_treatmentCheckmatePhases of clinical researchEsophageal adenocarcinomaCancerGastroesophageal Junctionmedicine.diseaseGastroenterologyOncologyInternal medicineMedicineNivolumabbusinessJournal of Clinical Oncology
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Checkmate 577:Health-related quality of life (HRQoL) in a randomized, double-blind phase III study of nivolumab (NIVO) versus placebo (PBO) as adjuva…

2021

167 Background: NIVO is the first adjuvant therapy to provide a statistically significant and clinically meaningful improvement in disease-free survival (DFS) versus PBO in resected EC/GEJC following neoadjuvant chemoradiotherapy as demonstrated by CheckMate 577. NIVO was well tolerated with an acceptable safety profile. This analysis provides additional information on the exploratory HRQoL endpoints in this clinical trial. Methods: The effect of NIVO versus PBO on HRQoL, including general and disease-related symptoms, functioning, disease burden, and overall QoL, was assessed using FACT-E and EQ-5D-3L patient-reported outcome (PRO) questionnaires administered at baseline (BL), every 4 wee…

Health related quality of lifeCancer Researchmedicine.medical_specialtybusiness.industrymedicine.medical_treatmentCancerGastroesophageal JunctionPlacebomedicine.diseaseGastroenterologyDouble blindOncologyInternal medicinemedicineAdjuvant therapyNivolumabbusinessAdjuvantJournal of Clinical Oncology
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De novo 13q deletions in two patients with mild anorectal malformations as part of VATER/VACTERL and VATER/VACTERL-like association and analysis of E…

2013

Item does not contain fulltext Anorectal malformations (ARMs) comprise a broad spectrum of conditions ranging from mild anal anomalies to complex cloacal malformations. In 40-50% of cases, ARM occurs within the context of defined genetic syndromes or complex multiple congenital anomalies, such as VATER/VACTERL (vertebral defects [V], ARMs [A], cardiac defects [C], tracheoesophageal fistula with or without esophageal atresia [TE], renal malformations [R], and limb defects [L]) association. Here, we report the identification of deletions at chromosome 13q using single nucleotide polymorphism-based array analysis in two patients with mild ARM as part of VATER/VACTERL and VATER/VACTERL-like ass…

Heart Defects CongenitalMalemedicine.medical_specialtyCandidate geneLimb Deformities CongenitalTracheoesophageal fistulaSingle-nucleotide polymorphismContext (language use)Chromosome DisordersEphrin-B2BiologyGastroenterologyAnus ImperforateMiceEsophagusInternal medicineGeneticsmedicineAnimalsHumansIn patientGenetics (clinical)Mice KnockoutChromosomes Human Pair 13Infant NewbornChromosomeAnatomymedicine.diseaseAnorectal MalformationsSpineTracheaDisease Models AnimalRadiusHuman Reproduction Renal disorder [NCEBP 12]Evaluation of complex medical interventions [NCEBP 2]AtresiaChild PreschoolMutationMutation testingFemaleChromosome DeletionGenetics and epigenetic pathways of disease Genomic disorders and inherited multi-system disorders [NCMLS 6]American Journal of Medical Genetics. Part A
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