Search results for "Class II"

showing 10 items of 194 documents

IL-10 down-regulates T cell activation by antigen-presenting liver sinusoidal endothelial cells through decreased antigen uptake via the mannose rece…

1998

SUMMARYOur study demonstrates that antigen-presenting liver sinusoidal endothelial cells (LSEC) induce production of interferon-gamma (IFN-γ) from cloned Th1 CD4+ T cells. We show that LSEC used the mannose receptor for antigen uptake, which further strengthened the role of LSEC as antigen-presenting cell (APC) population in the liver. The ability of LSEC to activate cloned CD4+ T cells antigen-specifically was down-regulated by exogenous prostaglandin E2 (PGE2) and by IL-10. We identify two separate mechanisms by which IL-10 down-regulated T cell activation through LSEC. IL-10 decreased the constitutive surface expression of MHC class II as well as of the accessory molecules CD80 and CD86 …

Liver cytologyT cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsDown-RegulationReceptors Cell SurfaceBiologyLymphocyte ActivationDinoprostoneMiceAntigenAntigens CDmedicineImmunology and AllergyAnimalsLectins C-TypeCD86Antigen PresentationMice Inbred BALB CMembrane GlycoproteinsHistocompatibility Antigens Class IIOriginal ArticlesInterleukin-10Interleukin 10medicine.anatomical_structureMannose-Binding LectinsLiverImmunologyB7-1 AntigenCytokinesFemaleB7-2 AntigenEndothelium VascularMannoseCD80Mannose receptorMannose ReceptorClinical and experimental immunology
researchProduct

Intrathymic tolerance induction: determination of tolerance to class II major histocompatibility complex antigens in maturing T lymphocytes by a bone…

1987

Two new experimental approaches were established to analyse the influence of the thymus on tolerance induction to major histocompatibility complex (MHC) antigens: The aim of the first experiment was to perform successful transplantation of adult allogeneic thymus tissue into nude mice, an attempt that has been unsuccessful in the past. Tolerance for the MHC genotype of a prospective thymus graft recipient (A) was induced in mice of strain B by injection of (A X B) splenocytes during the neonatal period. Adult thymic tissue obtained from these allogeneic donors (B) were grafted into the nude mice of strain A. The allogeneic thymus was accepted by the nude mice and immunoreconstitution was ac…

LymphocyteT-LymphocytesImmunologyMice NudeBone Marrow CellsThymus GlandBiologyMajor histocompatibility complexImmune toleranceMiceAntigenmedicineImmune ToleranceAnimalsTransplantation HomologousMice Inbred BALB CAge FactorsHistocompatibility Antigens Class IIGeneral MedicineDendritic cellTransplantationMice Inbred C57BLThymic TissueTolerance inductionmedicine.anatomical_structureMice Inbred DBARadiation ChimeraImmunologybiology.proteinScandinavian journal of immunology
researchProduct

Antigen-processing machinery breakdown and tumor growth.

2000

Defects in the major histocompatibility complex (MHC) class I antigen-processing machinery (APM) have been described in tumors of different histology. Murine data suggest that defects in the MHC class II APM might also be associated with malignant transformation of human cells. This article describes the pathophysiology of the MHC class I and II APM, reviews APM abnormalities in tumor cells and discusses their role in the escape of tumor cells from in vitro recognition by T cells.

MHC class IIAntigen PresentationProteasome Endopeptidase ComplexbiologyAntigen processingImmunologyAntigen presentationHistocompatibility Antigens Class IHistocompatibility Antigens Class IIATP-binding cassette transporterMajor histocompatibility complexIn vitroMalignant transformationCysteine EndopeptidasesATP Binding Cassette Transporter Subfamily B Member 3Multienzyme ComplexesNeoplasmsMHC class IImmunologybiology.proteinCancer researchAnimalsHumansATP-Binding Cassette TransportersImmunology today
researchProduct

Increased Level of Phosphotyrosine in Human Dendritic Cells under Stimulation with Contact Sensitizers but Not Irritants

1997

In the last years evidence was presented for the activation of dendritic cells (DC) under stimulation with contact sensitizers. Most data were obtained for murine Langerhans cells (LC) whereas in man blood-derived dendritic cells were found to be a more suitable model to study the mechanism of DC activation by haptens. The first observation was the upregulation of MHC class II molecules on murine Langerhans cells in vivo1 followed by their migration from the epidermis into regional lymph nodes2. Very early events during this activation include the upregulation of IL-1s in murine LC3 as well as the endocytotic activation of this cell type4. Based on the last observation attempts were made to…

MHC class IIEpidermis (botany)biologyChemistryCellProtein tyrosine phosphataseIn vitroCell biologymedicine.anatomical_structureDownregulation and upregulationImmunologymedicinebiology.proteinPhosphorylationSignal transduction
researchProduct

Modulation of MHC Class II Determinants on Rat Langerhans Cells During Short Term Culture

1993

Epidermal Langerhans cells (LC) are regarded as the most peripheral outpost of the immune system. They play a pivotal role during the onset of an immune response in the skin. One of the principal functions of LC is reflected in their extraordinary potency to present antigen to high activation requiring naive T cells.

MHC class IIImmune systemintegumentary systemAntigenImmunologybiology.proteinPotencyHigh activationBiologyInvariant chain
researchProduct

Major histocompatibility complex class I-restricted activation of cloned T cells by a soluble protein in the absence of accessory cells.

1989

A T-cell clone, 10BK.1, was established from the draining lymph nodes of (B10 x B10.BR)F1 mice immunized with ovalbumin (OVA) according to standard protocols. Upon coculture with the antigen, 10BK.1 cells reacted by production of lymphokines and by proliferation despite the absence of additional antigen-presenting cells. These T cells do not express major histocompatibility complex (MHC) class II molecules on the cell surface as assessed on the basis of several criteria: by cytofluorometric analysis I-A and I-E determinants were not detectable; 10BK.1 cells could not act as antigen-presenting cells for long-term-cultured MHC class II-restricted T-cell clones; and monoclonal antibodies direc…

MHC class IIMultidisciplinarybiologyOvalbuminT-LymphocytesHistocompatibility Antigens Class IAntigen presentationCD1chemical and pharmacologic phenomenaMHC restrictionLymphocyte ActivationVirologyMolecular biologyAntibodiesCell LineClone CellsMiceAntigenMHC class Ibiology.proteinAnimalsCytotoxic T cellAntigen-presenting cellResearch ArticleProceedings of the National Academy of Sciences
researchProduct

Evidence for T cell receptor-HLA class II molecule interaction in the response to superantigenic bacterial toxins

1991

The staphylococcal enterotoxins and related microbial T cell mitogens stimulate T cells by cross-linking variable parts of the T cell receptor (TcR) with MHC class II molecules on accessory or target cells. In this report we describe that a given combination of T cell, accessory cell (AC) and toxin can be non-stimulatory. However, the same T cell can respond to the same toxin on another AC and the same AC can present the same toxin to another T cell. This indicates that in the complex formed between TcR, toxin and class II molecule an interaction between TcR and class II molecule takes place.

MHC class IIT-LymphocytesT cellBacterial ToxinsImmunologyT-cell receptorAntigen presentationHistocompatibility Antigens Class IIReceptors Antigen T-CellAntigen-Presenting Cellsfood and beveragesT lymphocyteBiologyLymphocyte ActivationMicrobiologyCell biologymedicine.anatomical_structuremedicinebiology.proteinHumansImmunology and AllergyCytotoxic T cellAntigen-presenting cellCD8European Journal of Immunology
researchProduct

Requirements of Exogenous Protein Antigens for Presentation to CD4+ T lymphocytes By MHC Class II-Positive APC

1993

The antigen-specific activation of CD4-positive T helper cells depends on the recognition of a complex of MHC class II molecules and an antigen-derived peptide on the surface of antigen-presenting cells (APC). For most antigens generation of this MHC/peptide complex requires the uptake of the respective antigen by APC, followed by intracellular processing. The latter leads to suitable peptides of the antigen which are able to bind to MHC class ll-molecules. Subsequently the resulting complexes are transported to the cell surface. Evidence supporting this concept came mainly from the finding that agents such as chloroquine1, interfering with the function of endosomes and lysosomes, can block…

MHC class IIbiologyAntigenChemistryAntigen processingMHC class IImmunologyAntigen presentationbiology.proteinMHC restrictionMajor histocompatibility complexPan-T antigensCell biology
researchProduct

Processing and Presentation of Protein and Parasite-Derived Antigens by 4F7+ Dendritic Cells

1995

The dendritic cell (DC), a trace component of splenocytes is the principal cell type required for a primary mixed lymphocyte reaction.1 Splenic DC are described as antigen presenting cells (APC) capable to generate immunogenic fragments of intact protein antigens for presentation to MHC class II restricted T cells,2 in contrast to former findings.3 Langerhans cells (LC) as potent APC are members of the dendritic cell lineage forming a system of potent APC, first identified by Steinman and Conn.4 Importanly Schuler and Steinman have subsequently presented experimental evidence suggesting that LC represent immature DC.3 During infectious diseases caused by parasites of the genus Leishmania LC…

MHC class IIbiologyAntigenFollicular dendritic cellsChemistryAntigen processingAntigen presentationbiology.proteinDendritic cellAntigen-presenting cellMolecular biologyEpitope
researchProduct

Toxoplasma gondii down-regulates MHC class II gene expression and antigen presentation by murine macrophages via interference with nuclear translocat…

2001

The obligate intracellular protozoan parasite Toxoplasma gondii is able to establish persistent infections within human and animal hosts. We have shown recently that T. gondii down-regulates IFN-γ-induced MHC class II expression in murine bone marrow-derived macrophages (BMMΦ). As shown in this study, the capacity of IFN-γ-activated murine BMMΦ to present ovalbumin to CD4+ T cell hybridomas was dose-dependently inhibited by T. gondii. IFN-γ-induced up-regulation of H2-Aa, H2-Ab, H2-Eb, H2-Ma, H2-Mb, H2-Oa and invariant chain transcripts was prominently down-regulated by T. gondii. Furthermore, mRNA levels of class II transactivator and interferon-regulatory factor-1 were significantly dimin…

MHC class IIbiologyT cellImmunologyAntigen presentationToxoplasma gondiibiology.organism_classificationMolecular biologyMHC Class II Genemedicine.anatomical_structureparasitic diseasesbiology.proteinSTAT proteinmedicineImmunology and AllergySTAT1Signal transductionEuropean Journal of Immunology
researchProduct