0000000000874816

AUTHOR

J. Majdandzic

showing 3 related works from this author

Hypokalemic rhabdomyolysis associated with Bartter's syndrome.

1983

Severe potassium deficiency is an uncommon cause of rhabdomyolysis. We recently treated a 45-year-old patient with myalgia, serious generalized weakness, increased serum creatine kinase and myoglobin level as well as excessive hypokalemia. Histological examination of deltoid muscle biopsy showed rhabdomyolysis. After complete recovery of muscle damage by potassium substitution Bartter's syndrome proved to be the cause of initial and persistent hypokalemia.

myalgiaMalemedicine.medical_specialtyendocrine system diseasesHypokalemiaurologic and male genital diseasesGastroenterologyNecrosisInternal medicineDeltoid muscleDrug DiscoveryBiopsyHyperaldosteronismmedicineHumansCreatine KinaseGenetics (clinical)Histological examinationmedicine.diagnostic_testbusiness.industryMyoglobinMusclesMyoglobinuriaBartter SyndromeGeneral MedicineMiddle Agedmedicine.diseaseHypokalemiaBartter's syndromeAnesthesiaPotassiumMolecular MedicinePotassium deficiencymedicine.symptombusinessRhabdomyolysisKlinische Wochenschrift
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Verbesserung der Prognose bei Langzeitbeatmung durch frühzeitigen Einsatz von PEEP

1981

Bei 127 Patienten mit beatmungsbedurftiger akuter respiratorischer Insuffizienz wurde der Zusammenhang zwischen Zeitpunkt des Einsatzes der PEEP Beatmung und Behandlungergebnis untersucht. Bei 61 Patienten wurde in konventioneller Weise zunachst mit ZEEP beatmet und auf PEEP ubergegangen, wenn die inspiratorisch-arterielle O2-Differenz sich vergroserte, PaO2 bei FiO2≧0,5 unter 60 mm Hg lag, die radiologischen Lungenveranderungen zunahmen, oder eine Verschlechterung des Allgemeinzustandes offenkundig war (Gruppe I=sekundarer PEEP, zeitliche Latenz zwischen Intubation und PEEP im Mittel 46±47 Std., Medianwert 33 h). 66 Patienten wurden bei gegebener Indikation zur Beatmung unabhangig von dies…

Gynecologymedicine.medical_specialtybusiness.industryDrug DiscoveryMolecular MedicineMedicineGeneral MedicinebusinessGenetics (clinical)Klinische Wochenschrift
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Successful treatment of paraquat poisoning: activated charcoal per os and "continuous hemoperfusion".

1982

AbstractIngestion of paraquat results in an extremely dangerous poisoning. The first aim is to clear the gastrointestinal tract by inducing emesis and performing gastric/gut lavage; as much activated charcoal as possible should be administered per os and as quickly as possible. The best measure to eliminate paraquat from blood and tissue is hemoperfusion with coated activated charcoal; it has to be performed in the sense of “continuous hemoperfusion” about 8 h/d over a period of 2–3 weeks. These measures give a chance to lower the lethality of paraquat poisoning.

inorganic chemicalsMaleParaquatTime FactorsAdolescentHealth Toxicology and Mutagenesismedicine.medical_treatmentMagnesium CompoundsToxicologychemistry.chemical_compoundParaquatMedicineAnimalsHumansAluminum CompoundsKaolinTherapeutic IrrigationGastrointestinal tractbusiness.industrySilicatesRats Inbred StrainsHemoperfusionPARAQUAT POISONINGRatsHemoperfusionchemistryActivated charcoalAnesthesiaCharcoalBentoniteAluminum SilicatesFemaleAdsorptionbusinessJournal of toxicology. Clinical toxicology
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