Search results for "Nephritis"

showing 10 items of 101 documents

Colonic Conduit in Children

1996

Complication rates of up to 86.6% have been reported after creation of an ileal conduit. In contrast to others, we construct an antirefluxing isoperistaltic colonic for incontinent diversion in children.Between 1968 and 1989 a colonic conduit was created in 105 patients up to age 20 years for neurogenic bladder (76), bladder exstrophy (16) and other reasons (13). Long-term followup (minimum 5 years, mean 16.3 years, range 5 to 26) was possible in 84 patients (159 renal units).Early and late stenosis at the ureterocolic anastomosis, and stomal stenosis occurred in 7.6 and 15.5% of the cases, respectively. Renal calculi developed in 8.2% of the renal units. Eight kidneys without function afte…

AdultMalemedicine.medical_specialtyTime FactorsAdolescentColonUrologyUrinary Diversionurologic and male genital diseasesStomal stenosisPostoperative ComplicationsRecurrent pyelonephritisHumansMedicineChildUpper urinary tractbusiness.industryUrinary Bladder DiseasesInfantColonic conduitUreterocolic anastomosismedicine.diseaseSurgeryBladder exstrophyStenosisChild PreschoolFemalebusinessComplicationFollow-Up StudiesThe Journal of Urology
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Tubulointerstitielle-Nephritis-Uveitis-Syndrom (TINU-Syndrom)

1995

Abstract For 10 weeks a 25-year-old man had been suffering from tiredness, fatigue, nausea and a 16 kg weight loss. Erythrocyte sedimentation rate (83/133 mm), serum C-reactive protein (5.5 mg/dl) and creatinine (5.05 mg/dl) were all elevated. He also had proteinuria (1120 mg daily), sterile leukocytosis and a creatinine clearance of 10 ml/min. Renal biopsy showed interstitial nephritis and bone marrow biopsy revealed non-caseous epithelioid-cell granulomas. 14 days after admission he developed acute iritis in the right eye. Other causes having been excluded, the diagnosis of tubulo-intestinal nephritis with uveitis (TINU syndrome) was made. The clinical symptoms and laboratory findings imp…

AdultMalemedicine.medical_specialtyTime FactorsBiopsyInterstitial nephritisIritisRenal functionKidneyKidney Function TestsGastroenterologyDiagnosis DifferentialUveitischemistry.chemical_compoundPrednisoneInternal medicineHumansMedicineLeukocytosisCreatininemedicine.diagnostic_testbusiness.industrySyndromeGeneral Medicinemedicine.diseaseEndocrinologychemistryErythrocyte sedimentation rateAcute DiseaseNephritis InterstitialPrednisoneRenal biopsymedicine.symptombusinessNephritismedicine.drugDMW - Deutsche Medizinische Wochenschrift
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Blood and urinary abnormalities induced during and after 24-hour continuous running: A case report

2016

In this reported clinical case, a healthy and well-trained male subject [aged 37 years, maximal oxygen uptake (V[Combining Dot Above]O2max) 64 mL·kg·min] ran for 23 hours and 35 minutes covering 160 km (6.7 km/h average running speed). The analysis of hematological and biochemical parameters 3 days before the event, just after termination of exercise, and after 24 and 48 hours of recovery revealed important changes on muscle and liver function, and hemolysis. The analysis of urine sediments showed an increment of red and white blood cells filtrations, compatible with transient nephritis. After 48 hours, most of these alterations were recovered. Physicians and health professionals who monito…

AdultMalemedicine.medical_specialtyUrinary systemPhysiologyPhysical Therapy Sports Therapy and RehabilitationUrine030204 cardiovascular system & hematologyHemolysisRunning03 medical and health sciences0302 clinical medicineAtletamedicineHumansOrthopedics and Sports MedicineMuscle SkeletalPathologicalNephritisbusiness.industryMedicina deportivaVO2 max030229 sport sciencesAtletasmedicine.diseaseHemolysisSurgeryLiverClinical caseLiver functionbusinessNephritisBiomarkers
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Treatment with essential amino acids in patients on chronic hemodialysis: a double blind cross-over study.

1978

Patients on chronic hemodialysis may suffer from a latent protein deficiency, and therapy with essential amino acids has been recommended. In a double blind cross-over study, 13 hemodialysis patients received orally 15.7 g of essential amino acids daily over a 3-month period. Patients were on a liberal diet, containing 1 g of protein per kilogram of body weight per day. Hemodialysis was adequate. Therapy resulted in an increase in urea, uric acid, C3 c complement factor and a fall in C4. Lysine levels increased and phenylalanine fell. Malnutrition could not account for the observed metabolic changes, which are more likely due to uremic metabolic disturbances. A liberal diet of 1 g of protei…

AdultMalemedicine.medical_specialtymedicine.medical_treatmentMedicine (miscellaneous)PhenylalanineComplement factor Ichemistry.chemical_compoundGlomerulonephritisRenal DialysisInternal medicineProtein DeficiencyMedicineHumansPrealbuminchemistry.chemical_classificationClinical Trials as TopicNutrition and DieteticsPyelonephritisbusiness.industryTransferrinComplement C3Middle Agedmedicine.diseaseCrossover studyAmino acidRetinol-Binding ProteinsMalnutritionEndocrinologychemistryBiochemistryUreaUric acidFemaleHemodialysisAmino Acids EssentialbusinessThe American journal of clinical nutrition
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Immunoregulatory role of Jα281 T cells in aged mice developing lupus-like nephritis

2007

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the emergence of autoreactive T cells. Humans and mice with SLE have reduced numbers of CD1d-restricted invariant natural killer T (iNKT) cells, suggesting a key role for these cells in its immunopathogenesis. This subset uses an invariant TCR constituted by Valpha14 Jalpha281 chains paired with some Vbeta domains. The regulatory role for iNKT cells in non-autoimmune mice was suggested by our previous results showing that aged Jalpha281 knockout (KO) mice produce anti-dsDNA. Here we show that old Jalpha281 KO mice have proteinuria and antibodies against dsDNA and cardiolipin. Histological analysis of Jalpha281 KO m…

AgingImmunologyReceptors Antigen T-CellEnzyme-Linked Immunosorbent AssayLymphocyte Activationmedicine.disease_causeAutoimmunity Knockout NKT cellsAutoimmunityMicemedicineAnimalsLupus Erythematosus SystemicImmunology and AllergyAutoantibodiesMice KnockoutSettore MED/04 - Patologia GeneraleB-LymphocytesSystemic lupus erythematosusbiologyT-cell receptorAutoantibodyNatural killer T cellMarginal zonemedicine.diseaseImmunohistochemistryLupus NephritisKiller Cells NaturalImmunologybiology.proteinAntibodyNephritisSpleenEuropean Journal of Immunology
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Inducible Co-Stimulator Null MRL-Fas lpr Mice

2005

MRL/MpJ-Tnfrsf6lpr (MRL-Faslpr) mice develop a spontaneous T cell-dependent autoimmune disease that shares features with human lupus, including fatal nephritis, systemic pathology, and autoantibodies (autoAb). The inducible co-stimulator (ICOS) is upregulated on activated T cells and modulates T cell-mediated responses. To investigate whether ICOS has an essential role in regulating autoimmune lupus nephritis and the systemic illness in MRL-Faslpr mice, ICOS null (-/-) MRL Faslpr and ICOS intact (+/+) MRL-Faslpr strains (wild-type [WT]) were generated and compared. It was determined that in ICOS-/- MRL-Faslpr as compared with the WT strain, (1) there is a significant reduction in circulatin…

Antigens Differentiation T-LymphocyteMice Inbred MRL lprT-LymphocytesT cellLupus nephritismedicine.disease_causeBlood Urea NitrogenAutoimmunityInducible T-Cell Co-Stimulator ProteinInterferon-gammaMiceImmune systemimmune system diseasesmedicineAnimalsskin and connective tissue diseasesAutoantibodiesMice Inbred C3HSystemic lupus erythematosusTumor Necrosis Factor-alphabusiness.industryAutoantibodyGeneral Medicinemedicine.diseaseLupus NephritisIsotypeInterleukin-10Mice Inbred C57BLProteinuriamedicine.anatomical_structureNephrologyImmunoglobulin GImmunologyInterleukin-4businessNephritisJournal of the American Society of Nephrology
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PS5:100 Patophysiological role of type i and iii interferons in systemic lupus erythematosus (sle)

2018

Systemic Lupus erythematosus (SLE) is an autoimmune disease characterised by activated autoreactive lymphocytes and autoantibodies, resulting in tissue damage in multiple organs. An important factor for the disease´s mortality is the development of Lupus nephritis (LN). Type I and III interferons, which are both part of the antiviral defense, have both been associated with the disease´s activity. In sera and urine of SLE patients an enhanced level of IL28/29 was described, but their distinct functional role in the course of disease need to be further investigated. To determine the role of type I and III interferons during onset and progression of autoimmunity – with focus on the development…

Autoimmune diseaseSystemic lupus erythematosusbusiness.industryLupus nephritisAutoantibodyGlomerulonephritisSpleenmedicine.diseasemedicine.disease_causeAutoimmunitymedicine.anatomical_structureimmune system diseasesImmunologyMedicineskin and connective tissue diseasesbusinessReceptorPoster session 5: Innate and adaptive immunity
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Re: The Clinical Impact of Bacteremia on Outcomes in Elderly Patients with Pyelonephritis or Urinary Sepsis: A Prospective Multicenter Study

2018

Background Bacteremia is common in severe urinary infections, but its influence on the outcomes is not well established. The aim of this study was to assess the association of bacteremia with outcomes in elderly patients admitted to hospital with pyelonephritis or urinary sepsis. Methods This prospective muticenter observational study was conducted at 5 Spanish hospitals. All patients aged >65 years with pyelonephritis or urinary sepsis admitted to the departments of internal medicine and with urine and blood cultures obtained at admission to hospital were eligible. Transfer to ICU, length of hospital stay, hospital mortality and all cause 30-day mortality in bacteremic and non-bacteremic g…

Bacterial DiseasesMaleMultivariate analysisPhysiologylcsh:MedicineBacteremiaUrineUrinePathology and Laboratory MedicineElderly0302 clinical medicineMedicine and Health Sciences030212 general & internal medicineProspective Studieslcsh:ScienceProspective cohort studyAged 80 and overMultidisciplinaryPyelonephritisHospitalsBody FluidsInfectious DiseasesBloodCohortUrinary Tract InfectionsFemaleAnatomyResearch Articlemedicine.medical_specialtyUrologyUrinary systemMEDLINESepsis03 medical and health sciencesSigns and SymptomsDiagnostic MedicineSepsisInternal medicinemedicineHumansAgedSeptic shockbusiness.industrylcsh:RBiology and Life Sciencesmedicine.diseaseHealth Care030228 respiratory systemMulticenter studyHealth Care FacilitiesGeriatricsAge GroupsBacteremiaPeople and PlacesPopulation Groupingslcsh:QbusinessJournal of Urology
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Characterization of cells involved in the formation of granuloma. An ultrastructural study on macrophages, epitheloid cells, and giant cells in exper…

1981

In experimental tubulo-interstitial (anti-basement membrane) nephritis of the rat, granulomatous inflammation develops around immunologically altered tubular basement membranes. The present light- and electron microscopic studies indicate that in the course of the granulomatous reaction, tissue monocytes evolve from blood monocytes and pursue two independent pathways of differentation. On the one hand they may differentiate into macrophages ("distant from tubules") or, alternatively, into epitheloid cells ("adjacent to tubules"). The latter, through cell fusion, develop into multinucleated giant cells of the Langhans' type. The cytoplasmic components of the epitheloid cells and the multinuc…

Basement membraneMaleCell fusionGranulomaInterstitial nephritisMacrophagesLanghans giant cellGeneral MedicineBiologymedicine.diseaseBasement MembraneMonocytesCell biologyRatsCell FusionMicroscopy Electronmedicine.anatomical_structureKidney TubulesGiant cellGranulomamedicineUltrastructureAnimalsNephritis InterstitialNephritisVirchows Archiv. B, Cell pathology including molecular pathology
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Formation of basement membrane in extracapillary proliferates in rapidly progressive glomerulonephritis

1977

In the extracapillary proliferations (crescents) of the glomeruli in glomerulonephritis, basement membranes appear and in addition "secretory bodies" are formed in the cisternae of the rough endoplasmatic reticulum. The findings permit the conclusion that proliferated visceral epithelial cells are involved in the crescent formation to a greater extent than previously assumed.

Basement membranePathologymedicine.medical_specialtyurogenital systemChemistryKidney Glomerulusfood and beveragesGlomerulonephritisAnatomyEndoplasmic Reticulumurologic and male genital diseasesmedicine.diseaseBasement Membranefemale genital diseases and pregnancy complicationsPathology and Forensic MedicineGlomerulonephritismedicine.anatomical_structuremedicineHumansRapidly progressive glomerulonephritisReticulumVirchows Archiv B Cell Pathology
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