Search results for "Adaptive Immunity"

showing 10 items of 69 documents

Regulatory T Cells and IL-10 Independently Counterregulate Cytotoxic T Lymphocyte Responses Induced by Transcutaneous Immunization

2011

Background: The imidazoquinoline derivate imiquimod induces inflammatory responses and protection against transplanted tumors when applied to the skin in combination with a cognate peptide epitope (transcutaneous immunization, TCI). Here we investigated the role of regulatory T cells (Treg) and the suppressive cytokine IL-10 in restricting TCI-induced cytotoxic T lymphocyte (CTL) responses. Methodology/Principal Findings: TCI was performed with an ointment containing the TLR7 agonist imiquimod and a CTL epitope was applied to the depilated back skin of C57BL/6 mice. Using specific antibodies and FoxP3-diphteria toxin receptor transgenic (DEREG) mice, we interrogated inhibiting factors after…

Mouselcsh:MedicineEpitopes T-LymphocyteAdaptive ImmunityT-Lymphocytes RegulatoryImmune toleranceMiceMedicineCytotoxic T celllcsh:ScienceImmune ResponseSkinMice KnockoutB-LymphocytesMultidisciplinaryImiquimodFOXP3hemic and immune systemsForkhead Transcription FactorsAnimal ModelsFlow CytometryInterleukin-10Interleukin 10medicine.anatomical_structureAminoquinolinesCytokinesIntercellular Signaling Peptides and ProteinsImmunotherapyResearch ArticleHeparin-binding EGF-like Growth FactorT cellImmune CellsImmunologychemical and pharmacologic phenomenaImmune SuppressionImmunomodulationImmune systemModel OrganismsImmune ToleranceAnimalsBiologyB cellbusiness.industrylcsh:RImmunityMice Inbred C57BLCTL*Immune SystemImmunologyImmunologic Techniqueslcsh:QImmunizationbusinessT-Lymphocytes CytotoxicPLoS ONE
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Neurons as targets for T cells in the nervous system

2013

International audience; Accumulating evidence shows that T cells penetrate the central nervous system (CNS) parenchyma in several autoimmune, infectious, and degenerative neurological diseases. The structural and functional consequences for CNS neurons of their encounter with activated T cells have been investigated in several experimental systems, including ex vivo co-cultures, electrophysiology, and in vivo imaging. Here, we review the modalities of neuron/T cell interactions. We substantiate the contention that T cells are directly responsible for neuronal damage in a large number of neurological diseases and discuss mechanisms of neuronal damage mediated by distinct T cell subsets, the …

Nervous systemMultiple SclerosisT cell[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyAntigen presentationCentral nervous systemInflammationAdaptive ImmunityBiology[SDV.BC.IC] Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Nervous System03 medical and health sciences0302 clinical medicineT-Lymphocyte Subsets[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]medicineAnimalsHumansEncephalitis Viral030304 developmental biologyNeuronsAntigen PresentationImmunity Cellular0303 health sciencesGeneral NeuroscienceHistocompatibility Antigens Class Iapoptosis[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyT cellNeurodegenerative DiseasesAcquired immune systemcentral nervous systemneuron3. Good healthmedicine.anatomical_structurenervous system[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyinflammation[SDV.IMM.IA] Life Sciences [q-bio]/Immunology/Adaptive immunologyencephalomyelitisNeuronNervous System Diseasesmedicine.symptomNeuroscience030217 neurology & neurosurgeryEx vivo
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Oligodendrocyte ablation triggers central pain independently of innate or adaptive immune responses in mice.

2014

Mechanisms underlying central neuropathic pain are poorly understood. Although glial dysfunction has been functionally linked with neuropathic pain, very little is known about modulation of pain by oligodendrocytes. Here we report that genetic ablation of oligodendrocytes rapidly triggers a pattern of sensory changes that closely resemble central neuropathic pain, which are manifest before overt demyelination. Primary oligodendrocyte loss is not associated with autoreactive T- and B-cell infiltration in the spinal cord and neither activation of microglia nor reactive astrogliosis contribute functionally to central pain evoked by ablation of oligodendrocytes. Instead, light and electron micr…

NociceptionSpinothalamic tractSpinal Cord Dorsal HornSpinothalamic TractsT-LymphocytesGeneral Physics and AstronomyAdaptive ImmunityGeneral Biochemistry Genetics and Molecular BiologyArticleMicemedicineAnimalsOligodendrocyte; central painB-LymphocytesMultidisciplinaryMicrogliabusiness.industryGeneral Chemistrymedicine.diseaseSpinal cordOligodendrocyteAxonsImmunity InnateAstrogliosisMicroscopy ElectronOligodendrogliamedicine.anatomical_structureNociceptionSpinal CordAstrocytesNeuropathic painNeuralgiaNeuralgiaMicrogliabusinessNeuroscienceNature communications
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Biological properties of extracellular vesicles and their physiological functions

2015

The authors wish to thank Dr R Simpson and Dr D Taylor for critical reading of the manuscript and acknowledge the Horizon 2020 European Cooperation in Science and Technology programme and its support of our European Network on Microvesicles and Exosomes in Health & Disease (ME-HaD; BM1202 www.cost.eu/COST_Actions/bmbs/Actions/BM1202). In the past decade, extracellular vesicles (EVs) have been recognized as potent vehicles of intercellular communication, both in prokaryotes and eukaryotes. This is due to their capacity to transfer proteins, lipids and nucleic acids, thereby influencing various physiological and pathological functions of both recipient and parent cells. While intensive invest…

ProteomicsCellular distributionMATURE DENDRITIC CELLSReviewReview ArticleUrineEmbryo developmentMonocyteProtein processingVascular biologyFecesVesícules seminalsSYNCYTIOTROPHOBLAST MICROVILLOUS MEMBRANESCell selectionPregnancyT lymphocyteBileCELL-DERIVED EXOSOMESBiogenesisLung lavageUterus fluidInnate immunityMale genital systemlcsh:CytologyMicrovesicleOUTER-MEMBRANE VESICLESBlood clottingprokaryoteEukaryotaExtracellular vesicleRNA analysisCell biologyBloodCerebrospinal fluidLiver metabolismmicrovesicleMorphogenHumanNervous systemCell signalingBreast milkNatural killer cellFisiologiaExtracellular vesiclesExosomelcsh:QH573-671SalivaBiologyBiology and Life SciencesDNAPlantRNA transportCell functionMacrophageMolecular biologyPhysiologyMedizinProteomicsFACTOR PATHWAY INHIBITOReukaryoteProtein glycosylationExtracellular spaceTissue repairEspai extracel·lularReticulocyteSeminal plasmaMesenchymal stem cellAntigen presenting cellSeminal vesiclesNose mucusBiofilmNeutrophilMicroRNAPLANT-MICROBE INTERACTIONSLipidAmnion fluidProkaryotamicroparticleCell interactionCell transporteukaryote exosome extracellular vesicle microparticle microvesicle physiology prokaryoteBone mineralizationMicroorganismHistologyAdaptive immunityMembrane vesicleComputational biologyMembrane receptorBiologyStressCell communicationMast cellMESENCHYMAL STEM-CELLSHUMAN ENDOTHELIAL-CELLSexosomeCytokineSynovial fluidCell BiologyNonhumanIMMUNE-MODULATORY FEATURESReview articleDNA contentphysiologyRNAINTESTINAL EPITHELIAL-CELLSextracellular vesicleBody fluidLectinBiogenesis
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Bacterial and viral infections and related inflammatory responses in chronic obstructive pulmonary disease

2021

Abstract In chronic obstructive pulmonary disease (COPD) patients, bacterial and viral infections play a relevant role in worsening lung function and, therefore, favour disease progression. The inflammatory response to lung infections may become a specific indication of the bacterial and viral infections. We here review data on the bacterial–viral infections and related airways and lung parenchyma inflammation in stable and exacerbated COPD, focussing our attention on the prevalent molecular pathways in these different clinical conditions. The roles of macrophages, autophagy and NETosis are also briefly discussed in the context of lung infections in COPD. Controlling their combined response…

Review ArticleNK cells030204 cardiovascular system & hematologyAdaptive Immunitymedicine.disease_causeAutoimmunityPulmonary Disease Chronic Obstructive0302 clinical medicineNETosiPulmonary Medicine030212 general & internal medicineLungRespiratory Tract InfectionsT-lymphocytesCOPDB cellpyroptosisautoimmunityPyroptosisNETosisGeneral Medicinerespiratory systemAcquired immune systemmacrophagesmedicine.anatomical_structureautoimmunity; autophagy; B cells; dendritic cells; disability; ILCs; macrophages; NETosis; NK cells; outcome; pyroptosis; T-lymphocytesDisease Progressionoutcomemedicine.symptomSignal Transductionautophagydendritic cellILCsContext (language use)Inflammationmacrophage03 medical and health sciencesImmune systemmedicineHumansNK celldendritic cellsB cellsLungbusiness.industrymedicine.diseaseImmunity Innaterespiratory tract diseasespyroptosiILCdisabilityImmunologybusiness
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How Can We Improve the Vaccination Response in Older People? Part II: Targeting Immunosenescence of Adaptive Immunity Cells.

2022

The number of people that are 65 years old or older has been increasing due to the improvement in medicine and public health. However, this trend is not accompanied by an increase in quality of life, and this population is vulnerable to most illnesses, especially to infectious diseases. Vaccination is the best strategy to prevent this fact, but older people present a less efficient response, as their immune system is weaker due mainly to a phenomenon known as immunosenescence. The adaptive immune system is constituted by two types of lymphocytes, T and B cells, and the function and fitness of these cell populations are affected during ageing. Here, we review the impact of ageing on T and B …

Settore MED/04 - Patologia GeneraleB cellAgingImmunosenescenceOrganic ChemistryVaccinationT cellGeneral MedicinevaccinesAdaptive ImmunityCatalysisComputer Science ApplicationsInorganic ChemistryQuality of LifeHumansPhysical and Theoretical ChemistryMolecular BiologySpectroscopyAgedInternational journal of molecular sciences
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Host immune response to Cryptosporidium parvum infection

2010

Species of the genus Cryptosporidium are protozoan parasites (Apicomplexa) that cause gastroenteritis in animals and humans. Of these Cryptosporidium parvum and Cryptosporidium hominis are the major causative agents of human cryptosporidiosis. Whereas infection is self-limiting in the immunocompetent hosts, immunocompromised individuals develop a chronic, life-threatening disease. As specific therapeutic or preventive interventions are not yet available, better understanding of the immune response to the parasite is required. This minireview briefly summarizes the factors involved in the innate and acquired immune response in this pathogen-host interaction with an emphasis on more recent da…

T-Lymphocytesanimal diseasesAIDS-Related Opportunistic InfectionsImmunologyAntibodies ProtozoanCryptosporidiosisAdaptive ImmunityBiologyNitric OxideImmunocompromised HostMiceImmune systemIntestinal mucosaImmunityparasitic diseasesAnimalsHumansIntestinal MucosaCryptosporidium parvumB-LymphocytesPhagocytesAIDS-Related Opportunistic InfectionsComplement System ProteinsDendritic CellsGeneral MedicineAcquired immune systembiology.organism_classificationVirologyImmunity InnateKiller Cells NaturalDisease Models AnimalInfectious DiseasesCryptosporidium parvumImmunologyCytokinesParasitologyImmunocompetenceImmunocompetenceCryptosporidium hominisExperimental Parasitology
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Release of dendritic cells from cognate CD4 + T-cell recognition results in impaired peripheral tolerance and fatal cytotoxic T-cell mediated autoimm…

2012

Resting dendritic cells (DCs) induce tolerance of peripheral T cells that have escaped thymic negative selection and thus contribute significantly to protection against autoimmunity. We recently showed that CD4 + Foxp3 + regulatory T cells (Tregs) are important for maintaining the steady-state phenotype of DCs and their tolerizing capacity in vivo. We now provide evidence that DC activation in the absence of Tregs is a direct consequence of missing DC–Treg interactions rather than being secondary to generalized autoimmunity in Treg-less mice. We show that DCs that lack MHC class II and thus cannot make cognate interactions with CD4 + T cells are completely unable to induce peripheral CD8 +…

TransgeneGenes MHC Class IIAutoimmunityMice Transgenicchemical and pharmacologic phenomenaAdaptive ImmunityLymphocyte Activationmedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunityMicemedicineAnimalsCytotoxic T cellHomeodomain ProteinsMHC class IIMultidisciplinarybiologyPeripheral ToleranceBody WeightHistological TechniquesFOXP3Peripheral tolerancehemic and immune systemsDendritic CellsBiological SciencesFlow CytometryAcquired immune systemTamoxifenImmunologybiology.proteinCD8T-Lymphocytes CytotoxicProceedings of the National Academy of Sciences
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Vascular Endothelial Growth Factor, a Key Modulator of the Anti-Tumor Immune Response

2021

During tumor growth, angiogenesis is required to ensure oxygen and nutrient transport to the tumor. Vascular endothelial growth factor (VEGF) is the major inducer of angiogenesis and appears to be a key modulator of the anti-tumor immune response. Indeed, VEGF modulates innate and adaptive immune responses through direct interactions and indirectly by modulating protein expressions on endothelial cells or vascular permeability. The inhibition of the VEGF signaling pathway is clinically approved for the treatment of several cancers. Therapies targeting VEGF can modulate the tumor vasculature and the immune response. In this review, we discuss the roles of VEGF in the anti-tumor immune respon…

Vascular Endothelial Growth Factor AAngiogenesisAngiogenesis InhibitorsVascular permeabilityReviewAdaptive Immunityimmune responsechemistry.chemical_compoundangiogenesisNeoplasmsVEGF Signaling PathwayInducerBiology (General)SpectroscopyNeovascularization Pathologicvascular endothelial growth factorGeneral MedicineSorafenibComputer Science ApplicationsBevacizumabGene Expression Regulation NeoplasticVascular endothelial growth factorVascular endothelial growth factor AChemistrySignal TransductionQH301-705.5Recombinant Fusion ProteinsAntibodies Monoclonal HumanizedCatalysisCapillary PermeabilityInorganic ChemistryImmune systemmedicineHumansImmunologic FactorscancerPhysical and Theoretical ChemistryMolecular BiologyQD1-999Vascular Endothelial Growth Factor Receptor-1business.industryOrganic ChemistryEndothelial CellsCancermedicine.diseaseImmunity InnateReceptors Vascular Endothelial Growth FactorchemistryCancer researchbusinessInternational Journal of Molecular Sciences
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Regulation of T-Cell Immune Responses by Pro-Resolving Lipid Mediators

2021

Both the initiation and the resolution of inflammatory responses are governed by the sequential activation, migration, and control/suppression of immune cells at the site of injury. Bioactive lipids play a major role in the fine-tuning of this dynamic process in a timely manner. During inflammation and its resolution, polymorphonuclear cells (PMNs) and macrophages switch from producing pro-inflammatory prostaglandins and leukotrienes to specialized pro-resolving lipid mediators (SPMs), namely, lipoxins, resolvins, protectins, and maresins, which are operative at the local level to limit further inflammation and tissue injury and restore homeostasis. Accumulating evidences expand now the rol…

chronic inflammationT cellT-LymphocytesImmunologyInflammationReviewBiologymedicine.disease_causeT-Lymphocytes RegulatoryAutoimmunity03 medical and health sciencesImmunomodulating Agents0302 clinical medicineImmune systemmedicineHumansImmunology and Allergy030304 developmental biology0303 health sciencestherapyEffectorautoimmunityresolutionT cellLipid signalingadaptive immunityRC581-607Acquired immune systemCellular Reprogramming3. Good healthLipoxinsmedicine.anatomical_structureEicosapentaenoic AcidImmunologyspecialized pro-resolving lipid mediators (SPMs)Eicosanoidsmedicine.symptomImmunologic diseases. AllergyInflammation MediatorsHomeostasis030215 immunologyFrontiers in Immunology
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