Search results for "Helium"

showing 10 items of 1689 documents

Endothelin receptor expression in idiopathic pulmonary arterial hypertension: effect of bosentan and epoprostenol treatment.

2011

Endothelin receptor antagonists are used to treat idiopathic pulmonary arterial hypertension (IPAH), but human pulmonary arterial endothelin receptor expression is not well defined. We hypothesised that disease and treatment would modify normal receptor distribution in pulmonary resistance arteries of children. Using immunohistochemistry and semiquantitative analysis, we investigated endothelin receptor subtypes A and B (ET(A) and ET(B), respectively), and endothelial nitric oxide synthase (eNOS) expression in peripheral pulmonary arteries of tissue from untreated children with IPAH (n=7), following extended combined bosentan and epoprostenol therapy (n=5) and from normal subjects (n=5). Cl…

Pulmonary and Respiratory MedicineMalePathologymedicine.medical_specialtyEndotheliumAdolescentNifedipineNitric Oxide Synthase Type IIIEndothelin A Receptor AntagonistsEndothelin B Receptor AntagonistsHypertension PulmonaryVasodilator AgentsPulmonary ArteryMuscle Smooth VascularPiperazinesSildenafil CitrateNitric oxidechemistry.chemical_compoundEnosInternal medicinemedicineHumansFamilial Primary Pulmonary HypertensionSulfonesReceptorChildAntihypertensive AgentsSulfonamidesbiologybusiness.industryBosentanbiology.organism_classificationReceptor Endothelin AEpoprostenolReceptor Endothelin BEndothelin A Receptor AntagonistsBosentanEndothelin B Receptor Antagonistsmedicine.anatomical_structureEndocrinologychemistryPurinesChild PreschoolDrug Therapy CombinationFemaleEndothelium VascularEndothelin receptorbusinessmedicine.drugThe European respiratory journal
researchProduct

Authors' response to: epithelial mesenchymal transition (EMT) in small airways of COPD patient.

2013

We understand the reservations highlighted by Sohal and Walters in their letter in response to our recent work published.1 In their letter, Sohal and Walters argue that E-cadherin and ZO-1 are absent in our immunohistochemistry analysis of small bronchi of smokers and COPD patients, and that if disappeared completely epithelium would fall apart.1 In our study, we detected downregulation of E-cadherin and ZO-1 as well as a change of intercellular and apical distribution to diffuse cytoplasmic redistribution (figure 3B; in our work published in Thorax on 7 January 2013),2 but not …

Pulmonary and Respiratory MedicineMalePathologymedicine.medical_specialtyEpithelial-Mesenchymal TransitionCopd patientsCOPD PathologyBronchiPulmonary Disease Chronic ObstructiveDownregulation and upregulationAirway EpitheliumSmokemedicineHumansEpithelial–mesenchymal transition1506COPDSmall airwaysbusiness.industrySmokingEpithelial CellsPostScriptmedicine.diseaseEpitheliumrespiratory tract diseasesmedicine.anatomical_structureRespiratory epitheliumImmunohistochemistryFemalebusinessThorax
researchProduct

Endurance training damages small airway epithelium in mice.

2007

RATIONALE: In athletes, airway inflammatory cells were found to be increased in induced sputum or bronchial biopsies. Most data were obtained after exposure to cold and dry air at rest or during exercise. Whether training affects epithelial and inflammatory cells in small airways is unknown. OBJECTIVES: To test whether endurance training under standard environmental conditions causes epithelial damage and inflammation in the small airways of mice. METHODS AND MEASUREMENTS: Formalin-fixed, paraffin-embedded lung sections were obtained in sedentary (n = 14) and endurance-trained (n = 16) Swiss mice at baseline and after 15, 30, and 45 days of training. The following variables were assessed (m…

Pulmonary and Respiratory MedicineMalePathologymedicine.medical_specialtyInflammationApoptosisCritical Care and Intensive Care MedicineSettore BIO/09 - FisiologiaEpitheliumEpithelial DamageLeukocyte CountMiceEndurance trainingIntensive carePhysical Conditioning AnimalProliferating Cell Nuclear AntigenmedicineLeukocytesAnimalsBronchitisCell ProliferationBasement membraneLungAerobic exercise bronchial responsivenes methacholine deep inspiration leukotrienesbusiness.industryNF-kappa Brespiratory systemImmunohistochemistryEpitheliumrespiratory tract diseasesDisease Models Animalmedicine.anatomical_structureRespiratory epitheliummedicine.symptombusinessAmerican journal of respiratory and critical care medicine
researchProduct

Bronchial epithelial damage after a half-marathon in nonasthmatic amateur runners.

2010

Am J Physiol Lung Cell Mol Physiol. 2010 Jun;298(6):L857-62. Epub 2010 Apr 2. Bronchial epithelial damage after a half-marathon in nonasthmatic amateur runners. Chimenti L, Morici G, Paternò A, Santagata R, Bonanno A, Profita M, Riccobono L, Bellia V, Bonsignore MR. SourceDept. Biomedico Di Medicina Interna & Specialistica, Section of Pneumology, Univ. of Palermo, Via Trabucco 180, 90146 Palermo, Italy. laurachimenti@yahoo.it Abstract High neutrophil counts in induced sputum have been found in nonasthmatic amateur runners at rest and after a marathon, but the pathogenesis of airway neutrophilia in athletes is still poorly understood. Bronchial epithelial damage may occur during intense exer…

Pulmonary and Respiratory MedicineMaleSputum CytologyPhysiologyNeutrophilsinflammatory mediatorsInduced sputumBronchiendurance exerciseSettore MED/10 - Malattie Dell'Apparato RespiratorioSettore BIO/09 - FisiologiaEpitheliumRunningEpithelial DamageLeukocyte CountEndurance trainingPhysiology (medical)bronchial epithelial cellMedicineHumansUteroglobinAir Pollutantsbusiness.industryInterleukin-8neutrophils; endurance exercise; apoptosis; bronchial epithelial cells; inflammatory mediatorsSputumneutrophilCell Biologyrespiratory systemapoptosiNeutrophiliarespiratory tract diseasesImmunologyPhysical Endurancemedicine.symptomBronchial Hyperreactivitybusinesshuman activitiesAmateurAmerican journal of physiology. Lung cellular and molecular physiology
researchProduct

Pulmonary vascular endothelium: the orchestra conductor in respiratory diseases

2017

The European Respiratory Society (ERS) Research Seminar entitled “Pulmonary vascular endothelium: orchestra conductor in respiratory diseases - highlights from basic research to therapy” brought together international experts in dysfunctional pulmonary endothelium, from basic science to translational medicine, to discuss several important aspects in acute and chronic lung diseases. This review will briefly sum up the different topics of discussion from this meeting which was held in Paris, France on October 27–28, 2016. It is important to consider that this paper does not address all aspects of endothelial dysfunction but focuses on specific themes such as: 1) the complex role of the pulmon…

Pulmonary and Respiratory MedicineParismedicine.medical_specialtyEndotheliumRespiratory Tract DiseasesRespiratory SystemDysfunctional familyDiseasePulmonary ArteryVascular Remodeling030204 cardiovascular system & hematologyLung injury03 medical and health sciences0302 clinical medicinemedicineHumansEndothelial dysfunctionIntensive care medicineLungbusiness.industryTranslational medicine11 Medical And Health SciencesCongresses as Topicmedicine.diseasePulmonary hypertensionmedicine.anatomical_structure030228 respiratory systemDrug DesignEndothelium VascularbusinessEuropean Respiratory Journal
researchProduct

Chronological expression of Ciliated Bronchial Epithelium 1 during pulmonary development

2009

Ciliated Bronchial Epithelium (CBE) 1 is a novel gene, which is expressed in ciliated cells. As cilia are important during embryogenesis, the present authors characterised the murine homologue of CBE1 (Cbe1) and compared its temporal expression during murine and human lung development. Cbe1 cDNA was cloned and characterised using sequencing, standard PCR and Western blotting. Mouse and human embryonic/fetal lungs (HELs) were harvested for mRNA analysis and protein localisation in vivo and in vitro using RT-PCR and immunohistochemistry. The Cbe1 amino acid sequence was >75% identical with CBE1 and its alternative splicing and tissue distribution were highly conserved. Pulmonary expression of…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtyDNA ComplementaryTime FactorsBlotting WesternDNA Mutational AnalysisBiologyTransfectionStatistics NonparametricImmunoenzyme TechniquesMiceOpen Reading FramesCiliogenesisGene expressionmedicineAnimalsHumansAmino Acid SequenceRNA MessengerCloning MolecularLungDNA PrimersFetusMessenger RNALungReverse Transcriptase Polymerase Chain ReactionEmbryogenesisAlternative splicingNuclear ProteinsCell DifferentiationMolecular biologyEpitheliumDNA-Binding Proteinsmedicine.anatomical_structureBronchial epithelium Asthma DevelopmentTranscription FactorsEuropean Respiratory Journal
researchProduct

GM-CSF expression by human lung microvascular endothelial cells: in vitro and in vivo findings.

2002

Recently, many findings indicate that granulocyte-macrophage colony-stimulating factor (GM-CSF) plays an important role in the pathogenesis of acute and chronic lung diseases. In the present paper, the production of this cytokine in human pulmonary microvascular endothelial cells (HPMEC) is investigated. In an in vitro study, quiescent HPMEC did not express GM-CSF, either at the transcriptional or at the protein level. After activation for 4 h with tumor necrosis factor (TNF)-α (30/300 U/ml), lipopolysaccharide (LPS; 0.1/1 μg/ml), or interleukin (IL)-1β (100 U/ml), a significant release of GM-CSF was measured by enzyme-linked immunosorbent assay, with a time-dependent increase over 72 h. IL…

Pulmonary and Respiratory MedicinePathologymedicine.medical_specialtyPulmonary CirculationUmbilical VeinsEndotheliumPhysiologymedicine.medical_treatmentHemangiosarcomaEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesPathogenesisIn vivoPhysiology (medical)medicineHumansCells CulturedLungbusiness.industryReverse Transcriptase Polymerase Chain ReactionMicrocirculationRespiratory diseaseGranulocyte-Macrophage Colony-Stimulating FactorCell Biologymedicine.diseaseImmunohistochemistryEndothelial stem cellGranulocyte macrophage colony-stimulating factorCytokinemedicine.anatomical_structureEndothelium Vascularbusinessmedicine.drugAmerican journal of physiology. Lung cellular and molecular physiology
researchProduct

Assessment of lung microstructure with magnetic resonance imaging of hyperpolarized Helium-3

2005

Magnetic resonance imaging of the apparent diffusion coefficient (ADC) of hyperpolarized Helium-3 is a new technique for probing pulmonary microstructure in vivo. The aim of this study was the assessment of potential sources of systematic errors of the ADC measurement. The influence of macroscopic motion was determined by measurements at two different delays after initiating the breath-hold, and before and after cardiac arrest. An intercentre comparison was performed in two age- and lung function-matched groups of lung-healthy volunteers at two research sites. Moreover, measurements of diffusion anisotropy were performed. We found no dependency of the ADC as a function of the delay after st…

Pulmonary and Respiratory MedicineTime FactorsSwinePhysiologyHyperpolarized Helium 3HeliumDiffusion AnisotropyNuclear magnetic resonanceIsotopesImage Processing Computer-AssistedmedicineAnimalsHumansEffective diffusion coefficientTissue DistributionDiffusion (business)LungLungmedicine.diagnostic_testChemistryGeneral NeuroscienceMagnetic resonance imagingMiddle AgedMicrostructureHeart ArrestDiffusion Magnetic Resonance Imagingmedicine.anatomical_structureBreath TestsCase-Control StudiesAnisotropyDiffusion MRIRespiratory Physiology & Neurobiology
researchProduct

Lung fixation for the preservation of air spaces.

2004

The procedures for the fixation of entire lungs of small rodents are presented together with various techniques used to verify the structural integrity of the lung tissue. To achieve this, the lungs were dissected out from rats and mice killed by ether overdose. The specimens were rinsed with isotonic saline and fixation solution under low vacuum conditions. After fixation, they were dried using alcohol and stored in a noncollapsed state (i.e., state of inhalation). Light and scanning electron microscopy as well as magnetic resonance imaging using hyperpolarized 3He were employed to verify the intact state on interalveolar septa and walls of smaller bronchi as well as accessibility of the a…

Pulmonary and Respiratory MedicineTissue FixationIsotonic salineClinical BiochemistryIn Vitro TechniquesSuctionHeliumRats Sprague-DawleyFixativesMiceLow vacuumFormaldehydemedicineAnimalsRespiratory systemMolecular BiologyFixation (histology)LungInhalationChemistryAirSyringesStructural integrityAnatomyrespiratory systemMagnetic Resonance ImagingRatsMice Inbred C57BLPulmonary Alveolimedicine.anatomical_structureAlcoholsMicroscopy Electron ScanningLung tissueExperimental lung research
researchProduct

Induction of stress proteins in human endothelial cells by heavy metal ions and heat shock.

1999

In the present study, we compared the induction of heat shock proteins (HSPs) by heat and heavy metal ions in three different endothelial cell types, namely, human umbilical vein endothelial cells, human pulmonary microvascular endothelial cells, and the cell line EA.hy 926. Our results show that especially Zn2+and Cd2+are inducers of 70-kDa (HSP70), 60-kDa (HSP60), 32-kDa (HSP32), and 27-kDa (HSP27) HSPs. The strength of inducibility is specific for each HSP. Ni2+and Co2+only show an inducible effect at very high concentrations, that is, in the clearly cytotoxic range. Furthermore, we investigated the time course of HSP expression and the involvement of heat shock factor-1. Our study demon…

Pulmonary and Respiratory MedicineUmbilical VeinsPhysiologyMetal ions in aqueous solutionBlotting WesternGene ExpressionBiologyUmbilical veinPhysiology (medical)Heat shock proteinMetals HeavyGene expressionmedicineHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsRNA MessengerFluorescent Antibody Technique IndirectCells CulturedHeat-Shock ProteinsCell BiologyChaperonin 60Endothelial stem cellmedicine.anatomical_structureCell cultureShock (circulatory)ImmunologyBiophysicsEndothelium Vascularmedicine.symptomHeat-Shock ResponseBlood vesselThe American journal of physiology
researchProduct