Search results for "Innate"

showing 10 items of 638 documents

Defective Induction of COX-2 Expression by Psoriatic Fibroblasts Promotes Pro-inflammatory Activation of Macrophages

2019

Fibroblasts play an important role as members of the innate immune system through the secretion of COX-2-derived inflammatory mediators such as prostaglandin E-2 (PGE(2)). However, it has been described that dermal fibroblasts behave like mesenchymal stem cells reducing lymphocyte recruitment and dendritic cell activation through PGE(2) release. As the role of fibroblasts in psoriasis remains poorly characterized, in the present study we have evaluated the possible influence of PGE(2) derived from dermal fibroblasts as modulator of the immune response in psoriatic skin. Our results indicate that under inflammatory conditions, psoriatic fibroblasts showed defective induction of COX-2, which …

lcsh:Immunologic diseases. AllergyAdultMale0301 basic medicineTHP-1 CellsLymphocyteImmunologyInflammationDinoprostoneYoung Adult03 medical and health sciences0302 clinical medicineImmune systemPsoriasismedicineHumansImmunology and AllergyPsoriasisProstaglandin E2SkinOriginal ResearchInflammationInnate immune systemChemistryMacrophagesMesenchymal stem cellpsoriasisDendritic cellMiddle AgedFibroblastsmedicine.diseaseCyclooxygenasecyclooxygenase030104 developmental biologymedicine.anatomical_structureCyclooxygenase 2inflammationCancer researchFemalemedicine.symptomlcsh:RC581-607030215 immunologymedicine.drug
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INNATE IMMUNITY AND INFLAMMATION IN AGEING: A KEY FOR UNDERSTANDING AGE-RELATED DISEASES.

2005

Abstract The process of maintaining life for the individual is a constant struggle to preserve his/her integrity. This can come at a price when immunity is involved, namely systemic inflammation. Inflammation is not per se a negative phenomenon: it is the response of the immune system to the invasion of viruses or bacteria and other pathogens. During evolution the human organism was set to live 40 or 50 years; today, however, the immune system must remain active for much a longer time. This very long activity leads to a chronic inflammation that slowly but inexorably damages one or several organs: this is a typical phenomenon linked to ageing and it is considered the major risk factor for a…

lcsh:Immunologic diseases. AllergyAgingmedia_common.quotation_subjectLongevityImmunologyInflammationReviewDiseaselcsh:GeriatricsSystemic inflammationImmune systemImmunitymedicinePermissiveCytokinemedia_commonInflammationInnate immune systembusiness.industryLongevityInnate Immunitylcsh:RC952-954.6Immunologymedicine.symptomlcsh:RC581-607businessAge-related diseases
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Innate Immune Cells' Contribution to Systemic Lupus Erythematosus

2019

Systemic lupus erythematosus (SLE) is a chronic autoimmune disease characterized by the presence of autoantibodies against nuclear antigens, immune complex deposition, and tissue damage in the kidneys, skin, heart and lung. Because of the pathogenic role of antinuclear antibodies and autoreactive T cells in SLE, extensive efforts have been made to demonstrate how B cells act as antibody-producing or as antigen-presenting cells that can prime autoreactive T cell activation. With the discovery of new innate immune cells and inflammatory mediators, innate immunity is emerging as a key player in disease pathologies. Recent work over the last decade has highlighted the importance of innate immun…

lcsh:Immunologic diseases. AllergyAnti-nuclear antibodyMini ReviewT cellImmunologyPathogenesisAntigenimmune system diseasesmedicineAnimalsHumansLupus Erythematosus SystemicImmunology and Allergydendritic cellsskin and connective tissue diseasesinnate immunitylupus (SLE)Autoimmune diseaseInnate immune systembusiness.industryInnate lymphoid cellAutoantibodymedicine.diseaseImmunity Innatemacrophage-cellmedicine.anatomical_structureImmunologyinnate lymphoid celllcsh:RC581-607businessFrontiers in Immunology
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Heat-Shock Proteins in Autoimmunity

2013

Heat shock proteins (HSPs), also known as “stress proteins,” are among the highly conserved and immunogenic proteins shared among diverse groups of microbial agents and mammals [1]. Heat and other types of stressful stimuli can increase the cellular expression of HSPs. These proteins have been categorized into different families according to their molecular mass, for example, HSP110, HSP90, HSP70, HSP60, HSP40, HSP20-30, and HSP10 [1–3]. For uniformity, guidelines for the nomenclature of various human HSP families have been proposed [4]. Under physiological conditions, the ubiquitously distributed HSPs maintain the integrity and function of other cellular proteins in stressful conditions. H…

lcsh:Immunologic diseases. AllergyArticle SubjectImmunologychemical and pharmacologic phenomenaBiologymedicine.disease_causeAutoimmunity03 medical and health sciences0302 clinical medicineImmune systemImmunology and Microbiology (miscellaneous)Heat shock proteinmedicineImmunology and Allergy030304 developmental biology0303 health sciencesInnate immune systemFOXP3Acquired immune system3. Good healthMolecular mimicryEditorialImmunologyHSP60lcsh:RC581-607030217 neurology & neurosurgeryAutoimmune Diseases
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Are Toll-like receptors and decoy receptors involved in the immunopathogenesis of systemic lupus erythematosus and lupus-like syndromes?

2011

In this paper we focus our attention on the role of two families of receptors, Toll-like receptors (TLR) and decoy receptors (DcR) involved in the generation of systemic lupus erythematosus (SLE) and lupus-like syndromes in human and mouse models. To date, these molecules were described in several autoimmune disorders such as rheumatoid arthritis, antiphospholipids syndrome, bowel inflammation, and SLE. Here, we summarize the findings of recent investigations on TLR and DcR and their role in the immunopathogenesis of the SLE.

lcsh:Immunologic diseases. AllergyChemokineImmunologyInflammationAutoimmunityReview ArticleCell Communicationmedicine.disease_causeAutoantigensAutoimmunityMiceimmune system diseasesToll-like receptormedicineImmunology and AllergyAnimalsHumansLupus Erythematosus SystemicDecoy receptorsReceptorskin and connective tissue diseasesSettore MED/04 - Patologia GeneraleToll-like receptors decoy receptors systemicic erythematous lupusSystemic lupus erythematosusbiologybusiness.industryToll-Like ReceptorsGeneral Medicinemedicine.diseaseImmunity Innatedecoy receptorDisease Models AnimalTumor Necrosis Factor Decoy ReceptorsRheumatoid arthritisImmunologybiology.proteinsystemicic erythematous lupusmedicine.symptomChemokinesbusinesslcsh:RC581-607Tumor Necrosis Factor Decoy ReceptorsSignal Transduction
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Antigen-specific T cells and cytokines detection as useful tool for understanding immunity against zoonotic infections.

2012

Zoonoses include a broad range of diseases, that are becoming of great interest, due to the climate changing, that cause the adaptation of vectors to new niches and environments. Host immune responses play a crucial role in determining the outcome of infections, as documented by expansion of antigen-specific T cells during several zoonotic infections. Thus, understanding of the contribution of antigen-specific T-cell subsets in the host immune response is a powerful tool to evaluate the different immunological mechanisms involved in zoonotic infections and for the development of effective vaccines. In this paper we discuss the role of T cells in some eukaryotic and prokaryotic infectious mo…

lcsh:Immunologic diseases. AllergyNematodaT-LymphocytesImmunologyReview ArticleAdaptive ImmunityBiologyHost-Parasite InteractionsImmune systemT-Lymphocyte SubsetsAntigen specificImmunityZoonosesAnimalsHumansImmunology and AllergyAntigensTh1-Th2 BalanceZoonoses antigen specific T-cells animal immunology.VaccinesBacteriaZoonotic InfectionGeneral MedicineAcquired immune systemVirologyImmunity InnateHost-Pathogen InteractionsImmunologyCytokinesAdaptationlcsh:RC581-607
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New Insight into Immunity and Immunopathology of Rickettsial Diseases

2011

Human rickettsial diseases comprise a variety of clinical entities caused by microorganisms belonging to the generaRickettsia,Orientia,Ehrlichia, andAnaplasma. These microorganisms are characterized by a strictly intracellular location which has, for long, impaired their detailed study. In this paper, the critical steps taken by these microorganisms to play their pathogenic roles are discussed in detail on the basis of recent advances in our understanding of molecularRickettsia-host interactions, preferential target cells, virulence mechanisms, three-dimensional structures of bacteria effector proteins, upstream signalling pathways and signal transduction systems, and modulation of gene exp…

lcsh:Immunologic diseases. AllergySettore MED/09 - Medicina InternaVirulence FactorsRickettsial diseasesImmunologyRickettsiaceae InfectionsVirulenceImmunopathologyReview ArticleAdaptive ImmunityHost SpecificityMicrobiologyImmune systemBacterial ProteinsImmunityAnimalsHumansImmunology and AllergyAnaplasmaMolecular Targeted TherapyRickettsiaArthropodsPathogenRickettsieaeGeneticsImmunopathology; Rickettsial diseasesbiologyEffectorGeneral Medicinebacterial infections and mycosesbiology.organism_classificationAcquired immune systemOrientiaImmunity InnateGene Expression RegulationHost-Pathogen Interactionslcsh:RC581-607Signal TransductionClinical and Developmental Immunology
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γδ T Cells Cross-Link Innate and Adaptive Immunity in Mycobacterium tuberculosis Infection

2011

Protective immunity against mycobacterial infections such asMycobacterium tuberculosisis mediated by interactions between specific T cells and activated antigen presenting cells. To date, many aspects of mycobacterial immunity have shown that innate cells could be the key elements that substantially may influence the subsequent adaptive host response. During the early phases of infection, innate lymphocyte subsets play a pivotal role in this context. Here we summarize the findings of recent investigations onγδT lymphocytes and their role in tuberculosis immunity.

lcsh:Immunologic diseases. AllergyT-LymphocytesT cellImmunologyReview ArticleAdaptive ImmunityLymphocyte ActivationMycobacterium tuberculosisImmune systemAntigenImmunitymedicineAnimalsHumansTuberculosisImmunology and AllergyIL-2 receptorAntigen-presenting cellbiologyReceptors Antigen T-Cell gamma-deltaMycobacterium tuberculosisGeneral MedicineAcquired immune systembiology.organism_classificationVirologyImmunity Innategamma delta T cells Mycobacterium tuberculosismedicine.anatomical_structureImmunologylcsh:RC581-607Immunologic Memory
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Macrophage: SHIP of Immunity

2014

Immunology. Why does it exist? Two words. Cure disease. People get diseases. “Test tubes” do not. People fund immunologists for solutions to their health problems. But, immunologists often study leukocytes in test tubes – the laboratory – away from diseases. Why? Because much can be learned from analyzing cellular biochemistry and behaviors in vitro that cannot be ascertained when leukocytes are in animals. At the same time, isolated leukocyte reactions often do not reflect how the immune system operates as a unit. So, it is critical to verify in vitro observations in vivo. Among leukocytes, macrophages are the central initiating and directing element in immune systems, and serve this role …

lcsh:Immunologic diseases. AllergywoundImmunologyM1DiseasemacrophageBiologyM2Immune systemAntigenIn vivoImmunitynitric oxideImmunology and AllergyMacrophagecancerCancerWound HealingInnate immune systemMacrophagesarginaseOpinion ArticleIn vitroiNOSImmunologylcsh:RC581-607Frontiers in Immunology
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Diversity of lectin-sugar recognition dystems in the evolution of fish innate immunity

2014

lectin fish innate immunity D. labrax S. aurata
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