Search results for "wistar"

showing 10 items of 1094 documents

Modulation of aflatoxin B1 carcinogenicity, genotoxicity and metabolism in rat liver by dietary carotenoids: evidence for a protective effect of CYP1…

1997

The effects of several carotenoids of vitamin A and of 3-methylcholanthrene have been tested on the initiation of hepatocarcinogenesis by aflatoxin B1, using the sequential protocol of Solt and Farber. AFB1-induced DNA single-strand breaks and AFB1-metabolism were also assessed. The P4501A inducer carotenoids (canthaxanthin, astaxanthin, beta-apo-8'-carotenal) and 3-methylcholanthrene reduce the carcinogenicity of AFB1, divert AFB1-metabolism into the less genotoxic aflatoxin M1 and reduce AFB1-induced DNA single-strand breaks: we conclude that these carotenoids exert their protective effect through the deviation of AFB1 metabolism towards detoxification pathways. beta-Carotene decreased AF…

MaleVitaminCancer ResearchAflatoxinAflatoxin B1[SDV]Life Sciences [q-bio]MutagenBiologymedicine.disease_causeAntioxidants03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyAstaxanthinmedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACarotenoidCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesINDUCTIONfood and beverages04 agricultural and veterinary sciencesCarotenoids040401 food scienceDietRats3. Good health[SDV] Life Sciences [q-bio]LiverOncologychemistryBiochemistryCarcinogensRATCARCINOGENESEGenotoxicityDNA DamageMethylcholanthrene
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Dietary carotenoids inhibit aflatoxin B1-induced liver preneoplastic foci and DNA damage in the rat: Role of the modulation of aflatoxin B1 metabolism

1998

To study the effects of carotenoids on the initiation of liver carcinogenesis by aflatoxin B1 (AFB1), male weanling rats were fed beta-carotene, beta-apo-8'-carotenal, canthaxanthin, astaxanthin or lycopene (300 mg/kg diet), or an excess of vitamin A (21000 RE/kg diet), or were injected i.p. with 3-methylcholanthrene (3-MC) (6 x 20 mg/kg body wt) before and during i.p. treatment with AFB1 (2 x 1 mg/kg body wt). The rats were later submitted to 2-acetylaminofluorene treatment and partial hepatectomy, and placental glutathione S-transferase-positive liver foci were detected and quantified. The in vivo effects of carotenoids or of 3-MC on AFB1-induced liver DNA damage were evaluated using diff…

MaleVitaminCancer Researchmedicine.medical_specialtyAflatoxinAflatoxin B1DNA damage[SDV]Life Sciences [q-bio]Biology03 medical and health scienceschemistry.chemical_compoundLiver Neoplasms Experimental0302 clinical medicineAstaxanthinInternal medicinemedicineAnimalsAnticarcinogenic AgentsCanthaxanthinRats WistarVitamin ACYTOCHROME P 450CarotenoidSerum Albumin030304 developmental biologychemistry.chemical_classification0303 health sciencesRetinolGeneral MedicineCarotenoidsDietRats3. Good health[SDV] Life Sciences [q-bio]Endocrinologychemistry030220 oncology & carcinogenesisCarcinogensMicrosomes LiverRAT2-AcetylaminofluoreneCARCINOGENESEPrecancerous ConditionsDNA DamageMethylcholanthrene
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Dietary lycopene decreases the initiation of liver preneoplastic foci by diethylnitrosamine in the rat

1997

To test whether carotenoids can modulate the initiation of liver preneoplasia by diethylnitrosamine (DEN) or by 2-nitropropane (2-NP) in a sequential protocol of hepatocarcinogenesis, male weanling rats were fed for three or four weeks (respectively) diets containing beta-carotene, canthaxanthin, astaxanthin, or lycopene (300 mg/kg diet) or an excess of vitamin A (15,000 retinol equivalents/kg diet) or were treated intraperitoneally with 3-methylcholanthrene. During this period, all rats were injected intraperitoneally with the initiator carcinogen, either 2-NP (6 times at 100 mg/kg body wt) or DEN (once at 100 mg/kg body wt). Three weeks after the termination of carotenoid or vitamin A fee…

MaleVitaminCancer Researchmedicine.medical_specialtySTRUCTURE[SDV]Life Sciences [q-bio]Medicine (miscellaneous)WeanlingBiologyNitroparaffinsPropane03 medical and health scienceschemistry.chemical_compoundLycopene0302 clinical medicinebeta-CaroteneAstaxanthinInternal medicinemedicineAnimalsAnticarcinogenic AgentsDiethylnitrosamineCanthaxanthinRats Wistar030304 developmental biology0303 health sciencesNutrition and DieteticsLiver NeoplasmsRetinolRetinol EquivalentCarotenoidsLycopeneRats3. Good health[SDV] Life Sciences [q-bio]EndocrinologyOncologychemistry030220 oncology & carcinogenesisCarcinogensRATPrecancerous Conditions
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Vitamin E deficiency induces liver nuclear factor-κB DNA-binding activity and changes in related genes

2005

The biological functions of vitamin E have been classically attributed to its property as a potent inhibitor of lipid peroxidation in cellular membranes. However, in 1991, Azzi's group first described that alpha-tocopherol inhibits smooth muscle cell proliferation in a protein kinase C (PKC)-dependent way, demonstrating a non-antioxidant cell signalling function for vitamin E. More recently, the capacity of alpha-tocopherol to modulate gene expression with the implication of different transcription factors, beyond its antioxidant properties, has also been established. This study was to determine the effect of vitamin E-deficiency on liver nuclear factor-kappa B (NF-kappaB) DNA-binding activ…

MaleVitaminChromatin ImmunoprecipitationGlutamate-Cysteine Ligasemedicine.medical_treatmentBlotting WesternBiologyBiochemistrychemistry.chemical_compoundCyclin D1CyclinsMalondialdehydemedicineAnimalsVitamin EVitamin E DeficiencyRNA MessengerRats WistarTranscription factorVitamin EBody WeightNF-kappa BPromoterDNAGeneral MedicineCell cycleGlutathioneRatsCell biologyGene Expression RegulationLiverBiochemistrychemistryVitamin E deficiencyChromatin immunoprecipitationFree Radical Research
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Formulation, process conditions, and biological evaluation of dairy mixed gels containing fava bean and milk proteins: Effect on protein retention in…

2019

International audience; Food formulation and process conditions can indirectly influence AA digestibility and bioavailability. Here we investigated the effects of formulation and process conditions used in the manufacture of novel blended dairy gels (called "mixed gels" here) containing fava bean (Vicia faba) globular proteins on both protein composition and metabolism when given to young rats. Three mixed dairy gels containing casein micelles and fava bean proteins were produced either by chemical acidification (A) with glucono-δ-lactone (GDL) or by lactic acid fermentation. Fermented gels containing casein and fava bean proteins were produced without (F) or with (FW) whey proteins. The AA…

MaleWhey proteinProtein efficiency ratioFood Handling[SDV]Life Sciences [q-bio]chemistry.chemical_compoundCaseinLeguminDenaturation (biochemistry)Food scienceAmino AcidsPlant Proteins2. Zero hunger0303 health sciencesChemistry[SDV.BA]Life Sciences [q-bio]/Animal biologyCaseinsfood and beverages04 agricultural and veterinary sciencesHydrogen-Ion ConcentrationMilk ProteinsLactic acidVicia fabaProtéines de fèvesDigestionDietary ProteinsNutritive ValueLactic acid fermentationQualité des protéines alimentairefava bean proteinBiological AvailabilitygelationMélange de protéinesprotein aggregation03 medical and health sciencesGélificationmilk proteinGeneticsAnimalsRats Wistar030304 developmental biology0402 animal and dairy sciencedietary protein quality040201 dairy & animal scienceRatsWhey ProteinsFermentationAnimal Science and ZoologyDairy ProductsProtéines du lait[SDV.AEN]Life Sciences [q-bio]/Food and NutritionProtein qualityGelsFood Science
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Inhibition of Xanthine Oxidase by Phenolic Conjugates of Methylated Quinic Acid

2003

The caffeoyl conjugates of prenylhydroquinone glucoside and of quinic acid, either in the carboxyl-free or carboxymethyl forms, isolated from Phagnalon rupestre (Asteraceae), showed inhibitory activity on lipid peroxidation induced by Fe 2+/ascorbate and by CCl4/NADPH in rat liver microsomes, with IC50 values ranging from 3 to 11 microM. After having demonstrated their effect on the xanthine oxidase-regulated superoxide production, the active compounds were tested for the direct inhibition of this enzyme. Methylated dicaffeoylquinic conjugates competitively inhibited the enzyme and the highest potency was obtained for the 4,5-diester, with an IC50 value of 3.6 microM, nearly ten times lower…

MaleXanthine OxidaseAntioxidantStereochemistrymedicine.medical_treatmentQuinic AcidPharmaceutical ScienceAsteraceaeAntioxidantsAnalytical ChemistryLipid peroxidationInhibitory Concentration 50chemistry.chemical_compoundPhenolsGlucosideDrug DiscoverymedicineAnimalsEnzyme InhibitorsRats WistarXanthine oxidasePharmacologybiologyPlant ExtractsSuperoxideOrganic ChemistryQuinic acidXanthineHydroquinonesRatsComplementary and alternative medicinechemistryBiochemistryEnzyme inhibitorMicrosomes Liverbiology.proteinMolecular MedicineLipid PeroxidationPhytotherapyPlanta Medica
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Expression and differential localization of xenobiotic transporters in the rat olfactory neuro-epithelium.

2011

International audience; Transporters, such as multidrug resistance P-glycoproteins (MDR), multidrug resistance-related proteins (MRP) and organic anion transporters (OATs), are involved in xenobiotic metabolism, particularly the cellular uptake or efflux of xenobiotics (and endobiotics) or their metabolites. The olfactory epithelium is exposed to both inhaled xenobiotics and those coming from systemic circulation. This tissue has been described as a pathway for xenobiotics to the brain via olfactory perineural space. Thereby, olfactory transporters and xenobiotic metabolizing enzymes, dedicated to the inactivation and the elimination of xenobiotics, have been involved in the toxicological p…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Multidrug Resistance-Associated Proteinsp glycoproteinATP-binding cassette transporterMESH : HepatocytesReceptors OdorantMESH : P-GlycoproteinMESH: HepatocytesMESH : Lymphatic Vessels0302 clinical medicineMESH : Protein Transportugt2a1MESH: SmellMESH: Receptors OdorantMESH: AnimalsReceptorxenobiotic metabolizingmucosa0303 health sciencesMESH : Gene Expression RegulationMESH : RatsGeneral NeuroscienceMESH : OdorsMESH: Gene Expression RegulationSmellProtein Transportmedicine.anatomical_structureBiochemistryLivertransporterbarrierEffluxMultidrug Resistance-Associated ProteinsMESH: Multidrug Resistance-Associated ProteinsMESH: XenobioticsMESH: Protein TransportMESH: P-GlycoproteinMESH: RatsMESH: Lymphatic VesselsMESH : Maleodorant clearancebrainMESH : XenobioticsxenobioticBiologysystemMESH : Rats WistarOlfactory Receptor NeuronsXenobiotics03 medical and health sciencesbulbOlfactory Mucosamultidrug resistanceMESH : Receptors OdorantmedicineAnimalsATP Binding Cassette Transporter Subfamily B Member 1Rats WistardetoxificationMESH: Olfactory Mucosa030304 developmental biologyLymphatic VesselsMESH : Olfactory MucosaMESH: OdorsMESH : LiverTransporterMESH: Rats WistarMESH: Olfactory Receptor NeuronsEpitheliumMESH: MaleOlfactory bulbRatsenzymeGene Expression RegulationOdorantsHepatocytesMESH : SmellMESH : Olfactory Receptor NeuronsMESH : Animalsolfactory epitheliumOlfactory epitheliumperireceptor event[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDrug metabolismMESH: Liver
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A multi-generational study on low-dose BPA exposure in Wistar rats: Effects on maternal behavior, flavor intake and development

2012

Bisphenol A (BPA) is a common endocrine disruptor found as an environmental and food contaminant. It exerts both developmental and behavioral effects, mainly when exposure occurs in early life. The aim of this study was to determine the multi-generational effects of chronic, human-relevant low-dose exposure to BPA on development, maternal behavior and flavor preference in Wistar rats. BPA was orally administered at a daily dose of 5 mu g/kg body weight to FO pregnant dams from the first day of gestation (GD 1) until the last day of lactation (LD 21), and then to Fl offspring from weaning (PND 21) to adulthood (PND 100). F2 offspring were not exposed. Development and clinical signs of toxici…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutrition[ SDV.TOX ] Life Sciences [q-bio]/ToxicologyToxicology[ SDV.BA ] Life Sciences [q-bio]/Animal biologyEatingPregnancyLactationBirth RateMaternal BehaviorPerinatal ExposureChemistryTaste preferencesBISPHENOL-A EXPOSURE[SDV.BA]Life Sciences [q-bio]/Animal biologyAnogenital distanceAge FactorsDIETARY EXPOSUREmedicine.anatomical_structureEndocrine disruptorEndocrine disruptorPrenatal Exposure Delayed EffectsENVIRONMENTALLY RELEVANT LEVELS[SDV.TOX]Life Sciences [q-bio]/ToxicologyToxicityMalformationsFemaleCD-1 MICEReproductive toxicityPERINATAL EXPOSUREmedicine.medical_specialtyendocrine systemSEX-DIFFERENCESOffspringGestational AgeAir Pollutants OccupationalREPRODUCTIVE TOXICITYSEXUALLY DIMORPHIC BEHAVIORSFood PreferencesCellular and Molecular NeurosciencePhenolsDevelopmental NeuroscienceInternal medicinemedicineAnimalsWeaningSex RatioBenzhydryl CompoundsRats WistarSPRAGUE-DAWLEY RATSOFFSPRING TOXICITYBody WeightRatsFlavoring AgentsEndocrinologyAnimals NewbornF2 body weight change[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Induction of cytochrome P450 and/or detoxication enzymes by various extracts or rosemary: description of specific patterns

2001

The ability of rosemary to modulate cytochrome P450 (CYP) and detoxication enzymes in rat liver was evaluated by comparing the effects of dried leaves and leaf extracts with different chemical compositions: essential oil (EO) containing monoterpenes, a dichloromethane extract (DCME) containing phenolic diterpenes and a water-soluble extract (WSE) containing phenolic compounds such as rosmarinic acid and flavonoids. Chemical analyses were done in order to characterize the composition of extracts. Male Wistar rats received the leaves or extracts of rosemary in their diet at 0.5% (w/w) for 2 weeks. The effects of such treatments were evaluated for CYP (1A, 2B, 2E1), glutathione S-transferase (…

Male[SDV]Life Sciences [q-bio]ReductaseToxicologychemistry.chemical_compoundCytosol0302 clinical medicineCytochrome P-450 Enzyme System[SDV.IDA]Life Sciences [q-bio]/Food engineeringCYTOCHROME P 450AnticarcinogenComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationGLUTATHIONE S-TRANSFERASE0303 health sciencesbiologyReverse Transcriptase Polymerase Chain ReactionChemistryRosmarinic acidOrgan SizeGeneral Medicine[SDV.IDA] Life Sciences [q-bio]/Food engineeringSpecific Pathogen-Free Organisms[SDV] Life Sciences [q-bio]LiverBiochemistryEnzyme Induction030220 oncology & carcinogenesisMicrosomes Liver[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringImmunoblottingChemopreventiondigestive system03 medical and health sciencesAnimals[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringRNA MessengerRats Wistar030304 developmental biologyLamiaceaePlant ExtractsBody WeightROMARINCytochrome P450GlutathioneUDP-GLUCURONOSYLTRANSFERASENAD(P)H Dehydrogenase (Quinone)RatsEnzymeMicrosomebiology.proteinRATFood Science
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Fibroblast growth factor-2 and its receptor expression in proliferating precursor cells of the subventricular zone in the adult rat brain

2008

Several findings have suggested the existence in the subventricular zone (SVZ) from sagittal sections of adult rat brain of a trophic mechanism, mediated by fibroblast growth factor-2 (FGF-2) and its multiple high-affinity FGF receptors (FGFRs), regulating neurogenesis mainly by controlling precursor cell proliferation. However, no clear data are available on the expression of FGF-2 and FGFRs in proliferating precursor cells of the SVZ. To address these questions we examined FGF-2 mRNA and its FGFR mRNA expression in proliferating precursor cells of the SVZ by using a double labeling procedure, combining in situ hybridization for FGF-2 and its FGFR mRNA with immunohistochemistry for bromode…

Maleanimal diseasesReceptor expressionGene ExpressionFGF-2Subventricular zoneSVZBiologySettore BIO/09 - FisiologiaCerebral Ventricleschemistry.chemical_compoundPrecursor cellmedicineAnimalsReceptor Fibroblast Growth Factor Type 3RNA MessengerReceptor Fibroblast Growth Factor Type 1Rats WistarReceptor Fibroblast Growth Factor Type 2BrdUCell ProliferationFGF receptorGeneral NeuroscienceFibroblast growth factor receptor 1NeurogenesisBrainPrecursor cellsFibroblast growth factor receptor 4RatsCell biologyAdult Stem CellsFGF receptorsmedicine.anatomical_structureBromodeoxyuridinenervous systemchemistryFibroblast growth factor receptorFibroblast Growth Factor 2NeuroscienceBromodeoxyuridineBrdU; FGF receptors; FGF-2Neuroscience Letters
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