Differential expression of PGC-1α and metabolic sensors suggest age-dependent induction of mitochondrial biogenesis in Friedreich ataxia fibroblasts.

6533b82bfe1ef96bd128e121

RESEARCH PRODUCT

Differential expression of PGC-1α and metabolic sensors suggest age-dependent induction of mitochondrial biogenesis in Friedreich ataxia fibroblasts.

Belén Mollá Federico V. Pallardó Pilar Gonzalez-cabo Arantxa Bolinches-amorós Francisco Dasí José Luis García-jiménez Francesc Palau Amparo Gimeno

subject

Male Aging Mitochondrial Diseases Mitochondrial Myopathy Ubiquinone Cardiomyopathy lcsh:Medicine Mitochondrion AMP-Activated Protein Kinases p38 Mitogen-Activated Protein Kinases Antioxidants Adenosine Triphosphate AMP-activated protein kinase Trinucleotide Repeats Fibrosis Molecular Cell Biology Child lcsh:Science Heat-Shock Proteins Regulation of gene expression Multidisciplinary Movement Disorders biology Neuromuscular Diseases Middle Aged Catalase Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha Cell biology Mitochondria DNA-Binding Proteins Neurology Disease Progression Medicine Female medicine.symptom Signal Transduction Research Article Adult congenital hereditary and neonatal diseases and abnormalities Ataxia Adolescent Mitochondrial Proteins medicine Genetics Humans Biology Alleles Glutathione Peroxidase Superoxide Dismutase lcsh:R Human Genetics Fibroblasts medicine.disease Molecular biology Oxidative Stress Mitochondrial biogenesis Gene Expression Regulation Friedreich Ataxia biology.protein Frataxin lcsh:Q Energy Metabolism Reactive Oxygen Species Transcription Factors

description

11 pages, 6 figures. PMID:21687738[PubMed] PMCID: PMC3110204

year	journal	country	edition	language
2011-06-07	PLoS ONE

10.1371/journal.pone.0020666 http://europepmc.org/articles/PMC3110204?pdf=render

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