Search results for "protein"

showing 10 items of 21431 documents

Crosstalk between receptor tyrosine kinases (RTKs) and G protein-coupled receptors (GPCR) in the brain: Focus on heteroreceptor complexes and related…

2019

Neuronal events are regulated by the integration of several complex signaling networks in which G protein-coupled receptors (GPCRs) and receptor tyrosine kinases (RTKs) are considered key players of an intense bidirectional cross-communication in the cell, generating signaling mechanisms that, at the same time, connect and diversify the traditional signal transduction pathways activated by the single receptor. For this receptor-receptor crosstalk, the two classes of receptors form heteroreceptor complexes resulting in RTKs transactivation and in growth-promoting signals. In this review, we describe heteroreceptor complexes between GPCR and RTKs in the central nervous system (CNS) and their …

0301 basic medicineG proteinRTKHeteroreceptorSettore BIO/09 - FisiologiaReceptor tyrosine kinaseReceptors G-Protein-Coupled03 medical and health sciencesCellular and Molecular NeuroscienceTransactivation0302 clinical medicineGPCRReceptor Fibroblast Growth Factor Type 1Receptor Fibroblast Growth Factor Type 2ReceptorG protein-coupled receptorPharmacologyTransactivationbiologyChemistryReceptor Protein-Tyrosine KinasesBrainReceptor Cross-TalkCrosstalk (biology)030104 developmental biologyHeteroreceptor complexebiology.proteinSignal transductionNeuroscience030217 neurology & neurosurgerySignal Transduction
researchProduct

The Amino-Terminal Domain of GRK5 Inhibits Cardiac Hypertrophy through the Regulation of Calcium-Calmodulin Dependent Transcription Factors.

2018

We have recently demonstrated that the amino-terminal domain of G protein coupled receptor kinase (GRK) type 5, (GRK5-NT) inhibits NFκB activity in cardiac cells leading to a significant amelioration of LVH. Since GRK5-NT is known to bind calmodulin, this study aimed to evaluate the functional role of GRK5-NT in the regulation of calcium-calmodulin-dependent transcription factors. We found that the overexpression of GRK5-NT in cardiomyoblasts significantly reduced the activation and the nuclear translocation of NFAT and its cofactor GATA-4 in response to phenylephrine (PE). These results were confirmed in vivo in spontaneously hypertensive rats (SHR), in which intramyocardial adenovirus-med…

0301 basic medicineG-Protein-Coupled Receptor Kinase 5MalecalmodulinMutantWistarPlasma protein binding030204 cardiovascular system & hematologyCatalysilcsh:ChemistryPhenylephrine0302 clinical medicineRats Inbred SHRMyocytes Cardiaclcsh:QH301-705.5SpectroscopybiologyChemistrycardiac hypertrophyNFATComputer Science Applications1707 Computer Vision and Pattern RecognitionGeneral MedicineLeft VentricularComputer Science ApplicationsCell biologycardiac hypertrophy; transcription factors; calmodulin; GRKGRKHypertrophy Left VentricularCardiacProtein BindingInbred SHRCalmodulinCalmodulin; Cardiac hypertrophy; GRK; Transcription factors; Animals; Binding Sites; Calmodulin; Cell Line; G-Protein-Coupled Receptor Kinase 5; GATA4 Transcription Factor; Hypertrophy Left Ventricular; Male; Myocytes Cardiac; NFATC Transcription Factors; Phenylephrine; Protein Binding; Rats; Rats Inbred SHR; Rats Wistar; Catalysis; Molecular Biology; Spectroscopy; Computer Science Applications1707 Computer Vision and Pattern Recognition; Physical and Theoretical Chemistry; Organic Chemistry; Inorganic ChemistryCatalysisArticleCell LineInorganic Chemistry03 medical and health sciencesG-Protein-Coupled Receptor Kinase 5transcription factorsAnimalsPhysical and Theoretical ChemistryRats WistarTranscription factorMolecular BiologyG protein-coupled receptor kinaseMyocytesBinding SitesNFATC Transcription FactorsOrganic ChemistryHypertrophyNFATC Transcription FactorsGATA4 Transcription FactorRats030104 developmental biologylcsh:Biology (General)lcsh:QD1-999biology.proteinTranscription factorInternational journal of molecular sciences
researchProduct

Transcriptional Changes after Enniatins A, A1, B and B1 Ingestion in Rat Stomach, Liver, Kidney and Lower Intestine

2021

Enniatins (ENs) are depsipeptide mycotoxins produced by Fusarium fungi. They are known for their capacity to modulate cell membrane permeability and disruption of ionic gradients, affecting cell homeostasis and initiating oxidative stress mechanisms. The effect of the acute toxicity of ENs A, A1, B and B1 at two different concentrations after 8 h of exposure was analysed in Wistar rats by a transcriptional approach. The following key mitochondrial and nuclear codified genes related to the electron transport chain were considered for gene expression analysis in stomach, liver, kidney and lower intestine by quantitative Real-Time PCR: mitochondrially encoded NADH dehydrogenase 1 (MT-ND1), mit…

0301 basic medicineGPX1Health (social science)oxidative phosphorylationPlant ScienceOxidative phosphorylationTP1-1185medicine.disease_causeOccludinHealth Professions (miscellaneous)Microbiologyquantitative Real-Time PCR (qPCR)Article03 medical and health sciences0404 agricultural biotechnologyenniatinsGene expressionmedicineCytochrome c oxidasebiologyChemistryenniatins; oxidative phosphorylation; in vivo; quantitative Real-Time PCR (qPCR)Succinate dehydrogenaseChemical technology04 agricultural and veterinary sciencesSalut pública040401 food scienceMolecular biologyHeme oxygenasein vivo030104 developmental biologybiology.proteinOxidative stressFood ScienceFoods
researchProduct

FRET biosensor allows spatio-temporal observation of shear stress-induced polar RhoGDIα activation

2018

Rho GDP-dissociation inhibitor α (RhoGDIα) is a known negative regulator of the Rho family that shuts off GDP/GTP cycling and cytoplasm/membrane translocation to regulate cell migration. However, to our knowledge, no reports are available that focus on how the RhoGDIα-Rho GTPases complex is activated by laminar flow through exploring the activation of RhoGDIα itself. Here, we constructed a new biosensor using fluorescence resonance energy transfer (FRET) technology to measure the spatio-temporal activation of RhoGDIα in its binding with Rho GTPases in living HeLa cells. Using this biosensor, we find that the dissociation of the RhoGDIα-Rho GTPases complex is increased by shear stress, and i…

0301 basic medicineGTP'ChemistryMedicine (miscellaneous)Cell migrationGTPasebiosensoritbiosensorsArticleGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences030104 developmental biology0302 clinical medicineFörster resonance energy transferlcsh:Biology (General)Cytoplasm030220 oncology & carcinogenesisMembrane fluidityBiophysicsGeneral Agricultural and Biological SciencesCytoskeletonlcsh:QH301-705.5Proto-oncogene tyrosine-protein kinase SrcCommunications Biology
researchProduct

Photoactivatable Adhesive Ligands for Light-Guided Neuronal Growth

2018

Neuro-regeneration after trauma requires growth and reconnection of neurons to reestablish information flow in particular directions across the damaged tissue. To support this process, biomaterials for nerve tissue regeneration need to provide spatial information to adhesion receptors on the cell membrane and to provide directionality to growing neurites. Here, photoactivatable adhesive peptides based on the CASIKVAVSADR laminin peptidomimetic are presented and applied to spatiotemporal control of neuronal growth to biomaterials in vitro. The introduction of a photoremovable group [6-nitroveratryl (NVOC), 3-(4,5-dimethoxy-2-nitrophenyl)butan-2-yl (DMNPB), or 2,2′-((3′-(1-hydroxypropan-2-yl)…

0301 basic medicineGUIDED AXON GROWTHNeuritePeptidomimeticNeurogenesisPeptideBiocompatible Materials02 engineering and technologyNEUROCHEMISTRYLigandsBiochemistryPHOTOTRIGGERED CELL ADHESIONCell membrane03 medical and health sciencesLamininmedicineCell AdhesionAnimalsAmino Acid SequenceCell adhesionMolecular BiologyCells Culturedchemistry.chemical_classificationNeuronsPhotolysisbiologyChemistryCELL ADHESIONOrganic ChemistryCiencias QuímicasAdhesion021001 nanoscience & nanotechnologyIn vitroLAMININ PEPTIDOMIMETICSMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureQuímica OrgánicaBiophysicsbiology.proteinMolecular MedicineLaminin0210 nano-technologyPeptidesCIENCIAS NATURALES Y EXACTAS
researchProduct

Redox signaling in the gastrointestinal tract.

2017

Redox signaling regulates physiological self-renewal, proliferation, migration and differentiation in gastrointestinal epithelium by modulating Wnt/β-catenin and Notch signaling pathways mainly through NADPH oxidases (NOXs). In the intestine, intracellular and extracellular thiol redox status modulates the proliferative potential of epithelial cells. Furthermore, commensal bacteria contribute to intestine epithelial homeostasis through NOX1- and dual oxidase 2-derived reactive oxygen species (ROS). The loss of redox homeostasis is involved in the pathogenesis and development of a wide diversity of gastrointestinal disorders, such as Barrett's esophagus, esophageal adenocarcinoma, peptic ulc…

0301 basic medicineGastrointestinal DiseasesNotch signaling pathwaymedicine.disease_causeBiochemistryGastrointestinal epitheliumSuperoxide dismutase03 medical and health scienceschemistry.chemical_compoundPhysiology (medical)medicineHumansSulfhydryl CompoundsIntestinal MucosaWnt Signaling PathwayCell Proliferationchemistry.chemical_classificationReactive oxygen speciesbiologySuperoxideWnt signaling pathwayNADPH OxidasesDual oxidase 2digestive system diseasesGastrointestinal TractIntestinesOxidative Stress030104 developmental biologychemistryImmunologybiology.proteinCancer researchReactive Oxygen SpeciesOxidation-ReductionOxidative stressFree radical biologymedicine
researchProduct

Bioactive potential of silica coatings and its effect on the adhesion of proteins to titanium implants

2018

There is an ever-increasing need to develop dental implants with ideal characteristics to achieve specific and desired biological response in the scope of improve the healing process post-implantation. Following that premise, enhancing and optimizing titanium implants through superficial treatments, like silica sol-gel hybrid coatings, are regarded as a route of future research in this area. These coatings change the physicochemical properties of the implant, ultimately affecting its biological characteristics. Sandblasted acid-etched titanium (SAE-Ti) and a silica hybrid sol-gel coating (35M35G30T) applied onto the Ti substrate were examined. The results of in vitro and in vivo tests and t…

0301 basic medicineGene Expression02 engineering and technologychemistry.chemical_compoundMiceColloid and Surface ChemistryCoatingCoated Materials Biocompatiblebone regenerationOsteogenesisTitaniumChemistrySurfaces and InterfacesGeneral MedicineAdhesion021001 nanoscience & nanotechnologySilicon DioxideRabbits0210 nano-technologyBiotechnologyTitaniumSilicon dioxideSurface Propertieschemistry.chemical_elementengineering.materialOsseointegrationPhase Transitionosteogenesis03 medical and health sciencesproteomicsIn vivoOsseointegrationCell Line Tumordental implantsAnimalsPhysical and Theoretical ChemistryBone regenerationDental ImplantsosteoimmunologyOsteoblastsTibiaInterleukin-6Complement System ProteinsAlkaline Phosphatase030104 developmental biologyengineeringBiophysicsImplantapolipoproteinsBiomarkers
researchProduct

ESC-Derived BDNF-Overexpressing Neural Progenitors Differentially Promote Recovery in Huntington's Disease Models by Enhanced Striatal Differentiation

2016

Summary Huntington's disease (HD) is characterized by fatal motoric failures induced by loss of striatal medium spiny neurons. Neuronal cell death has been linked to impaired expression and axonal transport of the neurotrophin BDNF (brain-derived neurotrophic factor). By transplanting embryonic stem cell-derived neural progenitors overexpressing BDNF, we combined cell replacement and BDNF supply as a potential HD therapy approach. Transplantation of purified neural progenitors was analyzed in a quinolinic acid (QA) chemical and two genetic HD mouse models (R6/2 and N171-82Q) on the basis of distinct behavioral parameters, including CatWalk gait analysis. Explicit rescue of motor function by…

0301 basic medicineGene ExpressionBiochemistrychemistry.chemical_compoundMice0302 clinical medicineNeural Stem CellsNeurotrophic factorsGenes Reporterlcsh:QH301-705.5Neuronslcsh:R5-920NeurogenesisCell DifferentiationAnatomyembryonic stem cellsHuntington Diseaselcsh:Medicine (General)NeurogliaLocomotionNeurotrophinHuntington’s diseaseCell SurvivalBiologyMedium spiny neuronArticle03 medical and health sciencesHuntington's diseaseGeneticsmedicinestriatal differentiationAnimalsBrain-derived neurotrophic factorBrain-Derived Neurotrophic FactorCell Biologymedicine.diseaseCorpus StriatumTransplantationDisease Models Animal030104 developmental biologylcsh:Biology (General)chemistrynervous systembiology.proteinNeuroscience030217 neurology & neurosurgeryBiomarkersDevelopmental BiologyQuinolinic acidStem Cell TransplantationStem Cell Reports
researchProduct

Oxidative stress-mediated alterations in histone post-translational modifications

2021

Abstract Epigenetic regulation of gene expression provides a finely tuned response capacity for cells when undergoing environmental changes. However, in the context of human physiology or disease, any cellular imbalance that modulates homeostasis has the potential to trigger molecular changes that result either in physiological adaptation to a new situation or pathological conditions. These effects are partly due to alterations in the functionality of epigenetic regulators, which cause long-term and often heritable changes in cell lineages. As such, free radicals resulting from unbalanced/extended oxidative stress have been proved to act as modulators of epigenetic agents, resulting in alte…

0301 basic medicineGene ExpressionContext (language use)Biologymedicine.disease_causeBiochemistryEpigenesis GeneticHistones03 medical and health sciences0302 clinical medicinePhysiology (medical)Gene expressionmedicineHumansHistone codeEpigeneticsRegulation of gene expressionDNA MethylationChromatinCell biologyOxidative Stress030104 developmental biologyHistonebiology.proteinProtein Processing Post-Translational030217 neurology & neurosurgeryOxidative stressFree Radical Biology and Medicine
researchProduct

Unicellular ancestry and mechanisms of diversification of Goodpasture antigen-binding protein.

2018

The emergence of the basement membrane (BM), a specialized form of extracellular matrix, was essential in the unicellular transition to multicellularity. However, the mechanism is unknown. Goodpasture antigen–binding protein (GPBP), a BM protein, was uniquely poised to play diverse roles in this transition owing to its multiple isoforms (GPBP-1, -2, and -3) with varied intracellular and extracellular functions (ceramide trafficker and protein kinase). We sought to determine the evolutionary origin of GPBP isoforms. Our findings reveal the presence of GPBP in unicellular protists, with GPBP-2 as the most ancient isoform. In vertebrates, GPBP-1 assumed extracellular function that is further e…

0301 basic medicineGene isoformBasement membrane030102 biochemistry & molecular biologyCell BiologyBiologyProtein Serine-Threonine KinasesBiochemistryBasement MembraneCell biologyExtracellular matrixEvolution MolecularIsoenzymes03 medical and health sciencesMulticellular organism030104 developmental biologymedicine.anatomical_structuremedicineExtracellularHumansEditors' PicksProtein kinase AMolecular BiologyFunction (biology)IntracellularThe Journal of biological chemistry
researchProduct