Search results for "Lymphokines"

showing 10 items of 29 documents

Characterization of a T-cell-derived mast cell costimulatory activity (MCA) that acts synergistically with interleukin 3 and interleukin 4 on the gro…

1990

The proliferation of mucosal mast cells (MMC) depends on the presence of interleukin 3 (IL 3) and can be further enhanced by interleukin 4 (IL 4). The supernatant of a TH2 cell clone (ST2/K.9) stimulated by concanavalin A was found to contain a factor, provisionally termed mast cell costimulatory activity (MCA), that substantially enhances the proliferation of MMC promoted by a combination of IL 3 and IL 4. In comparison to other lymphokines MCA is rather resistant to tryptic digestion but is very sensitive to pH values lower than 6.0 and to organic solvents. Chromatographic fractionation of MCA revealed that activity is associated with protein(s) or glycoprotein(s) of 35 to 40 kDa. Partial…

Malemedicine.medical_specialtyT-LymphocytesImmunologyBone Marrow CellsBiologyBiochemistryMast cell proliferationCell LineBiological FactorsMiceEpidermal growth factorInternal medicinemedicineImmunology and AllergyAnimalsMast CellsMolecular BiologyInterleukin 4Interleukin 3LymphokinesLymphokineDrug SynergismHematologyMast cellHematopoietic Stem CellsMolecular biologyClone Cellsmedicine.anatomical_structureEndocrinologyMice Inbred DBAChromatography GelCytokinesTumor necrosis factor alphaFemaleInterleukin-3Interleukin-4Leukemia inhibitory factorCell DivisionCytokine
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B8, DR3 antigens and production of human leucocyte migration inhibitory factor (LIF) by mononuclear cells stimulated with concanavalin A (Con A)

2008

LIF release by Con A stimulated mononuclear cells was evaluated in 67 randomly selected healthy Sicilians typed for HLA antigens. The results show that B8 and/or DR3 positive subjects release less LIF than negative ones, suggesting that this immunological response might be controlled by HLA-linked immune response (Ir) gene(s).

Leukocyte Migration-Inhibitory FactorsImmunologyHuman leukocyte antigenInhibitory postsynaptic potentialBiochemistryPeripheral blood mononuclear cellMonocytesHLA-B8 AntigenHLA-DR3 AntigenImmune systemAntigenHLA AntigensConcanavalin AGeneticsHumansImmunology and AllergyGeneLymphokinesbiologyChemistryHistocompatibility Antigens Class IIGeneral MedicineMolecular biologyConcanavalin AImmunologybiology.proteinLeucocyte migrationTissue Antigens
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T-cell-derived helper factor allows in vivo induction of cytotoxic T cells in nu/nu mice

1980

T-cell immunocompetence and diversity are thought to be generated in the thymus1,2. This view is based on the findings that (1) T-cell ontogeny is thymus dependent3,4, (2) the major histocompatibility restrictions of T-cell interactions are phenotypically related to the H–2 type of the thymus5–9, and (3) the phenotypic manifestation of H–2-linked immune responsiveness parallels the restriction elements selected in thymus10–12. However, it is unclear whether pre-thymic cells programmed to develop into T cells do already express a receptor diversity, also whether pre-thymic cells have the potential to react against self-antigens, and whether the mechanism of self-tolerance is initiated in the…

Cytotoxicity ImmunologicInterleukin 2LymphokinesMultidisciplinaryT-LymphocytesT cellLymphocyte CooperationMice NudeCell DifferentiationThymus GlandBiologyCell biologyTransplantationMiceInterleukin 21medicine.anatomical_structureImmune systemmedicineAnimalsCytotoxic T cellImmunocompetenceAntigen-presenting cellmedicine.drugNature
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The role of accessory cells in polyclonal T cell activation. I. Both induction of interleukin 2 production and of interleukin 2 responsiveness by con…

1983

Recent studies from other laboratories have shown that concanavalin A (Con A) acts at two separate steps in polyclonal T cell activation: interleukin 2 (IL2) production, and induction of responsiveness to IL2. Using a combination of techniques for the depletion of accessory cells from lymph node T cells, we have investigated which of these steps, if not both, is responsible for the known requirement for accessory cells in the Con A response. It was found that with increasing T cell purification, first the ability is lost to produce sufficient levels of endogenous IL2, whereas induction of IL2 responsiveness can still take place. Further removal of accessory cells however yields a population…

Malemusculoskeletal diseasesInterleukin 2medicine.medical_specialtyComplement Activating EnzymesT-LymphocytesT cellLymphocyte CooperationImmunologyPopulationchemical and pharmacologic phenomenaLymphocyte ActivationMiceInterleukin 21immune system diseasesInternal medicineConcanavalin AmedicineAnimalsImmunology and AllergyAntigen-presenting celleducationInterleukin 3LymphokinesMice Inbred BALB Ceducation.field_of_studybiologyComplement C1qImmune SeraHistocompatibility Antigens Class IIhemic and immune systemsCell biologyKineticsstomatognathic diseasesEndocrinologymedicine.anatomical_structurePolyclonal antibodiesConcanavalin Abiology.proteinInterleukin-2FemaleLymph NodesSpleenmedicine.drugEuropean Journal of Immunology
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Spontaneous lymphokine synthesis by human blood mononuclear cells

1975

LYMPHOCYTES, after antigenic stimulation, may synthesise and release biologically active soluble factors other than antibodies. These mediators were termed lymphokines by Dumonde1, and the most extensively studied and best characterised are migration inhibitory factors which can inhibit the migration of macrophages or leukocytes: this is the property used for their in vitro bioassay. Apart from antigens, various other stimuli may trigger lymphokine synthesis by lymphocytes, for example, polyclonal mitogens2, anti-immunoglobulin or membrane Fc or C3-receptor reactions3,4. Furthermore, migration inhibitory activity has been found in the long term culture supernatants of some established lymph…

CellPeripheral blood mononuclear cellMonocytesAntigenmedicineHumansLymphocytesMacrophage Migration-Inhibitory FactorsLymphokinesMultidisciplinarybiologyChemistryLymphokineBiological activityIn vitroCell biologyCold TemperatureBloodmedicine.anatomical_structurePolyclonal antibodiesDepression ChemicalProtein BiosynthesisImmunologybiology.proteinPuromycinAntibodyNature
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3D microvascular architecture of pre-cancerous lesions and invasive carcinomas of the colon.

2001

Despite the significance of tumour neoangiogenesis and the extensive knowledge on the molecular basis of blood vessel formation currently no quantitative data exist on the 3D microvascular architecture in human primary tumours and their precursor lesions. This prompted us to examine the 3D vascular network of normal colon mucosa, adenomas and invasive carcinomas by means of quantitative microvascular corrosion casting. Fresh hemicolectomy specimens from 20 patients undergoing cancer or polyposis coli surgery were used for corrosion casting, factor VIII and VEGF immunostaining. In addition, immunostaining was done on colorectal tissue from 33 patients with metastatic and non-metastatic carci…

AdenomaMaleVascular Endothelial Growth Factor ACancer ResearchPathologymedicine.medical_specialtytumour vascular architectureAdenomaAngiogenesisAdenomatous polyposis coliEndothelial Growth FactorsMetastasisNeovascularizationangiogenesisImage Processing Computer-AssistedMedicineHumansNeoplasm InvasivenessGrading (tumors)AgedLymphokinesbiologybusiness.industryVascular Endothelial Growth FactorsMicrocirculationCancerRegular ArticleMiddle Agedmedicine.diseasecolorectal adenocarcinomapre-cancerous lesionsOncologyAdenomatous Polyposis ColiColonic Neoplasmsbiology.proteinFemalemedicine.symptombusinessColorectal NeoplasmsPrecancerous ConditionsImmunostainingBritish journal of cancer
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The in Vivo Effects of Interleukin 2 (TCGF)

1982

This brief review of our experiments concerning the in vivo activity of crude Il-2 led us to the following conclusion: The first is the existence, in vivo, of a cyclophosphamide-sensitive T-cell controlling the activity of a serum born Il-2 inhibitor in thymus-bearing normal mice. Under in vivo conditions which are characterized by high Il-2 inhibitor activities, locally applied Il-2 administered along with antigen amplified in vivo CTL-responsiveness, yet the effect observed was poor. Crude Il-2 proved to be a potent immuno-enhancing agent in the athymic (nu/nu) mouse, which lacks Il-2 inhibitor activity. It was found that together with antigen administration of Il-2 to nude mice results i…

Cytotoxicity ImmunologicInterleukin 2T-LymphocytesLymphocyte CooperationImmunologyMice NudeMice Inbred StrainsIn Vitro TechniquesPharmacologyEpitopeEpitopesMiceAntigenIn vivoAnimalsImmunology and AllergyMedicineMice nudeLymphokinesbusiness.industryLymphokineHematologyLymphocyte CooperationCTL*ImmunologyInterleukin-2businessmedicine.drugImmunobiology
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Oncostatin M, leukaemia-inhibitory factor and interleukin 6 trigger different effects on alpha1-proteinase inhibitor synthesis in human lung-derived …

1998

Interleukin 6 (IL-6), oncostatin M (OSM) and leukaemia-inhibitory factor (LIF) share a common signal-transducing subunit in each of their receptors and thus mediate an overlapping spectrum of biological activities. Although all of these cytokines stimulate the production of α1-proteinase inhibitor (α1-PI) in hepatocyte-derived cells, only OSM is able to up-regulate levels of this inhibitor in epithelial cells originating from the lung. In this study we characterized human lung-derived epithelial-like HTB58 cells for their ability to synthesize α1-PI after treatment with IL-6, OSM and LIF. The results demonstrate that the resistance of HTB58 cells to the effects of IL-6 and LIF was not becau…

medicine.medical_specialtyendocrine systemProtein subunitBlotting WesternOncostatin MInhibitory postsynaptic potentialBiochemistryLeukemia Inhibitory FactorInternal medicinemedicineTumor Cells CulturedHumansInterleukin 6ReceptorMolecular BiologyLungLymphokinesbiologyChemistryInterleukin-6fungiOncostatin MOncostatin M receptorEpithelial CellsCell BiologyGlycoprotein 130Blotting NorthernGrowth InhibitorsCell biologyInterleukin 31Endocrinologyalpha 1-Antitrypsinbiology.proteinPeptidesResearch ArticleThe Biochemical journal
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Tcgfiii/p40 is produced by naive murine cd4+ t cells but is not a general t cell growth factor*

1989

Several antigen-specific T cell lines were found to secrete a lymphokine upon activation by antigen or lectin that was provisionally termed T cell growth factor III (TCGF III) because it induced the proliferation of a CD4+ T cell clone independently from IL2 and IL4. Amino acid sequence analysis (and the functional properties of TCGF III) revealed that TCGF III was identical with a recently identified lymphokine termed P40. TCGF III/P40 was not only produced by long-term cultured T cell lines but also upon stimulation of freshly isolated Mlsa-reactive T cells. In addition, naive CD4+ T cells secreted TCGF III/P40 upon activation by lectin or allo-major histocompatibility complex structures.…

CD4-Positive T-LymphocytesT cellMolecular Sequence DataImmunologyMice Inbred StrainsBiologyMajor histocompatibility complexCell LineMiceAntigenmedicineAnimalsImmunology and AllergyCytotoxic T cellInterleukin 9Amino Acid SequenceGrowth SubstancesInterleukin 4GlycoproteinsLymphokinesInterleukin-9LymphokineT-Lymphocytes Helper-InducerT lymphocyteVirologyMolecular biologymedicine.anatomical_structurebiology.proteinInterleukin-2Interleukin-4Lymphocyte Culture Test MixedEuropean Journal of Immunology
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Lymphokine profile and activation pattern of two unrelated antigen- or idiotype-specific T suppressor cell clones.

1992

Two T suppressor (Ts) clones of different specificity have been analyzed for their lymphokine spectrum. BVI/5 is an I-Ek-restricted bovine serum albumin (BSA)-specific Ts cell clone from a CBA/J mouse tolerized by low doses of BSA. It affects directly or indirectly the function of BSA-specific T helper (Th) cells. The Ts cell clone 178-4 from a BALB/c mouse is I-Ed restricted and recognizes the public J558 Id on B cells. It prevents alpha(1----3)dextran B 1355S (Dex)-specific IgG antibody production and drives Dex-specific J558 idiotype-bearing B cells into an anergic B IgG memory cell state. Both Ts cell clones thus cause specific suppression, yet in different experimental systems using di…

IdiotypeMalemedicine.medical_treatmentImmunologyLymphocyte ActivationT-Lymphocytes RegulatoryInterferon-gammaMiceAntigenInterferonAntibody SpecificityCell ClonemedicineImmunology and AllergyAnimalsAntigensLymphokinesCD40biologyLymphokineHistocompatibility Antigens Class IISerum Albumin BovineT-Lymphocytes Helper-InducerMolecular biologyClone CellsCytokineImmunologybiology.proteinMice Inbred CBAClone (B-cell biology)medicine.drugInterleukin-1European journal of immunology
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