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showing 10 items of 6213 documents

Unraveling the Molecular Mechanism of Action of Empagliflozin in Heart Failure With Reduced Ejection Fraction With or Without Diabetes

2019

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0301 basic medicinelcsh:Diseases of the circulatory (Cardiovascular) systemmedicine.medical_specialtyCardiac & Cardiovascular Systemsempagliflozinheart failure030204 cardiovascular system & hematologySGLT2i sodium-glucose co-transporter 2 inhibitorHF heart failurePRECLINICAL RESEARCH03 medical and health sciences0302 clinical medicineDM diabetes mellitusDiabetes mellitusInternal medicinemedicineEmpagliflozinMI-HF post-infarct heart failureGlycemicScience & TechnologyEjection fractionbusiness.industryNHE sodium-hydrogen exchangerANN artificial neural networkmedicine.diseaseHFrEF HF with reduced ejection fractionBlockadeXIAPmachine learning030104 developmental biologyMechanism of actionlcsh:RC666-701Heart failureCardiovascular System & CardiologyCardiologyRNAseq RNA sequencingempagtiflozinmedicine.symptomCardiology and Cardiovascular MedicinebusinessLife Sciences & BiomedicineJACC: Basic to Translational Science
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Methods for RNA Modification Mapping Using Deep Sequencing: Established and New Emerging Technologies

2019

New analytics of post-transcriptional RNA modifications have paved the way for a tremendous upswing of the biological and biomedical research in this field. This especially applies to methods that included RNA-Seq techniques, and which typically result in what is termed global scale modification mapping. In this process, positions inside a cell`s transcriptome are receiving a status of potential modification sites (so called modification calling), typically based on a score of some kind that issues from the particular method applied. The resulting data are thought to represent information that goes beyond what is contained in typical transcriptome data, and hence the field has taken to use …

0301 basic medicinelcsh:QH426-470Computer scienceProcess (engineering)Emerging technologieschemical treatmentNext Generation Sequencingengineered Reverse Transcriptase enzymesRNA-SeqReviewcomputer.software_genreDeep sequencingField (computer science)deep sequencing03 medical and health sciences0302 clinical medicineepitranscriptome[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]GeneticsAnimalsHumansRNA-SeqRNA Processing Post-TranscriptionalComputingMilieux_MISCELLANEOUSGenetics (clinical)Sequence Analysis RNAbusiness.industryScale (chemistry)High-Throughput Nucleotide Sequencing[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyRNA modificationTerm (time)lcsh:Genetics030104 developmental biologyAnalyticsRNAData miningbusinesscomputer030217 neurology & neurosurgeryGenes
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RNA Modifications Modulate Activation of Innate Toll-Like Receptors

2019

Self/foreign discrimination by the innate immune system depends on receptors that identify molecular patterns as associated to pathogens. Among others, this group includes endosomal Toll-like receptors, among which Toll-like receptors (TLR) 3, 7, 8, and 13 recognize and discriminate mammalian from microbial, potentially pathogen-associated, RNA. One of the discriminatory principles is the recognition of endogenous RNA modifications. Previous work has identified a couple of RNA modifications that impede activation of TLR signaling when incorporated in synthetic RNA molecules. Of note, work that is more recent has now shown that RNA modifications in their naturally occurring context can have …

0301 basic medicinelcsh:QH426-470EndosomeContext (language use)ReviewBiology03 medical and health sciences0302 clinical medicineRNA modificationsGeneticsAnimalsHumansGenetics(clinical)RNA Processing Post-TranscriptionalReceptorGeneinnate immunityGenetics (clinical)Innate immune systemRNATLR7Immunity InnateCell biologyToll-like receptorslcsh:Genetics030104 developmental biologyTransfer RNAmethylation030215 immunologyGenes
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Smooth muscle and neural dysfunction contribute to different phases of murine postoperative ileus.

2015

Background Postoperative ileus (POI) is characterized by a transient inhibition of gastrointestinal (GI) motility after abdominal surgery mediated by the inflammation of the muscularis externa (ME). The aim of this study was to identify alterations in the enteric nervous system that may contribute to the pathogenesis of POI. Methods Gastrointestinal transit, contractility of isolated smooth muscle strips and inflammatory parameters were evaluated at different time points (1.5 h to 10 days) after intestinal manipulation (IM) in mice. Immune-labeling was used to visualize changes in myenteric neurons. Key Results Intestinal manipulation resulted in an immediate inhibition of GI transit recove…

0301 basic medicinemedicine.medical_specialtyCarbacholIleusPhysiologyInflammationBiologyEnteric Nervous SystemProinflammatory cytokineContractility03 medical and health sciencesMiceIleusOrgan Culture TechniquesPostoperative ComplicationsInternal medicinemedicineAnimalsEndocrine and Autonomic SystemsGastroenterologyMuscle SmoothSmooth muscle contractionmedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyAnesthesiaCholinergicEnteric nervous systemFemalemedicine.symptomInflammation MediatorsGastrointestinal Motilitymedicine.drugNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Early increased density of cyclooxygenase-2 (COX-2) immunoreactive neurons in Down syndrome

2017

iNeuroinflammation is one of the hallmarks of Alzheimer's disease. One of the enzymes involved in neuroinflammation, even in early stages of the disease, is COX-2, an inducible cyclooxygenase responsible for the generation of eicosanoids and for the generation of free radicals. Individuals with Down syndrome develop Alzheimer's disease early in life. Previous studies pointed to the possible overexpression of COX-2 and correlated it to brain regions affected by the disease. We analysed the COX-2 expression levels in individuals with Down syndrome and in young, adult and old mice of the Ts65Dn mouse model for Down syndrome. We have observed an overexpression of COX-2 in both, Down syndrome in…

0301 basic medicinemedicine.medical_specialtyDown syndromelcsh:MedicineMice TransgenicDiseasePathology and Forensic Medicineneuroinflammationmicroglia</i>Mice03 medical and health sciences0302 clinical medicineInternal medicinemedicineAnimalsHumansPostnatal dayNeuroinflammationNeuronschemistry.chemical_classificationMicrogliabiologybusiness.industryNeurodegenerationlcsh:RBrainmedicine.disease<i>Ts65Dn030104 developmental biologyEndocrinologymedicine.anatomical_structureEnzymechemistryCyclooxygenase 2biology.proteinNeurology (clinical)CyclooxygenaseDown SyndromebusinessNeuroscienceAlzheimer’s disease030217 neurology & neurosurgeryFolia Neuropathologica
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Mechanisms underlying the influence of oestrogen on cardiovascular physiology in women.

2019

Women show a lower incidence of cardiovascular diseases than age-matched men, but this benefit disappears after menopause. Oestrogen-mediated vascular actions are mainly attributed to oestradiol and exerted by oestrogen receptors (ERα, ERβ and G protein-coupled oestrogen receptor), through rapid and/or genomic mechanisms, but these effects depend on ageing and inflammation. A cardiovascular approach in women's health has arisen due to controversy regarding oestrogen's beneficial impact as reported in experimental and observational studies and large randomized trials. These can be explained, in part, by two mutually non-exclusive hypotheses. On the one hand, the timing hypothesis, which stat…

0301 basic medicinemedicine.medical_specialtyEndotheliumPhysiologyMedicinaEstrogen receptorInflammationProstacyclin03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansEndothelial dysfunctionReceptorskin and connective tissue diseasesMolecular Structurebusiness.industryEstrogen Receptor alphaEstrogensmedicine.diseaseCardiovascular physiologyPostmenopause030104 developmental biologymedicine.anatomical_structureEndocrinologyAgeingFemaleEndothelium Vascularmedicine.symptombusiness030217 neurology & neurosurgerymedicine.drugThe Journal of physiologyReferences
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Oral Unsaturated Fat Load Impairs Postprandial Systemic Inflammation in Primary Hypercholesterolemia Patients.

2021

Context: Primary hypercholesterolemia (PH) is a lipid disorder characterized by elevated levels of cholesterol and low-density lipoprotein (LDL). Low-grade systemic inflammation is associated with PH, which might explain the higher incidence of cardiovascular diseases in this setting.Objective: To evaluate the effect of an oral unsaturated fat load (OUFL) on different immune parameters and functional consequences in patients with PH in postprandial state.Design: A commercial liquid preparation of long-chain triglycerides (Supracal®; ω6/ω3 ratio &amp;gt;20/1, OUFL) was administered to 20 patients and 10 age-matched controls. Whole blood was collected before (fasting state) and 4 h after admi…

0301 basic medicinemedicine.medical_specialtyEndotheliumleukocytesinflammatory mediatorsRM1-950030204 cardiovascular system & hematologySystemic inflammationendothelial dysfunction03 medical and health scienceschemistry.chemical_compound0302 clinical medicineoral unsaturated fat loadInternal medicineprimary hypercholesterolemiamedicineplatelet activationPharmacology (medical)Platelet activationEndothelial dysfunctionOriginal ResearchPharmacologysystemic inflammationCholesterolbusiness.industryUnsaturated fatmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurePostprandialchemistryTherapeutics. Pharmacologymedicine.symptombusinessLipoproteinFrontiers in pharmacology
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Algorithm for the management of patients at low, high and very high risk of osteoporotic fractures

2019

Summary Guidance is provided in an international setting on the assessment and specific treatment of postmenopausal women at low, high and very high risk of fragility fractures. Introduction The International Osteoporosis Foundation and European Society for Clinical and Economic Aspects of Osteoporosis and Osteoarthritis published guidance for the diagnosis and management of osteoporosis in 2019. This manuscript seeks to apply this in an international setting, taking additional account of further categorisation of increased risk of fracture, which may inform choice of therapeutic approach. Methods Clinical perspective and updated literature search. Results The following areas are reviewed: …

0301 basic medicinemedicine.medical_specialtyFRAXEndocrinology Diabetes and MetabolismeducationOsteoporosisPsychological interventioninhibitors of bone resorption030209 endocrinology & metabolismRisk AssessmentAnabolic agents03 medical and health sciencesTherapeutic approach0302 clinical medicineBone DensityRisk FactorsTreatment of osteoporosisInternal medicinemedicinefracture risk assessmentHumansanabolic agentsIntensive care medicineOsteoporosis PostmenopausalAgedAnabolic agents Fracture risk assessment FRAX Inhibitors of bone resorption Treatment of osteoporosisddc:616Postmenopausal womenbusiness.industryCorrectionFracture risk assessmentInhibitors of bone resorptionMiddle Agedtreatment of osteoporosismedicine.diseaseRheumatologyIncreased riskFemalePosition Paper030101 anatomy & morphologybusinessVery high riskAlgorithmsOsteoporotic FracturesFRAXOsteoporosis International
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Membrane Attack Complex in Myocardial Ischemia/Reperfusion Injury: A Systematic Review for Post Mortem Applications

2020

The complement system has a significant role in myocardial ischemia/reperfusion injury, being responsible for cell lysis and amplification of inflammatory response. In this context, several studies highlight that terminal complement complex C5b-9, also known as the membrane attack complex (MAC), is a significant contributor. The MAC functions were studied by many researchers analyzing the characteristics of its activation in myocardial infarction. Here, a systematic literature review was reported to evaluate the principal features, advantages, and limits (regarding the application) of complement components and MAC in post mortem settings to perform the diagnosis of myocardial ischemia/infar…

0301 basic medicinemedicine.medical_specialtyForensic pathologyClinical BiochemistryInfarctionContext (language use)Reviewischemia/reperfusion injury03 medical and health sciences0302 clinical medicineSettore MED/43 - Medicina LegaleInternal medicinemedicine030216 legal & forensic medicineMyocardial infarctioncomplement systemlcsh:R5-920business.industryC5b-9schemia/reperfusion injuryforensic pathologymedicine.diseaseComplement systempost mortem investigation030104 developmental biologymyocardial infarctionInclusion and exclusion criteriaCardiologyC5b-9; complement system; forensic pathology; ischemia/reperfusion injury; myocardial infarction; post mortem investigationbusinessComplement membrane attack complexlcsh:Medicine (General)Reperfusion injury
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Decreases in Circulating Concentrations of Long-Chain Acylcarnitines and Free Fatty Acids During the Glucose Tolerance Test Represent Tissue-Specific…

2019

Background: Insulin plays a pivotal role in the regulation of both carbohydrate and lipid intermediate turnover and metabolism. In the transition from a fasted to fed state, insulin action inhibits lipolysis in adipocytes, and acylcarnitine synthesis in the muscles and heart. The aim of this study was to measure free fatty acid (FFA) and acylcarnitine levels during the glucose tolerance test as indicators of tissue-specific insulin resistance. Results: Insulin release in response to glucose administration decreased both FFA and long-chain acylcarnitine levels in plasma in healthy control animals by 30% (120 min). The glucose tolerance test and [3H]-deoxy-D-glucose uptake in tissues revealed…

0301 basic medicinemedicine.medical_specialtyGlucose uptakemedicine.medical_treatmentEndocrinology Diabetes and MetabolismAdipose tissuelong-chain acylcarnitines030209 endocrinology & metabolismType 2 diabetesglucose tolerance testlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineinsulin resistancemedicineLipolysisOriginal ResearchGlucose tolerance testlcsh:RC648-665medicine.diagnostic_testChemistryInsulinfree fatty acidsmedicine.disease030104 developmental biologyEndocrinologyPostprandialtype 2 diabetesFrontiers in Endocrinology
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