Search results for " microbiome"

showing 10 items of 253 documents

Health and Disease Imprinted in the Time Variability of the Human Microbiome

2017

The human microbiota correlates closely with the health status of its host. This article analyzes the microbial composition of several subjects under different conditions over time spans that ranged from days to months. Using the Langevin equation as the basis of our mathematical framework to evaluate microbial temporal stability, we proved that stable microbiotas can be distinguished from unstable microbiotas. This initial step will help us to determine how temporal microbiota stability is related to a subject’s health status and to develop a more comprehensive framework that will provide greater insight into this complex system.

0301 basic medicineScaling lawPhysiologySystems biologyPopulationlcsh:QR1-502microbiomeDiseaseGut floraBiochemistryMicrobiologylcsh:MicrobiologyHost-Microbe Biology03 medical and health sciences0302 clinical medicineGeneticsMicrobiomeeducationMolecular BiologyEcology Evolution Behavior and Systematicseducation.field_of_studymetagenomicsbiologyHuman microbiomesystems biologystabilitybiology.organism_classificationEditor's PickQR1-502Computer Science Applications030104 developmental biologyEvolutionary biologyMetagenomicsModeling and Simulationecological modeling030217 neurology & neurosurgeryResearch ArticlemSystems
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Gut microbiota and aging-A focus on centenarians.

2020

Gut microbiota (GM) is a dynamic organ throughout the lifespan. Aging is a complex process that comprises a plethora of mechanisms such as senescence, immunosenescence and inflammaging, representing important pathways of age-related diseases. GM structure could both influence and be influenced by aging occurring changes within the host. A unique category of long living individuals exists, namely centenarians that have the outstanding capacity to adapt to various challenges. Longevity seems to be associated with certain GM which, among other factors, might render individuals more resistant to age-related diseases and subsequently to long living. Diet, prebiotics, probiotics and synbiotics ma…

0301 basic medicineSenescenceAgingSynbioticsmedia_common.quotation_subjectLongevitySynbioticsGut microbiotaBiologyGut flora03 medical and health sciences0302 clinical medicineCentenariansHumansAging Centenarians Gut microbiota Prebiotics Probiotics Synbiotics Aged 80 and over Aging Gastrointestinal Microbiome Humans Longevity Prebiotics Probiotics SynbioticsMolecular Biologymedia_commonAged 80 and overProbioticsLongevityImmunosenescencebiology.organism_classificationGastrointestinal Microbiome030104 developmental biologyPrebioticsImmunologyMolecular Medicine030217 neurology & neurosurgery
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Gut microbiota and cancer: How gut microbiota modulates activity, efficacy and toxicity of antitumoral therapy

2019

Gut microbiota is involved in gastrointestinal carcinogenesis. Also, it modulates the activity, efficacy and toxicity of several chemotherapy agents, such as gemcitabine, cyclophosphamide, irinotecan, cisplatin and 5-Fluorouracil, and target therapy, such as tyrosine kinase inhibitors. More recently, accumulating data suggest that the composition of gut microbiota may also affect efficacy and toxicity of cancer immunotherapy. Therefore, the manipulation of gut microbiota through antibiotics, probiotics, prebiotics or fecal transplantation has been investigating with the aim to improve efficacy and mitigate toxicity of anticancer drugs.

0301 basic medicineSettore MED/06 - Oncologia Medicamedicine.drug_class5-Fluorouracilmedicine.medical_treatmentAntibioticsAntineoplastic AgentsImmune checkpoint inhibitorGut floraPharmacologyIrinotecandigestive systemImmune checkpoint inhibitors03 medical and health sciences0302 clinical medicineCancer immunotherapyNeoplasmsmedicineAnimalsHumansCyclophosphamide5-Fluorouracil; Cisplatin; Cyclophosphamide; Gemcitabine; Immune checkpoint inhibitors; Irinotecan; Microbiota; Tyrosine kinase inhibitorsTyrosine kinase inhibitorsChemotherapybiologybusiness.industryMicrobiotaCancerHematologyFecal Microbiota Transplantationbiology.organism_classificationmedicine.diseaseGemcitabineGemcitabineGastrointestinal MicrobiomeIrinotecan030104 developmental biologyOncology030220 oncology & carcinogenesisToxicityImmunotherapyCisplatinbusinessmedicine.drugCritical Reviews in Oncology/Hematology
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NUTRITION, OXIDATIVE STRESS AND INTESTINAL DYSBIOSIS: INFLUENCE OF DIET ON GUT MICROBIOTA IN INFLAMMATORY BOWEL DISEASES.

2016

Background: Microbiota refers to the population of microorganisms (bacteria, viruses and fungi) that inhabit the entire gastrointestinal tract, more particularly the colon whose role is to maintain the integrity of the intestinal mucosa and control the proliferation of pathogenic bacteria. Alteration in the composition of the gut microbiota is called dysbiosis. Dysbiosis redisposes to inflammatory bowel diseases such as ulcerative colitis, Crohn disease and indeterminate colitis. Methods: The purpose of this literature review is to elucidate the influence of diet on the composition of the gastrointestinal microbiota in the healthy gut and the role of diet in the development of dysbiosis. Co…

0301 basic medicineSettore MED/09 - Medicina InternaPopulationhealthy dietlcsh:MedicineNutritional Statuscolorectal cancerGut floramedicine.disease_causeintestinal dysbiosisInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyMicrobiology03 medical and health sciences0302 clinical medicineIntestinal mucosaInflammatory Bowel diseases colorectal cancer intestinal dysbiosis gut microbiota MALT healthy diet.medicineOily fishHumanseducationeducation.field_of_studybiologygut microbiotabusiness.industrylcsh:RPathogenic bacteriamedicine.diseasebiology.organism_classificationInflammatory Bowel DiseasesUlcerative colitisDietGastrointestinal MicrobiomeOxidative StressSettore MED/18 - Chirurgia Generale030104 developmental biology030220 oncology & carcinogenesisImmunologyDysbiosisbusinessDysbiosis
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The role of the gastrointestinal tract in the pathogenesis of rheumatic diseases

2016

Dysregulation of the intestinal epithelial barrier in genetically susceptible individuals may lead to both intestinal and extraintestinal autoimmune disorders. There is emerging literature on the role of microbiota changes in the pathogenesis of systemic rheumatic diseases such as rheumatoid arthritis, spondyloarthropathies, and connective tissue diseases. Although the role of the gastrointestinal tract in the pathogenesis of spondyloartropathies is well defined and many studies underline the importance of gastrointestinal inflammation in modulating local and systemic inflammation, the data are inconclusive regarding the effect of dysbiosis on rheumatoid arthritis and connective tissue dise…

0301 basic medicineSpondyloarthropathyConnective tissueSystemic inflammationPathogenesis03 medical and health sciences0302 clinical medicineRheumatologyRheumatic DiseasesmedicineHumans030203 arthritis & rheumatologyEpithelial barrierGastrointestinal tractbusiness.industryRheumatoid arthritis Spondyloarthropathy Connective tissue diseases Gastrointestinal tract Dysbiosismedicine.diseaseGastrointestinal MicrobiomeSettore MED/16 - Reumatologia030104 developmental biologymedicine.anatomical_structureRheumatoid arthritisImmunologyDysbiosismedicine.symptombusinessDysbiosis
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Bronchial inflammation and bacterial load in stable COPD is associated with TLR4 overexpression.

2017

Toll-like receptors (TLRs) and nucleotide-binding oligomerisation domain (NOD)-like receptors (NLRs) are two major forms of innate immune sensors but their role in the immunopathology of stable chronic obstructive pulmonary disease (COPD) is incompletely studied. Our objective here was to investigate TLR and NLR signalling pathways in the bronchial mucosa in stable COPD.Using immunohistochemistry, the expression levels of TLR2, TLR4, TLR9, NOD1, NOD2, CD14, myeloid differentiation primary response gene 88 (MyD88), Toll-interleukin-1 receptor domain-containing adaptor protein (TIRAP), and the interleukin-1 receptor-associated kinases phospho-IRAK1 and IRAK4 were measured in the bronchial muc…

0301 basic medicineTIRAPMaleRespiratory SystemVital CapacityHAEMOPHILUS-INFLUENZAELUNG MICROBIOMEPathogenesisPulmonary Disease Chronic Obstructive0302 clinical medicineNOD2ImmunopathologyForced Expiratory VolumeNod1 Signaling Adaptor ProteinNOD1PhosphorylationCOPDSmoking11 Medical And Health SciencesMiddle AgedCPG-DNAbronchial inflammationAnti-Bacterial AgentsStreptococcus pneumoniaePseudomonas aeruginosaMOUSE LUNGFemaleLife Sciences & BiomedicineMoraxella catarrhalisSignal TransductionEXPRESSIONPulmonary and Respiratory MedicineCD14BronchiRespiratory MucosaReal-Time Polymerase Chain ReactionOBSTRUCTIVE PULMONARY-DISEASETLRs NLR bronchial inflammationNLRDENDRITIC CELL SUBSETS03 medical and health sciencesProtein DomainsmedicineHumansTLRsAgedTOLL-LIKE RECEPTORSCOPD TLR4InflammationScience & TechnologyBacteriabusiness.industrymedicine.diseaseHaemophilus influenzaeBacterial Loadrespiratory tract diseasesToll-Like Receptor 4TLR2030104 developmental biology030228 respiratory systemImmunologyINNATE IMMUNITYT-CELLSbusinessThe European respiratory journal
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Resveratrol and the Interaction between Gut Microbiota and Arterial Remodelling

2020

Arterial remodelling refers to the alteration in the structure of blood vessel that contributes to the progression of hypertension and other cardiovascular complications. Arterial remodelling is orchestrated by the crosstalk between the endothelium and vascular smooth muscle cells (VSMC). Vascular inflammation participates in arterial remodelling. Resveratrol is a natural polyphenol that possesses anti-oxidant and anti-inflammatory properties and has beneficial effects in both the endothelium and VSMC. Resveratrol has been studied for the protective effects in arterial remodelling and gut microbiota, respectively. Gut microbiota plays a critical role in the immune system and inflammatory pr…

0301 basic medicineVascular smooth muscleEndotheliumMyocytes Smooth MusclePopulationlcsh:TX341-641InflammationReviewresveratrolVascular Remodeling030204 cardiovascular system & hematologyResveratrolGut floraPharmacologydigestive systemAntioxidantsMuscle Smooth VascularVascular remodelling in the embryo03 medical and health scienceschemistry.chemical_compound0302 clinical medicinemedicineAnimalsHumanseducationCell ProliferationNeointimal hyperplasiaeducation.field_of_studyNutrition and Dieteticsgut microbiotabiologybusiness.industryanti-oxidantSIRT1 arterial remodellingArteriesmedicine.diseasebiology.organism_classificationGastrointestinal Microbiome030104 developmental biologymedicine.anatomical_structurechemistryinflammationcardiovascular systemmedicine.symptombusinesslcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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A widely used sampling device in colorectal cancer screening programmes allows for large-scale microbiome studies.

2018

We read with interest the article by Passamonti et al ,1 reporting the performance of two different faecal immunochemical tests (FITs) highlighting the importance of standardisation and validation of screening methodologies. Conventionally, laboratory-based FIT is the preferred approach in testing for occult blood in faeces, which includes colorectal cancer screening programmes.2–4 The potential of preserving stable faecal samples in a widely used FIT buffer for microbiome research would enable prospective microbiome studies in generally healthy subjects undergoing colorectal cancer screening. For this purpose, we evaluated faecal sample stability in the commonly used OC-Sensor (Eiken Chemi…

0301 basic medicineVeterinary medicine2312BiologySampling device03 medical and health sciencesHemoglobins0302 clinical medicineHumansMass Screening1506Microbiomecolonic microfloraEarly Detection of CancerMicrobiotaGastroenterologyHealthy subjectsIllumina miseqIon semiconductor sequencingPostScriptSample stabilityGastrointestinal Microbiome030104 developmental biologyColorectal cancer screeningMetagenomicsOccult Bloodepidemiology030211 gastroenterology & hepatologyGuaiacColorectal NeoplasmsGut
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Systematic gene overexpression in Candida albicans identifies a regulator of early adaptation to the mammalian gut.

2018

International audience; Candida albicans is part of the human gastrointestinal (GI) microbiota. To better understand how C. albicans efficiently establishes GI colonisation, we competitively challenged growth of 572 signature-tagged strains (~10% genome coverage), each conditionally overexpressing a single gene, in the murine gut. We identified CRZ2, a transcription factor whose overexpression and deletion respectively increased and decreased early GI colonisation. Using clues from genome-wide expression and gene-set enrichment analyses, we found that the optimal activity of Crz2p occurs under hypoxia at 37°C, as evidenced by both phenotypic and transcriptomic analyses following CRZ2 geneti…

0301 basic medicine[SDV.MHEP.AHA] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]030106 microbiologyImmunologyMicrobiologyMannosyltransferasesBiological pathwayTranscriptomeFungal ProteinsMannans03 medical and health scienceschemistry.chemical_compoundtranscriptomicsregulatory networksCell WallVirologyGene Expression Regulation FungalCandida albicanssignature‐tagged overexpression[SDV.MHEP.AHA]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]AnimalsGene Regulatory NetworksCandida albicansPromoter Regions GeneticGeneTranscription factorResearch ArticlesFungal proteinMice Inbred BALB CCRZ2chromatin immunoprecipitation‐on‐chipbiologyCRZ2;Candida albicans;chromatin immunoprecipitation-on-chip;gastrointestinal colonisation;regulatory networks;signature-tagged overexpression;transcriptomicsTunicamycinTunicamycinHydrogen-Ion Concentrationbiology.organism_classificationPhenotypeCell biologyGastrointestinal MicrobiomeGastrointestinal Tractchemistrychromatin immunoprecipitation-on-chipFemalesignature-tagged overexpressionMicroorganisms Genetically-Modifiedgastrointestinal colonisationResearch Article
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N-3 polyunsaturated fatty acids: An innovative strategy against obesity and related metabolic disorders, intestinal alteration and gut microbiota dys…

2019

Obesity is now widely recognized to be associated with low-grade systemic inflammation. It has been shown that high-fat feeding modulates gut microbiota which strongly increased intestinal permeability leading to lipopolysaccharide absorption causing metabolic endotoxemia that triggers inflammation and metabolic disorders. N-3 polyunsaturated fatty acids (PUFAs) have been shown associated with anti-obesity properties, but results still remain heterogeneous and very few studies underlined the metabolic pathways involved. Thus, the use of Fat-1 transgenic mice allows to better understanding whether endogenous n-3 PUFAs enrichment contributes to obesity and associated metabolic disorders preve…

0301 basic medicine[SDV]Life Sciences [q-bio]PopulationGut floradigestive systemBiochemistry03 medical and health sciencesMiceMetabolic DiseasesFatty Acids Omega-3medicineAnimalsHumansObesityeducation2. Zero hungerchemistry.chemical_classificationeducation.field_of_studyIntestinal permeability030102 biochemistry & molecular biologybiologyAkkermansiaGeneral Medicinebiology.organism_classificationmedicine.disease3. Good healthGastrointestinal MicrobiomeMetabolic pathway030104 developmental biologychemistryImmunologyDysbiosisMetabolic syndromeDysbiosisPolyunsaturated fatty acid
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